History of NASH cirrhosis and hepatocellular carcinoma status post ablation. Interval follow-up exam. ABDOMEN:LIVER, BILIARY TRACT: Cirrhotic morphology liver with nodular surface contour and widened fissures. There is redemonstration of a segment 8 ablation cavity. There is no adjacent early arterial enhancement to indicate recurrent disease. No suspicious arterial enhancing lesions elsewhere in the liver.The portal vein is borderline enlarged measuring 13 mm in diameter. The portal veins are patent. The hepatic veins and hepatic arteries are also widely patent. There is recanalization of the umbilical vein as well as multiple prominent splenorenal collaterals measuring up to 9 mm.Cholelithiasis with chronic gallbladder wall thickening. No intrahepatic or extrahepatic biliary ductal dilatation.SPLEEN: Prominent splenomegaly with the spleen measuring 17.7 cm in length.PANCREAS: The pancreas has normal signal and morphology. The duct is within normal limits for size.ADRENAL GLANDS: No significant abnormality noted.KIDNEYS, URETERS: Bilateral simple renal cysts. No focal mass lesion. No hydronephrosis.RETROPERITONEUM, LYMPH NODES: No significant abnormality noted.BOWEL, MESENTERY: No significant abnormality noted.BONES, SOFT TISSUES: Note is made of abdominal wall venous collaterals.OTHER: Trace perihepatic ascites.
1.Hepatic cirrhosis. Unchanged ablation cavity in segment 8. No suspicious arterial enhancing lesions to indicate new or recurrent disease.2.Findings of portal hypertension including splenomegaly, perihepatic ascites, and splenorenal shunt formation.