Anoxic brain injury. There is extensive restricted diffusion involving the cortex of the occipital lobes, parietal lobes, posterior temporal lobes, and posterior frontal lobes bilaterally. There is more mild restricted diffusion within the bilateral basal ganglia. The pattern of injury is consistent with a global anoxic event. The cerebellum and brainstem structures are spared. There is no significant associated edema, mass effect, or midline shift.There is encephalomalacia related to a chronic right anterior thalamic lacunar infarct.The ventricles and basal cisterns are normal in size and configuration.The expected intracranial vascular flow voids are present.The paranasal sinuses are clear. The visualized mastoid air cells are clear. The orbits are unremarkable.Small focus of increased T1 and T2 signal in the posterior left parietal bone may represent a small hemangioma.
Acute to subacute ischemic changes involving the majority of the cerebral cortex as well as the basal ganglia. Findings are compatible with a global anoxic injury. No significant cerebral edema, midline shift, or herniation. 