Theoretical progress in understanding the dynamics of spreading processes on graphs suggests the existence of an epidemic threshold below which no epidemics form and above which epidemics spread to a significant fraction of the graph. We have observed information cascades on the social media site Digg that spread fast enough for one initial spreader to infect hundreds of people, yet end up affecting only 0.1 of the entire network. We find that two effects, previously studied in isolation, combine cooperatively to drastically limit the final size of cascades on Digg. First, because of the highly clustered structure of the Digg network, most people who are aware of a story have been exposed to it via multiple friends. This structure lowers the epidemic threshold while moderately slowing the overall growth of cascades. In addition, we find that the mechanism for social contagion on Digg points to a fundamental difference between information spread and other contagion processes: despite multiple opportunities for infection within a social group, people are less likely to become spreaders of information with repeated exposure. The consequences of this mechanism become more pronounced for more clustered graphs. Ultimately, this effect severely curtails the size of social epidemics on Digg.
@cite_22 modeled viral email cascades using branching processes like the Galton-Watson process and the Bellman-Harris process. They argued that the topology of the underlying social network is irrelevant in the prediction of cascade size. This may hold true in the tree-like cascades studied by the authors. However as stated previously, in Digg, dynamics of information propagation is not tree-like and these models do not hold. Future work includes studying the impact of activity patterns on the information diffusion dynamics.
@cite_22
2289705509 1937101755 1497522841 2109207218
Information cascades on social networks, such as retweet cascades on Twitter, have been often viewed as an epidemiological process, with the associated notion of virality to capture popular cascades that spread across the network. The notion of structural virality or average path length has been posited as a measure of global spread. In this paper, we argue that this simple epidemiological view, though analytically compelling, is not the entire story. We first show empirically that the classical SIR diffusion process on the Twitter graph, even with the best possible distribution of infectiousness parameter, cannot explain the nature of observed retweet cascades on Twitter. More specifically, rather than spreading further from the source as the SIR model would predict, many cascades that have several retweets from direct followers, die out quickly beyond that. We show that our empirical observations can be reconciled if we take interests of users and tweets into account. In particular, we consider a model where users have multi-dimensional interests, and connect to other users based on similarity in interests. Tweets are correspondingly labeled with interests, and propagate only in the subgraph of interested users via the SIR process. In this model, interests can be either narrow or broad, with the narrowest interest corresponding to a star graph on the interested users, with the root being the source of the tweet, and the broadest interest spanning the whole graph. We show that if tweets are generated using such a mix of interests, coupled with a varying infectiousness parameter, then we can qualitatively explain our observation that cascades die out much more quickly than is predicted by the SIR model. In the same breath, this model also explains how cascades can have large size, but low "structural virality" or average path length. Theoretical progress in understanding the dynamics of spreading processes on graphs suggests the existence of an epidemic threshold below which no epidemics form and above which epidemics spread to a significant fraction of the graph. We have observed information cascades on the social media site Digg that spread fast enough for one initial spreader to infect hundreds of people, yet end up affecting only 0.1 of the entire network. We find that two effects, previously studied in isolation, combine cooperatively to drastically limit the final size of cascades on Digg. First, because of the highly clustered structure of the Digg network, most people who are aware of a story have been exposed to it via multiple friends. This structure lowers the epidemic threshold while moderately slowing the overall growth of cascades. In addition, we find that the mechanism for social contagion on Digg points to a fundamental difference between information spread and other contagion processes: despite multiple opportunities for infection within a social group, people are less likely to become spreaders of information with repeated exposure. The consequences of this mechanism become more pronounced for more clustered graphs. Ultimately, this effect severely curtails the size of social epidemics on Digg. Cascades are ubiquitous in various network environments. How to predict these cascades is highly nontrivial in several vital applications, such as viral marketing, epidemic prevention and traffic management. Most previous works mainly focus on predicting the final cascade sizes. As cascades are typical dynamic processes, it is always interesting and important to predict the cascade size at any time, or predict the time when a cascade will reach a certain size (e.g. an threshold for outbreak). In this paper, we unify all these tasks into a fundamental problem: cascading process prediction. That is, given the early stage of a cascade, how to predict its cumulative cascade size of any later time? For such a challenging problem, how to understand the micro mechanism that drives and generates the macro phenomena (i.e. cascading process) is essential. Here we introduce behavioral dynamics as the micro mechanism to describe the dynamic process of a node's neighbors getting infected by a cascade after this node getting infected (i.e. one-hop subcascades). Through data-driven analysis, we find out the common principles and patterns lying in behavioral dynamics and propose a novel Networked Weibull Regression model for behavioral dynamics modeling. After that we propose a novel method for predicting cascading processes by effectively aggregating behavioral dynamics, and present a scalable solution to approximate the cascading process with a theoretical guarantee. We extensively evaluate the proposed method on a large scale social network dataset. The results demonstrate that the proposed method can significantly outperform other state-of-the-art baselines in multiple tasks including cascade size prediction, outbreak time prediction and cascading process prediction. We study the impact of human activity patterns on information diffusion. To this end we ran a viral email experiment involving 31183 individuals in which we were able to track a specific piece of information through the social network. We found that, contrary to traditional models, information travels at an unexpectedly slow pace. By using a branching model which accurately describes the experiment, we show that the large heterogeneity found in the response time is responsible for the slow dynamics of information at the collective level. Given the generality of our result, we discuss the important implications of this finding while modeling human dynamical collective phenomena.