 There is a focus of T1 hyperintensity and T2/FLAIR hyperintensity involving the posterior limb of the internal capsule on the left abutting the lateral aspect of the thalamus. No associated susceptibility effect. Remainder of the brain parenchyma appears unremarkable without findings to suggest traumatic encephalomalacia or hemosiderin deposition in the brain related to prior trauma. No evidence of recent infarct or hemorrhage. No intracranial mass or mass effect. No extra-axial collections. No hydrocephalus. The major cerebral flow voids are intact. There is small focus of T2 hyperintensity and restricted diffusion involving the subcutaneous soft tissues in the left temporal occipital region which may represent a sebaceous cyst. The orbits, skull, paranasal sinuses, and scalp soft tissues are otherwise grossly unremarkable.
There is a focus of T1 and T2 hyperintensity involving the posterior limb of the internal capsule on the left which is nonspecific and of uncertain etiology. Finding may represent mineralization related to prior trauma if there was history of prior injury in this region. Prior inflammation or other toxic metabolic injury also remain a possibility. Comparison with prior studies is suggested if available.Remainder of the brain appears otherwise unremarkable without evidence of traumatic encephalomalacia or hemosiderin deposition related to chronic hemorrhage.