51 years Female (DOB:10/16/1964)Reason: STROKE W/UP History: L NUMBNESSPROVIDER/ATTENDING NAME: RICHARD KRAIG RICHARD KRAIG MRI of the brainNo diffusion weighted abnormalities are appreciated.The CSF spaces are appropriate for the patient's stated age with no midline shift. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses demonstrate mild mucosal thickening. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact.Exam is mildly degraded due to patient motion.MRA brain:Antegrade flow is present in the distal internal carotid arteries, the distal vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries.There is no evidence for intracranial aneurysm or cerebrovascular occlusion.The anterior communicating artery is identified. The posterior communicating arteries are intact. The right posterior communicating artery is slightly larger than the left posterior communicating artery. The right posterior communicating artery is small. The anterior communicating artery also appears to be small The vertebral arteries are similar in size.There is extracranial origin of the posterior inferior cerebellar artery bilaterally.MRA neck:There is opacification of the aortic arch, great vessels from the aortic arch and carotid arteries and vertebral arteries. There is no stenosis identified of the great vessels from the aortic arch. On the basis of NASCET criteria there is no significant stenosis at the carotid bifurcations. There is no significant stenosis along the course of the vertebral arteries. The left vertebral artery originates from the aortic arch.
1.There is no evidence for acute cerebral ischemic infarction.2.There is no evidence for intracranial or extracranial cerebrovascular occlusive disease.3.No intracranial mass lesion is appreciated.