treatment with aco , however , caused statistically significant ( p less than .05 ) and dose-related increases from control in ( 1 ) microsomal protein and cytochrome p-450 content , and nadph-cytochrome c reductase , aryl hydrocarbon hydroxylase ( ahh ) , and 7-ethoxycoumarin-o-deethylase ( 7-ecod ) activities , and ( 2 ) cytosolic glutathione transferase activity of liver .
C0003927
aryl hydrocarbon
C0000644
7-ethoxycoumarin-o-deethylase