rupture. In the setting of the scourging and crucifixion, with associated hypovolemia, hypoxemia, and perhaps and altered coagulable state, friable non-infective thrombotic vegetations could have formed on the aortic or mitral valve. These then could have dislodged and embolized into the coronary circulation and thereby produced an acute transmural myocardial infarction. Thrombotic valvular vegetations have been reported to develop under analogous acute traumatic conditions. Rupture of the left ventricular free wall may occur, though uncommonly, in the first few hours following infarction.

However, another explanation may be more likely. Jesus’ death may have been hastened simply by his state of exhaustion and by the severity of the scourging, with its resultant blood loss and preshock state. The fact that he could not carry his patibulum supports this interpretation. TheHistory of the discovery of the mode of transmission of yellow fever virus.
This essay documents and examines the historical circumstances and events surrounding the discovery of the mode of transmission of yellow fever virus in Cuba. Close scrutiny of the articles published by Walter Reed and his colleagues in 1900, 1901 and 1902 reveals their limitations as historic documents. Fortunately, other sources of information from that period survive in letters and documents written by individuals involved in the quest for the mode of transmission. Examination and comparison of those sources of information unveiled a fascinating story which reveals that misunderstandings engendered by published articles accorded merit where it was not fully due.