Unspecified fall, initial encounter [W19.XXXA] / Other symptoms and signs involving cognitive functions and awareness [R41.89], Reason for Study: ^Reason: eval for seizure cause History: possible seizures There is no evidence of acute ischemic or hemorrhagic lesion.There is no abnormal enhancement.Patchy scattered FLAIR/T2 high signal intensity lesions are seen on periventricular white matter and centrum semiovale indicating nonspecific small vessel ischemic disease.Very prominent dilated perivascular spaces are seen throughout the supratentorial brain parenchyma especially around bilateral temporal lobe. These findings are nonspecific and likely to be related to normal aging phenomenon. Although remote, differential diagnosis include perivascular myelin loss, atherosclerotic change, secondary to arterial pulsation especially due to hypertension, increased permeability and etc.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, or restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
1. No evidence of acute ischemic or hemorrhagic lesion. No abnormal enhancement.2. Nonspecific small vessel ischemic disease.3. Prominent perivascular spaces especially around bilateral temporal lobes, most likely related to aging process. 