cardiac muscle physiology , cardiac dynamics and energetics of normotensive and hypertensive rats [ goldblatt ii , spontaneously hypertensive rats ( shr ) ] are analyzed in the light of the question of whether a shift in the isoenzyme pattern of myosin in favour of the isoenzyme vm-3 -- i.e. , transformation towards a slower muscle -- is the essential factor for manifestation of cardiac insufficiency under chronic haemodynamic overload .
C0018787
cardiac
C0018801
cardiac