after a brief presentation of the pathogenetic scheme of renoparenchymal hypertension ( rph ) in acute glomerulonephritis , of the pathogenesis of not controlled by dialysis and renin-dependent rph , as well as of the more important causes for origination of arterial hypertension in patients with transplanted kidney , the author suggests the hypothesis , that the rph in patients with chri , prior to the inclusion of the chroniodialysis treatment , originates , is maintained and becomes more frequent and graver , very likely , due to the gradual and progressive accumulation of factors within the patient 's organism , elevating the arterial pressure , either by their reduced excretion through the affected kidneys or due to their increased production .
C0156221
acute glomerulonephritis
C0022646
kidneys