44 years Female (DOB:9/5/1971)Reason: left sided weakness etiology History: left sided weaknessPROVIDER/ATTENDING NAME: DIANE B CHANEY KOUROSH REZANIA MRI of the brainNo diffusion weighted abnormalities are appreciated.The CSF spaces are appropriate for the patient's stated age with no midline shift. No intracranial hemorrhage is identified. No edema is identified within the brain parenchyma.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits demonstrate a subtle old orbital floor blowout fracture.MRA brain:Antegrade flow is present in the distal internal carotid arteries, the distal vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries.There is no evidence for intracranial aneurysm or cerebrovascular occlusion.The anterior communicating artery is identified and is small. The posterior communicating arteries are small but slightly larger on the left than the right side.. The right vertebral artery is slightly larger than the left vertebral artery.. There is extracranial origin of the right posterior inferior cerebellar artery.MRA neck:There is opacification of the aortic arch, great vessels from the aortic arch and carotid arteries and vertebral arteries. There is no stenosis identified of the great vessels from the aortic arch. On the basis of NASCET criteria there is no significant stenosis at the carotid bifurcations. There is no significant stenosis along the course of the right vertebral artery. The left common carotid artery originates from the innominate artery.The left vertebral artery appears irregular along the V2 segment but not occluded..
1.No evidence for intracranial aneurysm.2.The left vertebral artery appears irregular along the V2 segment, however, does not appear occluded. This could be a sign of underlying vasculopathy such as fibromuscular dysplasia.3.No evidence for intracranial or extracranial cervicocerebral vascular occlusive disease.4.No evidence for acute ischemic cerebral infarction.5.No evidence for intracranial hemorrhage