text:
A fortunate fella hammers cutie in the butt snatch and unloads his blue balls giving a sticky facial! Feels like sex-hungry Secret Saturdays cunts haven't been screwed for the eternity! A bitch from Secret Saturdays shared between a two-way fuck with huge cocks that spatter her hot face with semen rain.
Drew Saturday shows her perfect big boobs
Hentai Picture: Drew Saturday shows her perfect big boobs
Secret Saturdays? Secret lezzies!
Hentai Picture: Secret Saturdays? Secret lezzies!
There's an abundance extreme and hardcore Secret Saturdays content for you from people being tied up to hole filling creampie content, gaping assholes with two cocks, group sex action and more. Let's follow the example of a babe from Secret Saturdays who takes cock directly on the cobblestones after making purchases just the other moment. The whore getting nude and filling her soaking twat with a red dildo…
Secret Saturdays Porn Story: Kylie Who – Chapter 8
Secret Saturdays Porn Story: Kylie Who – Chapter 8
yah this ch short but im sick so im kinda gtetting a headach enjoy
The Injury
Zaks POV
I woke up to a loud thud I then remembered that Kylies room was right next to mine and that is were I heard the noise I got up and walked sleepily over to Kylies door I knocked and it slowly swung open I walked in and I saw a figure on the ground I walked over to it and knelt down I saw it was Kylie I picked up her head and tried to shack her awake she didnt wake up and I felt something wet on my hand I put her back on the floor and looked on my hand I couldnt see anything so I got up and switched the lights on with my other hand. I looked back at my hand and I saw it was blood I ran back over to Kylie and looked at the back of her head it had a deep cut down the middle. I picked her up and I ran to the kitchen I knew my mom was going to be there.
Mom!!!!! I yelled as I ran into the kitchen my mom got up when she saw Kylie in my arms with a bleeding head
Secret Saturdays Porn Story: Kylie Who Chapter 3
Secret Saturdays Porn Story: Kylie Who Chapter 3
hey this is ch 3 PLEASE review
The Dinner
Zaks POV
I went to dinner it was me, mom, dad, Matt and a few other people. They were talking about Kylie and how smart she was. When mom heard this she asked
where is Kylie? Matt looked up and said
she wasnt hungry shes in her hammock me, mom, and dad all had the same look off confusion until my dad said
you mean room right?
No hammock, she shares a room with me and sleeps in a hammock near the roof.
Did she fall out like the way I did mom asked. Matt laughed and said
No, but even if she did she can catch herself with her snipper using her grappling arrow. She always keeps it on her belt even when she sleeps so never and I mean NEVER sneak up on her when she is sleeping because she will shoot you with her trapper arrow and you will be stuck to the wall or ground all day but luckily she always has it on stun so it wont hurt, you saw her Zak it is rare if she misses
Secret Saturdays Porn Story: Dreams Chapter 1
Secret Saturdays Porn Story: Dreams Chapter 1
Me: OOOH! My first Secret Saturdays fanfic!!!
Zak: GRATZ!!!!
Me: I OWN NOTHING! To the story!!
Chapter one
Another day in the life
Zak was lying on his bed, daydreaming, something he himself never thought he would do. He was thinking, however, about something most boys thought of. Girls. He heard the door to his room open and close. Someone sat on his bed, on the side his back was facing. Hey, little man, whats goin on? Zak immediately recognized the voice of his uncle, Doyle. Nothing. Just thinking. Zak mumbled. Right, like Im supposed to believe that? Especially when this is on your side table. Doyle held the book The Complete Works of William Shakespeare in front of Zaks face. He put it down on the table and stood again. Well, talk to ya later, bud. The door re-opened, then closed again.
Secret Saturdays Porn Story: Dreams – Chapter 2
Secret Saturdays Porn Story: Dreams – Chapter 2
Zak: This was for real….. well, part of this chapter, just the last two paragraphs
Me: I OWN NOTHING! To the story!!
Chapter two
Doc and Drew were saying goodbye as Doyle walked out the door. Thanks again for watching Zak. Drew said to her brother, hugging him goodbye. Doc shook his hand as he said Any time. Although.. Doyle stopped on the bottom porch step He was in his room, curled on his bed or reading for most of the day. Id try to see if I could get whats wrong out of him, If I were you. Drew and Doc looked at each other, worried. Thanks for keeping us informed, Doyle, well try to get it out of him. Drew said as her brother stated walking down the road, toward the gate. He looked over his shoulder and waved. Drew closed the door. Lets go check on Zak. She said. Doc nodded and followed her to their sons room. The door opened, and the two could see Zak sleeping. Drew walked in and sat on his bed. As she stroked his head softly, Doc spotted Zaks journal on the nightstand, key still in the lock. Well, whats this? He whispered, unlocking it. Doc! Thats Zaks personal stuff! Drew whispered back. So? Hes our son. And besides, The answer could be in here. Of course, she knew what he meant.
Secret Saturdays Porn Story: New Friends mean New Adventures Chapter 1
Secret Saturdays Porn Story: New Friends mean New Adventures Chapter 1
I dont own the Secret Saturdays. Cartoon Network does.
Zak! Wake up! Breakfast! Drew called out. Zak got up and woke up Fisk, Komodo and Zon. Race you guys down to the kitchen. Zak, Fisk, Komodo and Zon darted to the kitchen. I win Zak shouted Doc I wanna talk to you about Zak After breakfast Doc replied. After a few minutes So you want Zak to go to school. Not a bad idea. Zak needs some social skills. Lets do it. Doc and Drew told Zak about the plan What Ive plenty of friends. Zak protested Zak, Fiskerton, Komodo, and Zon dont count. You need normal non-cryptid friends. Its good for you. Drew replied. The next day the Saturdays checked out a school for gifted children. Im sure your child will love this school. All his needs is you two to fill out some papers and well be ready. an old lady gave Doc and Drew some papers and they left. The next day Zak went to the school. He heard something was going on and went to check it out. Out in the court yard a red hair boy with glasses, a green short sleeved shirt, green pants, a vest and tan shoes was fighting a black hair boy wearing a blue long sleeved shirt, blue pants and blue shoes. Okay you two break it up. If I catch you doing these again your suspended. Got that? A teacher shouted. The boys muttered Yes sir Then the two raced into the school You know they arent always like that? Zak turned his head to see a girl at leased a year older then himself standing next to him. She pulled out her hand. Im Toph Beifong you? Zak shook her hand Zak Saturday Later in class Zak met Mizore, a very shy girl. Hello my name is Mizore Im Zak. Then Zak saw the two boys he saw fighting earlier having a peaceful talk. See told they arent always like they were outside Toph said Im Tom and this Kaz The black hair boy pointed to himself then to the red hair boy Why were you two fighting? Zak asked. Because Chad and Max were out there. Their ravels and we dont know why Tom started Ya Chad takes care of me while Max takes care of Tom for our parents while were here. Kaz finished Okay get in your seats we have a new student joining us the teacher said as he came in. Zak walked to the front of the classroom and waved. You can sit next to Kaz. Zak took his seat and everything was pretty much okay. Zak was surprised to find out he shared the same schedule with his new friends. Later he met Cynder, Tophs neighbor, in science class. At math Zak noticed something familiar. Hello class Im Mr. Vook Ill be substituting today. To Zak his voice was familiar to Van Rooks but he didnt look like Rook so Zak brushed it off as coincidence. Nothing strange happen for the rest of the day. Soon everyone went home. As soon as Zak got home he was welcomed by Fisk and Komodo. Hey there guys wheres mom and dad? Zak asked. Zak went downstairs to the lab where they kept Kur Stone. Hi mom hi dad. Zak greeted What no hi for your uncle? a male voice said. Zak turned and shouted Uncle Doyle! Zak shouted and ran to hug his uncle. Hey Doc got something you might want Doyle reach into his pocket and the third piece of the Kur Stone. Trust me it wasnt easy Doyle said Thanks bro. So Zak how was school today? Drew asked Nothing big but made some friends Zak answered Good for you Zak Doc said Also the substitute teacher, Mr. Vook, sounded just like Van Rook Zak said Uh-oh Doyle went Whats uh-oh? Drew asked Mr. Vook is really one of Van Rooks fake names. You have to be careful his probably after you, Zak Doyle explained Whatever you do Zak, dont leave the room alone. Dont ever be in the alone in the hallway. Dont ever get in trouble with him. And dont ever stay at school by yourself. Hell most likely want you alone to capture you. Okay? Doyle explained Okay, Ill be careful. Zak replied. The next day Zak was greeted by hearing Tom and Kazs fake argument and Mizore coming outta nowhere. Ahh, do you always do that? Zak asked Sorry bad habit Mizore replied. After school, Zak went home as soon as possible. At home Zak was tackled by Fisk, Zon, and Komodo. Okay guys, wheres mom and dad? Fighting Argost without you so Im in charge Zak saw Doyle What? Ya they said it was too dangerous for you Doyle said. That night Doyle and Zak were watching some funny videos on T.V. At three in the morning Zak and Doyle fell asleep on the couch when the doorbell rang waking them up. Mom and dad are back Zak said Wait Zak your folks said that they wont be home till tomorrow besides remember, Van Rook after you. Komodo, Zon, watch my back, Fisk, watch Zak, and Zak, go hide Doyle ordered What? Go, now, Ill take care of this Doyle pulled out a gun. Zak went to his room, grabbed the Claw, ran to his parents room and crawled under the bed. Fiskerton followed him. Zak held the Claw close as he heard someone come in the room. Dont worry I know how to handle my former apprentice. The Saturday boy has to be around here somewhere Zak knew it was Van Rook But if he not here we still bill the guy? an unfamiliar voice said. Must be new apprentice Zak thought Now your learning, a little slower then my last apprentice whose now laying on the floor stabbed three times in the gut Van Rook said. A chill down Zaks spine. Soon he heard foot steps leaving the room. After a minute or two, Zak crawl out of hiding but then *whack*. Everything went black. Doyle, on the other hand, mange to contact Doc and Drew, despite the stab wounds. They came as soon as they could. Give up Van Rook your outmatched. Doc warned Where Zak? Drew demanded Who? That little punk whose out cold in your room or the guy whose throat I just cut? the apprentice asked. That made Doc and Drew really mad but Van Rook and his apprentice got away with smoke bombs. Drew, help Zak, Ill get your bother. Doc shouted. Doc found Doyle barely conscious at a door near Zons nest. Drew looked for Zak but she found was a broken window and some broken glass. Nothing else. The next day, Doc and Drew alerted the other Secret Scientists about their missing son. Dont worry sis, I doubt Van Rooks client likes damage goods. It clamed Doc and Drew a little knowing that Zak would be okay sort of. At Weird World Headquarters. Zak woke up in a cell with chains on his wrist and a figure standing in front of him. Greetings and Bienvenue, young Saturday. I hope youll like your new home not that you have much a choice Argost laughed. Wha what are talking about? Zak asked Well young Saturday you hold the power to control cryptids, so I need you to help me control Kur. So you can either join me or rot down here Zak spilt in Argosts eye Have it your way then, Munya teach him some manners Argost as he left the cell. Munya came and tied some cloth over Zaks eyes and mouth. Zak began to squirm when he felt a punch in his gut. Then another and an another until he couldnt move due to the pain. He look miserable. Give him some company Munya nodded grabbed a crate near him and ripped it open. Inside was a small black dragon. Munya tossed her inside and locked the cell. The dragon got to feet and noticed Zak. She pulled the cloth off and licked him. Thanks, I guess Zak said. The dragon laid her hear on his lap hoping he was right.
It seems that Drew Saturday has seen bigger monsters before…
Hentai Picture: It seems that Drew Saturday has seen bigger monsters before…
By the way,by the bye, aint'it a Secret Saturdays love-making one can spot there? Comely voluptuous bitch losing her clothes, massaging on her plump tits, stretching her cunt and giving her pussy a fuck with a thick fuck tool… This batch of wild porn toon will make you drool over all those fuck-crazed Secret Saturdays infatuation weirdoes enjoying themselves…
Drew Saturday – clad and not so much!
Hentai Picture: Drew Saturday – clad and not so much!
Drew Saturday taking all of her clothes off and fucking her leaky cunt with a scarlet sex toy. Secret Saturdays personages are so fuck-starved that they are ready to fuck anywhere, 24/7 and in every way you can think of. Crazy girl from Secret Saturdays is ready for the hardest fucking task of all her previous experience!
Drew Saturday knows that she is one marvelous cougar
Hentai Picture: Drew Saturday knows that she is one marvelous cougar
You've always dreamed to watch the Secret Saturdays hotties who are ever hungry for cocks above all. Drew Saturday can't wait to get shagged in her snatch and anal slit and when she gets it at last she squirts hard. Heavily jugged slut from Secret Saturdays is bobbing with a massive vaginal invasion here in the report!


