Unpacking Terrorism, Revolution and Insurgency in Yemen: Real and Imagined Threats to Regional Security
Recent months have seen a seeming escalation in the international threat posed by Al Qaeda in the Arabian Peninsula (AQAP), a terrorist network that has taken Yemen as its regional base of operations. In light of recent attacks, and resulting embassy closures, Yemen is a rising priority in the Western-led War on Terror. However, this has resulted in a side-lining of other security threats in Yemen, which may cause serious challenges to the authority of the Yemeni Government. In reality, the role of AQAP has been heavily manipulated throughout Yemen’s contemporary history: this was most evident during the 2011 Arab Spring, when both sides in the conflict claimed that Al Qaeda operatives were working with members of the other. Two years later, the true nature of the AQAP threat in Yemen is rarely questioned by external observers, yet remains largely shrouded in mystery. There is a need for more critical approaches to the AQAP challenges, which take the broader context of Yemeni security into account.
1 The Horn of Africa and Yemen Diminishing the Threat of Terrorism
T greater Horn of Africa thrusts itself toward Yemen and hence the heart of Arabia and the Persian/Arab Gulf. Within the complex region of northeastern Africa that extends from the peaks of Kilimanjaro to the depression of Djibouti and from the deserts of Chad to the Red Sea and on southward, past Cape Guardafui, to the barren coastline of Punt, there are 149 million people, more than half of whom are Muslims. For geostrategic reasons, especially in an era of terror, Yemen belongs naturally to this greater Horn of Africa region, adding another 20 million people, virtually all Muslims. Although not necessarily cohesive physically, despite the unifying Rift Valley theme (from the Sudan and Djibouti south through Ethiopia and into Kenya), in the global battles for freedom and democracy and against terrorism these seven nation-states (Djibouti, Eritrea, Ethiopia, Kenya, Somalia, the Sudan, and Yemen) astride the Red Sea, the Gulf of Aden, and the Indian Ocean share a common enemy. They also roughly share a paucity of resources and unfulfilled desires for rapid economic advancement. Al Qaeda can strike anywhere. It has already struck twice in Kenya, at least once in Somalia, and once (with at least two
Alterations in spontaneous single unit activity of striatal subdivisions during ontogenesis in mutant dystonic hamsters
The pathophysiology of idiopathic dystonia, characterized by sustained twisting movements and postures, is still unknown. Clinically, however, the basal ganglia are thought to be the main causative origin of idiopathic dystonia. In the dtsz hamster, a genetic animal model for idiopathic paroxysmal dystonia, the attacks occur in response to mild stress and the severity of dystonia is age-dependent. Previous autoradiographic studies in the dtsz hamster revealed a decreased dopamine D1 and D2 receptor binding and an increased [3H]-2-deoxyglucose uptake in the dorsomedial caudate-putamen (CPu), a region supposed to be critically involved in dystonia. Therefore, we were interested whether the spontaneous firing rate of dorsomedial striatal neurons is age-dependently altered in comparison to age-matched non-dystonic control hamsters. Extracellular recordings of spontaneous single unit activity of dorsomedial and ventromedial Type II striatal neurons, i.e., biphasic positive-negative action potentials, from fentanyl anesthetized animals revealed a drastically increased firing rate in the dorsomedial CPu of mutants during age of maximum severity of dystonia. In post-dystonic dtsz hamsters, i.e., after remission of stress-inducible dystonia, no significant differences regarding the dorsomedial CPu could be obtained. We conclude that the dorsomedial subregion of the CPu seems to be critically involved in the dystonic syndrome of dtsz hamsters and that a transiently reduced inhibitory control over excitatory cortico-striatal processes, possibly due to an altered development of GABAergic inhibition, occurs during ontogenesis in dtsz hamsters.