History of AML and pancreatitis. Evaluate pancreatic lesion. History of hepatitis B. AML. ABDOMEN:LIVER, BILIARY TRACT: Loss of signal on in phase imaging compatible with iron deposition. The liver is of normal morphology and size. No focal lesion biliary ductal dilatation or vascular abnormality.SPLEEN: The spleen is low in signal intensity compatible with iron deposition.PANCREAS: Lobular cystic lesion in the head of the pancreas measures approximately 1.9 x 1.8 cm and appears to be multilocular and/or septated. The lesion appears to communicate with a normal caliber main pancreatic duct (series 7/100) which measures up to 3 mm in caliber. No suspicious postcontrast enhancement or mural nodularity is apparent. The pancreas is otherwise normal in morphology and enhancement.ADRENAL GLANDS: No significant abnormality noted.KIDNEYS, URETERS: No significant abnormality noted.RETROPERITONEUM, LYMPH NODES: No significant abnormality noted.BOWEL, MESENTERY: No significant abnormality noted.BONES, SOFT TISSUES: No significant abnormality noted.OTHER: No significant abnormality noted.
1.1.9 x 1.8 cm multilobular multiloculated/septated pancreatic head cystic lesion may represent a branch type IPMN without suspicious enhancing features. The main pancreatic duct measures up to 3 mm.2.Hemosiderosis.3.No suspicious liver lesion or specific morphologic stigmata of cirrhosis. Surveillance imaging is recommended in the setting of Hepatitis B.