Progressive vision loss left eye after head trauma, in context of poor visual acuity with retinitis pigmentosa. Brain: There is no evidence of intracranial hemorrhage, mass, or acute infarct. There are scattered areas of high T2 signal in the cerebral white matter, particularly in a periventricular distribution. There is also a more focal punctate area of T2 hyperintensity in the left caudate head. There is no abnormal intracranial enhancement. The ventricles and basal cisterns are normal in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. The skull, paranasal sinuses, and scalp soft tissues are grossly unremarkable. However, there is an enhancing subcentimeter nodule in the superficial right parotid gland.Orbits: There are bilateral lens implants, The globe and ocular adnexa are otherwise grossly unremarkable. There is no evidence of intraorbital mass lesions or abnormal enhancement in the orbits. The bilateral optic nerves display normal signal characteristics.
1. Probable chronic small vessel white matter ischemic disease and a chronic lacunar infarct versus prominent perivascular space in the left caudate, but no evidence of intracranial hemorrhage, mass, or acute infarct. 2. Aside from bilateral lens implants, the orbital contents appear grossly unremarkable. 3. An enhancing subcentimeter nodule in the right parotid gland may represent a lymph node or salivary gland neoplasm, for example.