Memory difficulty No intracranial mass or mass-effect. No restricted diffusion to suggest acute ischemia. No intracranial hemorrhage. There is mild global parenchymal volume loss which appears slightly prominent for age. No hydrocephalus.Several foci of T2/FLAIR hyperintensity are seen in the bilateral subcortical and periventricular white matter, including a relatively more prominent focus in the left frontal corona radiata and nonspecific. Brain parenchyma is otherwise unremarkable. No abnormal parenchymal or meningeal enhancement. Major flow-voids are preserved.Sella and orbits are grossly within normal limits. Paranasal sinuses and mastoid cells are clear. There is expansile, abnormal T1 hypointensity and enhancement involving the left skull base including the jugular tubercle extending into the upper left occipital condyle. There is a subcentimeter left parotid nodule, which is nonspecific, possibly small lymph node. Bone marrow signal and extracranial soft tissues are within normal limits.
1. No intracranial mass or mass-effect. 2. Mild global parenchymal volume loss which appears prominent for age.3. Several foci of T2/FLAIR hyperintensity are seen in the bilateral subcortical and periventricular white matter, including a relatively more prominent focus in the left frontal corona radiata. These are nonspecific but compatible with mild chronic small vessel ischemic changes, sequela of prior inflammation/infection, or less likely demyelination.4. Expansile, abnormal T1 hypointensity and enhancement involving the left skull base including the jugular tubercle extending into the upper left occipital condyle. Finding may represent a benign osseous lesions such as fibrous dysplasia, but recommend CT for further assessment to better exclude other processes including metastasis.