58 years Female (DOB:11/17/1957)Reason: Increased vertigo and episodes of loss of consciousness and h/o posterior circulation aneurysm. Eval for structural lesion/aneurysm change History: vertigo, loss of consciousnessPROVIDER/ATTENDING NAME: SANDRA LYNN ROSE SANDRA LYNN ROSE MRI of the brainNo diffusion weighted abnormalities are appreciated.The CSF spaces are appropriate for the patient's stated age with no midline shift. No abnormal mass lesions are appreciated intracranially. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact.MRA brain:The left posterior communicating artery has a sizable infundibulum measuring approximately 3 mm in diameter. There is a 3.5 mm aneurysm originating from the infundibulum and projecting posteriorly, laterally and inferiorly when compared to prior exam of 11/18/2014 it appears slightly enlarged.Antegrade flow is present in the distal internal carotid arteries, the distal vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries.There is no evidence for intracranial cerebrovascular occlusion.The anterior communicating artery is identified. The posterior communicating arteries are not readily identified. The vertebral arteries are similar in size.
1.There is redemonstration of a left posterior communicating artery aneurysm associated with an infundibulum of the left PCOMA which appears to be perhaps a millimeter larger on the current versus the prior exam. 2.No evidence for cerebrovascular occlusive disease.3.No evidence for acute ischemic cerebral infarction.