Female, 81 years old, with dysphagia, dysphonia and weakness. Brain:No evidence of restricted diffusion is seen. Scattered patchy foci of T2 hyperintensity are demonstrated in the subcortical and periventricular white matter. No evidence of parenchymal edema or mass effect is seen. No pathologic parenchymal or extra-axial enhancement is detected. There is no evidence of intracranial hemorrhage or any other abnormal extra-axial fluid. Ventricular system is normal in size and morphology.C-spine:A mild reversal of the normal cervical lordosis is demonstrated. Spinal alignment is unremarkable.Vertebral body heights are preserved. Degenerative endplate signal alteration is seen, but there is no evidence of any concerning marrow replacement or significant marrow edema.The visualized spinal cord shows no evidence of any pathologic signal abnormality and is morphologically unremarkable.No evidence of any pathologic spinal or paraspinal enhancement is seen.C2-3: Left facet hypertrophy. No significant spinal canal or neuroforaminal stenosis. C3-4: Facet hypertrophy. Mild disc osteophyte formation. Mild spinal canal narrowing. No significant foraminal narrowing. C4-5: No significant spinal canal or foraminal narrowing. C5-6: Disc osteophyte formation. Mild spinal canal narrowing. Mild bilateral foraminal narrowing. C6-7: Facet hypertrophy. Disc osteophyte formation. Mild spinal canal narrowing. Mild bilateral foraminal narrowing. C7-T1: Facet hypertrophy. No significant spinal canal or neuroforaminal stenosis.
1.No acute intracranial abnormality. Scattered small white matter lesions likely reflect chronic microvascular ischemic disease.2.Mild degenerative findings in the cervical spine without evidence of any high-grade spinal canal or neuroforaminal stenosis.