the present study was initiated to determine if dna damage induced by the bifunctional anti-tumor alkylating agents melphalan , nitrogen mustard , a spontaneously activated derivative of cyclophosphamide or chlorambucil inhibits transcription in vitro , and to determine if the potential sites of transcription termination correlate with the sites of n7 guanine adducts predominantly formed by these agents .
C0040649
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C0597600
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