52-year-old female with history of paroxysmal atrial fib with intermittent vision changes, facial numbness, evaluate for stroke, mass or other There is no restricted diffusion on DWI sequences. The CSF spaces are appropriate for patient's age. There is faint vascular enhancement traversing the white matter from the left frontal horn of the left lateral ventricle anteriorly to the left frontal cortex consistent with a development vascular anomaly of no clinical significance. Otherwise no areas of abnormal intraparenchymal enhancement, mass lesions or mass effect are noted. There are very few punctate foci of FLAIR hyperintensity in the subcortical white matter, nonspecific. There is mild prominence of the cerebellar and vermin folia for age which are of unknown clinical significance.There is a tiny focus of T2 and FLAIR hyperintensity in the mid pons posteriorly which on post enhanced images demonstrates diffuse ill-defined faint enhancement and definite susceptibility signal changes. This focus measures 6 mm x 5 mm on sagittal post enhanced images (series1203, image 95) and most likely represents a form of vascular malformation such as a capillary telangiectasia.The visualized paranasal sinuses are clear. The visualized orbits are unremarkable. The mastoid air cells are well-pneumatized.
1. Overall unremarkable examination. 2. Findings most likely represent a capillary telangiectasia at the mid pons and likely a developmental vascular anomaly in the left frontal cortex as described above. 3. Other findings as above. 