Female, 59 years old, with pan right sided hemisensory loss. Symmetric T2 hyperintensity is evident within the bilateral globus pallidus. The central portion of this T2 hyperintensity suppresses on FLAIR images, but the rim remains hyperintense. Symmetric bilateral susceptibility effect is evident within the globus pallidus, but within normal limits for age.No discrete thalamic lesions are seen. Scattered small foci of T2 hyperintensity are evident within the bilateral cerebral white matter. These are nonspecific both in their morphology and distribution. No pathologic parenchymal or extra-axial enhancement is seen.No evidence of intracranial hemorrhage or any abnormal extra axial fluid collection is seen. The ventricular system is normal in size and shape. Incidental note is made of a partially empty sella.
1.Bilateral T2 hyperintensity within the globus pallidus is seen with imaging findings suggesting chronic cavitation and minimal surrounding gliosis. The differential would include chronic injury from anoxia, toxic exposure (e.g. carbon dioxide, cyanide), and certain metabolic derangements. This may be an incidental finding unrelated to the patient's presenting symptoms.2.No evidence of any thalamic lesions or other specific abnormalities which would account for the patient's hemisensory deficits.3.No acute intracranial abnormalities are seen. Scattered nonspecific small white matter lesions are demonstrated which most commonly represent chronic microvascular ischemic disease though other etiologies including demyelination and sequelae of inflammation are also possible.