Paresthesia of skin [R20.2], Reason for Study: ^Reason: stroke/ History: stroke? There is no evidence of acute ischemic or hemorrhagic lesion.Previously seen left frontal lobe precentral gyrus ischemic infarct with hemorrhagic transformation is again seen, demonstrate interval encephalomalacia.Various sized tissue defects involving bilateral basal ganglia and right thalamus indicating chronic lacunar infarctions.Underlying brain shows patchy FLAIR/T2 high signal intensity lesions on bilateral periventricular white matter and centrum semiovale indicating nonspecific small vessel ischemic disease, unchanged.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, or restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
1. No evidence of acute ischemic or hemorrhagic lesion.2. Interval development of minimal encephalomalacia on previously diagnosed as acute ischemic infarct involving the left frontal lobe frontal operculum (precentral gyrus) with evidence of prior hemorrhagic conversion.3. Nonspecific small vessel ischemic disease, unchanged since prior scan.4. Multiple lacunar infarctions involving bilateral basal ganglia and right thalamus, unchanged since prior scan.