New diagnosis of cirrhosis, ascites, elevated LFTs ABDOMEN:LIVER, BILIARY TRACT: Micronodular liver contour, suggesting cirrhosis. Minimal intrahepatic biliary duct prominence. Dilated proximal common bile duct, measuring up to 11 mm, with gradual distal tapering seen. No focus of intraluminal signal void seen to suggest choledocholithiasis. Distended cystic duct. Findings suggestive of underlying benign stricture, no definite obstructing masslesion seen. However, evaluation suboptimal due to inability to perform dynamic postcontrast imaging. Suboptimal evaluation of liver parenchyma secondary to lack of dynamic postcontrast imaging, no suspicious liver lesion seen within these limitations.SPLEEN: No significant abnormality noted.PANCREAS: Mildly prominent pancreatic duct, measuring up her limits of normal, 3 mm. Mildly decreased intrinsic T1 signal pancreas, may be sequela of chronic pancreatitis.ADRENAL GLANDS: Bilateral diffusely enlarged adrenal glands, may reflect underlying adenomatous hyperplasia. KIDNEYS, URETERS: T2 hyperintense renal foci, most likely cysts. No hydronephrosis.RETROPERITONEUM, LYMPH NODES: Flattened configuration of the IVC.BOWEL, MESENTERY: Thickened small bowel, likely reactive in the setting of ascites.BONES, SOFT TISSUES, OTHER: Small pleural effusions. Large ascites.
1. Minimal intrahepatic biliary duct prominence and dilated proximal common bile duct, measuring up to 11 mm, with gradual distal tapering seen. No focus of intraluminal signal void seen to suggest choledocholithiasis. These findings favor underlying benign stricture, no definite obstructing masslesion seen. However, evaluation suboptimal due to inability to perform dynamic postcontrast imaging. Per MRI technologist, patient was claustrophobic and unable to tolerate dynamic series acquisition. Correlation with patient's clinical history/laboratory values recommended and confirmation of benign findings with ERCP should be considered.2. Underdistended and thick walled sludge-containing gallbladder, nonspecific in setting of ascites. 3. Micronodular liver contour, suggesting cirrhosis.4. Mildly decreased intrinsic T1 signal pancreas, may be sequela of chronic pancreatitis.5. Paraaortic and retrocaval soft tissue thickening, may reflect small adenopathy or chronic scarring, further evaluation with CT imaging recommended. 6. Large ascites, small pleural effusions. 