Dysphagia for 2 years. Sjogren syndrome and prior optic neuritis. The images are generally degraded by patient motion to a considerable degree. There is no evidence of intracranial hemorrhage, mass, or acute infarct. There is a punctate focus of encephalomalacia in the left thalamus. There is also mild periventricular white matter T2 hyperintensity. The brainstem appears to be unremarkable. There is no abnormal intracranial enhancement. There is mild diffuse cerebral volume loss. There is no midline shift or herniation. The major cerebral flow voids are intact. The skull, paranasal sinuses, and scalp soft tissues are grossly unremarkable. There is a left lens implant. There is a miliary nodule appearance of the bilateral parotid glands.
1. Chronic lacunar infarct in the left thalamus and mild probable chronic small vessel ischemia in the bilateral cerebral hemispheres, but no evidence of intracranial hemorrhage, mass, or acute infarct. The brainstem appears to be unremarkable, although the exam is limited by patient motion.2. Findings related to parotid Sjogren.