these results are consistent with the following hypothesis : altered binding by the tumor of cyclic nucleotides to putative nuclear ' r-gulatory ' proteins ( e.g . protein kinase subunits , or possibly other high affinity cyclic nucleotide-binding proteins unrelated to protein kinases ) contributes to atative nuclear 'regularory ' proteins ( e.g . protein kinase subunits , or possibly other high affinity cyclic nucleotide-binding proteins unrelated to protein kinases ) contributes to and may be responsible for some of the differences in response to cyclic nucleotides that were observed .
C0031727
kinase
C0033640
protein kinases