verapamil alone had no protective effect on reperfused myocardium , whereas dibunol , and particularly its combination with verapamil , essentially limited the size of necrosis , reduced plasma cpk activity and animal mortality rate and maintained ultrastructural integrity of cardiomyocytes , which was accompanied by a decrease of plasma lipid peroxidation products and a stabilization of cardiomyocyte sarcolemma .
C0027061
myocardium
C0225828
cardiomyocyte