the drop in the level of atp reserves that induces the poor deformability of the erythrocytes in atherosclerosis patients would appear to be caused by two consecutive shortages : first a shortage that seems to be related to a deficiency of erythrocyte adenosine as the atp shortage disappears with dipyridamole treatment and then a shortage induced by the lack of erythrocyte glucose and suppressed by the addition of ticlopidine to the dipyridamole treatment .
C0001443
adenosine
C0001480
atp