72 years Female (DOB:7/21/1944)Reason: evaluate for focal ischemic deficits History: L facial droop (transient), c/f TIAPROVIDER/ATTENDING NAME: JAFAR AL-SADIR JAFAR AL-SADIR MRI of the brainNo diffusion weighted abnormalities are appreciated.The CSF spaces are appropriate for the patient's stated age with no midline shift. There is a mild to moderate degree of periventricular and subcortical punctate hyperintense white matter lesions present identified on the FLAIR and T2 images.No abnormal mass lesions are appreciated intracranially. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.On susceptibility imaging there are dictated foci of signal loss in the left cerebellar hemisphere as well as the right cerebellar hemisphere.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact. Signal asymmetry from the patient size is suspected to be related to artifactMRA brain:Antegrade flow is present in the distal internal carotid arteries, the distal vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries. Some mild apparent narrowing at the right carotid siphon is suspected to be related to susceptibility effect from the adjacent clinoid processThere is no evidence for intracranial aneurysm or cerebrovascular occlusion.The anterior communicating artery is identified and is small. The posterior communicating arteries are very small. There is extracranial origin of the left posterior inferior cerebellar artery. The right vertebral artery slightly larger than the left vertebral artery
1.No evidence for cerebrovascular occlusive disease.2.No evidence for intracranial aneurysm. 3.Periventricular and subcortical white matter lesions of a mild to moderate degree are nonspecific. At this age they are most likely vascular related. 4.There are several microhemorrhages present within the cerebellum which are nonspecific.