title: Serie A

text: Prior to 2005–06 a play-off would immediately be used if teams were tied in a championship, European qualification, or relegation spot, but between 2006–07 and 2021–22 the tiebreakers currently used for all places besides first were in place to decide the scudetto winner, though this was never needed. In some past years, the playoff was a single game at a neutral site while in others it was a two-legged tie decided by aggregate score. A playoff game has never been needed since the tiebreaking format changed.


title: Serie A

text: The only time a playoff was used to decide the champion occurred in the 1963–64 season when Bologna and Inter both finished on 54 points. Bologna won the playoff 2–0 at the Stadio Olimpico in Rome to win the scudetto. Playoff games have been used on multiple occasions to decide European competition qualifications (most recently in 1999–2000) and relegation (most recently in 2004–05).


title: Serie A

text: Before 1929, many clubs competed in the top level of Italian football as the earlier rounds were competed up to 1922 on a regional basis then interregional up to 1929. Below is a list of Serie A clubs who have competed in the competition since it has been a league format (68 in total).


title: Serie A

text: There are 68 teams that have taken part in 91 Serie A championships in a single round that was played from the 1929–30 season until the 2022–23 season. The teams in bold compete in Serie A currently. Inter Milan is the only team that has played Serie A football in every season.


title: Serie A

text: Serie A had logos that featured its sponsor Telecom Italia (TIM). The logo that was introduced in 2010 had a minor change in 2016 due to the change of the logo of Telecom Italia itself. In August 2018, a new logo was announced, and another one in August 2019.


title: Serie A

text: In the past, individual clubs competing in the league had the rights to sell their broadcast rights to specific channels throughout Italy, unlike in most other European countries. Currently, the two broadcasters in Italy are the satellite broadcaster Sky Italia and streaming platform DAZN for its own pay television networks; RAI is allowed to broadcast only highlights (in exclusive from 13:30 to 22:30 CET).


title: Serie A

text: Since the 2010–11 season, Serie A clubs have negotiated television rights collectively rather than on an individual club basis, having previously abandoned collective negotiation at the end of the 1998–99 season.


title: Serie A

text: In the 1990s, Serie A was at its most popular in the United Kingdom when it was shown on "Football Italia" on Channel 4, although it has actually appeared on more UK channels than any other league, rarely staying in one place for long since 2002. Serie A has appeared in the UK on BSB's The Sports Channel (1990–91), Sky Sports (1991–1992), Channel 4 (1992–2002), Eurosport (2002–2004), Setanta Sports and Bravo (2004–2007), Channel 5 (2007–2008), ESPN (2009–2013), Eleven Sports Network (2018), Premier, FreeSports (2019–2021) and currently BT Sport (2013–2018; 2021–present).


title: Serie A

text: Boldface indicates a player still active in Serie A. "Italics" indicates a player active outside Serie A.


title: Serie A

text: Unlike La Liga, which imposed a quota on the number of non-EU players on each club, Serie A clubs could sign as many non-EU players as available on domestic transfer.


title: Serie A

text: During the 1980s and 1990s, most Serie A clubs signed a large number of players from foreign nations (both EU and non-EU members). Notable foreign players to play in Serie A during this era included Irish international Liam Brady, England internationals Paul Gascoigne and David Platt, France's Michel Platini and Laurent Blanc, Lothar Matthäus and Jürgen Klinsmann from Germany, Dutchmen Ruud Gullit and Dennis Bergkamp, and Argentina's Diego Maradona.


title: Serie A

text: But since the 2003–04 season, a quota has been imposed on each of the clubs limiting the number of non-EU, non-EFTA and non-Swiss players who may be signed from abroad each season, following provisional measures introduced in the 2002–03 season, which allowed Serie A and B clubs to sign only one non-EU player in the 2002 summer transfer window.


title: Serie A

text: In the middle of the 2000–01 season, the old quota system was abolished, which no longer limited each team to having more than five non-EU players and using no more than three in each match. Concurrent with the abolishment of the quota, the FIGC had investigated footballers that used fake passports. Alberto and Warley, Alejandro Da Silva and Jorginho Paulista of Udinese; Fábio Júnior and Gustavo Bartelt of Roma; Dida of Milan; Álvaro Recoba of Inter; Thomas Job, Francis Zé, Jean Ondoa of Sampdoria; and Jeda and Dede of Vicenza were all banned in July 2001 for lengths ranging from six months to one year. However, most of the bans were subsequently reduced.


title: Serie A

text: The number of non-EU players was reduced from 265 in 2002–03 season to 166 in 2006–07 season. It also included players who received EU status after their respective countries joined the EU (see 2004 and 2007 enlargement), which made players such as Adrian Mutu, Valeri Bojinov, Marek Jankulovski and Marius Stankevičius EU players.


title: Serie A

text: Since the 2008–09 season, three quotas have been awarded to clubs that do not have non-EU players in their squad (previously only newly promoted clubs could have three quotas); clubs that have one non-EU player have two quotas. Those clubs that have two non-EU players, are awarded one quota and one conditional quota, which is awarded after: 1) Transferred 1 non-EU player abroad, or 2) Release 1 non-EU player as free agent, or 3) A non-EU player received EU nationality. Clubs with three or more non-EU players, have two conditional quotas, but releasing two non-EU players as free agent, will only have one quota instead of two. Serie B and Lega Pro clubs cannot sign non-EU player from abroad, except those followed the club promoted from Serie D.


title: Serie A

text: Large clubs with many foreigners usually borrow quotas from other clubs that have few foreigners or no foreigners in order to sign more non-EU players. For example, Adrian Mutu joined Juventus via Livorno in 2005, as at the time Romania was not a member of the EU. Other examples include Júlio César, Victor Obinna and Maxwell, who joined Inter from Chievo (first two) and Empoli respectively.


title: Serie A

text: On 2 July 2010, the above conditional quota reduced back to one, though if a team did not have any non-EU players, that team could still sign up to three non-EU players. In 2011 the signing quota reverted to two.


title: Serie A

text: Serie A also imposed Homegrown players rule, a modification of Homegrown Player Rule (UEFA). Unlike UEFA, Serie A at first did not cap the number of players in first team squad at 25, meaning the club could employ more foreigners by increasing the size of the squad. However, a cap of 25 (under-21 players were excluded) was introduced to 2015–16 season (in 2015–16 season, squad simply require 8 homegrown players but not require 4 of them from their own youth team). In the 2016–17 season, the FIGC sanctioned Sassuolo for fielding ineligible player, Antonino Ragusa. Although the club did not exceed the capacity of 21 players that were not from their own youth team (only Domenico Berardi was eligible as youth product of their own) as well as under 21 of age (born 1995 or after, of which four players were eligible) in their 24-men call-up, It was reported that on Lega Serie A side the squad list was not updated.


title: Tuberculosis

text: Tuberculosis (TB) is an infectious disease usually caused by "Mycobacterium tuberculosis" (MTB) bacteria. Tuberculosis generally affects the lungs, but it can also affect other parts of the body. Most infections show no symptoms, in which case it is known as latent tuberculosis. Around 10% of latent infections progress to active disease which, if left untreated, kill about half of those affected. Typical symptoms of active TB are chronic cough with blood-containing mucus, fever, night sweats, and weight loss. It was historically referred to as consumption due to the weight loss associated with the disease. Infection of other organs can cause a wide range of symptoms.


title: Tuberculosis

text: Tuberculosis is spread from one person to the next through the air when people who have active TB in their lungs cough, spit, speak, or sneeze. People with Latent TB do not spread the disease. Active infection occurs more often in people with HIV/AIDS and in those who smoke. Diagnosis of active TB is based on chest X-rays, as well as microscopic examination and culture of body fluids. Diagnosis of Latent TB relies on the tuberculin skin test (TST) or blood tests.


title: Tuberculosis

text: Prevention of TB involves screening those at high risk, early detection and treatment of cases, and vaccination with the bacillus Calmette-Guérin (BCG) vaccine. Those at high risk include household, workplace, and social contacts of people with active TB. Treatment requires the use of multiple antibiotics over a long period of time. Antibiotic resistance is a growing problem with increasing rates of multiple drug-resistant tuberculosis (MDR-TB).


title: Tuberculosis

text: In 2018, one quarter of the world's population was thought to have a latent infection of TB. New infections occur in about 1% of the population each year. In 2020, an estimated 10 million people developed active TB, resulting in 1.5 million deaths, making it the second leading cause of death from an infectious disease after COVID-19. As of 2018, most TB cases occurred in the regions of South-East Asia (44%), Africa (24%), and the Western Pacific (18%), with more than 50% of cases being diagnosed in seven countries: India (27%), China (9%), Indonesia (8%), the Philippines (6%), Pakistan (6%), Nigeria (4%), and Bangladesh (4%). By 2021 the number of new cases each year was decreasing by around 2% annually. About 80% of people in many Asian and African countries test positive while 5–10% of people in the United States population test positive via the tuberculin test. Tuberculosis has been present in humans since ancient times.


title: Tuberculosis

text: Tuberculosis may infect any part of the body, but most commonly occurs in the lungs (known as pulmonary tuberculosis). Extrapulmonary TB occurs when tuberculosis develops outside of the lungs, although extrapulmonary TB may coexist with pulmonary TB.


title: Tuberculosis

text: General signs and symptoms include fever, chills, night sweats, loss of appetite, weight loss, and fatigue. Significant nail clubbing may also occur.


title: Tuberculosis

text: If a tuberculosis infection does become active, it most commonly involves the lungs (in about 90% of cases). Symptoms may include chest pain and a prolonged cough producing sputum. About 25% of people may not have any symptoms (i.e., they remain asymptomatic). Occasionally, people may cough up blood in small amounts, and in very rare cases, the infection may erode into the pulmonary artery or a Rasmussen's aneurysm, resulting in massive bleeding. Tuberculosis may become a chronic illness and cause extensive scarring in the upper lobes of the lungs. The upper lung lobes are more frequently affected by tuberculosis than the lower ones. The reason for this difference is not clear. It may be due to either better air flow, or poor lymph drainage within the upper lungs.


