Stroke progression: somnolence. Many of the sequences are degraded by patient motion. There is interval evolution of a recent left internal capsule posterior limb infarct, with signs of early Wallerian degeneration along the corticospinal track. There is no evidence of intracranial hemorrhage and there is no evidence of new acute infarcts. There is a background of extensive cerebral white matter T2 hyperintensity. There are also small foci of encephalomalacia in the right cerebellar hemisphere and right corona radiata. The ventricles and basal cisterns are unchanged in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. The skull is grossly unremarkable. There is a right lens implant. There is a 10 mm thick right suboccipital subcutaneous lipoma.
Interval evolution of a recent left internal capsule posterior limb infarct, with signs of early Wallerian degeneration along the corticospinal track, but no evidence of hemorrhagic transformation, superimposed upon a background of chronic small vessel ischemic disease and lacunar infarcts.