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Best Basketball Movies Not Named 'Hoosiers' or 'Teen Wolf'
When it comes to basketball in the movies, a handful of hands down, no-brainer, blue chip flicks hold the top seed rankings of all time in the genre: Hoosiers. Teen Wolf. BASEketball. (Okay, maybe not BASEketball. ) But why do so many basketball movies -- including this week's Just Wright, in which Queen Latifah romances an injured NBA player played by rapper-turned-thespian Common, who TOTALLY, REALISTICALLY chooses her over Paula Patton -- combine the team sport with other random themes, like race relations and "Jeopardy!" (White Men Can't Jump), fathers and sons and America and religion (He Got Game), or Will Ferrell (Semi-Pro)?
Maybe it's the myriad number of metaphors a good, emotion-laden game of one-on-one can contain. Or the meaning in which team you back, right down to the shared fandom that can bring two people together in lurve -- like say, a physical therapist like Queen Latifah and a New Jersey Net like Common. Either way, why hate when you can celebrate? Here are five of the best "____ & Basketball" movies around:
Theme: Love & Basketball (Love & Basketball)
Let's start with the obvious one. Former collegiate track athlete Gina Prince-Bythewood made her feature-length debut with this romantic drama about Monica (Sanaa Lathan) and Quincy (Omar Epps), neighbors and college-bound basketball standouts that fall in love while navigating school and sports in late '80s-early '90s Los Angeles. In addition to avoiding the clichés that haunt both the sports and romance genres, Love & Basketball is one of very few sports flicks that shows attention to the ins and outs of balancing competition, love, and life as a female athlete -- and much of the credit goes to Lathan, whose turn as the driven ladies hoops player helped her break out as a lead actress. The film's Big Important Game, of course, is one of the most emotional games of one-on-one you'll ever see, a nighttime showdown of hurt feelings set to the sounds of Meshell Ndegeocello's "Fool of Me" where the prize at hand is -- what else? -- LOVE. Omar Epps = Best trophy ever.
Latest Entertainment Videos
Theme: Drugs & Basketball (The Basketball Diaries)
Catholic school teen Jim Carroll (Leonardo Di Caprio) is like a cheetah on the basketball court -- and an amoral troublemaker off of it. But for the meantime, life is good, and as long as the wins keep on coming, Jim and his pals at St. Vitus High are the kings of their world. When his terminally ill BFF passes away, Jim's downward spiral kicks off with an anarchic game of hoops in the rain with Marky Mark and their other hooligan buddies. (Does it look just a tad like that one Backstreet Boys music video? Maybe,) Then come the harder drugs. When he hits rock bottom, beat up and strung out, who nurses Jim back to health? Ernie Hudson, the wise former addict who used he used to play friendly pick-up games with at the park. By that point, basketball is replaced by drugs and pretentious voice-overs as the main motivating forces in Jim's life, and "pass the rock" takes on new meaning.
Theme: Literary Geniuses & Basketball (Finding Forrester)
If you're like me, the first thing you think of when you think about Finding Forrester is a white-bearded Sean Connery barking, "You're the man now, dawg!" Let's just agree, it wasn't 007's finest hour. The second thing you might think of then, is basketball -- more specifically, how basketball-as-salvation is a tired movie cliché that Gus Van Sant's drama turns on its head. Sixteen-year-old Jamal (Rob Brown) is an inner-city teen who's gifted both on and off the court, only he likes to keep his intellectual smarts secret. After striking up a friendship with a reclusive writer (Connery) who begins mentoring him, Jamal's inner writer emerges and he throws a key game in favor of earning his way in the world academically. And Sean Connery comes out of hiding to read an essay about friendship. So basketball is like writing because… it's like finding new teammates! In the game of life!
Theme: Statutory Rape & Basketball (Coach)
Look, back in the '70s, a high school kid who scored with the hot teacher was a campus hero in the teen sex comedy genre. And so, Michael Biehn circa 1978 became every high school kid's idol when he hooked up with Coach Randy Rawlings (Cathy Lee Crosby), the very female former Olympian hired on to whip a boys' basketball team into championship form in the Crown International picture, Coach. In Crosby's defense, her Coach Rawlings was as excellent at her job as she was unafraid of tackling the progress-resistant school board that hired her, and besides -- have you seen the young Michael Biehn?
Theme: Hip Hop & Basketball (Above the Rim)
Basketball and hip-hop culture have long shared a mutual acquaintance, but that particular link never was as strong as it was in the 1994 sports drama Above the Rim. Duane Martin (who was reportedly once drafted by the Knicks) starred as promising high school basketball player Kyle Watson, who must make a simple choice: play for the good guys in a local street ball tournament, or go over to the dark side and play for Tupac Shakur. Reviews were mixed, but the real historic impact of Above the Rim came in its soundtrack, packed with songs by the likes of 2Pac himself, Snoop Dogg, SWV, and Warren G., the latter of whom debuted the now-classic "Regulate" before officially releasing it on his own album months later. Above the Rim was even co-conceived by recording exec Benny Medina, who took a producing and story credit, and its last act tournament sequence features quite possibly the highest number of ostentatious street ball dunks and flashy moves seen in a single basketball movie to date.