title: Tuberculosis

text: In 15–20% of active cases, the infection spreads outside the lungs, causing other kinds of TB. These are collectively denoted as extrapulmonary tuberculosis. Extrapulmonary TB occurs more commonly in people with a weakened immune system and young children. In those with HIV, this occurs in more than 50% of cases. Notable extrapulmonary infection sites include the pleura (in tuberculous pleurisy), the central nervous system (in tuberculous meningitis), the lymphatic system (in scrofula of the neck), the genitourinary system (in urogenital tuberculosis), and the bones and joints (in Pott disease of the spine), among others. A potentially more serious, widespread form of TB is called "disseminated tuberculosis", it is also known as miliary tuberculosis. Miliary TB currently makes up about 10% of extrapulmonary cases.


title: Tuberculosis

text: The main cause of TB is "Mycobacterium tuberculosis" (MTB), a small, aerobic, nonmotile bacillus. The high lipid content of this pathogen accounts for many of its unique clinical characteristics. It divides every 16 to 20 hours, which is an extremely slow rate compared with other bacteria, which usually divide in less than an hour. Mycobacteria have an outer membrane lipid bilayer. If a Gram stain is performed, MTB either stains very weakly "Gram-positive" or does not retain dye as a result of the high lipid and mycolic acid content of its cell wall. MTB can withstand weak disinfectants and survive in a dry state for weeks. In nature, the bacterium can grow only within the cells of a host organism, but "M. tuberculosis" can be cultured in the laboratory.


title: Tuberculosis

text: Using histological stains on expectorated samples from phlegm (also called sputum), scientists can identify MTB under a microscope. Since MTB retains certain stains even after being treated with acidic solution, it is classified as an acid-fast bacillus. The most common acid-fast staining techniques are the Ziehl–Neelsen stain and the Kinyoun stain, which dye acid-fast bacilli a bright red that stands out against a blue background. Auramine-rhodamine staining and fluorescence microscopy are also used.


title: Tuberculosis

text: The "M. tuberculosis" complex (MTBC) includes four other TB-causing mycobacteria: "M. bovis", "M. africanum", "M. canetti", and "M. microti". "M. africanum" is not widespread, but it is a significant cause of tuberculosis in parts of Africa. "M. bovis" was once a common cause of tuberculosis, but the introduction of pasteurized milk has almost eliminated this as a public health problem in developed countries. "M. canetti" is rare and seems to be limited to the Horn of Africa, although a few cases have been seen in African emigrants. "M. microti" is also rare and is seen almost only in immunodeficient people, although its prevalence may be significantly underestimated.


title: Tuberculosis

text: Other known pathogenic mycobacteria include "M. leprae", "M. avium", and "M. kansasii". The latter two species are classified as "nontuberculous mycobacteria" (NTM) or atypical mycobacteria. NTM cause neither TB nor leprosy, but they do cause lung diseases that resemble TB.


title: Tuberculosis

text: When people with active pulmonary TB cough, sneeze, speak, sing, or spit, they expel infectious aerosol droplets 0.5 to 5.0 µm in diameter. A single sneeze can release up to 40,000 droplets. Each one of these droplets may transmit the disease, since the infectious dose of tuberculosis is very small (the inhalation of fewer than 10 bacteria may cause an infection).


title: Tuberculosis

text: People with prolonged, frequent, or close contact with people with TB are at particularly high risk of becoming infected, with an estimated 22% infection rate. A person with active but untreated tuberculosis may infect 10–15 (or more) other people per year. Transmission should occur from only people with active TB – those with latent infection are not thought to be contagious. The probability of transmission from one person to another depends upon several factors, including the number of infectious droplets expelled by the carrier, the effectiveness of ventilation, the duration of exposure, the virulence of the "M. tuberculosis" strain, the level of immunity in the uninfected person, and others. The cascade of person-to-person spread can be circumvented by segregating those with active ("overt") TB and putting them on anti-TB drug regimens. After about two weeks of effective treatment, subjects with nonresistant active infections generally do not remain contagious to others. If someone does become infected, it typically takes three to four weeks before the newly infected person becomes infectious enough to transmit the disease to others.


title: Tuberculosis

text: The most important risk factor globally for developing active TB is concurrent HIV infection; 13% of those with TB are also infected with HIV. This is a particular problem in sub-Saharan Africa, where HIV infection rates are high. Of those without HIV infection who are infected with tuberculosis, about 5–10% develop active disease during their lifetimes; in contrast, 30% of those co-infected with HIV develop the active disease.


title: Tuberculosis

text: Use of certain medications, such as corticosteroids and infliximab (an anti-αTNF monoclonal antibody), is another important risk factor, especially in the developed world.


title: Tuberculosis

text: Other risk factors include: alcoholism, diabetes mellitus (3-fold increased risk), silicosis (30-fold increased risk), tobacco smoking (2-fold increased risk), indoor air pollution, malnutrition, young age, recently acquired TB infection, recreational drug use, severe kidney disease, low body weight, organ transplant, head and neck cancer, and genetic susceptibility (the overall importance of genetic risk factors remains undefined).


title: Tuberculosis

text: Tobacco smoking increases the risk of infections (in addition to increasing the risk of active disease and death). Additional factors increasing infection susceptibility include young age.


title: Tuberculosis

text: About 90% of those infected with "M. tuberculosis" have asymptomatic, latent TB infections (sometimes called LTBI), with only a 10% lifetime chance that the latent infection will progress to overt, active tuberculous disease. In those with HIV, the risk of developing active TB increases to nearly 10% a year. If effective treatment is not given, the death rate for active TB cases is up to 66%.


title: Tuberculosis

text: TB infection begins when the mycobacteria reach the alveolar air sacs of the lungs, where they invade and replicate within endosomes of alveolar macrophages. Macrophages identify the bacterium as foreign and attempt to eliminate it by phagocytosis. During this process, the bacterium is enveloped by the macrophage and stored temporarily in a membrane-bound vesicle called a phagosome. The phagosome then combines with a lysosome to create a phagolysosome. In the phagolysosome, the cell attempts to use reactive oxygen species and acid to kill the bacterium. However, "M. tuberculosis" has a thick, waxy mycolic acid capsule that protects it from these toxic substances. "M. tuberculosis" is able to reproduce inside the macrophage and will eventually kill the immune cell.


title: Tuberculosis

text: The primary site of infection in the lungs, known as the Ghon focus, is generally located in either the upper part of the lower lobe, or the lower part of the upper lobe. Tuberculosis of the lungs may also occur via infection from the blood stream. This is known as a Simon focus and is typically found in the top of the lung. This hematogenous transmission can also spread infection to more distant sites, such as peripheral lymph nodes, the kidneys, the brain, and the bones. All parts of the body can be affected by the disease, though for unknown reasons it rarely affects the heart, skeletal muscles, pancreas, or thyroid.


title: Tuberculosis

text: Tuberculosis is classified as one of the granulomatous inflammatory diseases. Macrophages, epithelioid cells, T lymphocytes, B lymphocytes, and fibroblasts aggregate to form granulomas, with lymphocytes surrounding the infected macrophages. When other macrophages attack the infected macrophage, they fuse together to form a giant multinucleated cell in the alveolar lumen. The granuloma may prevent dissemination of the mycobacteria and provide a local environment for interaction of cells of the immune system. However, more recent evidence suggests that the bacteria use the granulomas to avoid destruction by the host's immune system. Macrophages and dendritic cells in the granulomas are unable to present antigen to lymphocytes; thus the immune response is suppressed. Bacteria inside the granuloma can become dormant, resulting in latent infection. Another feature of the granulomas is the development of abnormal cell death (necrosis) in the center of tubercles. To the naked eye, this has the texture of soft, white cheese and is termed caseous necrosis.


title: Tuberculosis

text: If TB bacteria gain entry to the blood stream from an area of damaged tissue, they can spread throughout the body and set up many foci of infection, all appearing as tiny, white tubercles in the tissues. This severe form of TB disease, most common in young children and those with HIV, is called miliary tuberculosis. People with this disseminated TB have a high fatality rate even with treatment (about 30%).


title: Tuberculosis

text: In many people, the infection waxes and wanes. Tissue destruction and necrosis are often balanced by healing and fibrosis. Affected tissue is replaced by scarring and cavities filled with caseous necrotic material. During active disease, some of these cavities are joined to the air passages (bronchi) and this material can be coughed up. It contains living bacteria and thus can spread the infection. Treatment with appropriate antibiotics kills bacteria and allows healing to take place. Upon cure, affected areas are eventually replaced by scar tissue.


title: Tuberculosis

text: Diagnosing active tuberculosis based only on signs and symptoms is difficult, as is diagnosing the disease in those who have a weakened immune system. A diagnosis of TB should, however, be considered in those with signs of lung disease or constitutional symptoms lasting longer than two weeks. A chest X-ray and multiple sputum cultures for acid-fast bacilli are typically part of the initial evaluation. Interferon-γ release assays (IGRA) and tuberculin skin tests are of little use in most of the developing world. IGRA have similar limitations in those with HIV.


title: Tuberculosis

text: A definitive diagnosis of TB is made by identifying "M. tuberculosis" in a clinical sample (e.g., sputum, pus, or a tissue biopsy). However, the difficult culture process for this slow-growing organism can take two to six weeks for blood or sputum culture. Thus, treatment is often begun before cultures are confirmed.


title: Tuberculosis

text: Nucleic acid amplification tests and adenosine deaminase testing may allow rapid diagnosis of TB. Blood tests to detect antibodies are not specific or sensitive, so they are not recommended.


title: Tuberculosis

text: The Mantoux tuberculin skin test is often used to screen people at high risk for TB. Those who have been previously immunized with the Bacille Calmette-Guerin vaccine may have a false-positive test result. The test may be falsely negative in those with sarcoidosis, Hodgkin's lymphoma, malnutrition, and most notably, active tuberculosis. Interferon gamma release assays, on a blood sample, are recommended in those who are positive to the Mantoux test. These are not affected by immunization or most environmental mycobacteria, so they generate fewer false-positive results. However, they are affected by "M. szulgai", "M. marinum", and "M. kansasii". IGRAs may increase sensitivity when used in addition to the skin test, but may be less sensitive than the skin test when used alone.


title: Tuberculosis

text: The US Preventive Services Task Force (USPSTF) has recommended screening people who are at high risk for latent tuberculosis with either tuberculin skin tests or interferon-gamma release assays. While some have recommend testing health care workers, evidence of benefit for this is poor . The Centers for Disease Control and Prevention (CDC) stopped recommending yearly testing of health care workers without known exposure in 2019.


