44 years, Female, Reason: h/o Chronic pancreatitis with PRSS1 gene mutation. Pt needs secretin stimulating MRCP. History: Abdominal Pain. ABDOMEN:LUNG BASES: No significant abnormality notedLIVER, BILIARY TRACT: There is diffuse hepatic steatosis with regions of fatty sparing. A wedge-shaped segment 6 focus shows diffusion restriction, edema and progressive enhancement. Left hepatic subcentimeter simple cystsStatus post cholecystectomy. The common bile duct is mildly dilated to 9 mm with a small amount of stricturing in the distal common bile duct which is likely benign, no obstructing lesion delineated. Mild intrahepatic biliary ductal dilatation.SPLEEN: Multiple peri-splenic collateral vessels. The splenic vein is not visualized and likely thrombosed .PANCREAS: There are areas of narrowing involving the mid pancreatic duct with multiple dilated side branches. A blunted response to secretin is observed, compatible with element of exocrine dysfunction. There is diffusely decreased signal on T1-weighted images. Overall these findings suggest sequela of chronic pancreatitis.ADRENAL GLANDS: Two left adrenal lesions measuring 8 mm and 7 mm appear to lose signal on phase images, likely adenomas, are unchanged.KIDNEYS, URETERS: No significant abnormality noted.RETROPERITONEUM, LYMPH NODES: No significant abnormality noted.BOWEL, MESENTERY: No significant abnormality noted.BONES, SOFT TISSUES: No significant abnormality noted.
1.Findings of chronic pancreatitis.2.Thrombosis of the splenic vein.3.Wedge-shaped region of diffusion restriction, edema and enhancement within the liver. A vascular etiology is suspected, such as a small portal venous thrombus which is not directly visualized.4.Likely benign stricturing of the distal common bile duct with mild intrahepatic and extrahepatic biliary ductal dilatation.5.Left adrenal nodules are stable, likely adenomas.6.Contrast extravasation as above.