[{"from": "human", "value": "Could you give me a brief overview of this molecule? [C][Branch1][=C][C][Branch1][#Branch1][O][C][Branch1][C][F][F][Branch1][C][F][F][Branch1][C][F][Cl]"}, {"from": "gpt", "value": "The molecule is only found in individuals that have used or taken this drug. It is an extremely stable inhalation anesthetic that allows rapid adjustments of anesthesia depth with little change in pulse or respiratory rate. Enflurane induces a reduction in junctional conductance by decreasing gap junction channel opening times and increasing gap junction channel closing times. Enflurane also activates calcium dependent ATPase in the sarcoplasmic reticulum by increasing the fluidity of the lipid membrane. It also appears to bind the D subunit of ATP synthase and NADH dehydogenase. Enflurane also binds to and angonizes the GABA receptor, the large conductance Ca<sup>2+</sup> activated potassium channel, the glycine receptor, and antagonizes the glutamate receptor receptor. These yield a decreased depolarization and therefore, tissue excitability which results in anesthesia."}]