81-year-old presenting with stroke symptoms. Evaluate for ischemia. No restricted diffusion to indicate an acute infarct. Redemonstration of extensive near confluent increased T2 signal in the subcortical and periventricular white matter. This appears slightly progressed compared to prior most notably in the left anterior temporal and left parietal subcortical white matter. Chronic infarct involving the left inferior frontal lobe again seen. Numerous foci of susceptibility artifact within the subcortical white matter throughout the supratentorial and to a lesser extent infratentorial brain parenchyma compatible with cerebral amyloid angiopathy.There is no evidence of acute intracranial hemorrhage, mass or edema. The ventricles and basal cisterns are normal in size and configuration for the patient's age. The expected intracranial vascular flow voids are present.The paranasal sinuses are grossly clear. Mild opacification of the left mastoid air cells.
1. No evidence of acute infarct.2. Extensive white matter signal abnormality which is mildly progressed since 2/17/2014, specifically the involvement of the left anterior temporal and left parietal subcortical white matter. Finding is nonspecific but favored to represent chronic ischemic injury. No associated diffusion abnormality.3. Numerous foci of susceptibility throughout the brain which are predominantly in a peripheral lobar distribution and likely representing amyloid angiopathy.I personally reviewed the Images and/or procedure with the Resident/Fellow and agree with this report.