Cervical radiculopathy Craniovertebral junction appears within normal limits. The cervical vertebral bodies are appropriate in height. Alignment is maintained. Bone marrow signal is within normal limits. There is no definite cord signal abnormality at any level aside from punctate focus involving the left aspect of the cord at the C5-C6 level.There is developmental narrowing of the cervical spinal canal with significant superimposed degenerative changes as detailed below: C2-3: Central disc protrusion with mild indentation on the ventral cord and ligamentum flavum thickening. There is overall moderate spinal canal stenosis at this level. Neural foramina are patent.C3-4: There is a disc osteophyte complex and ligamentum flavum thickening contributing to moderate spinal canal stenosis at this level and mild cord deformity. There is uncovertebral hypertrophy and mild facet arthropathy contributing to moderate bilateral neural foraminal stenosis, right greater than left.C4-5: There is disc osteophyte complex and ligamentum flavum thickening contributing to moderate spinal canal stenosis with mild cord deformity. There is uncovertebral hypertrophy and mild facet arthropathy contributing to moderate to severe right and moderate left neural foraminal stenosis.C5-6: There is disc osteophyte complex and ligamentum flavum thickening contributing to moderate to severe spinal canal stenosis with cord deformity, particularly on the left. There is uncovertebral hypertrophy and mild facet arthropathy contributing to moderate to severe right and moderate left neural foraminal stenosis. There is abnormal T2 signal involving the left lateral aspect of the cord at this level.C6-7: There is disc osteophyte complex which effaces the ventral thecal sac and contributes to mild to moderate spinal canal stenosis. There is minimal narrowing of the neural foramina related to uncovertebral hypertrophy.C7-T1: No significant compromise to the spinal canal or neural foramina.The vertebral artery flow voids appear to be intact. Multinodular goiter is noted and better assessed on recent ultrasound. Right submandibular gland is not definitively seen and may be absent or diminutive.
1. Developmental narrowing of the cervical spinal canal with superimposed degenerative changes resulting in significant multilevel spinal canal stenoses from the C2-C3 to the C6-C7 level with mild cord flattening, worst at the C5-C6 level. There is also significant neural foraminal stenoses from the C3-C4 to the C5-C6 levels as detailed above. 2. Focal cord signal abnormality involving the left aspect of the cord at the C5-C6 level favored to represent sequela of compressive myelopathy.