By histopathology, IPMN is characterized on light microscopy by Mucinous epithelial cells, and growth within the pancreatic ducts.
Mucin 5AC is a useful immunohistochemistry marker.
 !!
Microscopy findings !!
Micrograph !!
Immunohistochemistry markers Characteristic genetic alterations are those of KRAS and GNAS.

Further subtyping of IPMN can be done as either:
Gross pathology: Main duct, branch duct, and mixed duct lesions, which determines surgical management.
Main duct lesion is the segmental or diffuse dilatation of main pancreatic duct greater than 5 mm without other causes of obstruction.
Meanwhile, branch duct lesion is the pancreatic cyst more than 5 mm that communicates with the main duct.
The mixed duct lesions fulfills both criteria above.

By light microscopy and immunohistochemistry: Gastric, intestinal, pancreaticbiliary, and oncocytic.
By histopathology, IPMN is characterized on light microscopy by Mucinous epithelial cells, and growth within the pancreatic ducts. Mucin 5AC is a useful immunohistochemistry marker. !! Microscopy findings !! Micrograph !! Immunohistochemistry markers Characteristic genetic alterations are those of KRAS and GNAS.
Further subtyping of IPMN can be done as either:
Gross pathology: Main duct, branch duct, and mixed duct lesions, which determines surgical management. Main duct lesion is the segmental or diffuse dilatation of main pancreatic duct greater than 5 mm without other causes of obstruction. Meanwhile, branch duct lesion is the pancreatic cyst more than 5 mm that communicates with the main duct. The mixed duct lesions fulfills both criteria above.
By light microscopy and immunohistochemistry: Gastric, intestinal, pancreaticbiliary, and oncocytic.
Intraductal papillary mucinous neoplasm