 Please note the patient requested the study be finished before all sequence acquisitions could be completed. Unable to obtain the DTI, T1 3-D SPGR, and VenBOLD.Brain MRI:There are foci of T2 hyperintensity without restricted diffusion throughout the white matter, both subcortical and periventricular, as well as within the cerebellar hemispheres. Additionally, there is a small cortical focus of encephalomalacia with gliosis involving the right postcentral gyrus from past ischemia. Small foci of encephalomalacia within the cerebellar hemispheres are consistent with remote small infarcts.The ventricles and sulci are normal in size for age. The cerebellar tonsils are in normal position. There are no masses, mass effect or midline shift. The pituitary gland is normal in size. There is no evidence for acute cerebral, brainstem or cerebellar infarction. There are no extraaxial fluid collections or subdural hematomas. The paranasal sinuses and mastoid air cells are clear. Brain MRA:There is focal loss of flow related signal on the time-of-flight images involving both vertebral arteries (left greater than right) at the level of C1/2, where the vessels have an in-plane of acquisition position. Thus, this may be artifactual due to loss of in-plane flow signal although stenosis cannot be excluded.Otherwise, there is antegrade flow within the distal bilateral vertebral arteries, within the basilar artery, distal bilateral internal carotid arteries and carotid siphons and bilateral anterior, middle and posterior cerebral arteries. No arterial stenoses, occlusions or aneurysms are identified.
1.The patient requested the study be finished before all sequence acquisitions could be completed. Unable to obtain the DTI, T1 3-D SPGR, and VenBOLD.2.Chronic supratentorial and infratentorial ischemia without acute infarction.3.There is focal loss of flow related signal on the time-of-flight images involving both vertebral arteries (left greater than right) at the level of C1/2, where the vessels have an in-plane of acquisition position. Thus, this may be artifactual due to loss of in-plane flow signal although stenosis cannot be excluded.