our working hypothesis to account for these changes postulates that soluble globular forms are unchanged because they are primarily associated with intrinsic cortical neurons that are relatively unaffected by ad , that chat and membrane-associated g4 ache decrease because they are primarily associated with incoming axons of cholinergic neurons that are abnormal in ad , and that asymmetric forms of ache increase because of an acrylamide-type impairment of fast axonal transport in diseased incoming cholinergic axons .
C0027882
neurons
C0004461
axons