Unspecified dementia without behavioral disturbance [F03.90], Reason for Study: ^Reason: 69 year old with short-term memory loss, likely dementia, considering Alzheimer disease and vascular etiology. Please comment on hippocampus. History: memory loss. There is no evidence of acute ischemic or hemorrhagic lesion on this scan.Bilateral amygdala and bilateral parahippocampal gyri appear to be symmetrically small in size. There is no evidence of increased signal intensity of bilateral hippocampus on FLAIR or T2 images though this is not a dedicated temporal lobe MR scan. There is no evidence of preferential fronto-parietal lobar atrophy. Scattered FLAIR/T2 high signal intensity lesions on bilateral periventricular white matter and centrum semiovale indicating non specific small vessel ischemic disease.The ventricular system appears to be slightly enlarged but unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, or restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
1. Bilateral symmetric atrophic changes of both amygdala and parahippocampal gyri as described above, unchanged since prior scan.2. No evidence of acute ischemic or hemorrhagic lesion.3. Non specific small vessel ischemic disease.