Other malaise and fatigue [780.79], Reason for Study: ^Reason: please r/o new MS lesions or any other pathology History: new weakness Motion artifacts degraded exam quality.Limited exam due to patient's movement.Brain MRI:Only post contrast T1 weighted scans were obtained.There is no evidence of abnormal enhancement. The ventricles, sulci and cisterns appear to be symmetric and unremarkable. The midline structures and cranial-cervical junction are normal. If necessary, this exam should be repeated for thorough brain evaluation.C-Spine MRIOnly Post contrast T1 sagittal and axial scans were available.Normal cervical lordosis. Normal spinal cord signal intensity. No abnormal enhancement.Degenerative changes include mild neuroforaminal stenosis on bilateral C34 level, prominent posterior longitudinal ligament which abuts thecal sac at the level of C45 and C56.T-Spine MRI: Normal thoracic kyphotic angulation. Normal spinal cord signal intensity. No evidence of abnormal enhancement. Multifocal degenerative changes especially on the lower thoracic vertebral bodies without evidence of neuroforaminal stenosis or spinal canal stenosis.L-Spine MRI:There is grade 1 anterolisthesis of L5 on S1.Normal lumbar lordosis. Vertebral body heights were preserved.Spinal cord signal intensity is normal.The conus medullaris terminates at the L1 level.Degenerative changes are; T12-L1: no neuroforaminal narrowing or spinal stenosis. L1-L2: no neuroforaminal narrowing or spinal stenosis. L2-L3: diffuse bulging of disc with minimal to mild spinal canal stenosis. L3-L4: diffuse bulging of disc with moderate spinal canal stenosis. L4-L5: diffuse bulging of disc with minimal to moderate spinal canal stenosis. L5-S1: grade 1 anterolisthesis of L5 on S1. No neuroforaminal narrowing or spinal stenosis.
1. Provided very limited exam due to patient's motion and poor cooperation, no evidence of abnormal enhancement, nor evidence of spinal cord compression.2. Multi-level degenerative changes were seen on both cervical spine and lumbar spine level as described above without evidence of abnormal spinal cord signal intensity.