 There is decreased white matter volume adjacent to the occipital horns as well as increased T2 signal with resultant enlargement of the occipital horns. There is also subtle increased T2 signal in the posterior bilateral thalami. The corpus callosum is thinned posteriorly, compatible with Wallerian degeneration. The basal cisterns remain patent. There is no midline shift or mass effect. There is no diffusion abnormality. No extra-axial fluid collection is identified.Normal flow-voids are demonstrated in the major intracranial vascular structures. The craniocervical junction are within normal limits.
Periventricular white matter volume loss and signal abnormality is compatible with leukomalacia/encephalomalacia. Abnormal T2 signal is also noted in the posterior bilateral thalami.