as a result , we believe its pathogenesis to be the obstructive mechanism of pancreatic ductuli , caused by epithelial papillary projection or mucin secreted by epithelial cells of mucinous metaplasia , and that the course to the terminal state of isolated islets is as follows : for the first time , we report that the atrophy and disappearance of acinar cells by obstructive mechanism of ductuli ( described above ) occur with fibrosis , inflammatory cellular infiltration , and fatty infiltration around the site of isolated islets , accompanied by gathering of islets .
C1327616
secreted
C0007634
cellular