title: Tuberculosis

text: Tuberculosis prevention and control efforts rely primarily on the vaccination of infants and the detection and appropriate treatment of active cases. The World Health Organization (WHO) has achieved some success with improved treatment regimens, and a small decrease in case numbers. Some countries have legislation to involuntarily detain or examine those suspected to have tuberculosis, or involuntarily treat them if infected.


title: Tuberculosis

text: The only available vaccine is bacillus Calmette-Guérin (BCG). In children it decreases the risk of getting the infection by 20% and the risk of infection turning into active disease by nearly 60%.


title: Tuberculosis

text: It is the most widely used vaccine worldwide, with more than 90% of all children being vaccinated. The immunity it induces decreases after about ten years. As tuberculosis is uncommon in most of Canada, Western Europe, and the United States, BCG is administered to only those people at high risk. Part of the reasoning against the use of the vaccine is that it makes the tuberculin skin test falsely positive, reducing the test's usefulness as a screening tool. Several vaccines are being developed.


title: Tuberculosis

text: Public health campaigns which have focused on overcrowding, public spitting and regular sanitation (including hand washing) during the 1800s helped to either interrupt or slow spread which when combined with contact tracing, isolation and treatment helped to dramatically curb the transmission of both tuberculosis and other airborne diseases which led to the elimination of tuberculosis as a major public health issue in most developed economies. Other risk factors which worsened TB spread such as malnutrition were also ameliorated, but since the emergence of HIV a new population of immunocompromised individuals was available for TB to infect.


title: Tuberculosis

text: The World Health Organization (WHO) declared TB a "global health emergency" in 1993, and in 2006, the Stop TB Partnership developed a Global Plan to Stop Tuberculosis that aimed to save 14 million lives between its launch and 2015. A number of targets they set were not achieved by 2015, mostly due to the increase in HIV-associated tuberculosis and the emergence of multiple drug-resistant tuberculosis. A tuberculosis classification system developed by the American Thoracic Society is used primarily in public health programs. In 2015, it launched the End TB Strategy to reduce deaths by 95% and incidence by 90% before 2035. The goal of tuberculosis elimination is hampered by the lack of rapid testing, of short and effective treatment courses, and of completely effective vaccines.


title: Tuberculosis

text: The benefits and risks of giving anti-tubercular drugs in those exposed to MDR-TB is unclear. Making HAART therapy available to HIV-positive individuals significantly reduces the risk of progression to an active TB infection by up to 90% and can mitigate the spread through this population.


title: Tuberculosis

text: Treatment of TB uses antibiotics to kill the bacteria. Effective TB treatment is difficult, due to the unusual structure and chemical composition of the mycobacterial cell wall, which hinders the entry of drugs and makes many antibiotics ineffective.


title: Tuberculosis

text: Active TB is best treated with combinations of several antibiotics to reduce the risk of the bacteria developing antibiotic resistance. The routine use of rifabutin instead of rifampicin in HIV-positive people with tuberculosis is of unclear benefit .


title: Tuberculosis

text: Latent TB is treated with either isoniazid or rifampin alone, or a combination of isoniazid with either rifampicin or rifapentine.


title: Tuberculosis

text: The treatment takes three to nine months depending on the medications used. People with latent infections are treated to prevent them from progressing to active TB disease later in life.


title: Tuberculosis

text: The recommended treatment of new-onset pulmonary tuberculosis, , is six months of a combination of antibiotics containing rifampicin, isoniazid, pyrazinamide, and ethambutol for the first two months, and only rifampicin and isoniazid for the last four months. Where resistance to isoniazid is high, ethambutol may be added for the last four months as an alternative. Treatment with anti-TB drugs for at least 6 months results in higher success rates when compared with treatment less than 6 months, even though the difference is small. Shorter treatment regimen may be recommended for those with compliance issues. There is also no evidence to support shorter anti-tuberculosis treatment regimens when compared to a 6-month treatment regimen. However recently, results from an international, randomized, controlled clinical trial indicate that a four-month daily treatment regimen containing high-dose, or "optimized," rifapentine with moxifloxacin (2PHZM/2PHM) is as safe and effective as the existing standard six-month daily regimen at curing drug-susceptible tuberculosis (TB) disease.


title: Tuberculosis

text: If tuberculosis recurs, testing to determine which antibiotics it is sensitive to is important before determining treatment. If multiple drug-resistant TB (MDR-TB) is detected, treatment with at least four effective antibiotics for 18 to 24 months is recommended.


title: Tuberculosis

text: Directly observed therapy, i.e., having a health care provider watch the person take their medications, is recommended by the World Health Organization (WHO) in an effort to reduce the number of people not appropriately taking antibiotics. The evidence to support this practice over people simply taking their medications independently is of poor quality. There is no strong evidence indicating that directly observed therapy improves the number of people who were cured or the number of people who complete their medicine. Moderate quality evidence suggests that there is also no difference if people are observed at home versus at a clinic, or by a family member versus a health care worker. Methods to remind people of the importance of treatment and appointments may result in a small but important improvement. There is also not enough evidence to support intermittent rifampicin-containing therapy given two to three times a week has equal effectiveness as daily dose regimen on improving cure rates and reducing relapsing rates. There is also not enough evidence on effectiveness of giving intermittent twice or thrice weekly short course regimen compared to daily dosing regimen in treating children with tuberculosis.


title: Tuberculosis

text: Primary resistance occurs when a person becomes infected with a resistant strain of TB. A person with fully susceptible MTB may develop secondary (acquired) resistance during therapy because of inadequate treatment, not taking the prescribed regimen appropriately (lack of compliance), or using low-quality medication. Drug-resistant TB is a serious public health issue in many developing countries, as its treatment is longer and requires more expensive drugs. MDR-TB is defined as resistance to the two most effective first-line TB drugs: rifampicin and isoniazid. Extensively drug-resistant TB is also resistant to three or more of the six classes of second-line drugs. Totally drug-resistant TB is resistant to all currently used drugs. It was first observed in 2003 in Italy, but not widely reported until 2012, and has also been found in Iran and India. There is some efficacy for linezolid to treat those with XDR-TB but side effects and discontinuation of medications were common. Bedaquiline is tentatively supported for use in multiple drug-resistant TB.


title: Tuberculosis

text: XDR-TB is a term sometimes used to define "extensively resistant" TB, and constitutes one in ten cases of MDR-TB. Cases of XDR TB have been identified in more than 90% of countries.


title: Tuberculosis

text: For those with known rifampicin or MDR-TB, molecular tests such as the Genotype® MTBDRsl Assay (performed on culture isolates or smear positive specimens) may be useful to detect second-line anti-tubercular drug resistance.


title: Tuberculosis

text: Progression from TB infection to overt TB disease occurs when the bacilli overcome the immune system defenses and begin to multiply. In primary TB disease (some 1–5% of cases), this occurs soon after the initial infection. However, in the majority of cases, a latent infection occurs with no obvious symptoms. These dormant bacilli produce active tuberculosis in 5–10% of these latent cases, often many years after infection.


title: Tuberculosis

text: The risk of reactivation increases with immunosuppression, such as that caused by infection with HIV. In people coinfected with "M. tuberculosis" and HIV, the risk of reactivation increases to 10% per year. Studies using DNA fingerprinting of "M. tuberculosis" strains have shown reinfection contributes more substantially to recurrent TB than previously thought, with estimates that it might account for more than 50% of reactivated cases in areas where TB is common. The chance of death from a case of tuberculosis is about 4% , down from 8% in 1995.


title: Tuberculosis

text: In people with smear-positive pulmonary TB (without HIV co-infection), after 5 years without treatment, 50-60% die while 20-25% achieve spontaneous resolution (cure). TB is almost always fatal in those with untreated HIV co-infection and death rates are increased even with antiretroviral treatment of HIV.


title: Tuberculosis

text: Roughly one-quarter of the world's population has been infected with "M. tuberculosis", with new infections occurring in about 1% of the population each year. However, most infections with "M. tuberculosis" do not cause disease, and 90–95% of infections remain asymptomatic. In 2012, an estimated 8.6 million chronic cases were active. In 2010, 8.8 million new cases of tuberculosis were diagnosed, and 1.20–1.45 million deaths occurred (most of these occurring in developing countries). Of these, about 0.35 million occur in those also infected with HIV. In 2018, tuberculosis was the leading cause of death worldwide from a single infectious agent. The total number of tuberculosis cases has been decreasing since 2005, while new cases have decreased since 2002.


title: Tuberculosis

text: Tuberculosis incidence is seasonal, with peaks occurring every spring and summer. The reasons for this are unclear, but may be related to vitamin D deficiency during the winter. There are also studies linking tuberculosis to different weather conditions like low temperature, low humidity and low rainfall. It has been suggested that tuberculosis incidence rates may be connected to climate change.


title: Tuberculosis

text: Tuberculosis is closely linked to both overcrowding and malnutrition, making it one of the principal diseases of poverty. Those at high risk thus include: people who inject illicit drugs, inhabitants and employees of locales where vulnerable people gather (e.g., prisons and homeless shelters), medically underprivileged and resource-poor communities, high-risk ethnic minorities, children in close contact with high-risk category patients, and health-care providers serving these patients.


title: Tuberculosis

text: The rate of tuberculosis varies with age. In Africa, it primarily affects adolescents and young adults. However, in countries where incidence rates have declined dramatically (such as the United States), tuberculosis is mainly a disease of the elderly and immunocompromised (risk factors are listed above). Worldwide, 22 "high-burden" states or countries together experience 80% of cases as well as 83% of deaths.


title: Tuberculosis

text: In Canada and Australia, tuberculosis is many times more common among the aboriginal peoples, especially in remote areas. Factors contributing to this include higher prevalence of predisposing health conditions and behaviours, and overcrowding and poverty. In some Canadian aboriginal groups, genetic susceptibility may play a role.


