 Foci of T2 hyperintensity are present within subcortical white matter involving primarily the frontal lobes, also involving gray matter to some degree. There are also a few foci of T2 hyperintensity within the white matter along radial glial lines. Given differences in technique, these appear unchanged. Lateral ventricular subependymal nodules are again noted (right greater than left). One on the right demonstrates susceptibility from presumed calcification. These findings are unchanged.A 4 mm enhancing nodule is noted near the rightward foramen of Monroe.Susceptibility hypointensity is also noted within the left cerebellar hemisphere, with associated folia prominence of focal volume loss. The susceptibility appears to have increased (again given differences in technique), although the volume loss appears unchanged.The ventricles and sulci are normal in size. The cerebellar tonsils are in normal position. There is no mass effect or midline shift. The pituitary gland is normal in size. There is no evidence for acute cerebral, brainstem or cerebellar infarction. No diffusion-weighted abnormalities are identified. There are no extraaxial fluid collections or subdural hematomas. Flow voids are present within the major vessels indicating patency. The paranasal sinuses and mastoid air cells are clear. Incidental note is a prominent developmental venous anomaly arising within the left frontal white matter draining to the inferomedial frontal lobe.
1.Constellation of MRI findings of tuberous sclerosis including cortical tubers, radial glial lines, and subependymal nodules, including a 4 mm enhancing nodule near the rightward foramen of Monroe.2.Susceptibility hypointensity is also noted within the left cerebellar hemisphere, with associated folia prominence of focal volume loss. The susceptibility appears to have increased (given differences in technique), although the volume loss appears unchanged. This is of uncertain etiology or clinical significance.