History of FNH and secondary hemosiderosis. ABDOMEN:LIVER, BILIARY TRACT: There is a loss of signal intensity of the liver on out of phase imaging most suggestive of steatosis.Multiple faintly T2 weighted hyperintense foci are noted in both lobes of the liver, unchanged in size. These lesions are faintly T1 hyperintense. Following contrast administration these lesions demonstrate relatively homogeneous arterial phase enhancement and become iso-intense to mildly hyperintense on portal venous and equilibrium phase imaging. On hepatobiliary phase imaging the lesions are relatively hypointense to the liver.The index right hepatic lobe peripheral lesion measures 2.8 x 2.0 cm (series 8/35), compared to 2.1 x 1.5 cm previously.The index segment 4B lesion measures 3.8 x 3.9 cm (series 3/68) compared to 3.1 x 2.8 cm previously.Previously seen segment II heterogeneous lesion appears to have involuted (03/15/2015 series 401/82).Status post cholecystectomy.SPLEEN: No significant abnormality noted.PANCREAS: No significant abnormality noted.ADRENAL GLANDS: No significant abnormality noted.KIDNEYS, URETERS: No significant abnormality noted.RETROPERITONEUM, LYMPH NODES: No significant abnormality noted.BOWEL, MESENTERY: No significant abnormality noted.BONES, SOFT TISSUES: No significant abnormality noted.OTHER: No significant abnormality noted.
Mild increase in size of several of the bilobar liver lesions, previously characterized as FNHs. However, on today's hepatobiliary phase imaging the lesions are relatively hypointense to the liver, contrary to what would be expected in a typical FNH and changed from prior hepatobiliary phase imaging. These may represent multiple adenomas versus atypical FNHs.Hepatic steatosis.