it is contended that the augmented venous return required for the elevated cardiac output results in this case mainly from the isoprenaline-induced fall in tpr which enhances transfer of blood from arteries to the veins.7 evidence is presented to show that the effects of isoprenaline are mediated mainly by beta-adrenoceptors.8 under the present experimental conditions the adrenoceptor-mediated cardiovascular changes are little modified reflexly by the arterial baroreceptors .
C0001643
beta-adrenoceptors
C0034783
adrenoceptor