19 years Female (DOB:7/28/1997)Reason: AMS, ?hx of lupus cerebritis History: see abovePROVIDER/ATTENDING NAME: LEAH C ANDERSON The CSF spaces are appropriate for the patient's stated age with no midline shift. There is a mild degree of periventricular and subcortical punctate hyperintense white matter lesions present identified on the FLAIR and T2 images. These are stable since the prior exam.There is redemonstration of an ill-defined focus of enhancement in the left basal ganglia which is stable compared to prior exam.There is a tuft of enhancing serpiginous structures posteriorly with signal loss on susceptibility imaging located in the left frontal lobe adjacent to the left middle frontal gyrus which is suspected to represent a developmental venous anomalyNormal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact.
1.Periventricular and subcortical white matter signal changes of a mild degree are present which are nonspecific. They could be vascular related, related to demyelination, trauma, vasculitis, sarcoid. They are nonspecific. These are stable since the prior exam.2.There is an ill-defined lesion present in the left basal ganglia. One possibility is that this may represent capillary telangiectasia.3.There is an ill-defined lesion present in the left basal ganglia. One possibility is that this may represent capillary telangiectasia.