subset: commoncrawl


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January 11th, 2019, 16:16 Posted By: wraggsterThe long-awaited noir psychological horror game Dollhouse of Soedesco and Creazn Studio has finally received its newest update. Recently, publisher Soedesco revealed the PlayStation 4 box art for the much-awaited horror title which can be seen below:
Dollhouse was first revealed by Soedesco in 2013 and was originally scheduled for a 2016 release on PC, Mac, Linux, and PlayStation 4. Unfortunately, the game has been pushed back for a 2017 release that resulted in another delay.
In 2018, no words from both publisher and developer have been heard about the noir psychological horror game, Dollhouse. Finally, the silence has been shattered by Soedesco and Creazn Studio when they recently revealed the box art for the game and guess what? A new release date for the game has been uncovered and it is expected to make its debut this 2019.
But, what is the game all about?
Dollhouse brings you to the unexpected side of Hollywood. The game is set in the enticing environment of Hollywood in the late 1950s. Follow the role of a detective who suffers from amnesia named Marie. Take control of her mind and help her in the mysterious things that haunt her in this noir psychological horror game.
Keep in mind that Marie only has a certain memory that was left with her, and it is about her daughter who has just died. And now, something terrible is after her. Are you ready to dive into the depths of Maries mind and unravel the mysteries of her past? Are you ready to embark on a terrifying journey?
Where to Pre-order the Game?
Although a concrete release for the game is still unknown as of this moment, pre-order for the game is already available here at Playasia. So, if you want to reserve your copies as early as now, you can secure yours here on our website. Click the button below to reserve the US version of the game.
For more information and downloads, click here!
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May is wettest month on record in Texas
Texas has never had a wetter month than this one.
According to state climatologist John Nielsen-Gammon, Texas has so far received an average of 7.54 inches of rain in May, which smashes the previous record of 6.66 inches, set in June 2004. Texas rain records go back 120 years.
The amount of water falling onto Texas this month is staggering, amounting to roughly 35 trillion gallons. That's as much rain as the mighty Mississippi river discharges over the course of three months. And even if every Texan had 50 large swimming pools, the rain would have filled all of them.
According to Nielsen-Gammon, the 10 highest rainfall months in state history were all over six inches per month. So for a single month to come along and jump all the way to 7.54 inches, with a few days left in May, is remarkable.
And it comes just four years after 2011, the worst one-year drought experienced by the state.
"The monthly rainfall record was annihilated this May about as badly as 2011 annihilated the drought records," said Nielsen-Gammon, a professor of atmospheric science at Texas A&M University.
About 10 to 15 inches of rain have fallen across Houston this month, which got much of that during the Memorial Day Weekend. Dallas has been the state's high-water mark with about 20 inches in May. Parts of Central Texas have gotten 18 inches.
At the San Antonio International Airport, the National Weather Service has recorded 7.05 inches so far this month, about three inches above normal but far from the record of 14.07 inches set in 1935. Combined with April's 7.54 inches, rainfall for the year is about 10 inches over the average.
The Edwards Aquifer, the main water supply for the San Antonio area, is also at its highest mark in more than three years, reaching 662.6 feet above sea level at the J-17 index well.
An active El Niño in the Pacific Ocean has driven the persistent rains across Texas by helping to steer the jet stream, a current of fast-moving air in the upper atmosphere, over the state. This has brought a steady flow of moisture and upper-level low pressure systems, which create the lift to form thunderstorms.
A wet May has pulled most of Texas out of the drought that began in late 2010. A year ago more than 55 percent of Texas remained in at least a "severe" drought, based upon data from the U.S. Drought Monitor. Now about 3 percent is, and falling.
Overall the state's reservoirs are now 82 percent full, says the Texas Water Development Board. That's up from less than 65 percent a few months ago, and it's the highest level since 2010.
Summer, of course, is coming.
That means the northern hemisphere jet stream, which can swoop down as far south as Texas during cooler months, will pull back north. During the summer months, the jet stream will more typically move over southern Canada and the upper Midwestern United States.
El Niño's influence on rainfall in Texas will wane. June could still be wet, but there's no reason to believe the wet pattern will extend into July or August.
In fact, some long-range forecast models indicate higher pressures over much of Texas and the Gulf of Mexico during these warmer months, which would mean the state receives less rain than normal.
So rain may be a pain now, but by August it may be a fond memory.


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Adobe Extension Platform: Launch
Launch is an Adobe website tag management solution. It empowers the customer to easily deploy and manage analytics, marketing, and advertising integrations. It offers many benefits including dynamic & faster deployment, data centralization, and powerful experiences through the integration of data and marketing technology.
More details are available in this overview page.
Setup Adobe Launch JS Tagging
Log in to Adobe Launch
First of all, you need to set up your Adobe account and login to Adobe Launch.
Create a Property for your website
If you haven't Adobe JS Tagging Property set up for your website, set it up using this guide.
Next, you'll need to set up these Property parameters to make it a bridge between CNECT and Adobe Analytics.
Add "Adobe Analytics" Extension
If you haven't "Adobe Analytics" Extension added for your Adobe JS Tagging Property, set it up using this guide.
Create Data Elements
Next, you'll need to create Data Elements for values you want to pass from CNECT to Adobe Analytics. To do it, go to "Data Elements" section and click "Add Data Element".
You'll need to create 6 similar Data Elements: for "CNECT Company", "CNECT Batch", "CNECT Product", "CNECT Experience", "CNECT Tag", and "CNECT Device".
Configuration for Company:
Configuration for Batch:
Configuration for Product:
Configuration for Experience:
Configuration for Tag (it's important to call query parameter "tagId"):
Configuration for Device (it's important to call query parameter "device_id"):
After Data Elements setup their list should look like this:
Setup Rules that pass Data Elements from Launch to Analytics
Next, you'll need to set up Rules that pass Launch Data Elements to Analytics. Go to the "Rules" section and click "Create New Rule".
Send AA Beacon Rule
Setting up this rule is only required if you haven't it already set up. Please skip this step if you already have "Send AA Beacon" rule.
Create "Send AA Beacon" ("AA" for Adobe Analytics), add "Core - DOM Ready" event and "Adobe Analytics - Send Beacon" action.
"Core - DOM Ready" event configuration:
"Adobe Analytics - Send Beacon" action configuration:
Set AA
Create "Set AA Variables" ("AA" for Adobe Analytics), add "Core - Page Bottom" event and "Adobe Analytics - Set Variables" action.
"Core - Page Bottom" event configuration:
"Adobe Analytics - Set Variables" action configuration:
Build, test and deploy the changes using the Launch publishing workflow
Publishing the library involves setting up Adapter, Environment and building the Library.
Detailed instructions of these steps:
Setup CNECT Products/Batches/Experiences for Adobe Launch
Log in to CNECT
Setup tracked Product
Go to "Products" and click "CREATE PRODUCT"
Fill all required fields and in "Product URL" enter the URL according to the following pattern:
and check "Send tag ID as a URL parameter" and "Send device unique ID as a URL parameter" in "Advanced" URL settings.
Setup tracked Batch
Go to "Batches" and click "CREATE BATCH"
Fill all required fields and in "Batch URL" enter the URL according to the following pattern:
and check "Send tag ID as a URL parameter" and "Send device unique ID as a URL parameter" in "Advanced" URL settings.
Setup tracked Experience
Go to "Experience" and create Experience.
Fill all required fields
In "Experience URL" enter the URL according to the following pattern:
and check "Send tag ID as a URL parameter" and "Send device unique ID as a URL parameter" in "Advanced" URL settings.
Now you have your Product, Batch, and Experience set up for passing the data to Adobe Launch.
Setup Adobe Analytics reports
Open Adobe Analytics
Setup basic report for Data Element
Data Elements that you've set up during Launch JS Tagging Setup are Custom Insights in Analytics. To generate the basic report of some property, find it using search and click.
There are different visualization options, but basically, the report will look like this:
Create custom charts for Data Elements in Workspace
Adobe Analytics Workspace allows you to create visualizations of selected metrics/dimensions. Let's see several examples of visualizations you can create for the data from CNECT.
Drag and drop "Map" from "Visualizations" menu into the workspace. It'll create empty frame for the future chart.
Then in the "Components" menu find "Occurrences" and drag and drop it into the "Add metric" input, select "Geographic Dimension" below and click "Build".
You'll get the map with locations of CNECT tap events:
From the "Visualizations" menu drag and drop "Donut" to the workspace, then find Custom Insight for the corresponding prop created during Launch setup (for example, if during Launch setup you linked Company to prop2, Custom Insight 2 will mean Company in Analytics). Drag and drop selected Custom Insight into the Donut chart data source ("Area Stacked Data" in this example).
You'll get Donut chart that shows what part each Company took in the general number:
Setup Real-Time report
Adobe Analytics also allows you to create Real-Time reports for the data you set up in Launch. To create such report go to "Reports" and find "Real-Time" using search.
It will open Real-Time report page, there you need to click "Configure".
In configuration, you can create Real-Time reports for 3 Custom Insights that correspond to props configured in Adobe Launch Property Rules.
After saving it you'll get a 3-tab Real-Time report.


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Jumper Triplet Bleach
- i/small - Sold Out
- ii/medium - Sold Out
- iii/large
- Details
- Shipping
- Share
Double thick and a cozy blend of wool and alpaca, this high neck jumper fits close to the body - but not too close. The ribbing is left in a single layer, to not impede your movements and comfort, while the body is a double layer of yarn, giving it a pillowy texture. What else could you desire for the depths of winter?
- Made in Belgium
- 50% wool, 50% alpaca
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An explosive day for the McYongs…Halloween is finally here! We have had several months of intense discussions about what the Princess will be for this hallowed eve. Of course a princess would be way too obvious, so Addison decided she wanted to be Halema'uma'u. Yes for you geology geeks that is the crater in the Kilauea caldera on the Big Island of Hawaii. It is actively spewing lava right now and we were able to see the glow when we were there in July.
So I got to work with some googling of "DIY volcano costume" and was pleasantly surprised to see the internet erupting with possibilities. (Now I can't stop the volcano puns…don't you lava them?) Thanks to the skillful sewing of Auntie Jennifer I think we created something pretty smoking hot. Charlie and I are of course dressed as hula girls. Ran out of time to get Aaron a Hawaiian shirt so he is just a daddy. 🙂
Hope everyone has a safe and happy Halloween!
Elaine, Aaron, Addison and Charlie


