these data indicate that a modest increase in acetaminophen sulfation , occurring alone following n-acetylcysteine treatment , is insufficient to explain the profound efficacy of the antidote in mice , and further suggest that this holds true for other species , such as humans , that are comparatively poor in the sulfoconjugation of acetaminophen .
C0001047
n-acetylcysteine
C0003295
antidote