commercial mixtures of polychlorinated biphenyls ( pcbs , aroclor 1016 , 1254 , and 1260 ) differing in their degree of chlorination and their accumulation in the brain were employed along with a peripheral monoamine oxidase inhibitor , debrisoquin sulfate ( declinax , ds ) to determine whether the rise in urinary homovanillic acid ( uhva ) following exposure to these pcbs is derived from the peripheral or central nervous system .
C0700449
debrisoquin sulfate
C0591329
declinax