Evaluate progression of multiple sclerosis, optic neuritis. Brain:There are innumerable T2 hyperintense lesions throughout the supratentorial and infratentorial white matter compatible with the patient's history of multiple sclerosis. There are several new T2 hyperintense lesions. For example, one is located within the subcortical white matter adjacent to the right central sulcus (series 501, image 50) and another within the left caudothalamic groove (series 501, image 43). Most of the lesions demonstrate low T1 signal, unchanged. A previously seen ring-enhancing lesion in the right superior frontal gyrus has resolved with residual T1 low signal (series 1701, image 65). Nonspecific enhancing foci in the the left caudate head (series 1701, image 41) and subcortical white matter of the left temporal lobe (series 1701, image 35) are nonspecific and may even be potentially artifactual. There is no restricted diffusion to suggest acute ischemia. The ventricles and sulci are unchanged in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. The skull, paranasal sinuses, and scalp soft tissues are unremarkable.Orbits:The optic nerves and chiasm are normal in caliber and signal without abnormal enhancement. The globes are intact. The extraocular muscles are within normal limits. There is no periorbital soft tissue abnormality.
1.Innumerable supratentorial and infratentorial white matter lesions compatible patient's history of multiple sclerosis with several new lesions as described above.2.Interval resolution of ring enhancing lesion in the right superior frontal gyrus.3.No imaging evidence of optic neuritis.4.No evidence for an acute intracranial abnormality. 