 There are foci of T2/FLAIR hyperintensities in the subcortical and the periventricular white matter, which are nonspecific, but most likely consistent with chronic small vessel ischemic changes. There is no evidence of acute hemorrhage, mass effect, or edema. The ventricles and sulci are enlarged, which may be age related versus neurodegeneration. There is no midline shift. Corticomedullary differentiation is maintained. The basal cisterns are patent. There is a partial empty sella. There are no diffusion or susceptibility abnormalities. The T2 flow voids are unremarkable.
1. No evidence for acute intracranial hemorrhage, mass effect, or edema. No evidence of acute cerebral infarction.2. Brain parenchyma volume loss and ventriculomegaly is present. The cause for this is unknown based on this study alone. Differential considerations could include neurodegenerative disorder and normal pressure hydrocephalus3. There is a mild degree of nonspecific T2/FLAIR hyperintensities in the subcortical and periventricular white matter, at this age is most likely vascular related.