Mild cognitive impairment, so stated [G31.84], Reason for Study: ^Reason: Eval for MS History: fatigue, nonspecific symptoms Scattered somewhat patchy FLAIR/T2 high signal intensity lesions at bilateral subcortical and deep white matter lesions involving bilateral hemispheres and lower pons. Comparing to prior scan, these lesions are more conspicuous on today's scan, however, considering the difference in imaging technique (today's one has higher resolution and higher signal to noise ratio), there is no significant interval change in terms of number, distribution and size of above described lesions.There is no evidence of abnormal enhancement.Constellation of above findings are nonspecific white matter disease. Differential diagnosis including nonspecific small vessel ischemic disease and other demyelinating disease such as multiple sclerosis.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia, or abnormal contrast enhancement. Minimal downward displacement of cerebellar tonsils through foramen magnum (about 4mm).Normal flow voids are identified in the major intracranial vessels. The mastoid air cells are clear.There is interval development of sphenoid sinus mucosal thickening indicating sinusitis.
1. Multifocal scattered somewhat patch high FLAIR high signal intensity lesions at subcortical white matter and the tip white matter. These are nonspecific findings and differential diagnosis including nonspecific small vessel ischemic disease and demyelinating disease such as multiple sclerosis. Unchanged since prior scan.2. No evidence of acute ischemic or hemorrhagic lesion on the scan.