on the basis of these studies we conclude : ( i ) disorders of the enterohepatic circulation and/or acceleration of metabolism of calcitriol are not responsible for its diminished level in oncogenous osteomalacia , ( ii ) the von kossa stain is preferred to the modified masson in evaluating osteomalacia , ( iii ) avascular necrosis of the femoral head may be part of the syndrome , ( iv ) heterogeneity may be the hallmark of the responsible mesenchymal tumor and account for the different histological interpretations in the literature , ( v ) in compliant patients with oncogenous osteomalacia , calcitriol and pi therapy may be effective .
C0012634
disorders
C0039082
syndrome