Disturbance of skin sensation [782.0], Reason for Study: ^Reason: stroke? History: hx TIA, weakness No evidence of acute ischemic or hemorrhagic lesion.Several scattered high signal intensity lesions on bilateral hemispheric white matter on FLAIR imaging are nonspecific.No change of previously demonstrated right cerebellar hemispheric linear tissue defect since prior scan.Opacification of paranasal sinus appear to be improved since prior scan.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction. Normal flow voids are identified in the major intracranial vessels. The mastoid air cells are clear.3D time of flight MOTSA MRA brain images with maximum intensity projections of the anterior/posterior intracranial circulation demonstrate normal ICAs, MCAs and ACAs. Vertebrobasilar system appears to be normal. Bilateral PCAs are fetal origin. There is no intracranial aneurysm. No significant luminal stenosis.3D MRA neck post-gadolinium images with maximum intensity projections of the cervical vasculature demonstrate normal flow enhancement in a normal aortic arch origin of the right brachiocephalic, left common carotid, and left subclavian arteries. The vertebral artery origins are normal. There is normal flow enhancement through the bilateral common carotid, carotid bifurcations, internal/external carotid, and vertebral arteries.
1. No evidence of acute ischemic or hemorrhagic lesion.2. Right cerebellar hemispheric encephalomalacia and few non specific high signal foci on FLAIR images, no change since prior scan.3. Normal brain MR angiogram and neck MR angiogram. 