Other disorders of psychological development [F88], Reason for Study: ^Reason: re-evaluate hypoxic injury History: global developmental delay There are bilateral nearly symmetric FLAIR/T2 high signal intensity lesions on the posterior aspect of basal ganglia likely globus pallidi and bilateral thalami. There is no evidence of restricted diffusion or susceptibility artifacts indicating hemosiderin deposition. These lesions could represent prior ischemic lesions but are non specific.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, or restricted diffusion/acute ischemia. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The mastoid air cells are clear. Focal fluid collection on the left ethmoid sinus.
1. Bilateral nearly symmetric basal ganglia especially posterior aspect of globus pallidus and bilateral thalami as described above.2. No evidence of acute ischemic or hemorrhagic lesion. 