Recent investigations in western countries have revealed LTs may be involved in the pathogenesis of SDB, and accelerate the progress of the disease by exacerbating local inflammatory response, then promoting the proliferation of the upper airway lymphoid tissues. And in the later pediatric investigation, it was determined that LTs production emerged disease severity-dependent increases in exhaled breath condensate of SDB patients. Nevertheless, few studies have investigated the role of LTs in systemic inflammation of SDB, and whether LTs productions is a independent risk factor of SDB, or is a obesity-dependent or ATH -dependent risk factor of SDB remains controversial. Moreover, no research work in China has been carried out to investigate the LTs pathway in a large population of Chinese snoring children with SDB.