 There is no evidence of acute intracranial hemorrhage, mass, or acute infarct. There are multiple unchanged patchy foci of T2 hyperintensity within the supratentorial and infratentorial white matter that are most compatible with chronic small vessel ischemic disease. There is unchanged mild global volume loss. There are multiple unchanged small foci of susceptibility effect throughout the supratentorial white matter, in a mainly subcortical distribution. The ventricles and basal cisterns are normal in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. The skull, paranasal sinuses, and scalp soft tissues are unremarkable.
1.No evidence of acute infarct.2.Unchanged moderate small vessel ischemic disease.3.Multiple unchanged supratentorial chronic microhemorrhages in a mainly subcortical distribution, suggestive of amyloid angiopathy.