72-year-old female with cerebellar degeneration experiencing ataxia. Redemonstrated are foci of T2/Flair hyperintensity in the cerebral white matter, without associated restricted diffusion, mass effect, susceptibility abnormality, or enhancement. There are a few more lesions compared to the 2012 study, especially notable within a cluster identified in the left frontal lobe white matter.Redemonstrated is developmental venous anomaly in the left frontal lobe. The ventricles, sulci and cisterns are prominent, with a slightly disproportionately greater degree of cerebellar atrophy. However, there is no apparent adjacent pons atrophy. Overall, the degree of supratentorial and infratentorial atrophy is stable.The gray-white matter differentiation is maintained. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia, or abnormal contrast enhancement. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. Mucosal thickening and a small superimposed air-fluid level is present within the right maxillary sinus. The remaining paranasal sinuses and mastoid air cells are clear.
1.Progressive chronic small vessel ischemic disease.2.Stable mild supratentorial cerebral atrophy and stable mild to moderate cerebellar atrophy.3.Mucosal thickening and a small superimposed air-fluid level is present within the right maxillary sinus.