History of congenital cavernous malformation, yearly surveillance. There is redemonstration of multiple areas of susceptibility artifact compatible with cavernous malformations. The lesion in the medulla appears minimally larger, now measuring up to 10 mm, previously 9 mm and demonstrates a slightly changed internal composition with increased T1 signal. There is no associated perilesional edema. Several other lesions including the dominant 12 mm lesion in the left frontal lobe appear unchanged.The ventricles and sulci are within normal limits. The basal cisterns remain patent. There is no midline shift or mass effect. There is redemonstration of scattered punctate T2 hyperintensities in the bilateral supratentorial white matter, unchanged in appearance. There is no evidence of acute infarct. No abnormal pathological enhancement is identified. Normal flow-voids are demonstrated in the major intracranial vascular structures. There is a persistent right fetal origin PCA.The orbits, skull, paranasal sinuses, and scalp soft tissues are grossly unremarkable.
Minimal increase in size and change in composition of medullary cavernous malformation without evidence of perilesional edema. Additional cavernous malformations not significantly changed.