 Redemonstrated are diffuse patchy foci of increased T2 signal within the bilateral basal ganglia, periventricular white matter, the pons, and bilateral cerebral peduncles. These findings are unchanged compared to previous examination. No abnormal enhancement.There is no evidence for intracranial hemorrhage or acute cerebral, brainstem or cerebellar infarction. No diffusion-weighted abnormalities are identified. There are no extraaxial fluid collections or subdural hematomas.The ventricles and sulci are stable in size and configuration with mild cerebral volume loss. The cerebellar tonsils are in normal position. The pituitary gland is normal in size. Flow voids are present within the major vessels indicating patency. Bilateral optic nerves appear atrophic, left greater than right. The optic chiasm, and tracts appear unremarkable. There is no optic nerve sheath dilatation. The extraocular muscles are normal and symmetric with physiologic, homogeneous, and intense contrast enhancement. The globes appear normal with physiologic enhancement of the ciliary bodies and choroid. No retinal detachment or vitreous clouding. The lacrimal glands are symmetric and within normal limits for size. No orbital mass lesion. No enhancing pathology.
1.Stable abnormal nonenhancing T2 signal abnormality within the bilateral basal ganglia and brainstem, which is secondary to presumed neurosarcoidosis.2.Bilateral optic nerves appear atrophic, left greater than right.