Pain and edema dorsomedial midfoot and calcaneus after increased time on feet. Evaluate for stress fracture of navicular and calcaneus. Right ankle/midfoot: There is diffuse subcutaneous edema involving the ankle that extends distally along the dorsolateral aspect of the foot. I see no focal bone marrow edema to suggest a stress fracture. Specifically, the signal intensity of the calcaneus and navicular is normal. There is a small amount of fluid within the ankle and subtalar joints, but no large effusion. I see no osteochondral defects. Ankle ligaments appear normal. Ankle tendons appear normal. There is a peroneus quartus, a normal variant. There is mild edema of the flexor hallucis longus and extensor digitorum longus muscles which is nonspecific. There appears to be a small accessory muscle that lies medial to the flexor hallucis longus.Left ankle/midfoot: There is diffuse subcutaneous edema involving the ankle that extends distally along the dorsal aspect of the foot. On the fluid sensitive sequences, there are small scattered ill-defined patchy foci of mild edema-type signal within the talus, navicular, calcaneus, and base of the second metatarsal which are nonspecific, but may in part be degenerative in etiology; I see no linear focus of signal abnormality to suggest a stress fracture. Increased signal intensity on the fluid sensitive sequences within the distal tibial diaphysis isn't fully imaged on this study and may simply represent residual red marrow, but I cannot exclude the possibility of marrow edema. I see no linear focus of signal abnormality to suggest a stress fracture. There is a small amount of fluid within the tibiotalar and subtalar joints but no large effusion. I see no osteochondral defect. Ankle ligaments are unremarkable. Ankle tendons are unremarkable. There is a peroneus quartus, a normal variant. There is mild edema-type signal involving the flexor hallucis longus muscle which is nonspecific. I see no fluid collections to suggest an abscess.
Nonspecific subcutaneous edema and other findings as described above without stress fracture evident. Increased signal intensity within the left distal tibial diaphysis is incompletely imaged on this study and may simply represent residual red marrow; however if there is clinical concern for tibial stress fracture, dedicated tibial/fibular MRI may be considered.