Evaluate for chronic pancreatitis. History of recurrent acute pancreatitis. Daily abdominal pain nausea and vomiting. ABDOMEN:LIVER, BILIARY TRACT: The liver is normal in morphology and size. There is loss of signal intensity on in phase imaging suggesting iron deposition. Status post cholecystectomy. No intra or extrahepatic biliary ductal dilatation. The vessels are patent. No focal suspicious lesion.The vessels are patent.Perfusional anomaly in segment 4B, benign.SPLEEN: The spleen is normal in morphology and size without a focal lesion. Small splenule adjacent to splenic hilum.PANCREAS: The pancreas is normal in morphology. The main pancreatic duct is at the upper limits of normal measuring approximately 3 to 4 mm in maximal caliber. It is relatively smooth without focal stricture or dilatation. Following secretin administration there is a prompt excretion of pancreatic fluid with mild increase in main pancreatic duct caliber. No side-branch ectasia.The pancreas demonstrates decreased intrinsic T1 weighted signal intensity suggesting parenchymal dysfunction. It demonstrates homogeneous parenchymal enhancement. No peripancreatic inflammatory changes to suggest acute pancreatitis. No focal lesion.ADRENAL GLANDS: No significant abnormality noted.KIDNEYS, URETERS: No significant abnormality noted.RETROPERITONEUM, LYMPH NODES: No significant abnormality noted.BOWEL, MESENTERY: No significant abnormality noted.BONES, SOFT TISSUES: No significant abnormality noted.OTHER: No significant abnormality noted.
No evidence of acute pancreatitis. Decreased intrinsic T1-weighted signal intensity and mild prominence of the main pancreatic duct can be seen in the setting of mild chronic pancreatitis. Normal secretin response. No focal pancreatic lesion or significant duct stricture.