CNS nocardia, now with numbness and tingling in fingers and visual change. There is an unchanged avidly enhancing mass with susceptibility effect in the splenium of the corpus callosum, which measures up to approximately 25 mm. There is unchanged associated extensive vasogenic edema that extends into the bilateral posterior cerebral hemispheres. There are multiple unchanged punctate foci of susceptibility effect in the bilateral cerebral and cerebellar hemispheres. There is also an unchanged branching area of susceptibility effect in the right pons with associated patchy enhancement and corresponding calcifications depicted on CT. There are unchanged scattered foci of T2 hyperintensity in the cerebral white matter, which may represent chronic small vessel ischemic disease. There is no evidence of acute intracranial hemorrhage or acute infarct. The ventricles and basal cisterns are unchanged in size and configuration. There is a partially empty sella. There is no midline shift or herniation. The major cerebral flow voids are intact. The orbits, skull, paranasal sinuses, and scalp soft tissues are grossly unremarkable.
No significant interval change in the lesion within the splenium of the corpus callosum with associated extensive surrounding vasogenic edema and other stigmata of alleged central nervous system nocardiosis.