War Termination in Sri Lanka
This article has a central aim of explaining the catastrophic loss of civilian life in the last five months of the civil war in Sri Lanka. The article proposes that both the government of Sri Lanka (GoSL) and the LTTE are culpable for civilian deaths; the LTTE in an effort to stave off defeat so it might be able to re-constitute itself at a later time, and the GoSL in a determination not to allow the LTTE as an organization to survive the conflict. The article also argues that a preliminary explanation for the origins of the Sri Lankan civil war can be found in the GoSL program of transferring members of the Sinhalese population into the Northern and Eastern provinces of the country, regions held by the Tamil population to be their homeland territory. Key words : Population; Tamil; Sinhalese insurgency; Civilian; Territory
Theories of Secession
Approche systematique des differents types de theories normatives de la secession. Distinguant les arguments en faveur d'une justification d'un droit moral de la secession et les arguments en faveur d'une justification des prescriptions du droit international, l'A. compare les theories du droit reperateur et les theories du droit originaire en vertu du principe de l'integrite territoriale et de l'integrite de la participation politique en democratie
Chemical, biological, and immunological characterization of mono- and diiodothyrotropin releasing factor.
Abstract Iodination of thyrotropin releasing factor (TRF) with chloramine-T and NaI at pH 7.5 produces both mono- and diiodo-TRF as well as unreacted TRF and I − . The four compounds can be separated by ion-exchange chromatography on SP-Sephadex C-25, using a stepwise gradient of 0.01 m MH 4 OAc at pH 3.5, 5.2 and 7.2. The monoiodo-TRF was characterized by nuclear magnetic resonance, mass spectra, and elemental analysis as [Im-5-iodo-His 2 ]-TRF while the diiodo-TRF was similarly characterized as [Im-2,5-diiodo-His 2 ]-TRF. Both iodination products showed no thyrotropin releasing activity at doses up to 1 μg in the in vivo mouse bioassay. The specific binding of mono- and diiodo-TRF was measured against three different TRF antisera. In the absence of TRF, antisera A and C bound more diiodo-TRF than monoiodo-TRF while antiserum B seemed to bind both tracers equally. In all cases steeper curves were produced when diiodo-TRF was displaced by synthetic TRF.