The random walk is fundamental to modeling dynamic processes on networks. Metrics based on the random walk have been used in many applications from image processing to Web page ranking. However, how appropriate are random walks to modeling and analyzing social networks? We argue that unlike a random walk, which conserves the quantity diffusing on a network, many interesting social phenomena, such as the spread of information or disease on a social network, are fundamentally non-conservative. When an individual infects her neighbor with a virus, the total amount of infection increases. We classify diffusion processes as conservative and non-conservative and show how these differences impact the choice of metrics used for network analysis, as well as our understanding of network structure and behavior. We show that Alpha-Centrality, which mathematically describes non-conservative diffusion, leads to new insights into the behavior of spreading processes on networks. We give a scalable approximate algorithm for computing the Alpha-Centrality in a massive graph. We validate our approach on real-world online social networks of Digg. We show that a non-conservative metric, such as Alpha-Centrality, produces better agreement with empirical measure of influence than conservative metrics, such as PageRank. We hope that our investigation will inspire further exploration into the realms of conservative and non-conservative metrics in social network analysis.
The interplay of the structural properties of the underlying network with the diffusion processes occurring in it, contributes to the complexity of real-life networks. For example in epidemiology, the dynamics of disease spread on a network and the epidemic threshold is closely related to its spectral radius of the graph @cite_27 . Similarly, random walk on a graph is closely related Laplacian of the graph @cite_33 .
@cite_27 @cite_33
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The spread of epidemics and malware is commonly modeled by diffusion processes on networks. Protective interventions such as vaccinations or installing anti-virus software are used to contain their spread. Typically, each node in the network has to decide its own strategy of securing itself, and its benefit depends on which other nodes are secure, making this a natural game-theoretic setting. There has been a lot of work on network security game models, but most of the focus has been either on simplified epidemic models or homogeneous network structure. We develop a new formulation for an epidemic containment game, which relies on the characterization of the SIS model in terms of the spectral radius of the network. We show in this model that pure Nash equilibria (NE) always exist, and can be found by a best response strategy. We analyze the complexity of finding NE, and derive rigorous bounds on their costs and the Price of Anarchy or PoA (the ratio of the cost of the worst NE to the optimum social cost) in general graphs as well as in random graph models. In particular, for arbitrary power-law graphs with exponent β > 2, we show that the PoA is bounded by O(T2(β -1))), where T = γ α is the ratio of the recovery rate to the transmission rate in the SIS model. We prove that this bound is tight up to a constant factor for the Chung-Lu random power-law graph model. We study the characteristics of Nash equilibria empirically in different real communication and infrastructure networks, and find that our analytical results can help explain some of the empirical observations. The systematic study of large-scale networks has unveiled the ubiquitous presence of connectivity patterns characterized by large-scale heterogeneities and unbounded statistical fluctuations. These features affect dramatically the behavior of the diffusion processes occurring on networks, determining the ensuing statistical properties of their evolution pattern and dynamics. In this article, we present a stochastic computational framework for the forecast of global epidemics that considers the complete worldwide air travel infrastructure complemented with census population data. We address two basic issues in global epidemic modeling: (i) we study the role of the large scale properties of the airline transportation network in determining the global diffusion pattern of emerging diseases; and (ii) we evaluate the reliability of forecasts and outbreak scenarios with respect to the intrinsic stochasticity of disease transmission and traffic flows. To address these issues we define a set of quantitative measures able to characterize the level of heterogeneity and predictability of the epidemic pattern. These measures may be used for the analysis of containment policies and epidemic risk assessment. Learning about the structure of hidden or covert networks is a major challenge in epidemiology, sociology, and intelligence analysis. Vertices in hidden networks usually cannot be enumerated or sampled in a systematic way; they can only be revealed by tracing links emanating from already-observed vertices. Observers sometimes cannot follow links directly, and instead must rely on passive observation of a dynamic process to reveal vertices and edges. This paper outlines a framework for estimating network structures from partial observation of information diffusion through the network. Diffusion is modeled by a continuous-time Markov epidemic model. Edges are revealed by transmission events and new vertices are uncovered when information is transmitted to them. The approach is a generalization of tools developed to reconstruct drug-user networks from respondent-driven sampling studies in epidemiology. The likelihood of the diffusion process can be interpreted as an exponential random graph model. A Bayesian method for probabilistic reconstruction of the transmission-induced subgraph is described. The time variation of contacts in a networked system may fundamentally alter the properties of spreading processes and affect the condition for large-scale propagation, as encoded in the epidemic threshold. Despite the great interest in the problem for the physics, applied mathematics, computer science, and epidemiology communities, a full theoretical understanding is still missing and currently limited to the cases where the timescale separation holds between spreading and network dynamics or to specific temporal network models. We consider a Markov chain description of the susceptible-infectious-susceptible process on an arbitrary temporal network. By adopting a multilayer perspective, we develop a general analytical derivation of the epidemic threshold in terms of the spectral radius of a matrix that encodes both network structure and disease dynamics. The accuracy of the approach is confirmed on a set of temporal models and empirical networks and against numerical results. In addition, we explore how the threshold changes when varying the overall time of observation of the temporal network, so as to provide insights on the optimal time window for data collection of empirical temporal networked systems. Our framework is of both fundamental and practical interest, as it offers novel understanding of the interplay between temporal networks and spreading dynamics.