title: Tuberculosis

text: Socioeconomic status (SES) strongly affects TB risk. People of low SES are both more likely to contract TB and to be more severely affected by the disease. Those with low SES are more likely to be affected by risk factors for developing TB (e.g. malnutrition, indoor air pollution, HIV co-infection, etc.), and are additionally more likely to be exposed to crowded and poorly ventilated spaces. Inadequate healthcare also means that people with active disease who facilitate spread are not diagnosed and treated promptly; sick people thus remain in the infectious state and (continue to) spread the infection.


title: Tuberculosis

text: The distribution of tuberculosis is not uniform across the globe; about 80% of the population in many African, Caribbean, South Asian, and eastern European countries test positive in tuberculin tests, while only 5–10% of the U.S. population test positive. Hopes of totally controlling the disease have been dramatically dampened because of many factors, including the difficulty of developing an effective vaccine, the expensive and time-consuming diagnostic process, the necessity of many months of treatment, the increase in HIV-associated tuberculosis, and the emergence of drug-resistant cases in the 1980s.


title: Tuberculosis

text: In developed countries, tuberculosis is less common and is found mainly in urban areas. In Europe, deaths from TB fell from 500 out of 100,000 in 1850 to 50 out of 100,000 by 1950. Improvements in public health were reducing tuberculosis even before the arrival of antibiotics, although the disease remained a significant threat to public health, such that when the Medical Research Council was formed in Britain in 1913 its initial focus was tuberculosis research.


title: Tuberculosis

text: In 2010, rates per 100,000 people in different areas of the world were: globally 178, Africa 332, the Americas 36, Eastern Mediterranean 173, Europe 63, Southeast Asia 278, and Western Pacific 139.


title: Tuberculosis

text: Russia has achieved particularly dramatic progress with a decline in its TB mortality rate—from 61.9 per 100,000 in 1965 to 2.7 per 100,000 in 1993; however, mortality rate increased to 24 per 100,000 in 2005 and then recoiled to 11 per 100,000 by 2015.


title: Tuberculosis

text: China has achieved particularly dramatic progress, with about an 80% reduction in its TB mortality rate between 1990 and 2010. The number of new cases has declined by 17% between 2004 and 2014.


title: Tuberculosis

text: In 2007, the country with the highest estimated incidence rate of TB was Eswatini, with 1,200 cases per 100,000 people. In 2017, the country with the highest estimated incidence rate as a % of the population was Lesotho, with 665 cases per 100,000 people.


title: Tuberculosis

text: As of 2017, India had the largest total incidence, with an estimated 2,740,000 cases. According to the World Health Organization (WHO), in 2000–2015, India's estimated mortality rate dropped from 55 to 36 per 100,000 population per year with estimated 480 thousand people died of TB in 2015. In India a major proportion of tuberculosis patients are being treated by private partners and private hospitals. Evidence indicates that the tuberculosis national survey does not represent the number of cases that are diagnosed and recorded by private clinics and hospitals in India.


title: Tuberculosis

text: In the United States Native Americans have a fivefold greater mortality from TB, and racial and ethnic minorities accounted for 84% of all reported TB cases.


title: Tuberculosis

text: In the United States, the overall tuberculosis case rate was 3 per 100,000 persons in 2017. In Canada, tuberculosis is still endemic in some rural areas.


title: Tuberculosis

text: In 2017, in the United Kingdom, the national average was 9 per 100,000 and the highest incidence rates in Western Europe were 20 per 100,000 in Portugal.


title: Tuberculosis

text: Tuberculosis has existed since antiquity. The oldest unambiguously detected "M. tuberculosis" gives evidence of the disease in the remains of bison in Wyoming dated to around 17,000 years ago. However, whether tuberculosis originated in bovines, then transferred to humans, or whether both bovine and human tuberculosis diverged from a common ancestor, remains unclear. A comparison of the genes of "M. tuberculosis" complex (MTBC) in humans to MTBC in animals suggests humans did not acquire MTBC from animals during animal domestication, as researchers previously believed. Both strains of the tuberculosis bacteria share a common ancestor, which could have infected humans even before the Neolithic Revolution. Skeletal remains show some prehistoric humans (4000 BC) had TB, and researchers have found tubercular decay in the spines of Egyptian mummies dating from 3000 to 2400 BC. Genetic studies suggest the presence of TB in the Americas from about AD 100.


title: Tuberculosis

text: Before the Industrial Revolution, folklore often associated tuberculosis with vampires. When one member of a family died from the disease, the other infected members would lose their health slowly. People believed this was caused by the original person with TB draining the life from the other family members.


title: Tuberculosis

text: Although Richard Morton established the pulmonary form associated with tubercles as a pathology in 1689, due to the variety of its symptoms, TB was not identified as a single disease until the 1820s. Benjamin Marten conjectured in 1720 that consumptions were caused by microbes which were spread by people living close to each other. In 1819, René Laennec claimed that tubercles were the cause of pulmonary tuberculosis. J. L. Schönlein first published the name "tuberculosis" (German: "Tuberkulose") in 1832. Between 1838 and 1845, John Croghan, the owner of Mammoth Cave in Kentucky from 1839 onwards, brought a number of people with tuberculosis into the cave in the hope of curing the disease with the constant temperature and purity of the cave air; each died within a year. Hermann Brehmer opened the first TB sanatorium in 1859 in Görbersdorf (now Sokołowsko) in Silesia. In 1865, Jean Antoine Villemin demonstrated that tuberculosis could be transmitted, via inoculation, from humans to animals and among animals. (Villemin's findings were confirmed in 1867 and 1868 by John Burdon-Sanderson.)


title: Tuberculosis

text: Robert Koch identified and described the bacillus causing tuberculosis, "M. tuberculosis", on 24 March 1882. In 1905, he was awarded the Nobel Prize in Physiology or Medicine for this discovery. Koch did not believe the cattle and human tuberculosis diseases were similar, which delayed the recognition of infected milk as a source of infection. During the first half of the 1900s, the risk of transmission from this source was dramatically reduced after the application of the pasteurization process. Koch announced a glycerine extract of the tubercle bacilli as a "remedy" for tuberculosis in 1890, calling it "tuberculin". Although it was not effective, it was later successfully adapted as a screening test for the presence of pre-symptomatic tuberculosis. World Tuberculosis Day is marked on 24 March each year, the anniversary of Koch's original scientific announcement.


title: Tuberculosis

text: Albert Calmette and Camille Guérin achieved the first genuine success in immunization against tuberculosis in 1906, using attenuated bovine-strain tuberculosis. It was called bacille Calmette–Guérin (BCG). The BCG vaccine was first used on humans in 1921 in France, but achieved widespread acceptance in the US, Great Britain, and Germany only after World War II.


title: Tuberculosis

text: Tuberculosis caused widespread public concern in the 19th and early 20th centuries as the disease became common among the urban poor. In 1815, one in four deaths in England was due to "consumption". By 1918, TB still caused one in six deaths in France. After TB was determined to be contagious, in the 1880s, it was put on a notifiable-disease list in Britain; campaigns started to stop people from spitting in public places, and the infected poor were "encouraged" to enter sanatoria that resembled prisons (the sanatoria for the middle and upper classes offered excellent care and constant medical attention). Whatever the benefits of the "fresh air" and labor in the sanatoria, even under the best conditions, 50% of those who entered died within five years ( 1916). When the Medical Research Council formed in Britain in 1913, it initially focused on tuberculosis research.


title: Tuberculosis

text: In Europe, rates of tuberculosis began to rise in the early 1600s to a peak level in the 1800s, when it caused nearly 25% of all deaths. In the 18th and 19th century, tuberculosis had become epidemic in Europe, showing a seasonal pattern. By the 1950s mortality in Europe had decreased about 90%. Improvements in sanitation, vaccination, and other public-health measures began significantly reducing rates of tuberculosis even before the arrival of streptomycin and other antibiotics, although the disease remained a significant threat. In 1946, the development of the antibiotic streptomycin made effective treatment and cure of TB a reality. Prior to the introduction of this medication, the only treatment was surgical intervention, including the "pneumothorax technique", which involved collapsing an infected lung to "rest" it and to allow tuberculous lesions to heal.


title: Tuberculosis

text: Because of the emergence of multidrug-resistant tuberculosis (MDR-TB), surgery has been re-introduced for certain cases of TB infections. It involves the removal of infected chest cavities ("bullae") in the lungs to reduce the number of bacteria and to increase exposure of the remaining bacteria to antibiotics in the bloodstream. Hopes of eliminating TB ended with the rise of drug-resistant strains in the 1980s. The subsequent resurgence of tuberculosis resulted in the declaration of a global health emergency by the World Health Organization (WHO) in 1993.


title: Tuberculosis

text: Tuberculosis has been known by many names from the technical to the familiar. () is a Greek word for consumption, an old term for pulmonary tuberculosis; around 460 BCE, Hippocrates described phthisis as a disease of dry seasons. The abbreviation "TB" is short for "tubercle bacillus". "Consumption" was the most common nineteenth century English word for the disease. The Latin root meaning 'completely' is linked to meaning 'to take up from under'. In "The Life and Death of Mr Badman" by John Bunyan, the author calls consumption "the captain of all these men of death." "Great white plague" has also been used.


title: Tuberculosis

text: Tuberculosis was for centuries associated with poetic and artistic qualities among those infected, and was also known as "the romantic disease". Major artistic figures such as the poets John Keats, Percy Bysshe Shelley, and Edgar Allan Poe, the composer Frédéric Chopin, the playwright Anton Chekhov, the novelists Franz Kafka, Katherine Mansfield, Charlotte Brontë, Fyodor Dostoevsky, Thomas Mann, W. Somerset Maugham, George Orwell, and Robert Louis Stevenson, and the artists Alice Neel, Jean-Antoine Watteau, Elizabeth Siddal, Marie Bashkirtseff, Edvard Munch, Aubrey Beardsley and Amedeo Modigliani either had the disease or were surrounded by people who did. A widespread belief was that tuberculosis assisted artistic talent. Physical mechanisms proposed for this effect included the slight fever and toxaemia that it caused, allegedly helping them to see life more clearly and to act decisively.


title: Tuberculosis

text: Tuberculosis formed an often-reused theme in literature, as in Thomas Mann's "The Magic Mountain", set in a sanatorium; in music, as in Van Morrison's song "T.B. Sheets"; in opera, as in Puccini's "La bohème" and Verdi's "La Traviata"; in art, as in Monet's painting of his first wife Camille on her deathbed; and in film, such as the 1945 "The Bells of St. Mary's" starring Ingrid Bergman as a nun with tuberculosis.


