72 years, Male, Reason: neurodeg disease History: ataxia, cognetive. No restricted diffusion to suggest acute ischemia. No intracranial hemorrhage. No intracranial mass or mass-effect. There is global parenchymal volume loss supra and infratentorially without a distribution to suggest a particular neural degenerative process. There is volume loss involving the hippocampi bilaterally which is not disproportional. No focal signal abnormalities involving the basal ganglia or midbrain.Several foci of T2/FLAIR hyperintensity are seen in the bilateral subcortical and periventricular white matter as well as in the pons, which are nonspecific, but compatible with mild chronic small vessel ischemic disease. Brain parenchyma is otherwise unremarkable for age. No abnormal parenchymal or meningeal enhancement. Major flow-voids are preserved.Sella and orbits are grossly within normal limits. Paranasal sinuses and mastoid cells are clear. Bone marrow signal and extracranial soft tissues are within normal limits.
1. Global parenchymal volume loss. 2. No intracranial mass or mass effect. 3. Mild chronic small vessel ischemic disease.