64 years Female (DOB:9/6/1951)Reason: eval for causes of AMS History: as above, hx of schizophreniaPROVIDER/ATTENDING NAME: TIEN S DONG TIEN S DONG The CSF spaces are appropriate for the patient's stated age with no midline shift. There are symmetric subdural effusions present which are probably related to brain atrophy.No abnormal enhancing mass lesions are appreciated intracranially. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.There are several periventricular and subcortical punctate hyperintense white matter lesions present identified on the FLAIR and T2 images. These are likely of little clinical significanceThere is a punctate T2 and FLAIR signal hyperintensity present along the anterior aspect of the right thalamus. There is no associated susceptibility effect or diffusion restriction appreciated.There are small parafalcine calcifications present which create some susceptibility artifact on diffusion-weighted imaging. These can be identified on the CT scan from 1/28/2016. One measures 12 x 6 mm and another one measures 19 x 6 mm.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact.
1.Small lesion along the right thalamus is suspected represent an old lacunar infarct. 2.Periventricular and subcortical white matter lesions of a mild degree are nonspecific. At this age they are most likely vascular related. 3.There is generalized brain atrophy present.