Social networks play a fundamental role in the diffusion of information. However, there are two different ways of how information reaches a person in a network. Information reaches us through connections in our social networks, as well as through the influence of external out-of-network sources, like the mainstream media. While most present models of information adoption in networks assume information only passes from a node to node via the edges of the underlying network, the recent availability of massive online social media data allows us to study this process in more detail. We present a model in which information can reach a node via the links of the social network or through the influence of external sources. We then develop an efficient model parameter fitting technique and apply the model to the emergence of URL mentions in the Twitter network. Using a complete one month trace of Twitter we study how information reaches the nodes of the network. We quantify the external influences over time and describe how these influences affect the information adoption. We discover that the information tends to "jump" across the network, which can only be explained as an effect of an unobservable external influence on the network. We find that only about 71 of the information volume in Twitter can be attributed to network diffusion, and the remaining 29 is due to external events and factors outside the network.
Time plays an essential role in the diffusion of information, influence and disease over networks. In many cases we only observe when a node copies information, makes a decision or becomes infected -- but the connectivity, transmission rates between nodes and transmission sources are unknown. Inferring the underlying dynamics is of outstanding interest since it enables forecasting, influencing and retarding infections, broadly construed. To this end, we model diffusion processes as discrete networks of continuous temporal processes occurring at different rates. Given cascade data -- observed infection times of nodes -- we infer the edges of the global diffusion network and estimate the transmission rates of each edge that best explain the observed data. The optimization problem is convex. The model naturally (without heuristics) imposes sparse solutions and requires no parameter tuning. The problem decouples into a collection of independent smaller problems, thus scaling easily to networks on the order of hundreds of thousands of nodes. Experiments on real and synthetic data show that our algorithm both recovers the edges of diffusion networks and accurately estimates their transmission rates from cascade data. Theoretical progress in understanding the dynamics of spreading processes on graphs suggests the existence of an epidemic threshold below which no epidemics form and above which epidemics spread to a significant fraction of the graph. We have observed information cascades on the social media site Digg that spread fast enough for one initial spreader to infect hundreds of people, yet end up affecting only 0.1 of the entire network. We find that two effects, previously studied in isolation, combine cooperatively to drastically limit the final size of cascades on Digg. First, because of the highly clustered structure of the Digg network, most people who are aware of a story have been exposed to it via multiple friends. This structure lowers the epidemic threshold while moderately slowing the overall growth of cascades. In addition, we find that the mechanism for social contagion on Digg points to a fundamental difference between information spread and other contagion processes: despite multiple opportunities for infection within a social group, people are less likely to become spreaders of information with repeated exposure. The consequences of this mechanism become more pronounced for more clustered graphs. Ultimately, this effect severely curtails the size of social epidemics on Digg. We study the relaxation response of a social system after endogenous and exogenous bursts of activity using the time series of daily views for nearly 5 million videos on YouTube. We find that most activity can be described accurately as a Poisson process. However, we also find hundreds of thousands of examples in which a burst of activity is followed by an ubiquitous power-law relaxation governing the timing of views. We find that these relaxation exponents cluster into three distinct classes and allow for the classification of collective human dynamics. This is consistent with an epidemic model on a social network containing two ingredients: a power-law distribution of waiting times between cause and action and an epidemic cascade of actions becoming the cause of future actions. This model is a conceptual extension of the fluctuation-dissipation theorem to social systems [Ruelle, D (2004) Phys Today 57:48–53] and [Roehner BM, et al, (2004) Int J Mod Phys C 15:809–834], and provides a unique framework for the investigation of timing in complex systems.