to the extent that mnl beta 2-adrenergic receptors and adenylate cyclase activities reflect those of extravascular catecholamine target cells , these findings suggest that the heightened hyperglycemic response to epinephrine exhibited by patients with iddm is not due to increased sensitivity of cellular beta 2-adrenergic receptor-effector systems and is best attributed to the altered hormonal milieu of the insulin-deficient state .
C0011854
iddm
C0021641
insulin