 Several sequences are motion greater. Given this caveat:Brain MRI:T2 hyperintensity is present throughout the white matter, confluent in several locations, without associated mass effect, restricted diffusion, susceptibility abnormality, or enhancement. Gyriform susceptibility abnormality is noted within the right parietal lobe. The ventricles and sulci are normal in size. The cerebellar tonsils are in normal position. There are no masses, mass effect or midline shift. The pituitary gland is normal in size. There is no evidence for intracranial hemorrhage or acute cerebral, brainstem or cerebellar infarction. No diffusion-weighted abnormalities are identified. There are no extraaxial fluid collections or subdural hematomas. Flow voids are present within the major vessels indicating patency. Mild mucosal thickening is present within the left maxillary sinus, otherwise the paranasal sinuses are clear. Mild inflammatory changes are noted in bilateral mastoid air cells with a few containing fluid. There is no abnormal enhancement within the brain. Cervical spine MRI:The marrow signal is benign. The cervical and upper thoracic cord is normal in signal without abnormal enhancement. The cervicomedullary junction is normal. The cerebellar tonsils are in normal position. The visualized paraspinal contents are unremarkable.There has been multilevel laminectomy extending from C2 through C6.C2/3: There is moderate left facet hypertrophy with facet fusion as well as mild right facet hypertrophy. Additionally, there is mild posterior aspect disc complex eccentric to the left. There is no central or right neural foraminal stenosis, however there is moderate left neural foraminal stenosis.C3/4: Trace anterolisthesis C3 on C4, posterior osteophyte disc complex, bilateral uncinate hypertrophy, ligamentum flavum thickening, very severe left facet hypertrophy, and severe right facet hypertrophy. There is moderate central and severe bilateral neural foraminal stenosis.C4/5: Posterior osteophyte disc complex, bilateral uncinate hypertrophy, ligamentum flavum thickening, and very severe bilateral facet hypertrophy. There is associated focal cord atrophy with intrinsic T2 hyperintensity. There is mild to moderate central and severe bilateral neural foraminal stenosisC5/6: Minimal posterior osteophyte disc complex, bilateral uncinate hypertrophy, ligamentum flavum thickening, and severe bilateral facet hypertrophy with probable bilateral facet fusion. There is mild to moderate bilateral neural foraminal stenosis.C6/7: Minimal posterior osteophyte disc complex, bilateral facet hypertrophy, partial vertebral body fusion, and mild bilateral facet hypertrophy with facet fusion. There is moderate left neural foraminal stenosis.C7/T1: Trace anterolisthesis C7 on T1, minimal posterior osteophyte disc complex, and moderate bilateral facet hypertrophy. There is moderate bilateral neural foraminal stenosis.Multilevel facet hypertrophy is present in the visualized upper thoracic spine, encroaching the neural foramina, worse at T1/2 and T2/3.
1.Advanced chronic small vessel ischemic disease.2.Gyriform susceptibility abnormality within the right parietal lobe most likely represents hemosiderin sequela of remote small hemorrhagic cortical infarction.3.There has been multilevel laminectomy extending from C2 through C6.4.C2/3: Moderate left neural foraminal stenosis.5.C3/4: Moderate central and severe bilateral neural foraminal stenosis.6.C4/5: There is spondylitic-associated focal cord atrophy with intrinsic T2 hyperintensity as well as mild to moderate central and severe bilateral neural foraminal stenosis7.C5/6: Mild to moderate bilateral neural foraminal stenosis.8.C6/7: Moderate left neural foraminal stenosis.9.C7/T1: Moderate bilateral neural foraminal stenosis.10.Multilevel facet hypertrophy is present in the visualized upper thoracic spine, encroaching the neural foramina, worse at T1/2 and T2/3.