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Info of JSSC SBCCE Exam Date is reached to the applicants only through the JSSC Special Branch Constable Admit Card 2019. The details published in the www.jssc.nic.in SBCCE 2019 Admit Card varies for each applicant. Hence to know the JSSC Special Branch Constable Exam Date recommended, download the JSSC Special Branch admit card.
Jharkhand Staff Selection Commission (JSSC) when checking the requirement of 1012 Special Branch Constable Vacancy will come to the conclusion regarding the JSSC SBCCE 2019 Exam Date. Eventually, JSSC Special Branch Constable Admit Card 2019 also liberated as per the test dates.
Post Details
JSSC Special Branch Constable Admit Card 2019
JSSC before conducting the written exam to all the applicants of 1012 Special Branch Constable Vacancy, will hint the details like exam date, the venue through the Jharkhand Special Branch Constable Admit Card 2019. One who is having this admit card with them, they don't any guidance from anyone. Directly they can attend the examination as per the information printed in the admit card. JSSC Special Branch Constable Admit Card 2019 will be generated to each and every contender whose application got the status of accepted from the official recruitment board who takes the application forms. JSSC will decide a date regarding the release of the admit card. Candidates from that particular date can start the JSSC Special Branch admit card saving process.
Jharkhand Police Constable Recruitment
Jharkhand Special Branch Constable Admit Card
To get the attendance in the JSSC SBCCE Exam, this JSSC Special Branch Constable Admit Card 2019 is the permission ticket. In your prescribed for,m you cannot carry it to the exam hall. Only in the format that is recommended exam holders have to show it to the exam hall invigilators. Jharkhand Special Branch Constable Admit Card will release in online mode through a separate link of the JSSC Special Branch admit card. That released admit card has to be modified into the form of hard copy and must be brought by the competitor. If you don't show your admit card then you are ineligible to take the exam question paper also. In fact, permission to get into the exam center is quite impossible. Because of not following the rules instructed regarding the JSSC SBCCE Admit Card 2019 candidates may lose the best employment chance.
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|Organization Name
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|Special Branch Constable
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|1012 Posts
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|13th Aug 2019
|Official Site
JSSC SBCCE 2019 Exam Date
After seeing how many candidates are really interested to write the JSSC SBCCE Examination 2019 the board might schedule the JSSC Special Branch Constable Exam Date of either the written exam or physical tests. Special Branch Constable vacancy filling selection process have the rounds like a written exam, physical tests, interview, and document verification. The order of the selection rounds will be decided by JSSC after the application registration ended. Recruitmentindia.in be focussed on the www.jssc.nic.in page firstly to inform that Jharkhand Special Branch Constable Exam Date. Aware of the exam date is quite useful for the applicants for planning the time for syllabus preparation. Due to this reason, our first priority is to post the JSSC SBCCE Exam Date here.
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www.jssc.nic.in 2019 Hall Ticket
Jharkhand Staff Selection Commission (JSSC) when uploading the JSSC SBCCE 2019 Admit Card instructed the applicants to get the admit cards individually. For this individual admit card downloading process applicants required the login details. Those login credentials are nothing but the application registration id given to each one when you submit the form finally. And the password to that id is your date of birth in most of the cases. In the www.jssc.nic.in Special Branch Constable 2019 Hall Ticket, the board printed the set of rules and guidelines instructed to the exam attendees. Neglecting any one of the rule effects the candidate's attendance in the examination. Have an extra copy of JSSC Special Branch Constable Admit Card 2019 for result checking or any other usage.
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The first "genetic diet" may appear in 3-4 years, say experts from the United States. To date, there is no single diet that would help all people with obesity obesity. However, scientists plan to fix it soon.
Each person has an individual set of genes. Now genetic information is used for the selection of drugs against cancer. In the foreseeable future genetic analysis will help in selecting the optimal diet, which has high efficiency in combination with security.
This approach, I'm sure the experts will allow people to quickly deal with being overweight and not resign ourselves to additional risks associated with losing kilograms.


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Strong Hold Styling Paste is a balanced blend of lightweight ingredients to give all-day hold.The product maintains firm hold and texture through a blend of beeswax, castor seed oil and PVP. Perfect for structured, short hair styling. All day firm hold and matte texture.
To Use: Heat the product by rubbing it between your hands before applying. Apply to damp or dry hair and mould to create a defined and structured look.


