 There is a focus of multisequence hypointensity involving the lateral aspect of the right medulla measuring 6 mm AP x 5 mm transverse x 9 mm craniocaudal dimension (as measured via T2 technique). There is an associated parenchymal defect along its lateral most aspect, and there are postprocedural changes involving the occipital calvarium, consistent with the provided history of previous surgery. There is no perilesional edema or apparent associated developmental venous anomaly. There are no other foci of susceptibility abnormality.Incidental note is made of a development venous anomaly involving primarily the superior cerebellar vermis, without associated susceptibility abnormality.There are scattered foci of T2 hyperintensity primarily within the subcortical white matter without mass effect, restricted diffusion, susceptibility abnormality, or enhancement.The ventricles and sulci are normal in size. The cerebellar tonsils are in normal position. There is no mass effect or midline shift. There is no evidence for intracranial hemorrhage or acute cerebral, brainstem or cerebellar infarction. There are no extraaxial fluid collections or subdural hematomas. Flow voids are present within the major vessels indicating patency. Mucosal thickening is present within the inferior right maxillary sinus. Otherwise the remaining paranasal sinuses and mastoid air cells are clear. There is no abnormal enhancement within the brain.
1.There is a focus of multisequence hypointensity involving the lateral aspect of the right medulla measuring 6 mm AP x 5 mm transverse x 9 mm craniocaudal dimension (as measured via T2 technique). There is an associated parenchymal defect along its lateral most aspect, and there are postprocedural changes involving the occipital calvarium, consistent with the provided history of previous surgery of a cavernous malformation. There is no perilesional edema or apparent associated developmental venous anomaly. There are no other foci of susceptibility abnormality.2.There are scattered foci of T2 hyperintensity primarily within the subcortical white matter without mass effect, restricted diffusion, susceptibility abnormality, or enhancement. These are nonspecific in appearance, and statistically most likely secondary to chronic small vessel disease.