Unspecified sequelae of cerebral infarction [I69.30], Reason for Study: ^Reason: r/o stroke and reperfusion injury History: neurological deficit after CEA There interval development of new restricted diffusion lesions on the left parietal lobe inferior parietal lobule indicating acute ischemic infarction.Previously seen left hemispheric ischemic infarction lesions on the left parietal lobe inferior parietal lobule and left frontal lobe show decreased extent of restricted diffusion areas but some still show diffusion restriction. In addition, there are small scattered susceptibility lesions on the left inferior parietal lobule indicating petechial hemorrhages which are more conspicuous on today's scan. However, the extent of susceptibility lesions are unchanged and there is no associated surrounding edema.There are cortical linear T1 high signal intensity lesions on the left frontal and parietal lobes indicating chronic ischemic lesions with laminar necrosis. Scattered right hemispheric chronic ischemic infarctions are seen, unchanged.Right cavernous sinus enhancing lesions indicating meningioma is again seen, unchanged since prior scan.The ventricles, sulci and cisterns are unremarkable. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
1. Interval development of new scattered restricted diffusion lesions involving left parietal lobe (series 202, images 144, 136).2. Evolving acute/subacute ischemic lesions on the left frontal and parietal lobes. Petechial hemorrhagic transformation are suggested but grossly unchanged since prior scan.3. Unchanged right hemispheric chronic ischemic lesions.4. Unchanged right cavernous sinus enhancing mass, meningioma, since prior scan.