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1. Introduction
Adenosine is a nucleoside distributed throughout the entire organism as an intermediary metabolite. At the extracellular level, adenosine plays multiple physiologic roles, interacting with specific receptors: A1, A2A, A2B and A3 (Fredholm et al., 2001). While the A1Rs and A3Rs are coupled in an inhibitory way to adenylate cyclase through the Gαi/o protein, the A2Rs are coupled in a stimulatory way to this enzymatic activity through Gαs protein (Ralevic & Burnstock, 1998).
Adenosine levels are increased after ischemia, hypoxia, excitotoxicity, inflammation and cerebral lesions. In these situations, it is considered that high adenosine levels play a neuroprotective role (Ribeiro et al., 2002). Interestingly, adenosine regulates the release of glutamate, the main excitatory neurotransmitter of the nervous system (Sebastiao & Ribeiro, 1996). A1Rs are widely expressed in the brain and have been shown to modulate neuronal excitability by decreasing pre-synaptic release of various neurotransmitters (Fredholm & Dunwiddie, 1988). The most dramatic inhibitory actions are on the glutamatergic system (Masino et al., 2002). In the central nervous system (CNS), A1Rs are associated with neuroprotective processes (Angulo et al., 2003; Dunwiddie and Masino, 2001). Moreover, they are upregulated in human neurodegenerative diseases with abnormal protein aggregates and it is related to compensatory mechanisms (Albasanz et al., 2007, 2008; Angulo et al., 2003; Perez-Buira et al., 2007; Rodríguez et al., 2006). Regarding A2ARs, these receptors are concentrated in the striatum, modulating dopaminergic activity, but they are also present in the hippocampus and cerebral cortex, modulating the glutamate release in the brain. Adenosine activity through A2 receptors (A2ARs) can eventually give rise to neurotoxicity, neuronal damage and cellular death (de Mendoça et al., 2000). In fact, A2ARs activity is associated with the outcome of cerebral injury as well as the development of Aβ-induced synaptotoxicity (Canas et al., 2009; Cunha, 2005; Stone et al., 2009).
2. Human brain A2AR localization and implications in PD pathophysiology
As mentioned in the previous section, A2ARs are G protein-coupled receptors that stimulate adenylyl cyclase through Gαs proteins, promoting accumulation of intracellular cAMP (Van Calker et al., 1979). The activation of these receptors mediates multiple physiological effects of adenosine, both in the CNS and in peripheral tissues (Fredholm et al., 2001). Pharmacological activation of A2ARs promotes vasodilatation, immunosuppression, tissue protection, sleep promotion and depression (Cerqueira, 2004; El Yacoubi et al., 2003; Linden, 2001; Satoh et al., 1998).
A2ARs are widely expressed, but they are highly concentrated in spleen, thymus, leukocytes and blood platelets. A2ARs levels in immune cells play a critical role in the protection of normal tissues by attenuating inflammation and tissue damage in vivo (Ohta and Sitkovsky, 2001). In the CNS, A2ARs are highly expressed in the striatum (Peterfreund et al., 1996; Schiffmann et al., 1991). Most striatal neurons (95%) are GABAergic medium spiny neurons (MSNs) which can be divided into two subtypes. One subpopulation projects to the globus pallidus and contains enkephalin. The other subpopulation projects to the substantia nigra and contains substance P and dynorphin. These neurons receive inputs from glutamatergic afferents from cortical, thalamic and limbic areas and dopaminergic afferents from the substantia nigra pars compacta and the ventral tegmental area. MSNs promote two striatal efferent pathways, the "direct" and "indirect", affecting motor activation and inhibition, respectively. The MSNs of the direct pathway correspond to the subpopulation containing dynorphin and they also express dopamine D1 receptors, whereas indirect MSNs express enkephalin, dopamine D2 receptors (D2Rs) and A2ARs (Schiffmann et al., 2007). In these cells, A2ARs physically interact (oligomerize) with D2R, and this receptor-receptor interaction results in a tidy adenosine/dopamine functional interaction controlled by the A2AR/D2R oligomer. Consequently, two reciprocal antagonistic A2AR/D2R interactions have been described, namely an intermembrane interaction in which A2AR mediates the inhibition of D2R, thus modulating neuronal excitability and neurotransmitter release, and an interaction at the level of adenylyl-cyclase in which D2R inhibits A2AR-mediated protein phosphorylation and gene expression (for review see Ciruela et al., 2011). As a result of this interaction, antagonists of A2ARs have recently emerged as a leading candidate class of non-dopaminergic anti-parkinsonian agents, based in part on the unique CNS distribution of A2ARs and A2AR/D2R oligomers (Fuxe et al., 2003). Moreover, the metabotropic glutamate receptors 5 (mGluR5s) are co-localized in the same GABAergic striatal output neurons and in glutamatergic nerve terminals in the striatum, and they form heteromeric complexes with A2ARs (Ferré et al., 2002; Rodrigues et al., 2005). This co-localization provides a morphological framework for the existence of multiple mGlu5/A2A/D2 receptor interactions (Cabello et al., 2009). Thus, it is proposed that the increase in glutamate and adenosine extracellular levels activates A2AR and mGluR5, both synergizing and promoting the inhibition of D2Rs (Ferré et al., 2007).
Of note is the characterization of A1R-A2AR heteromers with antagonistic activities between the two receptors, preferentially at presynaptically level in glutamatergic terminals of cortico-striatal afferents to the MSNs (Ciruela et al., 2006; Quiroz et al., 2009). Under baseline conditions, reduced levels of extracellular adenosine stimulate the activity of A1Rs while glutamatergic neurotransmission is inhibited. Under conditions of neuronal excitability, the extracellular adenosine levels are increased, showing A2ARs affinity and inhibiting A1R activity, and promoting the release of glutamate, which in turn also increases activation of mGluR5 synergizing with A2ARs and thereby facilitating more glutamate release (Rodrigues et al., 2005).
Interestingly, striatal A2ARs expression levels have been found to be increased in PD patients with dyskinesias; this upregulation is attributed to the effect of levodopa (L-dopa) treatment (Calon et al., 2004). Recently, it has been proven that high A2ARs levels in the striatum and in lymphocytes correlate with motor symptoms in PD patients who were previously either not pharmacologically treated or were treated with a wide spectrum of drugs and not restricted to only L-dopa (Varani et al., 2010). Therefore, A2AR upregulation in PD, which tonically inhibits D2R (see above), together with the low dopamine content in the striatum, a consequence of the death of dopaminergic neurons from the substantia nigra, contribute to a synergistic impairment of D2R function.
It remains to be clarified whether upregulation of A2ARs levels in PD is a hallmark of the disease or is a consequence of dopaminergic terminal drop-off in the striatum. This issue has been quite controversial, as increased striatal levels of A2ARs were shown in 6-hydroxidopamine (6-OHDA)-treated rats, as a consequence of dopamine denervation (Pinna et al., 2002), and also in 6-OHDA-treated rats with intermittent L-dopa treatment (Tomiyama et al., 2004).
3. Clinical trials with A2AR antagonists in PD
L-dopa remains the most effective treatment for symptomatic relief of PD, although its pharmacological administration over time induces motor dysfunctions such as dyskinesias (Obeso et al., 2000). One of the strategies to reduce these is the administration of
non-dopaminergic drugs that modulate dopaminergic neurotransmission. Indeed, A2AR has emerged as a potential pharmacological target in PD, as its relationship with the dopaminergic system has been clearly demonstrated (Ferré et al., 2002). There have been several clinical trials with A2AR antagonists, such as istradefylline (also known as KW-6002), confirming that their administration to PD patients reduces the "OFF" time and dyskinesias induced by L-dopa treatment (Bara-Jimenez et al., 2003; Factor et al., 2010; Hauser et al., 2003, 2008; LeWitt et al., 2008b; Mizuno et al., 2010; Stacy et al., 2008). As A2ARs expression levels are nearly exclusive of the striatum, the use of specific antagonists for these receptors could promote a specific brain-area effect (Brooks et al., 2008). Interestingly, it has recently been described how KW-6002 preferentially targets the A2AR within the postsynaptic A2AR/D2R oligomer located at the MSNs, thus potentiating D2R-mediated motor activation (Orru et al., 2011). Therefore, although the use of istradefylline as a monotheraphy in PD has not been statistically demonstrated (Fernandez et al., 2010), its administration as a coadjuvant seems to allow a reduction in the L-dopa dose.
As mentioned before, there are heteromers formed by A2AR, D2R and mGluR5 in striatal GABAergic neurons or MSNs (Cabello et al., 2009). It is proposed that the increase in glutamate and adenosine extracellular levels activates A2AR and mGluR5, both synergizing and promoting the inhibition of D2Rs (Ferré et al., 2007). Interestingly, it has been demonstrated in a rat model of PD that the simultaneous inhibition of A2AR and mGluR5 synergistically reverses the parkinsonian deficits in these animals (Coccurello et al., 2004). In this line, the use of mGluR type I antagonists as a therapy for PD has been proposed (Bonsi et al., 2007). Overall, this explains why pharmaceutical companies are going after either single compounds or combinations of drugs that will simultaneously antagonize A2AR and mGluR5.
4. Brain DNA methylation
Studies in mice deficient in enzymes that control DNA methylation and the results of folate-free diets have established an important role for methylation in the development of the nervous system (Waterland & Jirtle, 2003). In fact, the manipulation of methylation and acetylation affects neuronal vulnerability in experimental models of neurodegenerative diseases (El-Maarri, 2003). It has also been proposed that memory processes are highly related with the level of neuronal methylation (Day & Sweatt, 2010; Liu et al., 2009).
DNA methylation is a normal process that occurs during mammal embryo development, and it is also implicated in X-chromosome inactivation and repression of proviral genes and endogenous transposons. This chemical modification is one of the most important epigenetic mechanisms in gene silencing in mammals. It is characterized by the methylation of cytosines that precede guanines in the well-known CpG sites. Those genome CpG-rich regions are called CpG islands (CGIs); they present a size between 200 bp and several kilobases. In general, these active gene promoter regions are not methylated, while the inactive gene promoter regions are fully methylated. Most CGIs are found in 5' UTR regions and in the first exon, although they can be found in regions distal to the transcription start site and in intronic regions. In normal tissues, CGIs are usually non-methylated, while they are methylated in tumorous cells, especially in tumor repressor genes (Illingworth & Bird, 2009; Jones, 1999).
Methylation of CpG sites is achieved by the action of DNA metyltransferases (Dnmt) which catalyze the transfer of a methyl group from S-adenosyl-L-methionine (SAM) to DNA. The human DNA Methylome map has recently been published, annotating those genes found methylated in normal tissues and in human diseases such as Alzheimer's disease (AD) and schizophrenia (SZ) (Ballestar & Esteller, 2008). DNA methylation in the CpG sites interferes with gene expression in two ways. The first is interference with the binding of transcription factors to DNA through the methyl group. The second is caused by the binding of specific proteins, such as MeCP2, MBD1 and MBD2, to methyl CpG sites (methyl-CpG-binding proteins, MBDs). These MBDs recruit histone-modifying and chromatin-remodeling complexes to methylated sites (Portela & Esteller, 2010). The importance of these proteins is demonstrated by Rett syndrome, a disease causing severe mental dysfunction and brought on by MeCP2 mutations (Amir et al., 1999).
The role of DNA methylation in the brain is an emerging field of research. Neuronal DNA methylation is modified with lifespan, and the analysis of 12 loci related with AD has revealed an age-specific epigenetic drift in the percentage of DNA methylation (Siegmund et al. 2007; Wang et al., 2008). Moreover, the degree of gene methylation varies among the different cerebral regions, and it has been reported that DNA methyltransferase 1 (Dnmt1) expression levels are different in the various cerebral regions (Ladd-Acosta et al., 2007). Interestingly, Dnmt 1 is increased in the cortical interneurons where the GAD67 gene is suppressed in SZ patients (Veldic et al., 2004, 2005).
Finally, epigenetic therapies such as the use of demethylating agents are widely established in the treatment of tumors (Herranz & Esteller, 2006), but their use in neurodegeneration is poorly studied. In this line, S-adenosylmethionine (SAM) is a methyl group donor molecule necessary for DNA methylation which is reduced in AD (Linnebank et al., 2010; Morrison et al., 1996). There have been proposals to use it as a therapy for AD (Scarpa et al., 2003). Its administration to cell lines down-regulates PSEN1 and reduces β-amyloid production. In contrast, deprivation of SAM up-regulates PSEN1, increasing β-amyloid deposits in APP transgenic mice (Fuso et al., 2005, 2008). Interestingly, mice treated with L-methionine downregulate GAD67 and reelin levels by increasing DNA methylation of their respective gene promoter regions (Tremolizzo et al., 2002).
5. Endogenous SAM biosynthesis cycle and alterations in PD
SAM is the main biological methyl donor molecule in the methionine metabolic cycle, which is involved in the methylation of DNA, and protein, lipid and polyamine synthesis. Moreover, it is a precursor of glutathione in the liver and also perhaps in the brain (Vitvitsky et al, 2006). When SAM is demethylated, it is transformed into S-adenosylhomocysteine (SAH) which in turn is hydrolysed into homocysteine (HCY) and adenosine. To prevent the accumulation of HCY, it is remethylated to form methionine (Chiang et al., 1996; Lu, 2000) (see Figure 1). The SAM/SAH ratio is also known as methylation potential and its endogenous maintenance is very important.
L-dopa is the conventional drug used in the treatment of PD to minimize the lack of endogenous dopamine in these patients (Lewitt, 2008a; Tolosa et al., 1998). However, chronic treatment with L-dopa has been associated with hyperhomocysteinemia in plasma, peripheral tissues and brain of PD patients, as L-dopa metabolism requires S-adenosylmethionine (SAM) as a methyl donor (Lu, 2000; Müller et al., 2009a; Nutt, 2008; O'Suilleabhain et al, 2004; Zoccolella et al., 2006, 2009, 2010). Interestingly, it has been shown that L-dopa treatment in mice depletes the brain SAM content (Liu et al., 2000). In addition, elevated plasma homocysteine (Hcy) levels have been related to cognitive and motor impairment and have also been associated with the pathogenesis of other neurological diseases such as stroke and AD (Morris, 2003; Quadri et al., 2004; Seshadri et al., 2002). Interestingly, some polymorphisms described in MTHFR gene (methylenetetrahydrofolate reductase) have been associated with a reduction in its enzymatic activity, promoting an increase in the Hcy levels in L-dopa-treated PD patients (Frosst et al., 1995; Yasui et al., 2000). Vitamin B6 enhances the direct flow of Hcy to cysteine, the precursor of gutathione. Deficits in vitamin B6 induce oxidative stress and, in turn, enhance Hcy (Obeid et al., 2009). Therefore, several factors have been related with hyperhomocysteinemia and a concomitant reduction in the SAM levels in blood and cerebrospinal fluid of L-dopa-treated PD patients (Cheng et al., 1997). These include the following: 1. An excessive production of S-adenosylhomocysteine (SAH) when L-dopa is metabolized by catechol-O-methyltransferase (COMT), depleting the SAM levels an in turn decreasing the SAM/SAH ratio; 2. Reduced MTHFR enzymatic activity; and 3. Vitamin B12 or folic acid deficits (Dos Santos et al., 2009; Miller et al., 2003; Müller et al., 2001, 2009b; Woitalla et al., 2004). In this context, some clinical trials have been carried out using decarboxylase and COMT inhibitors (for instance, carbidopa and entacapone/tolcapone, respectively) or vitamin B12 and folate supplementation to reduce Hcy levels in PD patients (Müller et al., 2003, 2006; Zoccolella et al., 2007).
6. Epigenetic study of ADORA2A
A2AR gene (ADORA2A) is localized to chromosome 22 (Le et al., 1996; MacCollin et al., 1994; Peterfreund et al., 1996). It consists of two coding exons (exon 2 and 3) separated by a single intron of nearly 7 Kb. The exon 1 is a non-coding exon which is located at 5′ upstream exon 2 and presents 6 tissue-specific isoforms: h1A-h1F (Yu et al., 2004) (Figure 2A). Interestingly, differential expression of these isoforms has been reported in granulocytes of patients suffering from sepsis, indicating that 5′ UTR plays an important regulatory role in A2AR expression (Kreth et al., 2008). We recently identified a functional CGI surrounding the h1E isoform, demonstrating that DNA methylation controls basal ADORA2A expression in several cell lines and that it is one of the molecular mechanisms responsible for A2ARs' differential expression levels in specific human brain areas, such as the putamen and the cerebellum (Buira et al., 2010a, 2010b). Interestingly, we showed that A2AR expression levels can be modulated by SAM treatment in SH-SY5Y (human neuroblastoma) and U87MG (human glioblastoma) cell lines. In this context, A2ARs levels have been reported to be upregulated in PD patients (Calon et al., 2004; Varani et al., 2010). Therefore, we postulated that SAM treatment could represent a therapeutical tool to reduce A2ARs levels in these patients. This is based on the fact that the DNA methylation profile of striatal ADORA2A in PD patients is lower than the one found in SH-SY5Y and U87MG cells (Figure 2B). Therefore, as A2ARs cell surface levels are reduced in these cells after SAM treatment, due to an increase in the DNA methylation profile of ADORA2A (Buira et al., 2010a), it is also plausible that the same mechanism of action could play a role in the striatum of PD patients.
7. Clinical trials with SAM
SAM has been widely used for the treatment of liver diseases, as it increases the glutathione content (Friedel et al. 1989). Interestingly, SAM has antidepressant properties and its long term tolerability is excellent, presenting few side effects (Bell et al., 1988; Bottiglieri & Hyland, 1994; Delle Chiaie et al., 2002; Kagan et al., 1990; Lipinski et al., 1984; Papakostas, 2009, 2010). Moreover, SAM administration in patients with depression and dementia, intravenously or orally, has shown that it crosses the blood-brain barrier, and as a result, it is detected at increased levels in the cerebrospinal fluid (Bottiglieri et al., 1990).
Impaired transmethylation potential in L-dopa-treated PD patients has been proposed (De Bonis et al., 2010). The authors argue that a possible global DNA hypomethylation in hyperhomocysteinemic PD patients could be responsible for a generalized gene expression dysregulation and for playing a role in the outcome of the pathology. In accordance with this, another study has shown improved cognitive function in PD patients with a higher SAM/SAH ratio and higher plasma vitamin B6 (Obeid et al., 2009). It is noteworthy that SAM treatment improved depression of PD patients in an open-label clinical trial (Di Rocco et al., 2000). In fact, vitamin dietary supplementation, including SAM, has been shown to be effective in patients with early and moderate stages of AD (Chan et al., 2008; Panza et al., 2009; Remington et al., 2009; Shea and Chan, 2008). Moreover, SAM supplementation also presented antioxidant properties in an AD animal model (Cavallaro et al., 2010). In line with this, oxidative stress is also present in early-stages of PD (Ferrer et al., 2010), which points up the benefits of SAM administration in this pathology as an adjunctive treatment.
8. Conclusions and proposals for future PD interventions
As mentioned in the introduction, it is noteworthy that inactivation of A2ARs enhances the affinity of D2Rs for dopamine, this being the probable mechanism underlying the prodopaminergic effect of A2ARs antagonists in several clinical trials with PD patients. In this chapter, we have examined the literature which, in combination with our studies based on ADORA2A transcriptional regulation, has led us to propose SAM treatment as an epigenetic tool to modulate the increased expression of A2ARs in PD.
It is obvious that SAM treatment presents a broad spectrum of gene targets, and not only tumor supressor genes. However, it has been reported that SAM treatment promotes a decrease in the growth of hepatocellular carcinoma cells and liver cancer in animal models, hypothesising the methylation and repression of oncogenic gene promoters by this drug (Cai et al., 1998; Pascale et al., 1992). However, the existence of several clinical trials with SAM and the reduced number of side-effects in its administration must be taken into account. Based on our studies, and bearing in mind the restrictive expression of A2ARs in the brain (mainly in 95% of striatal neurons), we postulate that SAM treatment would have a "specific" effect on A2ARs in the brain. This would be especially true in a cerebral region where it colocalizes with D2Rs, which in turn present reduced activity due to the low dopamine content in PD. Then, although SAM treatment would reduce the expression of hypomethylated genes, its effect on A2ARs might represent significant activation of D2Rs.
Thus, the possible beneficial role of SAM in these patients should be examined in randomized controlled studies, examining supplementation to L-dopa (allowing a reduction of its dose) and to A2ARs antagonists (such as istradefylline), or in triple administration with both current therapies.
We are very grateful to Dr. Sandra Pérez Buira, Dr. José Luis Albasanz, Guido Dentesano and Jesús Moreno for their contributions to the study of ADORA2A transcriptional regulation. We thank T. Yohannan for editorial help. The experimental work described in the present chapter was funded by grants from the Ministerio de Ciencia e Innovación, (PI05/1631, CP08/00095, BFU2008-00138), the European Union through the Marie-Curie Research Training Network PRAIRIES (Contract MRTN-CT-2006-035810), the Consejería de Educación y Ciencia (PCI08-0125), the Consejería de Sanidad-FISCAM (PI-2007/50 and G-2007-C/13) of the Junta de Comunidades de Castilla-La Mancha and the Fundació La Marató de TV3 (092330, 090331).


