 Redemonstrated are multiple foci of T2 hyperintensity primarily within the periventricular white matter, however also within the pons and bilateral middle cerebellar peduncles, appearing similar to the 2009 comparison and suggestive of multiple sclerosis. There is no associated enhancement. Pontine lesions are associated with small foci of susceptibility hypointensity and thus may represent a component of small vessel ischemic disease with microhemorrhage.The ventricles and sulci are normal in size. The cerebellar tonsils are in normal position. There are no masses, mass effect or midline shift. The pituitary gland is normal in size. There is no evidence for intracranial hemorrhage or acute cerebral, brainstem or cerebellar infarction. No diffusion-weighted abnormalities are identified. There are no extraaxial fluid collections or subdural hematomas. Flow voids are present within the major vessels indicating patency. The paranasal sinuses and mastoid air cells are clear. There is no abnormal enhancement within the brain.
Redemonstrated are multiple foci of T2 hyperintensity primarily within the periventricular white matter, however also within the pons and bilateral middle cerebellar peduncles, appearing similar to the 2009 comparison and suggestive of multiple sclerosis. There is no associated enhancement. Pontine lesions are associated with small foci of susceptibility hypointensity and thus may represent a component of small vessel ischemic disease with microhemorrhage.