Unspecified fall [E888.9], Reason for Study: ^Reason: better define intracranial lesion History: S/P fall; s/p R craniotomy 2013; R proptosis Motion artifact degraded exam quality.There is no evidence of acute ischemic or hemorrhagic lesion on the scanThere is evidence of right frontal parietotemporal craniotomy with right temporal occipital encephalomalacia. Approximately 12 mm in thickness of dural thickening and enhancement are seen. No significant interval change since prior scan. There is no other abnormal enhancement.Patchy high signal intensities on bilateral periventricular white matter indicate mixed age indeterminate ischemic infarct and nonspecific small vessel ischemic disease. Again no change since prior scan.Susceptibility artifact lesion on the left parieto-occipital sulcus as well as right frontal parietal subdural space indicate prior hemorrhage related blood products. No change since prior scan.The ventricles, sulci and cisterns are symmetric and unremarkable. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear. Bilateral inguinal lens is not seen, no change since prior scan.
1. No evidence of acute ischemic or hemorrhagic lesion.2. Prior right frontal parietotemporal craniotomy with the underlying dural thickening with enhancement, no significant interval change in terms of dural thickening and enhancement pattern since prior scan.3. No change of right temporal occipital encephalomalacia with periventricular white matter changes since prior scan.