from comparisons between the histologic and biochemical features of isografts and allografts , we suggest that ischemia-reperfusion injury initiates pancreatitis through oxygen-free radicals , and that the transformation to hemorrhagic pancreatic necrosis in allografts reflects the involvement of chemotactic immune factors and extracellular secretions from activated proteases .
C0030305
pancreatitis
C0030274
pancreatic