Reason: contusions, concussions, intracranial changes History: headache, personality change There is T2 hyperintensity and surrounding inflammatory changes of the right frontal scalp, which may represent fluid or blood products. There is a 11 mm x 5 mm peripherally enhancing collection (series 1201 image 124) s, which may represent sequela of prior hematoma but superimposed infection cannot be excluded. The underlying bone is intact without intracranial extension.No abnormal diffusion restriction. No intracranial hemorrhage. Mild nonspecific foci of bilateral frontal subcortical white matter T2 hyperintensities, which may be related to prior inflammation, ischemia, trauma, or migraines for example. No extra-axial fluid collections. The ventricles, sulci, and cisterns are normal in size without evidence of hydrocephalus. Gray-white matter differentiation is preserved. The imaged paranasal sinuses and mastoid air cells are clear. The imaged orbits are intact. The osseous structures are unremarkable.
1.Soft tissue edema and inflammatory changes of the right frontal scalp at the location of the scalp hematoma seen on prior study. There is a small peripherally enhancing scalp fluid collection, which may represent sequela of prior hematoma but superimposed infection/abscess cannot be excluded. The underlying bone is intact without intracranial extension.2.No evidence of intracranial hemorrhage, edema, or mass effect. No abnormal parenchymal or meningeal enhancement.3.Mild nonspecific foci of T2 hyperintensity in the bilateral frontal subcortical white matter, etiology of which may be prior inflammation, ischemia, trauma, or migraines, for example. 