Unspecified convulsions [R56.9], Reason for Study: ^Reason: Altered mental status, concern for hypercoagulable state, hx of pericardial effusion s/p window and hypotension History: AMS Brain MRIThere are focal high signal intensity lesions on the left cerebellar hemisphere on 2 consecutive images (series 4, image 7 and 8). The inferior lesion shows restricted diffusion on ADC map but the superior one does not. The T2 images show slit like craniocaudal direction tissue defect on coronal scan. Therefore, this lesion is most likely T2 shine through artifact with subtle misregistration instead of acute ischemic infarction.  There is no evidence of acute hemorrhagic lesion on this scan.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The mastoid air cells are clear. Fluid collections on bilateral maxillary, ethmoid and frontal sinuses.Brain MRA3D time of flight MOTSA MRA brain images with maximum intensity projections of the anterior/posterior intracranial circulation demonstrate normal ICAs, MCAs and ACAs. Vertebrobasilar system appears to be normal. Bilateral P-comm arteries are patent and a common artery is patent.There is no evidence of intracranial arterial stenosis. There is no evidence of intracranial arterial aneurysm.Brain MRV3D Phase contrast MRV with maximum intensity projections of the superficial/deep intracranial venous drainage demonstrate normal flow enhancement in the superior sagittal sinus, transverse sinuses, sigmoid sinuses, internal cerebral veins, vein of Galen and straight sinus.
1. No evidence of acute ischemic or hemorrhagic leison.2. Normal brain MRA and MRV