Female, 24 years old, with neurofibromatosis type I, hydrocephalus, aqueduct stenosis. Evaluate for changes, tumors, CSF flow through 3rd VC. A right frontal approach ventriculostomy catheter appears to be in stable position. Ventricular size and morphology are stable and within normal limits. Evidence of third ventriculocisternostomy is again seen with CSF flow demonstrated across the defect.An oval, T2 hyperintense, nonenhancing lesion within the left splenium of the corpus callosum has increased in size now measuring 21 x 13 mm, previously 11 x 6 mm. Several additional small T2 hyperintense lesions are also redemonstrated without evidence of progression. In fact, a lesion in the right cerebral peduncle may even be somewhat smaller. As before, some of these lesions enhance, including those within the left temporal lobe, at the left foramen of Monro, and in the tectum. The tectal lesion causes narrowing of the cerebral aqueduct. A nonenhancing potentially cystic focus is redemonstrated in the pineal gland without significant interval change.No significant generalized parenchymal mass effect is seen. No evidence of intracranial hemorrhage or any abnormal extra-axial fluid collection is detected. Diffusion-weighted images are within normal limits.
1.Interval increase in size of the lesion within the left aspect of the splenium of the corpus callosum.2.Multiple additional stigmata of neurofibromatosis including scattered enhancing and nonenhancing lesions are otherwise unchanged, with one lesion perhaps slightly smaller.3.Stable positioning of the right frontal ventricular catheter. Stable ventricular size and morphology. The third ventriculocisternostomy remains patent.