enhancement of cardiac sympathetic nerve activity appeared to be responsible for the ventricular arrhythmias provoked by digoxin as 1 ) a temporal relationship was observed between augmented nerve activity and arrhythmia development , 2 ) a centrally acting sympathetic nervous system depressant drug , clonidine , converted the ventricular arrhythmia to normal rhythm , and 3 ) removal of sympathetic influence to the heart by spinal cord transection decreased the sensitivity of the heart to the arrhythmogenic effect of digoxin .
C0003811
arrhythmia
C0085612
ventricular arrhythmia