title: Tuberculosis

text: In 2014, the WHO adopted the "End TB" strategy which aims to reduce TB incidence by 80% and TB deaths by 90% by 2030. The strategy contains a milestone to reduce TB incidence by 20% and TB deaths by 35% by 2020. However, by 2020 only a 9% reduction in incidence per population was achieved globally, with the European region achieving 19% and the African region achieving 16% reductions. Similarly, the number of deaths only fell by 14%, missing the 2020 milestone of a 35% reduction, with some regions making better progress (31% reduction in Europe and 19% in Africa). Correspondingly, also treatment, prevention and funding milestones were missed in 2020, for example only 6.3 million people were started on TB prevention short of the target of 30 million.


title: Tuberculosis

text: The World Health Organization (WHO), the Bill and Melinda Gates Foundation, and the U.S. government are subsidizing a fast-acting diagnostic tuberculosis test for use in low- and middle-income countries as of 2012. In addition to being fast-acting, the test can determine if there is resistance to the antibiotic rifampicin which may indicate multi-drug resistant tuberculosis and is accurate in those who are also infected with HIV. Many resource-poor places have access to only sputum microscopy.


title: Tuberculosis

text: India had the highest total number of TB cases worldwide in 2010, in part due to poor disease management within the private and public health care sector. Programs such as the Revised National Tuberculosis Control Program are working to reduce TB levels among people receiving public health care.


title: Tuberculosis

text: A 2014 EIU-healthcare report finds there is a need to address apathy and urges for increased funding. The report cites among others Lucica Ditui "[TB] is like an orphan. It has been neglected even in countries with a high burden and often forgotten by donors and those investing in health interventions."


title: Tuberculosis

text: Slow progress has led to frustration, expressed by the executive director of the Global Fund to Fight AIDS, Tuberculosis and Malaria – Mark Dybul: "we have the tools to end TB as a pandemic and public health threat on the planet, but we are not doing it." Several international organizations are pushing for more transparency in treatment, and more countries are implementing mandatory reporting of cases to the government as of 2014, although adherence is often variable. Commercial treatment providers may at times overprescribe second-line drugs as well as supplementary treatment, promoting demands for further regulations. The government of Brazil provides universal TB care, which reduces this problem. Conversely, falling rates of TB infection may not relate to the number of programs directed at reducing infection rates but may be tied to an increased level of education, income, and health of the population. Costs of the disease, as calculated by the World Bank in 2009 may exceed US$150 billion per year in "high burden" countries. Lack of progress eradicating the disease may also be due to lack of patient follow-up – as among the 250 million rural migrants in China.


title: Tuberculosis

text: There is insufficient data to show that active contact tracing helps to improve case detection rates for tuberculosis. Interventions such as house-to-house visits, educational leaflets, mass media strategies, educational sessions may increase tuberculosis detection rates in short-term. There is no study that compares new methods of contact tracing such as social network analysis with existing contact tracing methods.


title: Tuberculosis

text: Slow progress in preventing the disease may in part be due to stigma associated with TB. Stigma may be due to the fear of transmission from affected individuals. This stigma may additionally arise due to links between TB and poverty, and in Africa, AIDS. Such stigmatization may be both real and perceived; for example, in Ghana, individuals with TB are banned from attending public gatherings.


title: Tuberculosis

text: Stigma towards TB may result in delays in seeking treatment, lower treatment compliance, and family members keeping cause of death secret – allowing the disease to spread further. In contrast, in Russia stigma was associated with increased treatment compliance. TB stigma also affects socially marginalized individuals to a greater degree and varies between regions.


title: Tuberculosis

text: One way to decrease stigma may be through the promotion of "TB clubs", where those infected may share experiences and offer support, or through counseling. Some studies have shown TB education programs to be effective in decreasing stigma, and may thus be effective in increasing treatment adherence. Despite this, studies on the relationship between reduced stigma and mortality are lacking , and similar efforts to decrease stigma surrounding AIDS have been minimally effective. Some have claimed the stigma to be worse than the disease, and healthcare providers may unintentionally reinforce stigma, as those with TB are often perceived as difficult or otherwise undesirable. A greater understanding of the social and cultural dimensions of tuberculosis may also help with stigma reduction.


title: Tuberculosis

text: The BCG vaccine has limitations, and research to develop new TB vaccines is ongoing. A number of potential candidates are currently in phase I and II clinical trials. Two main approaches are used to attempt to improve the efficacy of available vaccines. One approach involves adding a subunit vaccine to BCG, while the other strategy is attempting to create new and better live vaccines. MVA85A, an example of a subunit vaccine, is in trials in South Africa as of 2006, is based on a genetically modified vaccinia virus. Vaccines are hoped to play a significant role in treatment of both latent and active disease.


title: Tuberculosis

text: To encourage further discovery, researchers and policymakers are promoting new economic models of vaccine development as of 2006, including prizes, tax incentives, and advance market commitments. A number of groups, including the Stop TB Partnership, the South African Tuberculosis Vaccine Initiative, and the Aeras Global TB Vaccine Foundation, are involved with research. Among these, the Aeras Global TB Vaccine Foundation received a gift of more than $280 million (US) from the Bill and Melinda Gates Foundation to develop and license an improved vaccine against tuberculosis for use in high burden countries.


title: Tuberculosis

text: A number of medications are being studied as of 2012 for multidrug-resistant tuberculosis, including bedaquiline and delamanid. Bedaquiline received U.S. Food and Drug Administration (FDA) approval in late 2012. The safety and effectiveness of these new agents are uncertain as of 2012, because they are based on the results of relatively small studies. However, existing data suggest that patients taking bedaquiline in addition to standard TB therapy are five times more likely to die than those without the new drug, which has resulted in medical journal articles raising health policy questions about why the FDA approved the drug and whether financial ties to the company making bedaquiline influenced physicians' support for its use.


title: Tuberculosis

text: Mycobacteria infect many different animals, including birds, fish, rodents, and reptiles. The subspecies "Mycobacterium tuberculosis", though, is rarely present in wild animals. An effort to eradicate bovine tuberculosis caused by "Mycobacterium bovis" from the cattle and deer herds of New Zealand has been relatively successful. Efforts in Great Britain have been less successful.


title: Tuberculosis

text: , tuberculosis appears to be widespread among captive elephants in the US. It is believed that the animals originally acquired the disease from humans, a process called reverse zoonosis. Because the disease can spread through the air to infect both humans and other animals, it is a public health concern affecting circuses and zoos.


title: William the Conqueror

text: William I (– 9 September 1087), usually known as William the Conqueror and sometimes William the Bastard, was the first Norman king of England, reigning from 1066 until his death in 1087. A descendant of Rollo, he was Duke of Normandy from 1035 onward. By 1060, following a long struggle to establish his throne, his hold on Normandy was secure. In 1066, following the death of Edward the Confessor, William invaded England, leading an army of Normans to victory over the Anglo-Saxon forces of Harold Godwinson at the Battle of Hastings, and suppressed subsequent English revolts in what has become known as the Norman Conquest. The rest of his life was marked by struggles to consolidate his hold over England and his continental lands, and by difficulties with his eldest son, Robert Curthose.


title: William the Conqueror

text: William was the son of the unmarried Duke Robert I of Normandy and his mistress Herleva. His illegitimate status and his youth caused some difficulties for him after he succeeded his father, as did the anarchy which plagued the first years of his rule. During his childhood and adolescence, members of the Norman aristocracy battled each other, both for control of the child duke, and for their own ends. In 1047, William was able to quash a rebellion and begin to establish his authority over the duchy, a process that was not complete until about 1060. His marriage in the 1050s to Matilda of Flanders provided him with a powerful ally in the neighbouring county of Flanders. By the time of his marriage, William was able to arrange the appointment of his supporters as bishops and abbots in the Norman church. His consolidation of power allowed him to expand his horizons, and he secured control of the neighbouring county of Maine by 1062.


title: William the Conqueror

text: In the 1050s and early 1060s, William became a contender for the throne of England held by the childless Edward the Confessor, his first cousin once removed. There were other potential claimants, including the powerful English earl Harold Godwinson, whom Edward named as king on his deathbed in January 1066. Arguing that Edward had previously promised the throne to him and that Harold had sworn to support his claim, William built a large fleet and invaded England in September 1066. He decisively defeated and killed Harold at the Battle of Hastings on 14 October 1066. After further military efforts, William was crowned king on Christmas Day, 1066, in London. He made arrangements for the governance of England in early 1067 before returning to Normandy. Several unsuccessful rebellions followed, but William's hold was mostly secure on England by 1075, allowing him to spend the greater part of his reign in continental Europe.


title: William the Conqueror

text: William's final years were marked by difficulties in his continental domains, troubles with his son, Robert, and threatened invasions of England by the Danes. In 1086, he ordered the compilation of the "Domesday Book", a survey listing all the land-holdings in England along with their pre-Conquest and current holders. He died in September 1087 while leading a campaign in northern France, and was buried in Caen. His reign in England was marked by the construction of castles, settling a new Norman nobility on the land, and change in the composition of the English clergy. He did not try to integrate his various domains into one empire but continued to administer each part separately. His lands were divided after his death: Normandy went to Robert, and England went to his second surviving son, William Rufus.


title: William the Conqueror

text: Norsemen first began raiding in what became Normandy in the late 8th century. Permanent Scandinavian settlement occurred before 911, when Rollo, one of the Viking leaders, and King Charles the Simple of France reached an agreement ceding the county of Rouen to Rollo. The lands around Rouen became the core of the later duchy of Normandy. Normandy may have been used as a base when Scandinavian attacks on England were renewed at the end of the 10th century, which would have worsened relations between England and Normandy. In an effort to improve matters, King Æthelred the Unready took Emma, sister of Richard II, Duke of Normandy, as his second wife in 1002.


title: William the Conqueror

text: Danish raids on England continued, and Æthelred sought help from Richard, taking refuge in Normandy in 1013 when King Swein I of Denmark drove Æthelred and his family from England. Swein's death in 1014 allowed Æthelred to return home, but Swein's son Cnut contested Æthelred's return. Æthelred died unexpectedly in 1016, and Cnut became king of England. Æthelred and Emma's two sons, Edward and Alfred, went into exile in Normandy while their mother, Emma, became Cnut's second wife.


