55 years, Female, structural abnormality underlying epilepsy, seizure protocol.. Multiple sequences are motion degraded which limits assessment for subtle abnormalities. No focal lesion to suggest seizure focus is appreciated. No intracranial mass or mass effect. Bilateral hippocampi are symmetric in size and without signal abnormality. No evidence of infarct or hemorrhage. There is abnormal configuration of the lateral ventricles with dilation of the bodies and occipital horns of the lateral ventricles, right greater than left, which may be developmental or related to remote white matter injury. There is thinning of the adjacent white matter without discrete signal abnormality. No hydrocephalus. Several scattered foci of T2/FLAIR hyperintensity are seen in the bilateral subcortical and periventricular white matter, which are nonspecific and may represent mild chronic small vessel ischemic changes. Major flow-voids are preserved. There are bilateral lens implants. There is mild right maxillary sinus mucosal thickening and minimal mucosal thickening elsewhere. Calvarium and remainder of the extracranial soft tissues are grossly unremarkable.
1. No findings to suggest a seizure focus are appreciated within the limits of motion degraded study.2. Abnormal configuration of the lateral ventricles which are disproportionately enlarged in the parieto-occipital region (colpocephaly). This finding may be developmental or related to remote white matter injury.3. Scattered foci of T2/FLAIR hyperintensity in the white matter are nonspecific and may represent mild chronic small vessel ischemic changes, sequela of prior inflammation, or less likely demyelination. 