Other specified disorders of muscle [M62.89], Reason for Study: ^Reason: R sided weakness History: R face/arm/leg pure motor deficit Brain MRI:Motion artifacts significantly degraded image quality.Thus, only very limited sequences are obtained.There is evidence of midline suboccipital craniectomy.There are high signal intensity on DWI at bilateral cerebellar tonsils (left more than right) are seen which are corresponding to prior surgery.Patchy multifocal FLAIR high signal intensity lesions on bilateral periventricular white matter and bilateral trigone area (left more than right) are non-specific and could be related to small vessel ischemic disease.There is no evidence of acute ischemic or hemorrhagic lesionVentricular system appears to be slightly enlarged but no change since prior scan.There is no mass, mass effect, edema, midline shift, restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Brain MRA3D time of flight MOTSA MRA brain images with maximum intensity projections of the anterior/posterior intracranial circulation are performed.In flow related enhancement are seen on bilateral distal ICAs and vertebrobasilar system indicating patent and maintaining craniopetal flow. However, due to significant motion artifacts, this brain MRA is non-diagnostic especially in terms of arterial lumen evaluation and screening for intracranial arterial aneurysm.Brain Perfusion:Although there are motion artifacts during the perfusion scan, reasonable quality MR perfusion analysis is created.The arterial input function was measured at the right distal ICA.Calculated MTT, CBV, CBF and TTP maps show no evidence of asymmetry or focal perfusion defect.
1. Evidence of midline suboccipital craniectomy and bilateral cerebellar tonsilar surgery.2. No acute ischemic or hemorrhagic lesion in limited sequences.3. Mild non specific FLAIR high signal intensity lesions.4. Normal brain perfusion study.5. Non diagnostic brain MRA. 