these assumptions together with elementary hemodynamic considerations , allow formulation of a simple renal hemodynamic model whose quantitative predications regarding the characteristics of rbf , gfr and tgf control are remarkably consistent with the literature : ( 1 ) the magnitude of tgf response is mainly dependent upon the myogenic cooperative amplification and ( 2 ) although the tgf mechanism is not involved in the autoregulative control of rbf and gfr , changes of the tgf function may shift the autoregulation curve to higher or lower rbf and blood pressure levels .
C1521970
characteristics
C0005823
blood pressure