increases in steady-state h2o2 in liver homogenates of rats treated with dehp , di ( 2-ethylhexyl ) adipate , or nafenopin , a hypolipidemic drug , correlated well with the carcinogenic potential of these chemicals determined in previous carcinogenicity studies , and are consistent with but not definitive evidence for the involvement of peroxisome proliferation in the hepatocarcinogenesis of these compounds .
C0023884
liver
C1512409
hepatocarcinogenesis