76 years male with tinnitus.Provider Name: RIMAS V. LUKAS BRAINBilateral subdural effusions are present related to age-related atrophy. No midline shift or herniation.No abnormal enhancing mass lesions are appreciated intracranially. No intracranial hemorrhage is identified. Bilateral periventricular and subcortical white matter T2/FLAIR hyperintensity which are likely vascular related; additional areas of T2/FLAIR hyperintensity are present within the basal ganglia, thalami and brainstem also most likely vascular related.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits elongation the AP dimension of the eyes associated with scleral thickening posteriorly. The eyeball lenses are thin. IACThere is a lesion within the posterior aspect of the right internal auditory canal which demonstrates intermediate T1 and T2 signal intensity with enhancement measuring 4.4 x 3.9 mm (series 19, image 12). This is stable compared to prior exam. The left internal auditory canal is normal. The cochlea and semicircular canals appear intact. The cochlear nerve can be followed bilaterally tracking into the cochlea. The modiolus is well formed bilaterally.No abnormal enhancing lesions are appreciated at the cerebellopontine angle cisterns.
1.Stable small enhancing lesion within the right internal auditory canal likely a vestibular schwannoma.2.Multiple patchy lesions in the brainstem, thalami and basal ganglia are most likely vascular related.3.Periventricular and subcortical white matter lesions of a mild degree are nonspecific. At this age they are most likely vascular related. 4.Small Bilateral subdural effusions are present likely related to age-related atrophy.5.Bilateral staphylomas.