59 years, Female, grade 2 oligodendroglioma. Status post RT and now receiving PCV. Again seen is expansile T2 hyperintensity involving the left temporal lobe including the left middle and inferior temporal gyri as well as the fusiform gyrus consistent with known tumor. There is focal area of enhancement within this region which remains unchanged since 11/25/2015. Extent of FLAIR hyperintensity is also not significantly changed since 11/25/2015 but clearly decreased when compared to remote study from 5/9/2015, presumably related to decrease in the vasogenic edema component of the signal abnormality. There is mild serpiginous T1 hyperintensity and extensive susceptibility effect related to calcifications associated with the tumor. There is unchanged minimal local mass effect. No midline shift or herniation. No hydrocephalus. No evidence of acute ischemia or hemorrhage. Evidence of prior biopsy is noted via left temporal burr hole.There is unchanged subcentimeter focus of T2 hyperintensity involving the right cerebellum which is nonspecific and possibly related to prior infarct, although can be further assessed with future follow-up studies.There is partial opacification of the left mastoid air cells, similar to prior. There is trace mucosal thickening involving the paranasal sinuses. Remainder of the calvarium and extra cranial soft tissues are unremarkable.
1. No evidence of progression of left temporal oligodendroglioma compared to 11/25/2015. Compared to earliest available comparison study from 5/9/2015, there is decrease in extent of FLAIR signal abnormality in the left temporal lobe, presumably related to decrease in vasogenic edema.2. Subcentimeter focus of T2 hyperintensity involving the right cerebellum remains unchanged since 5/9/2015. This lesion is nonspecific and may represent prior infarct, although can be further assessed on future follow-up studies.