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We use GPS live driver tracking so you can be assured we know where and how far your order is. We endeavour to deliver your order at the selected time; please allow a 1-2 hour delivery window. A small fee applies for delivery within the Sydney & North CBD and other areas are determined by the delivery postcode.
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Ross Mitchell - Journalist
Ross is 19 years old and lives in Stewarton, East Ayrshire. He loves football and watches as many games as he can. He does not really follow any Scottish team but looks out for Kilmarnock and Celtic's results. He completed a HND in Sports Coaching and Development of Sport in May 2012.
Contact Ross Mitchell


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There is nothing more rewarding than to be able to answer the perennial questions that many of us ask ourselves: Where did our ancestors come from? Who are we? For many of us with Prussian roots, the spark for entertaining such questions often begins with looking at records.
Many years ago, I had seen "Prussia" listed as the country of origin for some of my ancestors on the U.S. Federal Census. From that moment on, I wanted to learn more about my Prussian roots, especially about my SECOND Great-Grandfather, Christian Wendt (see Fig. 1 below).1 Who was this man? What were my Wendt Family ancestors like? What was the homeland in Prussia like?
Christian Wendt first appeared on the 1850 U.S. Census with his parents, Martin and Maria Wendt, and his sisters. It was definitely exciting to learn a little more about him and his family while they resided in Niagara County, New York.2 However, I wanted to know when they arrived to the United States and where they came from in Prussia (now Germany). On the 1900 U.S. Census, it indicated that they had emigrated to the U.S. in 1846.3 This detail was a good starting point, but I was just at the beginning of my personal Prussian search. I needed more answers and proof.
A breakthrough occurred after spending what seemed like an eternity of searching for my Wendt ancestors on a passenger arrival list. I found the record on the popular Ancestry database under "New York, Passenger Lists, 1820-1957" (see Fig. 2 below).4
The passenger list indicated the following information, which I knew corresponded nicely with this immigrant Wendt Family. I had found out that:
- Christian Wendt and his family had arrived in New York on the 15th of June 1846
- Their port of departure was Hamburg
- The ship that they traveled on was the Howard
This passenger arrival record was like a dream come true! I was now able to trace my Wendt Family to their point of departure in Hamburg. Since the record did not indicate any other geographical details, including the name of the Prussian village, my search was far from over.
The next step was to look for emigration records. I did not have to search far! It just so happened that I had found the village information and more that I was looking for on Ancestry as well under the "Brandenburg, Prussia Emigration Records" (see Fig. 3 below).5
This emigration information marked a turning point in my Prussian family research. I had learned not only the name of the village of Damme, but about the Kreis (county) of Prenzlau as well. This information proved to be crucial in order to pinpoint exactly where the village was on a gazetteer (a kind of geographical dictionary).
My search did not end there. I wanted to verify whether everything on the transcript was factually correct. I was determined to look for a copy of the record. I did not have to wait long. During a time in which the LDS microfilm service was still in effect, I had the opportunity to borrow and view a copy of the record at my local Family History Center. I took two digital images of the immigration cards on microfiche containing information about my Wendt ancestors (see Fig. 4 and Fig. 5).6 On the first image, there is information concerning the parents, Martin and Maria (in this case, she is listed as Marie-Dorothee), the number of their children and their place of origin. Also, it lists Martin's age, his occupation and their travel destination to North America in 1846. On the second image, there is a list of the names of the children, including Christian, and their dates of birth.
It was a privilege to view a copy of the emigration record. A thorough review of the emigration record was proof enough that the information had been transcribed correctly. In my case, I was very fortunate to have been able to trace, online, my Wendt ancestors to their Prussian village of origin. In reality, though, one's Prussian village of origin is not as easy to find as some might think. Fortunately, there are other techniques that can be employed. In the next segment (How Can You Find Your Prussian Village? – Part 2), I will demonstrate how it is possible to find a Prussian village using other records.
1. Christian Wendt photograph, ca. 1900; digital image 2015, privately held by Stephen Wendt, Cleveland, Ohio, 2015.
2. Ancestry, "1850 United States Federal Census," database, Ancestry (https://search.ancestry.com/cgi-bin/sse.dll?indiv=1&dbid=8054&h=7656618&tid=&pid=&usePUB=true : accessed 1 July 2012), entry for Christian Wendt (b. 1834), Wheatfield, Niagara County, New York, p. 169B, fam. 361; citing "affiliate film number" M432_560. *The Wendt surname was incorrectly transcribed as "West" on the Ancestry database.
3. Ancestry, "1900 United States Federal Census," database, Ancestry (https://www.ancestry.com/interactive/7602/4119884_00243/76645495?backurl=https://www.ancestry.com/family-tree/person/tree/4137462/person/120011088967/facts/citation/540049711391/edit/record : accessed 1 July 2012), entry for Christian Wendt (b. 1834), Park, Deuel County, Nebraska, dist. 98, p. 2, fam. 33; citing "affiliate film number" T623.
4. "New York, Passenger Lists, 1820-1857, Roll 62: 1820-1957," database, Ancestry (https://search.ancestry.com/cgi-bin/sse.dll indiv=1&dbid=7488&h=1023032323&tid=&pid=&usePUB=true&_phsrc=XPV679&_phstart=successSource : accessed 31 July 2012), entry for C. Wendt age 12, arrived New York, New York, 15 June 1846 aboard the Howard.
5. Potsdam. Auswanderungskartei [Emigration documents], year 1846, Wendt Family; Brandenburgisches Landeshauptarchiv, Potsdam, Germany; FHL International Fiche 6109219, 6109220
6. Potsdam. Auswanderungskartei [Emigration documents], year 1846, Wendt Family.
© 2018 Tree Tidings Genealogy, Verubm LLC. All rights reserved.


