Weakness [R53.1] / Hyperlipidemia, unspecified [E78.5] / Cardiomyopathy, unspecified [I42.9], Reason for Study: ^Reason: question of ischemic stroke History: left sided clumsiness and weakness Brain MRIThere are scattered restricted diffusion lesions on the right pre central and post central gyri indicating possible acute/subacute ischemic infarction.There is no evidence of hemorrhagic conversion of those lesions.Scattered high signal lesions on FLAIR/T2 images on the left centrum semiovale and left occipital lobe are non-specific.The ventricles, sulci and cisterns are symmetric and unremarkable. The gray-white matter differentiation is normal. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.Brain MRA3D time of flight MOTSA MRA brain images with maximum intensity projections of the anterior/posterior intracranial circulation demonstrate normal ICAs, MCAs and ACAs. Vertebrobasilar system appears to be normal. No intracranial arterial luminal stenosis. No intracranial arterial aneurysm.Neck MRA3D MRA neck post-gadolinium images with maximum intensity projections of the cervical vasculature demonstrate normal flow enhancement in a normal aortic arch origin of the right brachiocephalic, left common carotid, and left subclavian arteries. The right vertebral artery appears to be hypoplastic. The vertebral artery origins are normal. There is normal flow enhancement through the bilateral common carotid, carotid bifurcations, internal/external carotid, and vertebral arteries.
1. Multiple acute/subacute ischemic infarction on the right pre-central and post-central gyri without evidence of hemorrhagic conversion.2. No evidence of intra and extra cranial arterial luminal stenosis. 