Unspecified cerebral artery occlusion with cerebral infarction. There is ventriculomegaly present out of proportion to sulcal width.There are patchy T2 and FLAIR signal hyperintensities along the pons and periventricular white matter. There is no focus of diffusion restriction present. Right there are small T2 hyperintense lesions in the cerebellum which are low signal on FLAIR. These probably represent enlarged periventricular spaces.No abnormal mass lesions are appreciated intracranially. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus. The left vertebral artery flow-void is very small.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact. The eyeball lenses are thin.Examination is somewhat degraded by artifact related to patient motion. Many fast sequences were used in an attempt to reduce artifact at the expense of lower contrast resolution.
1.There is no evidence for acute ischemic cerebral infarction.2.Periventricular and subcortical white matter changes of a mild degree as well as mild signal change in the pons are nonspecific. At this age they are most likely vascular related. 3.Some small cerebellar lesions may be related to prior microinfarcts but are nonspecific.4.There is generalized brain atrophy present.5.Examination is degraded by patient motion which may obscure more subtle abnormalities.