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Constitution Day is observed on September 17, the day in 1787 that delegates to the Constitutional Convention signed the United States Constitution in Philadelphia.
"The law establishing the present holiday was created in 2004 with the passage of an amendment by Senator Robert Byrd to the Omnibus spending bill of 2004. Before this law was enacted, the holiday was known as "Citizenship Day". In addition to renaming the holiday "Constitution Day and Citizenship Day," the act mandates that all publicly funded educational institutions, and all federal agencies, provide educational programming on the history of the American Constitution on that day. In May 2005, the United States Department of Education announced the enactment of this law and that it would apply to any school receiving federal funds of any kind." - Wikipedia.org
Included in Knowitall Media's Constitution Day resources are programs from the SC Hall of Fame series on each of South Carolina's four Founding Fathers, Charles Pinckney, Charles Cotesworth Pinckney, John Rutledge and Pierce Butler. We also are featuring the documentary from Carolina Stories, Forgotten Founder, about the life and work of Charles Pinckney.
Also in the Collection, we have the James Otis Lecture Series.
Find Teacher Resources to be utilized for teaching on Constitution Day or an adjacent weekday, if that date falls on a weekend or holiday.


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Secure Payment
My store is powered by Shopify who are certified Level 1 PCI DSS compliant.
The Payment Card Industry Data Security Standard (PCI DSS) is a security standard for organizations that handle credit and debit card information. The standard was created to increase controls around payment data to reduce fraud.
Shopify are very serious about securely hosting my store and have invested significant time and money to certify their solution is PCI compliant. From annual on-site assessments validating compliance to continuous risk management, they work hard to keep my shopping cart and ecommerce hosting secure.
I wouldn't trust any other way of taking payments for my products and services. You will have peace of mind that all your personal details are protected and the only detail i use is your email address only if you gave me permission to send you my monthly newsletter.


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Calgary.FoundLocally.com covers Calgary & Southern Alberta from Medicine Hat west to Bragg Creek, from the US border north to Red Deer. This is an extensive community information site, with information about politics, entertainment, shopping, business and sports…. with a business directory that combines their events, news announcements, coupons, ales & flyers, and jobs posting. The various communities in the FoundLocally network encompasses 30 communities from Victoria to St John's and includes a total about 100,000 pages of original content, and a business directory with over 250,0000 businesses. See the full network at http://FoundLocally.com
We conceived the site, programmed, researched, wrote, and photographed the content, and created and managed all the updates and all the various Social Media platforms and pages.
We revisit and even re-build the websites about EVERY TWO YEARS, based on one simple principle: "what might our competitors do, if they started from scratch today?" Then WE DO IT! Ourselves. and, In-house. Our methodologies are so efficient a small team can tackle that, and we continue to manage all the day-to-day stuff.
Over the years, we added many new communities, and new features and functionality. In 2010, we added a mobile version to our website(s) and in 2015 we switched to responsive design. In 2016 we switched to a more graphic "pinterest wall" layout. In 2017, our content management tools died (after 18 years of using this hyper-efficient tool) and we had to completely re-build our website's underlying technology. We completed that in 4 months.


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Hansika is superstitious
People from the film industry are always known to be superstitious. They meet astrologers to decide the right time to launch a film; they take advice from numerologists to decide upon a lucky name; they take every step hoping that the stars above will guide them to success.
Now Hansika Motwani, the child artist from Koi Mil Gaya who grew up into a sexy lass, also has her superstitions. She vows that she arrived upon them from past experiences. Hansika is very particular not to reveal anything about her new films. All she says is that she has some exciting films in hand but remains tight-lipped about the details. The reason? She does not want to reveal anything until the films are finalized. The pretty actress says she lost some films in the past when she revealed about them before finalizing them.


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Arkhi were appointed to develop a feasibility study for a parcel of residential development land in Great Stainton.
The study included developing house types that could be interchangeable and flexible depending on their location on the residential development land. There were also explorations into the surrounding context and how to ensure safety given the increase in vehicular traffic imposed on the settlement. Highway speed limits were evaluated and consequently reduced.
In conjunction with the development of the dwellings, recreational green space was provided alongside blue infrastructure provisions. To integrate the proposed development, carefully planned access routes linked the existing and proposed settlements with the aim to create a cohesive environment with high levels of pedestrian permeability.


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Search and Destroy – Down Syndrome
29 April 2007
Search and Destroy – Down Syndrome
Right to Life deplores the decision of the Minister of Health the Hon Pete Hodgson to request the National Screening Unit to advise him on how to implement a national screening programme for Down syndrome. A programme for screening all pregnant women for Down syndrome is a search and destroy mission. Those women who tested positive for Down syndrome would be encouraged to abort their child. The sole purpose of this programme would be to define who shall live and who shall die. This is unjust discrimination against the disabled. Every child at conception is endowed by its creator with an inalienable right to life. A child does not lose its right to life because it has a genetic condition.
The Minister of Health has a responsibility to protect life and to promote the health of the community; his action reveals that the government does not welcome the birth of children with Down syndrome. Its response is to sanction and fund the killing of children with Down syndrome; this is eugenics which promotes a humanistic quality of life ethic which decrees that only the perfect have a right to life. The Minister in this action is promoting a culture of death. The right to life is a sacred principle of civilisation; it is an indispensable guarantee of the individual worth of the persons within it, its universal denial would fail to recognise the dignity of man.
There are many people who are born with Down syndrome who have a good quality of life and who make a valuable contribution to the family and society.
Right to Life welcomes the Minister's decision to discontinue risky diagnostic tests that have been leading to the miscarriage of dozens of unborn children.
This proposed national screening programme should be seen in the context of the public debate on the government supported repeal of Section 59 of the Crimes Act that would outlaw the smacking of children. The Prime Minister has said that her government wishes to give a clear message to the community that violence against children is unacceptable. Violence against children begins in the womb. The government is giving the message that they support the killing of unborn children, including those with Down syndrome, but that after birth you may not smack them.


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Contact Information
Briarwood is managed by OMNI Management Services, Inc.
To create a Service Request, please use the OMNI HelpDesk.
To reach your Property Manager, Katy Swimm, call (843) 839-3131 or email her using the button below.
OMNI Management Services, Inc.
630 Chestnut Road
Myrtle Beach, SC 29572
Office: 843-839-3131
Fax: 843-626-9884
Association Dues should be mailed to:
The OMNI Payment Processing Center
P.O Box 98117
Las Vegas, NV 89193
*Please remember to include your account number on your check


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Book Tee Times
Tee Times
Input Score
18 hole regulation length course
72 par | 7,164 yards | 136 slope
75.1 rating | Bent grass
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The 18-hole "Port" course at the Harborside International Golf Center facility in Chicago, Illinois features 7,164 yards of golf from the longest tees for a par of 72. The course rating is 75.1 and it has a slope rating of 136 on Bent grass. Designed by Richard P. Nugent, ASGCA, the Port golf course opened in 1995. KemperSports Management, Inc. manages this facility, with Brian Dutler, PGA as the General Manager.
The location to the city is good. The practice facilty is perfect. There is a grass range, chipping area, sandtrap and putting green. Range balls are included in the price. The course is in good shape. It does not drain well. The greens are fast and in good shape. The negative is that outside of the par threes and the last few holes the rest of the course is the same. There are about 10 holes that are the same tee shot and same approach. Overall it is a good course but it is overpriced and a little boring at times.
Another Nugent gem ! Played on a weekend, in light rain, had the course mostly to ourselves, so I can not comment on the overall pace of play. Bev cart service was good, good brat at the turn. Nice clubhouse, well stocked pro shop. Looking at the map, one would not expect such a fine course and scenery, surrounded by blight neighborhoods and mostly industrial surroundings. $95 GF on weekend includes cart, small bucket at the range and a yardage book. $57 weekday twilight rate after 3 PM. Agree with earlier post, Port course is quite chalenging and would be frustrating for high handicappers.
Great golf course. Whether you're a great player or mid handicapper this one will challenge your game. Would not recommend for poor or beginning player. Pleasant staff. Anyone who thinks it's not a good value should go play equally groomed and challenging tracks in area for twice the price!!!
Harborside International Golf Center
Golf Only facility
Managed by KemperSports Management, Inc.
30 tees driving range
36 regulation holes
Golfweek Magazine's
America's Best Public-Access Courses for Illinois 2005
No local tournaments listed for this course. Add a tournament here.


