 Patchy and confluent areas of T2/FLAIR signal hyperintensity and diffusion restriction in the cortex and subcortical white matter in the medial aspect of the right posterior temporal lobe extending to the right occipital lobe have increased compared to prior examination and correlate with the area of hypoattenuation on recent CT head. There is subtle increased sulcal effacement in this area compared to prior MR without evidence of susceptibility to suggest hemorrhage.There are multiple additional foci of cortical and subcortical white matter T2/FLAIR signal hyperintensity in the left precentral gyrus and left frontal lobe, which appear decreased compared to prior MRI examination. There is improved signal abnormality in the right thalamus. There is subtle T2/FLAIR signal hyperintensity in the right greater than left brachium pontis of the cerebellum, decreased compared to prior MRI. No residual abnormality is appreciated in the cerebellar hemispheres.Postcontrast images demonstrate gyriform enhancement of the medial right occipital lobe in the area of increased T2/FLAIR signal. There are additional scattered foci of cortical enhancement overlying the convexity of the right frontal lobe. The ventricles and sulci are within normal limits. The cisterns remain patent. No extra-axial fluid collection is identified.Normal flow-voids are demonstrated in the major intracranial vascular structures. There is complete opacification of the bilateral mastoid air cells and inner ears.
1.Increased confluence of T2/FLAIR signal abnormality in the medial right posterior temporal lobe extending to the right occipital lobe as well as gyriform enhancement, with interval sulcal effacement. Additional foci of cortical enhancement are also seen over the convexity of the right frontal lobe. These findings are suggestive of evolving now subacute infarcts.2.Interval decrease or resolution of signal abnormality in the infratentorial and anterior white matter.