it is concluded that the sustained recovery in group i resulted from the resolution not only of the acute vascular lesions but also of tubular necrosis and microangiopathy , and the postrecovery deterioration in group ii is attributed to the more severe renal damage initially , the progression of the chronic vascular lesions in uncompliant patients , and possibly hyperfiltration damage in the remaining nephrons .
C0022646
renal
C0027713
nephrons