83 years, Male, transient alteration of awareness (unresponsive and aphasic). Image quality is moderately degraded due to motion. No restricted diffusion to suggest acute ischemia. No acute intracranial hemorrhage. Multiple foci of susceptibility effect are seen which appear to be predominantly in a peripheral distribution although assessment is limited with severely degraded susceptibility weighted sequence. No intracranial mass or mass-effect. The ventricles and other CSF spaces are proportionately enlarged, consistent with advanced cerebral volume loss.There are small areas of FLAIR hyperintensity with suggestion of volume loss in the posterior right frontal and right parietal lobes suggesting remote cortical infarcts. Confluent and patchy areas of T2/FLAIR hyperintensity are seen in the bilateral subcortical and periventricular white matter, which are nonspecific, but compatible with chronic small vessel ischemic changes. Major flow-voids are preserved on T2 weighted imaging.Sella and orbits are grossly within normal limits. Paranasal sinuses and mastoid cells are clear. Bone marrow signal and extracranial soft tissues are within normal limits.
Please note examination is moderately motion degraded which limits assessment.1. No evidence of acute infarct.2. Multiple predominantly peripheral foci of chronic microhemorrhage are suggested on the significantly degraded susceptibility weighted sequence which may be related to cerebral amyloid angiopathy.3. Remote cortical infarcts are suggested in the posterior right frontal and parietal lobes. 4. Moderate to severe chronic small vessel ischemic changes and cerebral volume loss. 