Obstructive sleep apnea (adult) (pediatric) [G47.33], Reason for Study: ^Reason: Patient with episodes of apnea requiring intubation History: Patient with episodes of apnea requiring intubation Brain MRINo evidence of acute ischemic or hemorrhagic lesion.A couple of T2/FLAIR high signal intensities are non specific. There are focal tissue losses on the bilateral thalami indicating chronic lacunar infarctions. Prominent sulci and ventricular system indicate mild brain atrophy which is age appropriate.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia, or abnormal contrast enhancement. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses are clear.Bilateral mastoid air cells are opacified.Brain MRAMotion artifacts degraded examination quality.3D time of flight MOTSA MRA brain images with maximum intensity projections of the anterior/posterior intracranial circulation demonstrate irregularities of bilateral MCAs and focal luminal narrowing on the proximal basilar artery. However, these lesions could well be related to motion artifacts. Overall, there is no evidence of significant luminal narrowing. Bilateral PCAs are fetal origin and Acom artery appears to be patent.No aneurysm was found.However, if clinically indicated, brain MRA can be repeated without additional charge for more precise evaluation.
1. No evidence of acute ischemic or hemorrhagic lesion.2. Motion artifacts degraded Brain MRA, so precise evaluation of intracranial arterial system is not possible. Grossly, there is no significant intracranial arterial luminal stenosis. 