the present study assessed the role of complement as a mediator of myocardial inflammation by quantifying products of complement activation including , the terminal complement complex ( tcc ) the cytolytic component of the complement system , c1rc1s-c1 inhibitor complex and c3bbbp complex , formed following activation of the classical and alternative pathway , respectively , and anaphylatoxins c3a and c5a in 41 patients following ami .
C0002791
anaphylatoxins
C0009510
c3a