History of epidural abscess, back pain No evidence of epidural abscess is appreciated. Previously demonstrated peripheral enhancing soft tissue in the left ventral epidural space at L1 level extending into the left L1-L2 neural foramen is significantly decreased in size and compatible with evolution of postsurgical change and/or resorption of minimal residual disc material.Unchanged compression deformity of L1 with approximately 70% loss of height. Unchanged associated bone marrow edema. There are mild compression fractures of L2 and L3 vertebral bodies also unchanged since 11/11/2014. Additional scattered areas of T1 hypointensity throughout the spine are nonspecific. Areas of T1 hyperintensity are compatible with focal fat or hemangiomas.Degenerative changes throughout the lumbar spine also again seen without evidence of high-grade spinal canal or neural foraminal stenosis. Again seen is mild spinal canal and mild-to-moderate left neural foramina stenosis at L4-L5 related to disk bulge, facet arthropathy, and ligamentum flavum buckling as well as mild left L3-4 neural foraminal narrowing. Multilevel facet arthropathy. No paraspinous fluid collections are demonstrated.
1. No evidence of epidural abscess or active infection. Previously demonstrated peripheral enhancing soft tissue in the left ventral epidural space at L1 level extending into the left L1-L2 neural foramen is significantly decreased in size and most compatible with evolution of postsurgical change and/or resorption of minimal residual disc material.2. Multiple compression fractures, worst at L1, are unchanged since 11/11/2014. Again seen is focal kyphosis at the L1-L2 level similar to prior. Please note L1-L3 compression fractures and L1-2 kyphosis is clearly progressed since 8/29/2014. Bone marrow signal remains heterogeneous which is nonspecific. Compression fractures are favored to be benign in etiology.