Sensory ganglia have been associated with infections from viruses like herpes simplex, which can exist in a dormant state within the ganglia for decades after the primary infection.
When the virus becomes reactivated, blisters on the skin and mucous membranes appear.
During the latent stage of the virus, the viruses are rarely located in the SGCs within the sensory ganglia, but the SGCs may still play an important role within the disease.
 It has been proposed that SGCs act to create walls to prevent the spread of the virus from infected to uninfected neurons.
If this wall of protection was to break down, then the infection could become more widespread.
This property may be explained by looking at the location and arrangement of the SGCs, as they are centered on the neurons, allowing them to protect the neurons.
It has also been proposed that SGCs may have a job in ridding the ganglia of the virus and in protecting and repairing the nervous system after the virus has left the dormant stage.
Sensory ganglia have been associated with infections from viruses like herpes simplex, which can exist in a dormant state within the ganglia for decades after the primary infection. When the virus becomes reactivated, blisters on the skin and mucous membranes appear. During the latent stage of the virus, the viruses are rarely located in the SGCs within the sensory ganglia, but the SGCs may still play an important role within the disease.  It has been proposed that SGCs act to create walls to prevent the spread of the virus from infected to uninfected neurons. If this wall of protection was to break down, then the infection could become more widespread. This property may be explained by looking at the location and arrangement of the SGCs, as they are centered on the neurons, allowing them to protect the neurons. It has also been proposed that SGCs may have a job in ridding the ganglia of the virus and in protecting and repairing the nervous system after the virus has left the dormant stage.
Satellite glial cell