Female, 91 years old, with a lesion in the corpus callosum, follow-up exam. Motion artifact degrades image quality on multiple sequences. T2 hyperintensity without mass effect is redemonstrated involving the inferior portion of the splenium, and to lesser degree the undersurface of the posterior body of the corpus callosum and the posterior fornices. No associated pathologic enhancement is seen. In comparison with multiple prior examinations, there has been no significant interval change in these findings. No evidence for restricted diffusion is seen. Numerous additional small foci of T2 hyperintensity are seen within the cerebral white matter including the periventricular tissues. These findings are also unchanged. No mass-effect or parenchymal edema is detected. No evidence of intracranial hemorrhage or any pathologic extra-axial fluid is seen. No enhancing lesions are detected. The ventricles remain within normal limits in size and shape for patient age.
Examination slightly limited due to patient motion. No significant interval change is seen in the appearance of signal abnormality involving the inferior aspect of the splenium, the undersurface of the posterior body of the corpus callosum and the posterior fornices. The differential remains wide and includes the chronic sequelae of a vascular, metabolic or toxic process (including medication-related toxicity), as well as demyelinating, postinfectious or postinflammatory etiologies.