we have investigated the presence of au ag on 70 patients whose diagnostic was ra `` '' classical ( according to the judgement of the american rheumatism association ) . we used immunoelectrophoresis ( counter immunoelectrophoresis-ciep- ) and radioimmunoassay ( ria ) techniques . we prepared individual records including clinical , epidemic , biochemical and personal data . au ag investigation both through different techniques gave negative results in 70 cases , of which only four referred a possible previous hepatitis b clinic history , not showing during the investigation suggestive clinical data of hepatitis or biochemical alterations . panus et al . ( 1970 ) ( 5 ) , desche-labarte et al . ( 1972 ) , trempo ( 1972 ) , burrel ( 1973 ) ( 6 ) , serre et al . ( 1973 ) ( 8 ) , obtained negative results in the matter , with similar or lower casuistics than ours . notwithstanding roques et al . ( 1975 ) ( 7 ) , in 300 cases of ra showed the presence of au ag in a significant number of cases ( 5 % ) as compared to au ag incidence in the french population ( estimated in about 0.22 % ) ( 6 ) . however , roques did not find in their au ag positive cases any evidence which enables to think that this antigen has played an important role on the origin and development of the rheumatic illness . these important discrepancies may be due to different reasons : a ) use of different techniques for au ag detection .
C0019163
hepatitis
C0019168
au a