[{"from": "human", "value": "Describe this molecule. [C][=C][C][=Branch1][=Branch2][=C][C][=Branch1][Ring2][=C][Ring1][=Branch1][F][C][=C][N][Branch1][Branch1][N][=N][Ring1][Branch1][C@H1][C@H1][Branch2][Branch1][=Branch2][C@H1][Branch2][Branch1][Ring1][O][C@H1][Branch1][Branch1][C@@H1][Ring1][=Branch1][O][S][C@H1][C@@H1][Branch2][Ring2][Ring2][C@H1][Branch1][=N][C@H1][Branch1][=Branch2][C@H1][Branch1][Ring2][O][Ring1][=Branch1][C][O][O][N][C][=C][Branch1][Branch1][N][=N][Ring1][Branch1][C][=C][C][=Branch1][=Branch1][=C][C][=C][Ring1][=Branch1][F][O][C][O][O]"}, {"from": "gpt", "value": "The molecule is an inhaled, small molecule inhibitor of galectin-3 (Gal-3), with potential anti-fibrotic activity. Upon administration, olitigaltin inhibits the activity of Gal-3. This may prevent the activation of alveolar macrophages and fibroblasts, and the resulting fibrosis in the lungs. Gal-3, a beta-galactoside-binding lectin, is expressed in multiple cell types including macrophages, fibroblasts, activated T-lymphocytes and epithelial cells. It is upregulated in fibrotic lung diseases including idiopathic pulmonary fibrosis (IPF) and interstitial pneumonia associated with collagen vascular disease (CVD-IP), and plays an important role in fibrosis in multiple organs. It is also often dysregulated in cancers, and may play a role in angiogenesis, cell proliferation and cell survival."}]