Syncope and collapse No evidence of acute ischemic or hemorrhagic lesion.Especially on gradient echo technique, there are small multiple scattered signal lordosis on cortical and subcortical white matter indicate possibility cerebral amyloid angiopathy. However, there is no evident local signal void area indicating acute or chronic hemorrhage lesion.There is no evidence of acute ischemic lesion either.On FLAIR imaging, skeletal somewhat patch high signal intensity lesions on bilateral periventricular white matter indicating nonspecific small vessel ischemic disease.Right eyeball is a prosthesis.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, or restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
No evidence of acute ischemic or hemorrhagic lesion.Multifocal skeletal small cortical and subcortical distributed signal voids indicating possibility of cerebral amyloid angiopathy.Mild on to minimal nonspecific small vessel ischemic disease.