Gliomatosis cerebri. The peripheral enhancement associated with a small lesion in the left thalamus has resolved and there is minimal residual T2 hyperintensity in this location. Otherwise, there are stable post-treatment findings in the brain and unchanged diffuse T2 hyperintensity involving the anterior cerebral hemispheres bilaterally with multiple cystic cavities. There is no evidence of associated abnormal intraparenchymal enhancement. There is a subcentimeter T1 hyperintense lesion in the left posterior body of the corpus callosum, which is compatible with a cavernous malformation. A punctate focus of susceptibility effect in the right posterior corpus callosum may also represent a cavernous malformation. There is no evidence of acute intracranial hemorrhage or acute infarct. The ventricles and basal cisterns are unchanged in size and configuration, with diffuse cerebral volume loss. There is no midline shift or herniation. The skull appears unchanged. There is scattered paranasal sinus mucosal thickening and secretions.
1. The peripheral enhancement associated with a small lesion in the left thalamus has resolved and there is minimal residual T2 hyperintensity in this location, suggesting that this may have been a prior subacute lacunar infarct or evolving radiation necrosis. Stable post-treatment findings without evidence of tumor progression.2. Suggestion of acute sinusitis. 