thus , glomerular injury in aa-ntsn , previously linked to prominent glomerular macrophage accumulation , is shown to be primarily dependent on a brisk antibody response to the planted heterologous ( sheep ) immunoglobulin , whereas cellular immunity , readily demonstrated in the splenic t cell compartment , is likely to be of lesser import in the pathogenesis of this lesion in the intact animal .
C0003241
antibody
C0021027
immunoglobulin