History of optic neuritis with left hemifacial spasm. Evaluate for demyelinating disease. There is no evidence of intracranial hemorrhage, mass effect, or acute infarct. There are minimal scattered foci of abnormal T2/FLAIR hyperintensity within the periventricular and subcortical white matter and along the undersurface of the corpus callosum. There is no abnormal leptomeningeal or parenchymal enhancement. No lesions within the brainstem. The ventricles and basal cisterns are unchanged in size and configuration. There is no midline shift or herniation. The skull and extracranial soft tissues are unremarkable. There is a right lens implant. Dedicated orbital study was not obtained however there is some T2 hyperintense signal involving the left optic nerve (coronal T2 image 9/34) which may represent sequela of prior optic neuritis.
1.No evidence of intracranial hemorrhage, mass effect, or acute infarct.2.Several scattered foci of abnormal T2/FLAIR hyperintensity within the periventricular and subcortical white matter are seen; while these are nonspecific, several of these lesions are in a periventricular distribution and at the calloso-septal interface raising suspicion for chronic demyelinating disease. No abnormal enhancement to suggest active demyelination.I personally reviewed the Images and/or procedure with the Resident/Fellow and agree with this report.