History of SLE, hyperreflexia, evaluate for brain lesions and myelopathy. Brain:There are scattered foci of susceptibility in the bilateral cerebral hemispheres in a cortical and subcortical distribution, which are nonspecific. There is no evidence of mass, mass effect, or midline shift. There is mild diffuse global parenchymal volume loss. The ventricles, sulci, and basal cisterns are otherwise normal in size. The midline structures are normal. There is no evidence for intracranial hemorrhage or extra-axial collection. No diffusion-weighted abnormalities are identified. Flow voids are present within the major vessels indicating patency. The paranasal sinuses and mastoid air cells are grossly aerated. Cervical Spine:The sagittal images extend inferiorly to visualize the superior endplate of the T4 vertebral body.The cervical spine alignment is anatomic. The vertebral body and intervertebral disc space heights are preserved. The bone marrow demonstrates low T1 signal suggestive of red marrow reconversion. There is no significant spinal canal stenosis or neural foraminal narrowing at any level. The cord is normal in signal and morphology. The prevertebral soft tissues are unremarkable.
1. No evidence of acute infarct, intracranial mass, or mass effect.2. Multiple scattered cortical and subcortical foci of susceptibility compatible with chronic microhemorrhages. These are nonspecific but can be seen with thrombotic microangiopathy for example. 3. No evidence of cervical spinal canal stenosis or cord signal abnormality.4. Diffuse low T1 signal in the visualized bone marrow which is nonspecific but can be seen with marrow reconversion related to chronic anemia for example.