together our results suggest that in lysed cone models , myosin phosphorylation is sufficient for activating cone contraction , even in the absence of other ca2+-mediated events , that camp inhibition of contraction is mediated by camp-dependent phosphorylation of mlck , and that 10-20 mm mg2+ can activate actin-myosin interaction to produce contraction in the absence of myosin phosphorylation .
C0027120
mlck
C0031715
phosphorylation