Cerebral infarction, unspecified [I63.9], Reason for Study: ^Reason: L temporal fullness ? Tumor or just a strange appearance of a prior stroke History: seizures There is no evidence of acute ischemic lesion.Left temporal occipital encephalomalacia is again seen with especially linear high signal intensities along the gray matter indicating laminar necrosis right evidence of susceptibility artifacts indicating prior hemorrhage. On post gadolinium enhancement, there is a subtle parenchymal enhancement with the chest at the dura which is not unusual for post ischemic stroke finding. There is no evidence of discrete intra or extra-axial abnormal enhancing mass lesion.Constellation of above findings indicate chronic left temporal occipital ischemic infarction with hemorrhagic transformation.Minimal nonspecific small vessel ischemic disease was seen on periventricular white matter high signal intensities on FLAIR imaging.The ventricles, sulci and cisterns are symmetric and unremarkable except ex focal changes of left lateral ventricle occipital horn. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.
1.No acute ischemic or hemorrhagic lesion.2.Encephalomalacia with hemosiderin deposition of the left temporal occipital lobe likely due to chronic ischemic infarct, no change since prior scan.3. No abnormal enhancement.