Increased seizure frequencies. History of developmental delay (language, gross motor, fine motor), failure to thrive, with complicated pre-natal and natal course (with suspected hypoxic ischemic injury), developmental delay (language, gross motor, fine motor), failure to thrive, with complicated pre-natal and natal course (with suspected hypoxic ischemic injury). There is no evidence of intracranial hemorrhage, mass, or acute infarct. The corpus callosum is relatively diffusely thin and there is mild paucity of periventricular white matter in the bilateral cerebral hemispheres bilaterally. There are diffuse confluent areas of increased T2 hyperintensity in the bilateral cerebral periventricular and deep white matter. There is no evidence of hydrocephalus. There is no midline shift or herniation. The major cerebral flow voids are intact. There is mild mucosal thickening involving the bilateral ethmoid and maxillary sinuses. The orbits, skull, and scalp soft tissues are grossly unremarkable. There is fluid within the left mastoid air cells and middle ear.
1. Diminished periventricular white matter bulk and signal abnormality likely represents periventricular leukomalacia.2. No evidence of acute intracranial hemorrhage, mass, or acute infarct.3. Left otomastoid effusion may represent mastoiditis.I personally reviewed the Images and/or procedure with the Resident/Fellow and agree with this report.