 There are a few patchy foci of T2 hyperintensity primarily involving the subcortical white matter without associated mass effect, restricted diffusion, susceptibility abnormality, or enhancement. The ventricles and sulci are normal in size. The cerebellar tonsils are in normal position. There are no masses, mass effect or midline shift. The pituitary gland is normal in size. There is no evidence for intracranial hemorrhage or acute cerebral, brainstem or cerebellar infarction. No diffusion-weighted abnormalities are identified. Myelination is appropriate for age. There are no extraaxial fluid collections or subdural hematomas. Flow voids are present within the major vessels indicating patency. Fluid is noted within a few mastoid air cells (left greater than right). Middle ear cavities are clear. There is no abnormal enhancement within the brain. Images of the internal auditory canals demonstrate the seventh and eighth nerves to be normal in size and symmetric bilaterally without masses or enhancing lesions along the course of these nerves or in the cerebellopontine angle cisterns bilaterally. The inner ear structures are normal in appearance and symmetric bilaterally without congenital inner ear anomalies identified on either side. There are no vascular anomalies.
1.There are a few patchy foci of T2 hyperintensity primarily involving the subcortical white matter without associated mass effect, restricted diffusion, susceptibility abnormality, or enhancement. Per Epic notes, the patient was born term via spontaneous vaginal delivery without complication. Given this constellation of imaging features, in the context of patient history, these findings are nonspecific and the differential diagnosis would include non-acute ischemic sequelae, inflammation, demyelination, dysmyelination, or perhaps a metabolic etiology (such as mitochondrial disorder).2.The seventh and eighth nerves are normal in size and are symmetric bilaterally.