 There is no evidence of acute infarct. There is a small 1 mm thick T1 hyperintense extra-axial fluid collection along the right cerebellar hemisphere compatible with subacute subdural blood product. There are a few scattered punctate foci of T2 hyperintensity supratentorially that likely represent age-related white matter changes/chronic small vessel ischemic disease. There is T1 hyperintensity within the basal ganglia compatible with manganese deposition related to chronic liver dysfuntion. The ventricles and basal cisterns are normal in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. There is minimal fluid within the mastoid air cells. There is a nasogastric tube and fluid within the nasopharynx.
1.No evidence of acute infarct.2.Trace subacute subdural hematoma along the right cerebellar hemisphere.3.Findings compatible with basal ganglia manganese deposition, related to chronic liver disease.4.Fluid within the nasopharynx surrounding a nasal gastric tube.