86 years Female (DOB:3/10/1930)Reason: Look for cause of dizziness History: vertigoPROVIDER/ATTENDING NAME: ADAM S CIFU IRSK JASON ANDERSON The CSF spaces are appropriate for the patient's stated age with no midline shift. There is vague T2 and FLAIR signal hyperintensity associated with T1 hypointensity present in the midbrain centrally at the expected location of the oculomotor nuclei, medial longitudinal fasciculus and periaqueductal gray which extends to the superior cerebellar peduncles suggesting that there is involvement of rubrocerebellar or cerebellothalamic tracts.There are several periventricular and subcortical punctate hyperintense white matter lesions present identified on the FLAIR and T2 images. These are likely of little clinical significance.No abnormal mass lesions are appreciated intracranially. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses demonstrate opacification of the left maxillary sinus and mucosal thickening in the right maxillary sinus with the partial opacification of the ethmoid air cells and frontal sinuses.. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact.The eyeball lenses are thin.
1.There is no evidence for acute ischemic cerebral infarction.2.There is no evidence for intracranial mass lesion3.There is subtle signal change present in the midbrain and superior cerebellar peduncles as detailed above which is nonspecific. It is felt not to be artifactual. This could represent a neurodegenerative process, metabolic process or even paraneoplastic process. It is nonspecific.