60 years Male (DOB:7/27/1955)Reason: multifocal infarctions, suspect septic embolization History: as abovePROVIDER/ATTENDING NAME: RICHARD KRAIG RICHARD KRAIG A small focus of gyriform T2 and FLAIR hyperintensity is present along the right occipital lobe posteriorly at the inferior aspect of the right cuneus. There is associated gyriform T1 signal hyperintensity. There is no diffusion restriction associated with this. There is associated susceptibility effect. Small focus of susceptibility effect is present in the left occipital lobe along the left cuneus.A punctate foci of susceptibility effect and small foci of susceptibility effect scattered in the cerebellar hemispheres bilaterally.There is a mild degree of periventricular and subcortical punctate hyperintense white matter lesions present identified on the FLAIR and T2 images.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact.
1.There are hemorrhagic lesions present in the occipital lobes bilaterally but more on the right occipital lobe and the left. There is gyriform pattern suggests these are related to a vascular event. I suspect the related to subacute embolic events.2.Multiple small foci of hemorrhage are present in the cerebellar hemispheres. It is possible these are related to embolic events with some hemorrhagic conversion.3.Periventricular and subcortical white matter lesions of a mild degree are nonspecific. At this age they are most likely vascular related. 