36-year-old female with new onset paresthesias after exercising, history of known cerebral cavernous malformation. Redemonstrated are changes from left subtemporal approach for pontine cavernous reformation resection. Redemonstrated is susceptibility hypointensity with associated T2 hypointensity within the left pons. A central linear focus of T1 hyperintensity, yet with demonstrable enhancement which extends to the adjacent dural thickening may represent postprocedural changes versus a small developmental venous anomaly. Persistent residual T2 hyperintensity is again noted within the middle cerebellar peduncles bilaterally. Postprocedural dural enhancement is again noted. These findings are stable.There is a small focus of ill-defined T2 hyperintensity with associated enhancement just anterior to the left precentral gyrus, in a subcortical location, most consistent with either a tiny developmental venous anomaly or a capillary telangiectasia, unchanged in appearance.Ventricular sizes are normal. Expected vascular flow voids are demonstrated. There is new minimal mucosal thickening within the right sphenoid sinus. The remaining paranasal sinuses and right mastoid air cells are clear. Fat packing is now noted within the expected region of left mastoid air cells.
SRedemonstrated is susceptibility hypointensity with associated T2 hypointensity within the left pons. A central linear focus of T1 hyperintensity, yet with demonstrable enhancement which extends to the adjacent dural thickening may represent postprocedural changes versus a small developmental venous anomaly. Persistent residual T2 hyperintensity is again noted within the middle cerebellar peduncles bilaterally. Postprocedural dural enhancement is again noted. These findings are stable.