Speech delay. There is no evidence of intracranial hemorrhage, mass, or acute infarct. There is mild thinning of the posterior body of the corpus callosum and mildly diminished posterior periventricular white matter bulk, with associated mild prominence of the bilateral lateral ventricle atria. There are punctate foci of susceptibility effect in the bilateral frontal and temporal lobes. The brain parenchyma otherwise appears unremarkable, with an adequate degree of myelination. There is no midline shift or herniation. The major cerebral flow voids are intact. The orbits, skull, paranasal sinuses, and scalp soft tissues are grossly unremarkable.
1. Mild thinning of the posterior body of the corpus callosum and mildly diminished posterior periventricular white matter bulk, but no partial agenesis of the corpus callosum. 2. Nonspecific scattered punctate foci of susceptibility in the bilateral cerebral hemispheres may represent chronic microhemorrhages or calcifications. 