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14 Questions with Ryan Martin of The Ryan Martin Foundation
14 Questions with Ryan Martin of The Ryan Martin Foundation
Justin Richardson, Director of Advocacy Strategy
This post is part of the series
14 Questions
, featuring interviews with disability advocates regarding their work and insights into the Complex Rehab Technology (CRT) industry. In our latest post, Numotion Director of Advocacy Strategy, Justin Richardson interviews Ryan Martin, Founder and President of
The Ryan Martin Foundation
Richardson: You discovered wheelchair basketball at age 12. Beyond the obvious physical benefits tied to involvement with athletics, what other benefits do you believe involvement in sports has on the lives of adaptive athletes?
Aside from the physical benefits, sports has increased my confidence, my ability to communicate, lead and work within diverse groups. These skills have allowed me to be successful beyond athletics.
Richardson: Have you tried/been involved with other adaptive sports or have you focused solely on basketball? If you tried other sports, what were they?
I grew up in a big family and always played sports (e.g., wiffle ball or football) although basketball was the sport I explored and excelled at (it was my first love). As an elite level athlete you incorporate cross training philosophies into your development. You incorporate track workouts to increase speed or lift weights for augmented strength. To make it at a professional level the sport becomes your job so the majority of your focus needs to be on what makes you a better basketball player (or whatever sport one choose). As you get older the challenge becomes recovery. How can you play at a high level more often? The science and discipline of high level athletics is fascinating.
Richardson: What is it about basketball that has continuously led you back to the court?
As a kid growing up with a disability it was the first thing that I remember got me noticed for the right reasons. Instead of being "the kid in the wheelchair" or "the kid with no legs" I was Ryan "the kid who can play basketball!" It gave me a sense of purpose and belonging at a time in my life when I sorely needed one. As I progressed in the sport, the competitive desire to get better and reach my potential was always my driving fuel.
Richardson: You spent 10 seasons in Europe playing professional wheelchair basketball. To most, professional wheelchair sports in the United States seems like a pipe dream. What about European culture allows for the type of environment that would support professional wheelchair sports? What would need to happen to allow for the presence of professional wheelchair sports in the United States?
Over the last several years this has been the most common question I've been asked. In Europe a lot of sports, including adaptive sports, are financed by government entities, major corporate sponsors or successful organizations. They've found a way in these various countries to market the sport, finance their leagues and create a fan base. That's the one thing I miss about playing in Europe vs. the U.S.A., the fan support was significantly better. The level of sport is there. My current team, the New York Rollin' Knicks, is as good of a roster as anywhere I've played. The product from a sport perspective is there but no one has found a business model to make it successful. Also there is a tokenistic level of respect given to adaptive sports all too often in the U.S. My hope is that down the road wheelchair basketball can become professional here in the U.S. Much like when Jackie Robinson broke the color barrier in baseball it changed a lot of things for African Americans. The ripple effect of a pro league here in the U.S. could create great change and move towards more inclusion in other spaces.
Richardson: As far as accessibility is concerned, what European countries did you find to be the most accessible and why? The least accessible?
When you travel you have to do your homework about accessibility and advocate for yourself. Overall I think the places I've lived in Europe (Spain and France) were reasonably accessible, but not quite to the standard of here in the states (post ADA). I found people in Europe very eager to accommodate persons with a disability in situations where access was an issue.
Richardson: Considering limited in-person access to custom athletic equipment manufacturer representatives, seating and positioning decisions for recreational athletes are often left up to the athlete. At best, there may be input from teammates and/or fellow competitors. If all athletes had access to a seating and mobility specialist trained in fitting sports chairs, do you think the quality of competition in adaptive athletics would improve?
Yes. Getting the right equipment for the athlete is so very important. Often times sports chairs are passed down from athlete to athlete. When an athlete makes that next step to being more competitive and gets their own equipment it brings the whole level of the sport up. There is a strong need for affordable equipment that is functional at the grass roots level. I hate to see when cost is a barrier to a young athlete getting their start!
Richardson: Speaking of seating and positioning, as a lifelong wheelchair user, what advice would you give to someone who is either newly diagnosed or injured and is in the process of obtaining a chair for the first time?
My advice is do your research and speak with end users. Someone with experience can get you past a lot of those first hurdles. Get the equipment that gives you the greatest sense of independence. The ideal chair/equipment will fit you well, get you active in the things you want to do, and seem like an extension of your body.
Richardson: You're currently acting as a consultant with the NCAA on their adaptive sports model. What is the current state of collegiate wheelchair sports and where do you hope to see adaptive collegiate athletics in 10 years?
There are many colleges/universities exploring adaptive sports programs on their respective campuses. I would like students with disabilities to be given the same opportunity that their able-bodied peers are offered. Our colleges and universities in 2018 are places where we should foster inclusion, awareness, diversity and promote equality. Including adaptive sports is long overdue and just the next step in that journey. There are parallels that can be made to women's collegiate athletes, pre Title 9. I would like to see more students who are being denied these opportunities to reach out to the Office of Civil Rights and create some precedent in this space. My hope over the next several years is that we see more programs emerging to the point where it becomes a normal part of the fabric of campuses around the country!
Richardson: You recently had the opportunity to attend ESPN's ESPY Awards in Los Angeles along with Numotion team members Scott Ritchotte and Mike Mowry. On a national stage, do you believe adaptive sports and the athletes playing those sports are being given the credit they deserve?
Yes and no. Yes because we are starting to see the inclusion of adaptive athletes in events like the ESPY's and move coverage of sporting events like the Paralympics on NBC. No doubt great strides have been made in this areas and there are some tremendous advocates fighting to increase awareness. I'd like to see more coverage of adaptive sports beyond recognition once a year at an award show or every four years on NBC. Progress has been made but the journey is far from compete!
Richardson: How has sport chair technology changed in your lifetime? Has it outpaced or stayed on level ground with advancements in everyday chair technology?
I remember the first chairs I had (sports and everyday) were big, heavy and clunky. The technology has drastically improved. Chairs are lighter, smaller, more stream line and more of an extension of the end user. No question the advancements in everyday chairs have made them lighter and allow for more independence. Advancements in the technology of sports chairs have made them lighter, faster, more responsive and allowed for the overall sports to improve.
Richardson: In the United States, sports chairs are not typically covered by insurance. What advice would you give to the family of a youth athlete who wants to participate but may be struggling to afford the cost of equipment?
Research grants or join established programs that can provide you a chair to get started in.
Challenged Athlete Foundation (CAF)
does a great job providing grants for adaptive athletes and there are other ones out there as well.
Richardson: The Ryan Martin Foundation helps youth players, adults, and soldiers living with new disabilities become better athletes through various camp opportunities throughout the year. I imagine each opportunity is rewarding in its own way. How is each type of camp different from the other?
Each camp is unique because you always have new athletes joining camps/programs. The best part is getting a kid started and seeing their face when they make their first basket, as well as seeing athletes who are making that leap. The leap of getting their own equipment, going to camps, practicing on their own and having the drive to excel! Working with the veterans is great as well because it provides these men and women a sense of normalcy.
Richardson: What do you do with your free time away from basketball and your work with your foundation?
Free time? What's that? I run a NPO lol! I love to travel, spend time with Lindsay (my better half) and my family. Usually I'm on the go quite a bit so I enjoy downtime!
Richardson: What would be the best way for those interested to learn more about the Ryan Martin Foundation?
The best way would be to follow us on social media (
, and
), visit our
or drop us a line!


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Dig Dug
Scored By: 8bitDeity (OSM)
100% Legit
My username is on the paper, zoom in if you can't see it that well, I know that it's not that high of a score but I wanted to post something that can compete with the current high score. More people need to play TI-99/4a games :p
It's very clear when zoomed indeed (most probably know to right click on the photo and then hit View Image, then the plus sign. But for those that don't...).
Nice score, great game! B.t.w. I found it quite interesting as I didn't know about the TI-99/4A so far and only now realised that in the early 80ies there was a competetion to C64 and Sinclair! I don't think the system was very common in Europe. Thanks for bringing this here!
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Sarah Hyland could not be happier about her romance with Bachelorette alum Wells Adams. The Modern Family star just celebrated her first year with Adams and told the world exactly how she felt about the anniversary with an adorable post on social media.
On Sunday, Hyland shared a pic of her and Adams on Instagram and told fans that he "gave me my last first kiss." Hyland also talked about the time she met Adams and how he immediately stole her heart on the dance floor.
Adams also gushed about their romance with a post of his own. The former Bachelorette contestant called Hyland a "beautiful woman" and said this past year has been the best ever.
According to Pop Culture, Hyland's romance with Adams actually started on social media with the pair exchanging tweets. The two went public with their relationship last Halloween when they showed up at a party in matching costumes based on the show Stranger Things.
Fast forward a year later and their relationship is stronger than ever, especially now that Adams is living with Hyland in Los Angeles.
With her romantic life in order, Hyland is also gearing up for the new season of Modern Family. As fans are well aware, the show's executive producer Christopher Lloyd recently teased that they will kill off an important character in Season 10, leaving fans in the dark about who will get the axe.
During a red-carpet appearance over the weekend, Hyland was asked about the major death next season. The actress unfortunately had no clue about the death but told fans that she hopes the dog survives.
"Fans of the show, if the dog dies, I leave," Hyland dished.
Sarah Hyland added that she was unaware that one of the characters will die next season. She also sounded a bit worried that her character, Haley, will be the one who is killed off, especially given how accident-prone she is.
Modern Family returns Sept. 26 on ABC.


text: Marc Posted October 18, 2018 Report Share Posted October 18, 2018 With Fright Nights well underway the 31st is fast approaching - the park have started some very early marketing for 2019. A sign has gone up over the exit and a page has been setup at https://www.thorpepark.com/2019 to sign up to be the first to hear more information on 2019. So what would you like to (realistically) see for 2019? I'm not expecting any new attractions for next season and that's probably a good thing - the park can really focus on what it has and going from there. For me id like to see: Clarity on Loggers Leap future - if it's returning or not I think this really needs to be decided / communicated. General tidy up - it's fairly obvious, but there's bits of the park which need a good tidy up, some queue lines are looking tired and need repairing / painting, buildings particularly around Angry Birds land could do with a lick of paint too. Fixing up of theming - the park over the last few years have come on leaps and bounds in this department, Infernos has its waterfalls back, the mist is working more times than it isn't after years of nothing, Tidal Waves sign, water tower work to a degree, the effects around The Swarm are mostly working and Saw finally saw its blades fixed, and seeing this continue would be great. Colossus could really do with seeing it's pit cleaned out and if possible a solution to the issues it's had with its fountain since it opened - it really looks great when it's working, more work over at tidal to get both the Sign and Water tower fully working would be great too. Audio - again, recent times we've seen a lot of good progress with audio, I know some replacement tracks are controversial but I'd say the park is sounding much better now than it did 3 years ago. I'd like to see more here, Stealth and Tidal Wave have seen a lot of work on their sound systems and both sound great, would like to see this continue, Colossus / Lost City has dead speakers which would be good to hear working again, the Bridge Speakers have had better days too and could do with replacing. Wouldn't mind The Swarm having a new IMAscore track too (hinted in the entrance track). From there would like to see general volumes abit louder, particularly at the entrance. Events - whilst the year of the walking dead / Love Island Lates have been fairly controversial on these parts I think events over the year is a great thing - even if the theme isn't for you (Love island isn't my thing but I loved the 10pm closes!) and more of this next year would be great. Fright Nights - obviously this is a hot subject at the moment, I hope next year we see more investment in Friht Nights, it would be great to see the big top return, along with at least one more all new indoor maze, personally am not against IP's for mazes if it's the right IP and is done well, but generally prefer original themes - the ideas they can come up with are usually just as good. It's also important to learn and evolve the good parts from this year too, The Big Top show time, whilst not anywhere near as good as the Big Top maze does a great job in the dome, the roaming actors this year in Amity and the clowns are also the best I can remember. That's my lot for what I'd like to see in 2019 - what would you like to see? Quote Link to post Share on other sites