 There is no evidence of acute infarct. There are multiple patchy foci of T2 hyperintensity within the supratentorial and infratentorial white matter that are most compatible with chronic small vessel ischemic disease. There are also a few small foci of T2 hyperintensity and enhancement with minimal associated susceptibility effect in the left cerebellar hemisphere.There is an ectatic distal intracranial right vertebral artery that makes a prominent impression upon the right aspect of the medulla of doubtful clinical significance. The major cerebral flow voids are intact. The ventricles and basal cisterns are normal in size and configuration. There is no midline shift or herniation.
1.No evidence of acute infarct, however, a few patchy foci of signal abnormality and enhancement within the left cerebellar hemisphere are most compatible with subacute lacunar infarcts. A follow-up MRI in 8 weeks may be helpful to ensure resolution of enhancement as clinically indicated.2.Supratentorial and infratentorial chronic small vessel ischemic disease.