Male, 69 years old, history of smouldering multiple myeloma, now with new lesions seen on skeletal survey. Assess for additional bony disease. Cervical:No areas of concerning marrow signal replacement, edema or enhancement are seen. Vertebral body morphology is normal. Spinal alignment is anatomic. Spinal cord signal intensity and morphology are within normal limits.Mild facet hypertrophy is seen within the cervical region, most severely affecting C7-T1 on the right. Small central disk osteophyte complexes are also present at C3-4 and C4-5. No high-grade spinal canal or neuroforaminal stenosis is suspected.Thoracic:No areas of concerning marrow signal replacement, edema or enhancement are seen. Vertebral body morphology is normal. Spinal alignment is anatomic. Spinal cord signal intensity and morphology are within normal limits.Scattered facet hypertrophy is seen through the thoracic spine. No significant degenerative disk disease, spinal canal or neuroforaminal stenosis is suspected.Lumbar:No areas of concerning marrow signal replacement, edema or enhancement are seen. Vertebral body morphology is normal allowing for small Schmorl's nodes the superior endplates of L2 and L3. Spinal alignment is anatomic. The visualized conus and cauda equina demonstrate normal signal intensity and morphology.L1-2: Mild facet hypertrophy and ligamentum flavum thickening. No significant spinal canal or neuroforaminal stenosis. L2-3: Moderate facet hypertrophy and ligamentum flavum thickening. Mild bulging disk. Mild generalized spinal canal stenosis. Moderate bilateral foraminal narrowing. L3-4: Moderate facet hypertrophy and ligamentum flavum thickening. Mild bulging disk. Mild generalized spinal canal stenosis. Moderate to severe bilateral foraminal narrowing. L4-5: Moderate facet hypertrophy and ligamentum flavum thickening. Mild bulging disk with a small superimposed central protrusion. No significant spinal canal stenosis. Moderate bilateral foraminal narrowing. L5-S1: Moderate facet hypertrophy. Mild bulging disk with small central annular fissure. No significant spinal canal or neuroforaminal stenosis.
No evidence to suggest myelomatous disease in the spine.