title: William the Conqueror

text: After Cnut's death in 1035, the English throne fell to Harold Harefoot, his son by his first wife, while Harthacnut, his son by Emma, became king in Denmark. England remained unstable. Alfred returned to England in 1036 to visit his mother and perhaps to challenge Harold as king. One story implicates Earl Godwin of Wessex in Alfred's subsequent death, but others blame Harold. Emma went into exile in Flanders until Harthacnut became king following Harold's death in 1040, and his half-brother Edward followed Harthacnut to England; Edward was proclaimed king after Harthacnut's death in June 1042.


title: William the Conqueror

text: William was born in 1027 or 1028 at Falaise, Duchy of Normandy, most likely towards the end of 1028. He was the only son of Robert I, son of Richard II. His mother Herleva was a daughter of Fulbert of Falaise; he may have been a tanner or embalmer. Herleva was possibly a member of the ducal household, but did not marry Robert. She later married Herluin de Conteville, with whom she had two sons – Odo of Bayeux and Count Robert of Mortain – and a daughter whose name is unknown. One of Herleva's brothers, Walter, became a supporter and protector of William during his minority. Robert I also had a daughter, Adelaide, by another mistress.


title: William the Conqueror

text: Robert I succeeded his elder brother Richard III as duke on 6 August 1027. The brothers had been at odds over the succession, and Richard's death was sudden. Robert was accused by some writers of killing Richard, a plausible but now unprovable charge. Conditions in Normandy were unsettled, as noble families despoiled the Church and Alan III of Brittany waged war against the duchy, possibly in an attempt to take control. By 1031 Robert had gathered considerable support from noblemen, many of whom would become prominent during William's life. They included the duke's uncle Robert, the archbishop of Rouen, who had originally opposed the duke; Osbern, a nephew of Gunnor the wife of Richard I; and Gilbert of Brionne, a grandson of Richard I. After his accession, Robert continued Norman support for the English princes Edward and Alfred, who were still in exile in northern France.


title: William the Conqueror

text: There are indications that Robert may have been briefly betrothed to a daughter of King Cnut, but no marriage took place. It is unclear whether William would have been supplanted in the ducal succession if Robert had had a legitimate son. Earlier dukes had been illegitimate, and William's association with his father on ducal charters appears to indicate that William was considered Robert's most likely heir. In 1034 the duke decided to go on pilgrimage to Jerusalem. Although some of his supporters tried to dissuade him from undertaking the journey, he convened a council in January 1035 and had the assembled Norman magnates swear fealty to William as his heir before leaving for Jerusalem. He died in early July at Nicea, on his way back to Normandy.


title: William the Conqueror

text: William faced several challenges on becoming duke, including his illegitimate birth and his youth: the evidence indicates that he was either seven or eight years old at the time. He enjoyed the support of his great-uncle, Archbishop Robert, as well as King Henry I of France, enabling him to succeed to his father's duchy. The support given to the exiled English princes in their attempt to return to England in 1036 shows that the new duke's guardians were attempting to continue his father's policies, but Archbishop Robert's death in March 1037 removed one of William's main supporters, and conditions in Normandy quickly descended into chaos.


title: William the Conqueror

text: The anarchy in the duchy lasted until 1047, and control of the young duke was one of the priorities of those contending for power. At first, Alan of Brittany had custody of the duke, but when Alan died in either late 1039 or October 1040, Gilbert of Brionne took charge of William. Gilbert was killed within months, and another guardian, Turchetil, was also killed around the time of Gilbert's death. Yet another guardian, Osbern, was slain in the early 1040s in William's chamber while the duke slept. It was said that Walter, William's maternal uncle, was occasionally forced to hide the young duke in the houses of peasants, although this story may be an embellishment by Orderic Vitalis. The historian Eleanor Searle speculates that William was raised with the three cousins who later became important in his career – William fitzOsbern, Roger de Beaumont, and Roger of Montgomery. Although many of the Norman nobles engaged in their own private wars and feuds during William's minority, the viscounts still acknowledged the ducal government, and the ecclesiastical hierarchy was supportive of William.


title: William the Conqueror

text: King Henry continued to support the young duke, but in late 1046 opponents of William came together in a rebellion centred in lower Normandy, led by Guy of Burgundy with support from Nigel, Viscount of the Cotentin, and Ranulf, Viscount of the Bessin. According to stories that may have legendary elements, an attempt was made to seize William at Valognes, but he escaped under cover of darkness, seeking refuge with King Henry. In early 1047 Henry and William returned to Normandy and were victorious at the Battle of Val-ès-Dunes near Caen, although few details of the actual fighting are recorded. William of Poitiers claimed that the battle was won mainly through William's efforts, but earlier accounts claim that King Henry's men and leadership also played an important part. William assumed power in Normandy, and shortly after the battle promulgated the Truce of God throughout his duchy, in an effort to limit warfare and violence by restricting the days of the year on which fighting was permitted. Although the Battle of Val-ès-Dunes marked a turning point in William's control of the duchy, it was not the end of his struggle to gain the upper hand over the nobility. The period from 1047 to 1054 saw almost continuous warfare, with lesser crises continuing until 1060.


title: William the Conqueror

text: William's next efforts were against Guy of Burgundy, who retreated to his castle at Brionne, which William besieged. After a long effort, the duke succeeded in exiling Guy in 1050. To address the growing power of the Count of Anjou, Geoffrey Martel, William joined with King Henry in a campaign against him, the last known cooperation between the two. They succeeded in capturing an Angevin fortress but accomplished little else. Geoffrey attempted to expand his authority into the county of Maine, especially after the death of Hugh IV of Maine in 1051. Central to the control of Maine were the holdings of the Bellême family, who held Bellême on the border of Maine and Normandy, as well as the fortresses at Alençon and Domfront. Bellême's overlord was the king of France, but Domfront was under the overlordship of Geoffrey Martel and Duke William was Alençon's overlord. The Bellême family, whose lands were quite strategically placed between their three different overlords, were able to play each of them against the other and secure virtual independence for themselves.


title: William the Conqueror

text: On the death of Hugh of Maine, Geoffrey Martel occupied Maine in a move contested by William and King Henry; eventually, they succeeded in driving Geoffrey from the county, and in the process, William had been able to secure the Bellême family strongholds at Alençon and Domfront for himself. He was thus able to assert his overlordship over the Bellême family and compel them to act consistently with Norman interests. However, in 1052 the king and Geoffrey Martel made common cause against William at the same time as some Norman nobles began to contest William's increasing power. Henry's about-face was probably motivated by a desire to retain dominance over Normandy, which was now threatened by William's growing mastery of his duchy. William was engaged in military actions against his own nobles throughout 1053, as well as with the new Archbishop of Rouen, Mauger.


title: William the Conqueror

text: In February 1054 the king and the Norman rebels launched a double invasion of the duchy. Henry led the main thrust through the county of Évreux, while the other wing, under the king's brother Odo, invaded eastern Normandy. William met the invasion by dividing his forces into two groups. The first, which he led, faced Henry. The second, which included some who became William's firm supporters, such as Robert, Count of Eu, Walter Giffard, Roger of Mortemer, and William de Warenne, faced the other invading force. This second force defeated the invaders at the Battle of Mortemer. In addition to ending both invasions, the battle allowed the duke's ecclesiastical supporters to depose Archbishop Mauger. Mortemer thus marked another turning point in William's growing control of the duchy, although his conflict with the French king and the Count of Anjou continued until 1060. Henry and Geoffrey led another invasion of Normandy in 1057 but were defeated by William at the Battle of Varaville. This was the last invasion of Normandy during William's lifetime. In 1058, William invaded the County of Dreux and took Tillières-sur-Avre and Thimert. Henry attempted to dislodge William, but the siege of Thimert dragged on for two years until Henry's death. The deaths of Count Geoffrey and the king in 1060 cemented the shift in the balance of power towards William.


title: William the Conqueror

text: One factor in William's favour was his marriage to Matilda of Flanders, the daughter of Count Baldwin V of Flanders. The union was arranged in 1049, but Pope Leo IX forbade the marriage at the Council of Rheims in October 1049. The marriage nevertheless went ahead sometime in the early 1050s, possibly unsanctioned by the pope. According to a late source not generally considered to be reliable, papal sanction was not secured until 1059, but as papal-Norman relations in the 1050s were generally good, and Norman clergy were able to visit Rome in 1050 without incident, it was probably secured earlier. Papal sanction of the marriage appears to have required the founding of two monasteries in Caen – one by William and one by Matilda. The marriage was important in bolstering William's status, as Flanders was one of the more powerful French territories, with ties to the French royal house and to the German emperors. Contemporary writers considered the marriage, which produced four sons and five or six daughters, to be a success.


title: William the Conqueror

text: No authentic portrait of William has been found; the contemporary depictions of him on the Bayeux Tapestry and on his seals and coins are conventional representations designed to assert his authority. There are some written descriptions of a burly and robust appearance, with a guttural voice. He enjoyed excellent health until old age, although he became quite fat in later life. He was strong enough to draw bows that others were unable to pull and had great stamina. Geoffrey Martel described him as without equal as a fighter and as a horseman. Examination of William's femur, the only bone to survive when the rest of his remains were destroyed, showed he was approximately in height.


title: William the Conqueror

text: There are records of two tutors for William during the late 1030s and early 1040s, but the extent of his literary education is unclear. He was not known as a patron of authors, and there is little evidence that he sponsored scholarships or other intellectual activities. Orderic Vitalis records that William tried to learn to read Old English late in life, but he was unable to devote sufficient time to the effort and quickly gave up. William's main hobby appears to have been hunting. His marriage to Matilda appears to have been quite affectionate, and there are no signs that he was unfaithful to her – unusual in a medieval monarch. Medieval writers criticised William for his greed and cruelty, but his personal piety was universally praised by contemporaries.


title: William the Conqueror

text: Norman government under William was similar to the government that had existed under earlier dukes. It was a fairly simple administrative system, built around the ducal household, which consisted of a group of officers including stewards, butlers, and marshals. The duke travelled constantly around the duchy, confirming charters and collecting revenues. Most of the income came from the ducal lands, as well as from tolls and a few taxes. This income was collected by the chamber, one of the household departments.


