Male, 41 years old, with problems walking and history of multiple sclerosis. Assess for changes from prior MRI. Brain:Numerous patchy areas of T2 hyperintensity are seen within the periventricular and juxtacortical white matter of the cerebral hemispheres. The cerebellum is also involved to a lesser degree. Since the prior examination, and accounting for differences in imaging technique, no significant interval change in the size or number of these lesions is appreciated.The above-mentioned lesions exert no significant mass effect. They are associated with no susceptibility effect or pathologic enhancement. No abnormal extra-axial collections are seen. The ventricular system is stable and within normal limits for size and morphology.Cervical spine:Abnormal T2 signal intensity is again observed at several locations in the cervical spinal cord. At C2, this signal abnormality is predominantly dorsal and extends for about 10 mm in the craniocaudal dimension. An additional area of signal abnormality is seen spanning from approximately C3 where the signal abnormality is holocord, down to approximately C4-5 where it is more right-sided. Evidence of cord volume loss is also seen at the C3 level. There has been no significant or convincing change in the appearance of these findings relative to the prior examination, again accounting for differences in imaging technique.Spinal alignment is anatomic. Vertebral body heights are preserved. No worrisome marrow signal replacement or marrow edema is seen. No significant disc herniation, spinal canal or neuroforaminal stenosis is identified.Thoracic spine:Areas of cord T2 signal abnormality are again observed on the right at T1, on the left at T3, on the right at T7-8, on the right at T9-10, and centrally at T11-12. Accounting for differences in imaging technique, and motion artifact on the present examination, no significant or convincing interval change is seen.Spinal alignment is anatomic. Vertebral body heights are preserved. Vertebral body hemangiomas are seen at T8 and T9. Marrow signal characteristics are otherwise unremarkable. No significant disc herniation, spinal canal or neuroforaminal stenosis is seen.
Numerous periventricular and juxtacortical white matter lesions are reidentified in the brain compatible with the stated history of multiple sclerosis. There has been no significant interval change in the size, number or distribution of these lesions.Multiple areas of pathologic spinal cord signal abnormality are identified in the cervical and thoracic regions, again without significant or convincing interval change.