Other malformations of cerebral vessels [Q28.3], Reason for Study: ^Reason: AVM, s/p SRS, 3T Scanner History: Surveillance Brain MRIThere is tangled vascular structure with enhancement at the inferior aspect of left post-central sulcus and left inferior parietal lobule caudal portion. The lesion is measured about 7.8mm x 8.6mm. There is associated parenchymal T2/FLAIR high signal intensity at the left inferior parietal lobule indicating gliotic changes more likely than brain edema. There is no evidence of acute ischemic or hemorrhagic lesion.The ventricles, sulci and cisterns are symmetric and unremarkable. The midline structures and cranial-cervical junction are normal. The paranasal sinuses and mastoid air cells are clear.Brain MRA3D time of flight MOTSA MRA brain images with maximum intensity projections of the anterior/posterior intracranial circulation demonstrate slightly prominent left MCA M2 and M3 segmental arteries as well as visualization of the right transverse-sigmoid sinuses indicating potential shunting of arterial flow. However, there is no definitive evidence of tangled vascular structure.There is no evidence of intracranial arterial aneurysm. No arterial luminal stenosis.The right ICA, MCA and bilateral ACAs appear to be normal. Vertebrobasilar system appears to be normal.
1. MRI evidence of enhancing lesion on the left post central sulcus and inferior parietal lobule caudal aspect indicating possible brain AVM (7.8mm x 8.6mm) with adjacent parenchymal gliosis. However, MRA did not show direct evidence (vascular tangling and enlarged feeding artery) of the lesion.2. No evidence of acute ischemic or hemorrhagic lesion.