Reason: is there any abnormality? History: term infant with hx perinatal depression and subarachnoid bleed, now s/p therapeutic hypothermia. There is a large, extra-periosteal fluid collection underlying the posterior scalp compatible with caput succedaneum with minimal hemorrhage, overall increased in size from 8/12/2016 CT. The ventricles and sulci are within normal limits. The basal cisterns remain patent. There is signal abnormality compatible with trace extra-axial blood product including scattered mild subarachnoid hemorrhage along the anterior temporal lobes as well as trace subdural blood along the parietal lobes, occipital lobes, and along the transverse sinuses. There is patchy T1 hyperintensity within the bilateral basal ganglia in a pattern that is not consistent with normal myelination and which is concerning for hypoxic ischemic encephalopathy. There is no associated restricted diffusion, edema, or mass effect. The brain parenchyma otherwise appears well formed; sulcation and myelination patterns are appropriate for age. The pituitary, brainstem, and cerebellum appear within normal limits. The paranasal sinuses and mastoid air cells are not yet aerated.
1.Scattered extra-axial hemorrhage without associated significant mass effect.2.Patchy signal abnormality in the bilateral basal ganglia concerning for sequela of hypoxic ischemic encephalopathy. Due to the timing of this examination at approximately 1 week postnatally, these imaging findings may underestimate the true extent of injury, and therefore follow-up imaging in one to two weeks can be considered as clinically warranted.3.Large caput succedaneum. 