Finite Element Evaluation of Stress Distribution in Mature and Immature Teeth
INTRODUCTION: Traumatic injuries may affect the vitality and development of tooth. So divergence or parallelism of canal walls induces an open apex which is very susceptible to fracture. The aim of this study was to determine the stress distribution pattern in immature teeth and compare it with mature teeth. MATERIALS AND METHODS: In order to analyze stress distribution using finite element method, models were first designed according to actual samples by "ANSYS" software. Two models of the maxillary central incisors were designed, one mature and one with open apex. Mature tooth was designed as having undergone root canal therapy with gutta-percha and the immature one with both MTA plug and gutta-percha. Samples were loaded in seven stages and then, the stress distribution in each model was measured, using the "ANSYS" software. RESULTS: During gutta-percha condensation, the immature tooth transfers the stress directly to the external root surface where force is directly applied, whereas during masticatory occlusal forces, the stress is transferred to middle third of buccal and lingual surfaces and to buccal cervix in lunar shape. During traumatic forces, stress concentration was on the cervical region of buccal surface and middle third of buccal and lingual surfaces. MTA plug prevents stress distribution toward apical region and forces concentrate in dentin at the point where stress terminates. CONCLUSION: Within the limitations of this simulated mechanical analysis, it was confirmed that the pattern of stress distribution in mature and immature teeth is different. Cervical area of the buccal surface in immature teeth is one of the stress concentration areas, which contribute to the high rate of fracture in this area.
Endodontic regeneration: hard shell, soft core
A loss of organs or the destruction of tissue leaves wounds to which organisms and living things react differently. Their response depends on the extent of damage, the functional impairment and the biological potential of the organism. Some can completely regenerate lost body parts or tissues, whereas others react by forming scars in the sense of a tissue repair. Overall, the regenerative capacities of the human body are limited and only a few tissues are fully restored when injured. Dental tissues may suffer severe damage due to various influences such as caries or trauma; however, dental care aims at preserving unharmed structures and, thus, the functionality of the teeth. The dentin–pulp complex, a vital compound tissue that is enclosed by enamel, holds many important functions and is particularly worth protecting. It reacts physiologically to deleterious impacts with an interplay of regenerative and reparative processes to ensure its functionality and facilitate healing. While there were initially no biological treatment options available for the irreversible destruction of dentin or pulp, many promising approaches for endodontic regeneration based on the principles of tissue engineering have been developed in recent years. This review describes the regenerative and reparative processes of the dentin–pulp complex as well as the morphological criteria of possible healing results. Furthermore, it summarizes the current knowledge on tissue engineering of dentin and pulp, and potential future developments in this thriving field.
Anomalous Annealing Behavior of Arsenic Implanted Silicon as a Function of Dose and Energy
Anomalous annealing behavior of damage in As implanted Si has been investigated by channeling effect measurements. Room temperature As implants with doses from 1014 to 2 × 1016cm-2 at energies ranging from 50 to 250 keV into Si were annealed over the range of 600°C to 1100°C. Anneal temperatures ≥ 950°C reduce the disorder for ion doses of 0.5 to 1 × 1016 cm-2 at energies between 50 and 250 keV. Anomalously high residual disorder was found for the intermediate doses of 2 × 1014 to 2 × 10/cm2 implanted above 100 keV. For this intermediate dose, annealing of implants performed at liquid nitrogen temperatures did not show significant reduction in this damage. However implants at 200° to 300°C indicate a lower amount of damage compared to the room temperature implants.