in order to investigate whether the high aerobic glycolysis of the cancer cell can be explained on the basis of a lack of the regulatory function of pfk due to an altered isozyme distribution pattern , we compared the activity and isozymic profile of the enzyme from malignant cells of human leukemias , lymphomas , virus-transformed cell lines , and established malignant cell lines of lymphoid , myeloid , erythroid , and fibroblastic origin and their normal counterparts .
C0007634
the
C0016030
fibroblastic