Foot pain There is increased signal intensity and possible thinning of the peroneus longus tendon as it courses along the lateral margin of the calcaneus anterior to the retrotrochlear eminence. This suggests tendinosis and attritional tearing. There is also mild edema in the adjacent soft tissues indicating mild peritendinitis. The signal intensity of the tendon normalizes as it enters the cuboid tunnel although a mild amount of fluid surrounds the tendon within the tunnel suggesting tenosynovitis. Proximally, the peroneus longus tendon appears intact. The peroneus brevis tendon is unremarkable. The remaining tendons of the ankle are normal.The anterior talofibular ligament appears thinned but otherwise intact. The posterior talofibular and calcaneofibular ligaments are intact. The distal tibiofibular syndesmotic complex is intact. There is loss of the normal fatty striation of the deep fibers of the deltoid suggesting prior sprain but there is no evidence of tear on this exam.There are small patchy foci of abnormal signal intensity within the cuboid which may reflect reactive edema secondary to the peroneal tendinopathy described above. There is no linear signal abnormality to confirm stress fracture however. There is minimally increased signal intensity within the talus on fluid sensitive sequences which is nonspecific and not necessarily of any clinical significance. An os trigonum is noted posterior to the talus. There is no osteochondral defect. A small amount of fluid signal intensity is noted beneath the second and third tarsometatarsal articulations as well as medial to the subtalar joint which may reflect small synovial cysts or ganglia.
Increased signal intensity and probable thinning of the peroneus longus tendon suggesting tendinosis and attritional tearing. There is no evidence of a cuboid stress fracture; additional findings are described above.