taken together , these data strongly suggest that ( i ) pma prevents cell-mediated cytotoxicity by modulating the disulfide-linked heterodimer associated to t3 and described as the receptor for antigen on the cell surface of major histocompatibility complex ( mhc ) and non-mhc specific ctl clones , without affecting the lytic mechanism per se , and ( ii ) the expression of the receptor for the antigen present on the tumor cell line k562 is not decreased on t3- nk clones after pma treatment and must be different from that on t3+ t-cell clones .
C0301896
cell-mediated cytotoxicity
C0003320
antigen