Headache, lupus cerebritis No evidence of acute ischemic lesion.There is localized high signal intensity on T1 weighted image on the left side of posterior pons (series 801, image 12/32) which shows susceptibility artifact on gradient echo images. There is no evidence of surrounding edema. The lesion is associated with linear enhancement which likely represent venous structure. Therefore, the diagnosis of cavernoma associated with developmental venous anomaly is most likely.On FLAIR images, multifocal scattered subcortical white matter high signal intensities on bilateral hemisphere.There is no evidence of abnormal enhancement. Considering the patient age, these lesions may represent inflammatory origin including lupus and multiple sclerosis.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, or midline shift. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
1. Left posterior pons cavernous malformation with associated developmental venous anomaly.2. Multiple scattered bihemispheric subcortical white matter high signal lesions on FLAIR images indicate inflammatory lesions such as lupus or multiple sclerosis. Follow up study is recommended.