 There tubular filling defect within the right transverse sinus which shows high signal intensity on T2, low signal intensity on T1, but best seen on postcontrast T1 coronal scan. The length was measured about 22mm and is connected to globular inferoposterior outpouching lesion from the dural sinus which may indicate involvement within arachnoid granulation. The signal characteristics of the lesion is likely represent chronic intra luminal thrombus. There is no evidence of redirection of venous flow or MR evidence of venous congestive encephalopathy.  Several punctate scattered high signal intensity foci on FLAIR on bilateral periventricular white matter indicate non specific small vessel disease. There is no evidence of acute ischemic or hemorrhagic lesion on this scan.Cerebellar sulci appear to be slightly more prominent comparing to those of cerebrum, but not that significant. The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia, or abnormal contrast enhancement. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.About 12.9mm sized left frontal epidermal inclusion cyst.
1. Chronic right transverse sinus intraluminal thrombus which extends toward adjacent arachnoid granulation without showing evidence of redirection of normal craniofugal flow nor evidence of venous congestive encephalopathy. 2. No evidence of acute ischemic or hemorrhagic lesion.3. Minimal non specific small vessel ischemic disease.