the ability of dna excision-repair processes in diploid human fibroblasts to eliminate potentially cytotoxic and mutagenic lesions induced by uv radiation ( 254 nm ) was demonstrated in two ways : ( 1 ) cells with normal rates of excision were compared with cells with an intermediate rate of excision ( xp2be ) and cells with an excision rate less than or equal to 1 % that of normal ( xp12be ) for sensitivity to the killing and mutagenic action of uv radiation .
C0016030
fibroblasts
C0007634
cells