Cirrhosis of liver, HCV, evaluate for blockage of biliary ducts, elevated alkaline phosphatase and GGT ABDOMEN:LIVER, BILIARY TRACT: Nodular liver contour and diffusely heterogeneous hepatic parenchymal signal. No suspicious arterially enhancing hepatic focus delineated. Mildly enlarged perihepatic and periportal lymphadenopathy, stable to mildly decreased in size compared to earlier exam and most likely reactive in etiology. Status post cholecystectomy. Not significantly changed from prior study is mild intrahepatic biliary duct dilatation. Common bile duct normal in size, distalmost CBD not well seen, similar in appearance to prior study. SPLEEN: Stable nonenhancing hypointense foci in spleen, may reflect sequela of prior ischemic insult.PANCREAS: No significant abnormality noted.ADRENAL GLANDS: Decreasing residual T1 hyperintensity seen in left adrenal region, for example, component measuring 3.8 x 2.1 cm on prior exam now measures 0.9 x 0.5 cm, persistent left adrenal nodularity, image 61 series 10.KIDNEYS, URETERS: Bilateral renal cysts.RETROPERITONEUM, LYMPH NODES: No significant abnormality noted.BOWEL, MESENTERY: No significant abnormality noted.BONES, SOFT TISSUES: Spinal degenerative disease and scoliosis.OTHER: Status post sternotomy. Cardiomegaly.
1. Not significantly changed from prior study is mild intrahepatic biliary duct dilatation. Common bile duct normal in size, distalmost CBD not well seen, similar in appearance to prior study. Findings may reflect combination of postcholecystectomy state/benign stricturing. No definite enhancing lesion seen.2. Cirrhotic liver. No suspicious liver lesion. Mildly enlarged perihepatic and periportal lymphadenopathy, stable to mildly decreased in size compared to earlier exam and most likely reactive in etiology. 3. Decreasing left adrenal hemorrhage, persistent left adrenal nodularity. 