the results of our phenotypic and functional testing of unmodified gic ( no enzyme treatment , no additional culture with or without interleukin 2 ) , show that t cells , especially okt8+ cells , are of paramount importance in the mechanism of this type of acute irreversible rejection of human renal allografts ( i.e. , to the point of allograft rupture ) , but other potential effector mechanisms are also present in situ .
C0021756
interleukin 2
C0086418
human