43 years Female (DOB:12/16/1973)Reason: evaluate for demyelinating disease History: abnormal non-contrast c-spine MRI; asymmetric hyper-reflexia PROVIDER/ATTENDING NAME: NAOUM ISSA SERVICES ANCILLARY Cervical spine:The cervical vertebral bodies are appropriate in overall alignment and height. There is redemonstration of a globular appearing T2 signal hyperintensity along the posterior midline of the spinal cord extending from approximately C2-3 disc space down to the C6 vertebral level. No abnormal enhancing lesions are appreciated in the cervical spine.At C2-3 there is no significant compromise to the spinal canal or neural foramina. There are small uncovertebral osteophytes at this level.At C3-4 there is no significant compromise to the spinal canal or neural foramina.At C4-5 there is no significant compromise to the spinal canal or neural foramina. There are small uncovertebral osteophytes at this level.At C5-6 there is no significant compromise to the spinal canal or neural foramina. There are small uncovertebral osteophytes present associated disc material layering in some mild encroachment of the exiting nerve roots.At C6-7 there is no significant compromise to the spinal canal or neural foramina.At C7-T1 there is no significant compromise to the spinal canal or neural foramina.The vertebral artery flow voids appear to be intact.MRI thoracic spine:The thoracic vertebral bodies are appropriate in the overall alignment and height. The thoracic spinal cord has normal signal characteristics and overall morphology. There is no compromise of thoracic spinal canal or exiting nerve roots. There are small disc bulges present at T6-7 T7-8 without significant compromise spinal canal or neural foramina.No abnormal enhancing lesions are appreciated in the thoracic spine.
1.There is a T2 hyperintense lesion along the posterior midline of the cervical spinal cord. It has not changed since the prior exam. It does not extend into the thoracic spinal cord. There is no abnormal enhancement to suggest breakdown barrier at this time. The differential diagnosis has not changed. Differential considerations could include demyelination subacute combined degeneration possible cord ischemia post transverse myelitis and vacuolar myelopathy.