experiments will be presented , and literature cited , that will allow us to make the following assumptions about the pathogenesis of fipv infection : 1 ) fipvs and fecvs represent a spectrum of viruses that differ only in infectivity ( ability to evoke seroconversion following oral infection ) and virulence ( ability to cause fip ) , 2 ) field isolates are generally nearer to fecvs in behavior than laboratory isolates made from animal passaged material , 3 ) immunity to fipv appears to be of the premunition type and is maintained for as long as the infection persists in a reactivatable form , 4 ) strains of feline coronaviruses that do not cause systemic disease , such as fecvs or low virulence fipvs , can actually sensitize cats to infection with virulent fipv strains , 5 ) felv infection interferes with established fip immunity and allows for the reactivation of disease in healthy carriers , 6 ) fipv may be passaged from queen to kitten either in utero or during neonatal life , and 7 ) kittens infected by their mothers with fipv do not usually develop fip but become immune carriers of the virus for a period of 5-6 months ;
C0009450
infection
C0012634
disease