experimental evidence of microvascular hyperreactivity ( spasm ) , both in the hereditary cardiomyopathy of the syrian hamster and in the acquired cardiomyopathy of the hypertensive-diabetic rat , suggests that transient spasm of the myocardial microcirculation may lead to focal myocellular necrosis and scarring , which then causes the unaffected myocardial cells to undergo compensatory hypertrophy in order to normalize the increased load per unit of myocardium .
C0878544
cardiomyopathy
C0853897
cardiomyopathy