L1 vertebral compression fracture Five lumbar type vertebral bodies are presumed to be present. There is compression fracture involving the L1 vertebral body with 80-90% loss of height grossly similar to prior radiograph from 6/16/2015. There is mild osseous retropulsion with effacement of the ventral thecal sac. No high-grade spinal canal stenosis. Remainder of the vertebral body heights are maintained. There is mild kyphotic angulation at the level of the compression fracture. Alignment of the lumbar spine is otherwise maintained. There is a lytic focus involving the posterior aspect of the superior endplate which may represent a Schmorl's node. No suspicious osseous lesion is definitively appreciated within the limits of the noncontrast study. There is minimal T2/STIR hyperintensity involving the L1 vertebral body. No signal abnormality involving the visualized spinal cord. Conus is borderline low at the L2-L3/upper L3 level which is of doubtful clinical significance. No evidence of filar lipoma or other lesions in the spinal canal.Mild degenerative changes are seen, as described below:L1-L2: No significant disc disease. No significant spinal canal or right neural foraminal stenosis. Minimal left neural foraminal stenosis.L2-L3: Minimal disc bulge and mild bilateral facet arthropathy. No significant spinal canal or neural foraminal stenosis.L3-L4: Minimal disc bulge and mild bilateral facet arthropathy. No significant spinal canal or neural foraminal stenosis.L4-L5: Minimal disc bulge and mild bilateral facet arthropathy. No significant spinal canal or neural foraminal stenosis.L5-S1: Minimal disc bulge and mild bilateral facet arthropathy. No significant spinal canal or neural foraminal stenosis.Paraspinous soft tissues are within normal limits.
1. Chronic L1 compression fracture with 80-90% height loss grossly similar to prior lumbar spine radiograph from June 20, 2015, allowing for differences in technique. There is mild STIR hyperintensity involving the bone marrow at this level. There is mild osseous retropulsion associated with the compression fracture which partially effaces the ventral thecal sac without high-grade spinal canal stenosis. Focal defect involving the posterior aspect of the superior L1 endplate may represent a Schmorl's node. No gross evidence of neoplastic disease in the lumbar spine; follow-up can be considered as clinically indicated.2. Minimal degenerative changes in the lumbar spine. Aside from the mild spinal canal stenosis related to compression fracture, there is no significant spinal canal or neural foraminal stenosis in the lumbar spine.