title: William the Conqueror

text: William cultivated close relations with the church in his duchy. He took part in church councils and made several appointments to the Norman episcopate, including the appointment of Maurilius as Archbishop of Rouen. Another important appointment was that of William's half-brother, Odo, as Bishop of Bayeux in either 1049 or 1050. He also relied on the clergy for advice, including Lanfranc, a non-Norman who rose to become one of William's prominent ecclesiastical advisors in the late 1040s and remained so throughout the 1050s and 1060s. William gave generously to the church; from 1035 to 1066, the Norman aristocracy founded at least twenty new monastic houses, including William's two monasteries in Caen, a remarkable expansion of religious life in the duchy.


title: William the Conqueror

text: In 1051 the childless King Edward of England appears to have chosen William as his successor. William was the grandson of Edward's maternal uncle, Richard II of Normandy.


title: William the Conqueror

text: The "Anglo-Saxon Chronicle", in the "D" version, states that William visited England in the later part of 1051, perhaps to secure confirmation of the succession, or perhaps William was attempting to secure aid for his troubles in Normandy. The trip is unlikely given William's absorption in warfare with Anjou at the time. Whatever Edward's wishes, it was likely that any claim by William would be opposed by Godwin, Earl of Wessex, a member of the most powerful family in England. Edward had married Edith, Godwin's daughter, in 1043, and Godwin appears to have been one of the main supporters of Edward's claim to the throne. By 1050, however, relations between the king and the earl had soured, culminating in a crisis in 1051 that led to the exile of Godwin and his family from England. It was during this exile that Edward offered the throne to William. Godwin returned from exile in 1052 with armed forces, and a settlement was reached between the king and the earl, restoring the earl and his family to their lands and replacing Robert of Jumièges, a Norman whom Edward had named Archbishop of Canterbury, with Stigand, the Bishop of Winchester. No English source mentions a supposed embassy by Archbishop Robert to William conveying the promise of the succession, and the two Norman sources that mention it, William of Jumièges and William of Poitiers, are not precise in their chronology of when this visit took place.


title: William the Conqueror

text: Count Herbert II of Maine died in 1062, and William, who had betrothed his eldest son Robert to Herbert's sister Margaret, claimed the county through his son. Local nobles resisted the claim, but William invaded and by 1064 had secured control of the area. William appointed a Norman to the bishopric of Le Mans in 1065. He also allowed his son Robert Curthose to do homage to the new Count of Anjou, Geoffrey the Bearded. William's western border was thus secured, but his border with Brittany remained insecure. In 1064 William invaded Brittany in a campaign that remains obscure in its details. Its effect, though, was to destabilise Brittany, forcing the duke, Conan II, to focus on internal problems rather than on expansion. Conan's death in 1066 further secured William's borders in Normandy. William also benefited from his campaign in Brittany by securing the support of some Breton nobles who went on to support the invasion of England in 1066.


title: William the Conqueror

text: In England, Earl Godwin died in 1053 and his sons were increasing in power: Harold succeeded to his father's earldom, and another son, Tostig, became Earl of Northumbria. Other sons were granted earldoms later: Gyrth as Earl of East Anglia in 1057 and Leofwine as Earl of Kent sometime between 1055 and 1057. Some sources claim that Harold took part in William's Breton campaign of 1064 and swore to uphold William's claim to the English throne at the end of the campaign, but no English source reports this trip, and it is unclear if it actually occurred. It may have been Norman propaganda designed to discredit Harold, who had emerged as the main contender to succeed King Edward. Meanwhile, another contender for the throne had emerged – Edward the Exile, son of Edmund Ironside and a grandson of Æthelred II, returned to England in 1057, and although he died shortly after his return, he brought with him his family, which included two daughters, Margaret and Christina, and a son, Edgar the Ætheling.


title: William the Conqueror

text: In 1065 Northumbria revolted against Tostig, and the rebels chose Morcar, the younger brother of Edwin, Earl of Mercia, as earl in place of Tostig. Harold, perhaps to secure the support of Edwin and Morcar in his bid for the throne, supported the rebels and persuaded King Edward to replace Tostig with Morcar. Tostig went into exile in Flanders, along with his wife Judith, who was the daughter of Baldwin IV, Count of Flanders. Edward was ailing, and he died on 5 January 1066. It is unclear what exactly happened at Edward's deathbed. One story, deriving from the "Vita Ædwardi", a biography of Edward, claims that he was attended by his wife Edith, Harold, Archbishop Stigand, and Robert FitzWimarc, and that the king named Harold as his successor. The Norman sources do not dispute the fact that Harold was named as the next king, but they declare that Harold's oath and Edward's earlier promise of the throne could not be changed on Edward's deathbed. Later English sources stated that Harold had been elected as king by the clergy and magnates of England.


title: William the Conqueror

text: Harold was crowned on 6 January 1066 in Edward's new Norman-style Westminster Abbey, although some controversy surrounds who performed the ceremony. English sources claim that Ealdred, the Archbishop of York, performed the ceremony, while Norman sources state that the coronation was performed by Stigand, who was considered a non-canonical archbishop by the papacy. Harold's claim to the throne was not entirely secure, as there were other claimants, perhaps including his exiled brother Tostig. King Harald Hardrada of Norway also had a claim to the throne as the uncle and heir of King Magnus I, who had made a pact with Harthacnut in about 1040 that if either Magnus or Harthacnut died without heirs, the other would succeed. The last claimant was William of Normandy, against whose anticipated invasion King Harold Godwinson made most of his preparations.


title: William the Conqueror

text: Harold's brother Tostig made probing attacks along the southern coast of England in May 1066, landing at the Isle of Wight using a fleet supplied by Baldwin of Flanders. Tostig appears to have received little local support, and further raids into Lincolnshire and near the River Humber met with no more success, so he retreated to Scotland, where he remained for a time. According to the Norman writer William of Jumièges, William had meanwhile sent an embassy to King Harold Godwinson to remind Harold of his oath to support William's claim, although whether this embassy actually occurred is unclear. Harold assembled an army and a fleet to repel William's anticipated invasion force, deploying troops and ships along the English Channel for most of the summer.


title: William the Conqueror

text: William of Poitiers describes a council called by Duke William, in which the writer gives an account of a great debate that took place between William's nobles and supporters over whether to risk an invasion of England. Although some sort of formal assembly probably was held, it is unlikely that any debate took place, as the duke had by then established control over his nobles, and most of those assembled would have been anxious to secure their share of the rewards from the conquest of England. William of Poitiers also relates that the duke obtained the consent of Pope Alexander II for the invasion, along with a papal banner. The chronicler also claimed that the duke secured the support of Henry IV, Holy Roman Emperor, and King Sweyn II of Denmark. Henry was still a minor, however, and Sweyn was more likely to support Harold, who could then help Sweyn against the Norwegian king, so these claims should be treated with caution. Although Alexander did give papal approval to the conquest after it succeeded, no other source claims papal support prior to the invasion. Events after the invasion, which included the penance William performed and statements by later popes, do lend circumstantial support to the claim of papal approval. To deal with Norman affairs, William put the government of Normandy into the hands of his wife for the duration of the invasion.


title: William the Conqueror

text: Throughout the summer, William assembled an army and an invasion fleet in Normandy. Although William of Jumièges's claim that the ducal fleet numbered 3,000 ships is clearly an exaggeration, it was probably large and mostly built from scratch. Although William of Poitiers and William of Jumièges disagree about where the fleet was built – Poitiers states it was constructed at the mouth of the River Dives, while Jumièges states it was built at Saint-Valery-sur-Somme – both agree that it eventually sailed from Valery-sur-Somme. The fleet carried an invasion force that included, in addition to troops from William's own territories of Normandy and Maine, large numbers of mercenaries, allies, and volunteers from Brittany, northeastern France, and Flanders, together with smaller numbers from other parts of Europe. Although the army and fleet were ready by early August, adverse winds kept the ships in Normandy until late September. There were probably other reasons for William's delay, including intelligence reports from England revealing that Harold's forces were deployed along the coast. William would have preferred to delay the invasion until he could make an unopposed landing. Harold kept his forces on alert throughout the summer, but with the arrival of the harvest season he disbanded his army on 8 September.


title: William the Conqueror

text: Tostig Godwinson and Harald Hardrada invaded Northumbria in September 1066 and defeated the local forces under Morcar and Edwin at the Battle of Fulford near York. King Harold received word of their invasion and marched north, defeating the invaders and killing Tostig and Hardrada on 25 September at the Battle of Stamford Bridge. The Norman fleet finally set sail two days later, landing in England at Pevensey Bay on 28 September. William then moved to Hastings, a few miles to the east, where he built a castle as a base of operations. From there, he ravaged the interior and waited for Harold's return from the north, refusing to venture far from the sea, his line of communication with Normandy.


title: William the Conqueror

text: After defeating Harald Hardrada and Tostig, Harold left much of his army in the north, including Morcar and Edwin, and marched the rest south to deal with the threatened Norman invasion. He probably learned of William's landing while he was travelling south. Harold stopped in London, and was there for about a week before marching to Hastings, so it is likely that he spent about a week on his march south, averaging about per day, for the distance of approximately . Although Harold attempted to surprise the Normans, William's scouts reported the English arrival to the duke. The exact events preceding the battle are obscure, with contradictory accounts in the sources, but all agree that William led his army from his castle and advanced towards the enemy. Harold had taken a defensive position at the top of Senlac Hill (present-day Battle, East Sussex), about from William's castle at Hastings.


title: William the Conqueror

text: The battle began at about 9 am on 14 October and lasted all day, but while a broad outline is known, the exact events are obscured by contradictory accounts in the sources. Although the numbers on each side were about equal, William had both cavalry and infantry, including many archers, while Harold had only foot soldiers and few, if any, archers. The English soldiers formed up as a shield wall along the ridge and were at first so effective that William's army was thrown back with heavy casualties. Some of William's Breton troops panicked and fled, and some of the English troops appear to have pursued the fleeing Bretons until they themselves were attacked and destroyed by Norman cavalry. During the Bretons' flight, rumours swept through the Norman forces that the duke had been killed, but William succeeded in rallying his troops. Two further Norman retreats were feigned, to once again draw the English into pursuit and expose them to repeated attacks by the Norman cavalry. The available sources are more confused about events in the afternoon, but it appears that the decisive event was Harold's death, about which differing stories are told. William of Jumièges claimed that Harold was killed by the duke. The Bayeux Tapestry has been claimed to show Harold's death by an arrow to the eye, but that may be a later reworking of the tapestry to conform to 12th-century stories in which Harold was slain by an arrow wound to the head.


