Intracerebral hemorrhage identified in March 2015. Evaluate stability. Breast cancer. Mild residual enhancement within the hemosiderin-stained hemorrhage bed within the left parietal lobe has diminished from prior, consistent with expected evolution of prior hemorrhage. There is confluent T2 hyperintensity within the left frontal and parietal white matter, consistent with evolving prior ischemic injury. There is left frontal encephalomalacia and gyriform intrinsic T1 hyperintensity along the middle frontal gyrus, consistent with evolution of ischemia. There is no evidence of new intracranial hemorrhage, mass, or acute infarction. There are scattered periventricular and subcortical T2 hyperintensities, which are nonspecific, but most likely secondary to chronic microvascular ischemic disease. There is unchanged T2 hyperintensity within the left basal ganglia and thalamus, probably chronic lacunar infarctions. The ventricles and basal cisterns are normal in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. The orbits, skull, paranasal sinuses, and scalp soft tissues are grossly unremarkable.
1.Evolving left parietal intraparenchymal hematoma with diminished mild residual enhancement in the affected parenchyma. No findings are seen to suggest an underlying lesion or mass.2.Evolving left frontal lobe encephalomalacia and areas of left frontoparietal T2 hyperintensity also consistent with evolution of prior ischemic injury.3.No definite evidence of metastatic disease or new pathologic enhancement.