Unexplained anorexia and dysgeusia. The postcontrast images are motion degraded. Confluent biparietal deep white matter T2/FLAIR hyperintense, T1-hypointense signal is not associated with diffusion restriction. There are scattered smaller T2/FLAIR hyperintense lesions throughout the cerebral white matter bilaterally also without diffusion restriction. Following contrast injection, there is no contrast enhancement in any of these lesions. An incidental left parietal developmental venous anomaly is present. There is no mass or mass effect. There is no midline shift. The CSF-containing spaces are symmetrically enlarged, reflecting mild volume loss. The vascular flow voids are present. Accounting for probable calcifications in the globi pallidi, there is no susceptibility artifact to indicate intracranial hemorrhage.The paranasal sinuses and mastoid air cells are clear. The orbits are notable for bilateral cataract surgery. There is no destructive skull lesion.
1.No evidence of acute infarct, intracranial mass, or mass effect. No abnormal enhancement.2.Patchy confluent areas and scattered foci of T2/FLAIR hyperintensity in the periventricular and subcortical white matter are nonspecific but compatible with moderate chronic small vessel ischemic disease.I personally reviewed the Images and/or procedure with the Resident/Fellow and agree with this report.