 Compared to 9/2/2015, there is unchanged appearance of the chronic infarct involving the right parietal, temporal, and occipital lobes with T1 hyperintensity indicative of laminar necrosis and enhancement demonstrated on prior study. There is subtle diffusion hyperintensity similar to prior. No findings to suggest a new infarct. There are unchanged scattered subcortical and periventricular white matter punctate T2/FLAIR hyperintensities, which are nonspecific but most likely representing chronic microvascular ischemic changes. No evidence of intracranial mass effect, midline shift, or herniation. No hydrocephalus.There is a redemonstration of left sellar and cavernous sinus mass that measures 11 mm in cc diameter, 13 mm in the transverse diameter, and 12 mm in the AP diameter and better assessed on prior post gadolinium studies. The mass is isointense in the T1 images and has intermediate intensity in T2 weighted images. There is no compression of the optic chiasm. The pituitary stalk is slightly deviated to the right.There is mild opacification of the bilateral ethmoid air cells. Remainder of the paranasal sinuses are clear. Bilateral lens prostheses is seen.
1. No evidence of acute infarct or intracranial mass effect.2. Unchanged appearance of the chronic infarct involving the right temporal, parietal, and occipital lobes. Diffusion restriction was demonstrated within this region on prior study from 12/16/2014.3. Left cavernous sinus/sellar mass (presumable meningioma) is again seen and better assessed on prior postcontrast studies.