### instruction:
If you are a doctor, please answer the medical questions based on the patient's description.

### input:
My dad, 90, passed on 3.15pm last Sunday in the hospital s Intermediate Care Area after being admitted late Saturday morning following paleness and abdominal discomfort ealier that morning at home. Heamoglobin level was dangerously low at 4 and blood transfusion was carried out with 40mg Furosemide initially by late evening, and subsequently Epoitine and the usual heart medications he has been on for years since heart bypass. He was previously diagnosed as having stage 4 renal impairment, suspectedly caused by Voltaren treatment following a fall in Nov 20102 which resulted in mild L2 fracture. His Creatinine level improved with Kestosterone and Epoitin (twice weekly) which was subsequently reduced to once weekly. I had stopped him from taking Furosemide, prescribed after his Edema event in March this year, because of adverse side effects and his condition improved with a diet of oats, multivatimins, kidney milk (Nepro for breakfast), fruit and vegetable juice (lunch) and soya milk (night/supper). Was death precipitated by iron insufficiency, leading to heart failure hence Edema and shutdown of kidneys after 40-80-80 doses of Furosemide did not release no more than 750ml of urine (on Saturday only)?

### output:

Hello because of the previous bypass surgery the heart was already week. Hemoglobin of 4 is severe anemia and hence it decreases the supply of oxygen making the heart pump harder ultimately causing it to decompensate and triggering an adverse cardiac event which could have led to his demise. His hemoglobin being on the lower side would have been because of iron deficiency and more importantly because of his chronic renal issues. Stopping lasix was not a wise choice and its CMS hold have been titrated to minimize its side effect. The treatment given by the doctor was appropriate and in accordance to international Guidelines. Regards Chat Doctor. 