Female, 64 years old, with multiple sclerosis. Evaluate for progression. Brain:Multiple scattered T2 hyperintense lesions are seen within the periventricular and subcortical white matter. Some of these are oriented radially with respect to the ventricles. The brainstem and cerebellum are relatively preserved. On comparison with the prior examination, no significant interval change in size or number of lesions is seen.No enhancing lesions are demonstrated. No evidence of significant local generalized edema is seen. No findings to suggest intracranial hemorrhage or any other abnormal extra axial fluid collection is detected. The ventricles are normal in size and morphology.C-spine:A T2 hyperintense lesion is evident within the dorsal cord at C2 with no corresponding pathologic enhancement demonstrated. Within the limitations of motion degradation, no definite additional spinal cord lesions are seen.There is a reversal of the cervical lordosis centered at C5-6. A trace anterolisthesis of C4 relative to C5 is noted. A trace retrolisthesis of C6 relative to C7 is noted. Vertebral body height and morphology are unremarkable.The visualized spinal cord is unremarkable except as discussed above. No pathologic cord or leptomeningeal enhancement is seen. No epidural abnormalities are suspected.Disc degeneration is seen most notably at C6-7 and to a lesser degree at C5-6. Additional level specific findings are as follows:C2-3: No spinal canal stenosis or neuroforaminal narrowing. C3-4: No spinal canal stenosis or neuroforaminal narrowing. C4-5: Right facet arthropathy. No spinal canal stenosis. Moderate right foraminal narrowing. C5-6: Mild posterior disc-osteophyte complex formation. No significant spinal canal stenosis. Mild right foraminal narrowing. C6-7: Posterior disc-osteophyte complex formation asymmetric to the left. No significant spinal canal stenosis. Moderate left and mild right foraminal narrowing. C7-T1: Left facet arthropathy. No significant spinal canal stenosis. Mild left foraminal narrowing.
1.Multiple lesions are redemonstrated in the brain compatible with demyelinating disease. There has been no significant interval change in lesion size or number. No enhancing lesions are identified.2.A lesion is evident within the ventral spinal cord at C2 with no associated enhancement. There is no prior examination for comparison.3.Level disc degeneration is seen. No high-grade spinal canal stenosis is noted. However, scattered mild to moderate foraminal narrowing is demonstrated.