the adp-ribosyltransferase ( or nad-glycohydrolase ) activity of the a-promoter ( the biggest subunit ) of iap , which is responsible for the principal action of the toxin , enhancing insulin secretory responses and thereby inhibiting epinephrine hyperglycemia , was not affected by acetamindination of lysine residues .
C0021641
insulin
C0020456
hyperglycemia