Male, 52 years old, with neurofibromatosis type I and a long-standing temporal lobe lesion, who recently had a seizure after being controlled for a few years. A multicystic lesion involving the left cavernous and choroidal fissure is redemonstrated showing no significant interval change in size, morphology or signal characteristics, comparing to exams dating back to 2007. The posterior component of the lesion continues to measure 18 x 11 mm (image 14 series 401), previously 18 x 11 mm. As before, no associated lesional enhancement is seen. There is a small vessel looping around and within the lesion, but this is unchanged.Brain parenchymal morphology is otherwise unremarkable. No areas of significant edema or mass effect is detected. At most, a punctate focus of T2 hyperintensity is seen within the right inferior parietal lobule, unchanged. No pathologic parenchymal or extra-axial enhancement is detected. No findings to suggest intracranial hemorrhage are seen. The ventricular system is normal in size and morphology.An ill-defined, enhancing cutaneous lesion is seen within the medial left periorbital soft tissues, unchanged. As before, vascular prominence is noted within the left masticator space. A chronic deformity of the left lamina papyracea is unchanged.
1.No change in size or morphology of a nonenhancing multicystic lesion involving the left hippocampus and choroidal fissure. The lesion is unchanged when compared to an examination dating back to 2007.2.No new intracranial lesions are seen to account for the patient's seizure.3.A left periorbital cutaneous lesion is unchanged perhaps representing a plexiform neurofibroma.