known CCM left frontal lobe, headache and fatigue. There is no evidence of acute ischemic lesion.There is, however, at least two low signal intensity lesions on T2 weighted images.The most prominent and largest one is on the left frontal lobe lesion (about 9mm) with low signal intensity rim indicating hemosiderin deposition. Relatively enlarge vascular structure on the superior aspect of the lesion indicate associated small developmental venous anomaly.Another suspicious low signal intensity lesion is on the left posterior inferior thalami (series 601, image 14/28). Since there is no susceptibility imaging sequence, it cannot be confirmed as another CCM lesion.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
Incomplete examination due to patient's intolerance.About 9mm sized CCM on the left frontal lobe associated with a small DVA.Suspicious lesion on the left thalami as described above. 