 MRI brain: There is no diffusion abnormality. There are a few scattered T2 and FLAIR hyperintensities in the periventricular and subcortical white matter which are nonspecific. The ventricles and sulci are within normal limits. The basal cisterns remain patent. There is no midline shift or mass effect. No extra-axial fluid collection is identified.Normal flow-voids are demonstrated in the major intracranial vascular structures. The midline structures and craniocervical junction are within normal limits.MRI spine: There is mild reversal of curvature of the spine, otherwise the cervical spine is in normal alignment. The vertebral body and disk heights are well-maintained. No worrisome focal marrow signal abnormality is appreciated. Central cord T2 signal abnormal at the C2 level is seen. No associated cord swelling is and in fact there is mild thinning of the cord at this level. No other area of significant abnormality is seen in the remaining cervical cord.The spinal canal is congenitally slender. Additional level specific findings are as follows.C2-C3: There is mild uncovertebral hypertrophy and facet arthropathy. There is no significant neural foraminal or spinal canal stenosis.C3-C4: There is uncovertebral hypertrophy and mild facet arthropathy resulting in moderate left neural foraminal narrowing. Minimal spinal canal stenosis is seen.C4-C5: There is uncovertebral hypertrophy and right paracentral disc osteophyte complex with effacement of ventral thecal sac. There is resultant moderate left neural foramina narrowing. No significant spinal canal stenosis.C5-C6: There is mild central disc osteophyte complex and mild left neuroforaminal narrowing. There is no spinal canal stenosis.C6-C7. There is osteophytic disc complex and mild uncovertebral hypertrophy resulting in mild bilateral neuroforaminal narrowing. There is no spinal canal stenosis.C7-T1: There is minimal disc osteophyte complex without significant spinal canal or neuroforaminal narrowing.Paraspinal soft tissue structures are grossly unremarkable.
1. Small foci of T2 hyperintensity are seen scattered in the cerebral white matter, which are nonspecific.2. Central spinal cord T2 signal abnormality at C2 with associated cord thinning suggesting a chronic lesion. The differential is broad and includes previous inflammation/infection as well as demyelination or post infectious/autoimmune conditions.3. Mild degenerative changes in the cervical spine without high-grade neural foraminal or spinal canal stenosis.