Paralysis agitans [332.0], Reason for Study: ^Reason: concern for stroke as cause for gait disorder History: hx Parkinsons, now worsening gait, left hemiparesis No evidence of acute ischemic or hemorrhagic lesion.Small age indeterminate likely chronic lacunar infarcts on the bilateral caudate nucleus and bilateral basal ganglia are seen.There are scattered multifocal patch high signal intensity lesions on bilateral periventricular white matter and deep white matter on FLAIR imaging indicating nonspecific small vessel ischemic disease.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, or restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
No evidence of acute ischemic or hemorrhagic lesion.Minimal nonspecific small vessel ischemic disease and several chronic lacunar infarctions.