title: William the Conqueror

text: Harold's body was identified the day after the battle, either through his armour or marks on his body. The English dead, who included some of Harold's brothers and his housecarls, were left on the battlefield. Gytha Thorkelsdóttir, Harold's mother, offered the victorious duke the weight of her son's body in gold for its custody, but her offer was refused. William ordered that the body was to be thrown into the sea, but whether that took place is unclear. Waltham Abbey, which had been founded by Harold, later claimed that his body had been secretly buried there.


title: William the Conqueror

text: William may have hoped the English would surrender following his victory, but they did not. Instead, some of the English clergy and magnates nominated Edgar the Ætheling as king, though their support for Edgar was only lukewarm. After waiting a short while, William secured Dover, parts of Kent, and Canterbury, while also sending a force to capture Winchester, where the royal treasury was. These captures secured William's rear areas and also his line of retreat to Normandy, if that was needed. William then marched to Southwark, across the Thames from London, which he reached in late November. Next, he led his forces around the south and west of London, burning along the way. He finally crossed the Thames at Wallingford in early December. Stigand submitted to William there, and when the duke moved on to Berkhamsted soon afterwards, Edgar the Ætheling, Morcar, Edwin, and Ealdred also submitted. William then sent forces into London to construct a castle; he was crowned at Westminster Abbey on Christmas Day 1066.


title: William the Conqueror

text: William remained in England after his coronation and tried to reconcile the native magnates. The remaining earls – Edwin (of Mercia), Morcar (of Northumbria), and Waltheof (of Northampton) – were confirmed in their lands and titles. Waltheof was married to William's niece Judith, daughter of his half-sister Adelaide, and a marriage between Edwin and one of William's daughters was proposed. Edgar the Ætheling also appears to have been given lands. Ecclesiastical offices continued to be held by the same bishops as before the invasion, including the uncanonical Stigand. But the families of Harold and his brothers lost their lands, as did some others who had fought against William at Hastings. By March, William was secure enough to return to Normandy, but he took with him Stigand, Morcar, Edwin, Edgar, and Waltheof. He left his half-brother Odo, the Bishop of Bayeux, in charge of England along with another influential supporter, William fitzOsbern, the son of his former guardian. Both men were also named to earldoms – fitzOsbern to Hereford (or Wessex) and Odo to Kent. Although he put two Normans in overall charge, he retained many of the native English sheriffs. Once in Normandy the new English king went to Rouen and the Abbey of Fecamp, and then attended the consecration of new churches at two Norman monasteries.


title: William the Conqueror

text: While William was in Normandy, a former ally, Eustace, the Count of Boulogne, invaded at Dover but was repulsed. English resistance had also begun, with Eadric the Wild attacking Hereford and revolts at Exeter, where Harold's mother Gytha was a focus of resistance. FitzOsbern and Odo found it difficult to control the native population and undertook a programme of castle building to maintain their hold on the kingdom. William returned to England in December 1067 and marched on Exeter, which he besieged. The town held out for 18 days, and after it fell to William he built a castle to secure his control. Harold's sons were meanwhile raiding the southwest of England from a base in Ireland. Their forces landed near Bristol but were defeated by Eadnoth. By Easter, William was at Winchester, where he was soon joined by his wife Matilda, who was crowned in May 1068.


title: William the Conqueror

text: In 1068 Edwin and Morcar revolted, supported by Gospatric, Earl of Northumbria. The chronicler Orderic Vitalis states that Edwin's reason for revolting was that the proposed marriage between himself and one of William's daughters had not taken place, but another reason probably included the increasing power of fitzOsbern in Herefordshire, which affected Edwin's power within his own earldom. The king marched through Edwin's lands and built Warwick Castle. Edwin and Morcar submitted, but William continued on to York, building York and Nottingham Castles before returning south. On his southbound journey, he began constructing Lincoln, Huntingdon, and Cambridge Castles. William placed supporters in charge of these new fortifications – among them William Peverel at Nottingham and Henry de Beaumont at Warwick. Then the king returned to Normandy late in 1068.


title: William the Conqueror

text: Early in 1069, Edgar the Ætheling rose in revolt and attacked York. Although William returned to York and built another castle, Edgar remained free, and in the autumn he joined up with King Sweyn. The Danish king had brought a large fleet to England and attacked not only York but Exeter and Shrewsbury. York was captured by the combined forces of Edgar and Sweyn. Edgar was proclaimed king by his supporters. William responded swiftly, ignoring a continental revolt in Maine, and symbolically wore his crown in the ruins of York on Christmas Day 1069. He then proceeded to buy off the Danes. He marched to the River Tees, ravaging the countryside as he went. Edgar, having lost much of his support, fled to Scotland, where King Malcolm III was married to Edgar's sister Margaret. Waltheof, who had joined the revolt, submitted, along with Gospatric, and both were allowed to retain their lands. But William was not finished; he marched over the Pennines during the winter and defeated the remaining rebels at Shrewsbury before building Chester and Stafford Castles. This campaign, which included the burning and destruction of part of the countryside that the royal forces marched through, is usually known as the "Harrying of the North"; it was over by April 1070, when William wore his crown ceremonially for Easter at Winchester.


title: William the Conqueror

text: While at Winchester in 1070, William met with three papal legates – John Minutus, Peter, and Ermenfrid of Sion – who had been sent by the pope. The legates ceremonially crowned William during the Easter court. The historian David Bates sees this coronation as the ceremonial papal "seal of approval" for William's conquest. The legates and the king then proceeded to hold a series of ecclesiastical councils dedicated to reforming and reorganising the English church. Stigand and his brother, Æthelmær, the Bishop of Elmham, were deposed from their bishoprics. Some of the native abbots were also deposed, both at the council held near Easter and at a further one near Whitsun. The Whitsun council saw the appointment of Lanfranc as the new Archbishop of Canterbury, and Thomas of Bayeux as the new Archbishop of York, to replace Ealdred, who had died in September 1069. William's half-brother Odo perhaps expected to be appointed to Canterbury, but William probably did not wish to give that much power to a family member. Another reason for the appointment may have been pressure from the papacy to appoint Lanfranc. Norman clergy were appointed to replace the deposed bishops and abbots, and at the end of the process, only two native English bishops remained in office, along with several continental prelates appointed by Edward the Confessor. In 1070 William also founded Battle Abbey, a new monastery at the site of the Battle of Hastings, partly as a penance for the deaths in the battle and partly as a memorial to the dead. At an ecclesiastical council held in Lillebonne in 1080, he was confirmed in his ultimate authority over the Norman church.


title: William the Conqueror

text: Although Sweyn had promised to leave England, he returned in early 1070, raiding along the Humber and East Anglia toward the Isle of Ely, where he joined up with Hereward the Wake, a local thegn. Hereward's forces attacked Peterborough Abbey, which they captured and looted. William was able to secure the departure of Sweyn and his fleet in 1070, allowing him to return to the continent to deal with troubles in Maine, where the town of Le Mans had revolted in 1069. Another concern was the death of Count Baldwin VI of Flanders in July 1070, which led to a succession crisis as his widow, Richilde, was ruling for their two young sons, Arnulf and Baldwin. Her rule, however, was contested by Robert, Baldwin's brother. Richilde proposed marriage to William fitzOsbern, who was in Normandy, and fitzOsbern accepted. But after he was killed in February 1071 at the Battle of Cassel, Robert became count. He was opposed to King William's power on the continent, thus the Battle of Cassel upset the balance of power in northern France in addition to costing William an important supporter.


title: William the Conqueror

text: In 1071 William defeated the last rebellion of the north. Earl Edwin was betrayed by his own men and killed, while William built a causeway to subdue the Isle of Ely, where Hereward the Wake and Morcar were hiding. Hereward escaped, but Morcar was captured, deprived of his earldom, and imprisoned. In 1072 William invaded Scotland, defeating Malcolm, who had recently invaded the north of England. William and Malcolm agreed to peace by signing the Treaty of Abernethy, and Malcolm probably gave up his son Duncan as a hostage for the peace. Perhaps another stipulation of the treaty was the expulsion of Edgar the Ætheling from Malcolm's court. William then turned his attention to the continent, returning to Normandy in early 1073 to deal with the invasion of Maine by Fulk le Rechin, the Count of Anjou. With a swift campaign, William seized Le Mans from Fulk's forces, completing the campaign by 30 March 1073. This made William's power more secure in northern France, but the new count of Flanders accepted Edgar the Ætheling into his court. Robert also married his half-sister Bertha to King Philip I of France, who was opposed to Norman power.


title: William the Conqueror

text: William returned to England to release his army from service in 1073 but quickly returned to Normandy, where he spent all of 1074. He left England in the hands of his supporters, including Richard fitzGilbert and William de Warenne, as well as Lanfranc. William's ability to leave England for an entire year was a sign that he felt that his control of the kingdom was secure. While William was in Normandy, Edgar the Ætheling returned to Scotland from Flanders. The French king, seeking a focus for those opposed to William's power, then proposed that Edgar be given the castle of Montreuil-sur-Mer on the Channel, which would have given Edgar a strategic advantage against William. Edgar was forced to submit to William shortly thereafter, however, and he returned to William's court. Philip, although thwarted in this attempt, turned his attentions to Brittany, leading to a revolt in 1075.


title: William the Conqueror

text: In 1075, during William's absence, Ralph de Gael, the Earl of Norfolk, and Roger de Breteuil, the Earl of Hereford, conspired to overthrow William in the "Revolt of the Earls". Ralph was at least part Breton and had spent most of his life prior to 1066 in Brittany, where he still had lands. Roger was a Norman, son of William fitzOsbern, but had inherited less authority than his father held. Ralph's authority seems also to have been less than his predecessors in the earldom, and this was likely the cause of his involvement in the revolt.