Dystonia in low back and left leg. L4-5 decompression via left-sided approach. The images are degraded by patient motion. There are interval postoperative findings related to L4-5 decompression via left-sided approach. There is apparent severe narrowing of the spinal canal at L4-5 due to what may be a combination of ligamentum flavum thickening, residual disc material, scar or granulation tissue, and perhaps even surgical fluid. There is also considerable effacement of the bilateral L4-5 neural foramina. There is mild retrolisthesis of L3 on L4, as well a degenerative spondylosis with mild narrowing of the spinal canal and left neural foramen. There is also degenerative spondylosis at the L5-S1 level, with moderate narrowing of the spinal canal and bilateral neural foramina. There is superimposed effacement of the left lateral recess at this level associated with a hemilaminectomy defect due to what may represent scar formation. There is degenerative bone marrow signal alteration in the L3 though S1 vertebrae with associated endplate irregularities. There is significant compromise to spinal canal or neural foramina at T12 through L3. The imaged portions of the spinal cord are unremarkable.
1. Interval postoperative findings related to L4-5 decompression via left-sided approach, with apparent severe narrowing of the spinal canal at L4-5 due to what may be a combination of ligamentum flavum thickening, residual disc material, scar or granulation tissue, and perhaps even surgical fluid. However, precise assessment is limit by patient motion artifacts.2. Mild retrolisthesis of L3 on L4, as well a degenerative spondylosis with mild narrowing of the spinal canal and left neural foramen. 3. Degenerative spondylosis at the L5-S1 level, with moderate narrowing of the spinal canal and bilateral neural foramina, as well as superimposed effacement of the left lateral recess at this level associated with a hemilaminectomy defect due to what may represent scar formation.