86 years Male (DOB:1/27/1930)Reason: CSF flow study History: ataxiaPROVIDER/ATTENDING NAME: PETER C WARNKE PETER C WARNKE There is redemonstration of ventriculomegaly. Biventricular diameter is 15 mm on the current exam and was 50 mm on the prior exam. The third ventricle width is 11 mm on the current exam and 11 mm on the prior exam.There are subdural effusions present bilaterally which were also present on the prior exam. These are also stable.There is a mild degree of periventricular and subcortical punctate hyperintense white matter lesions present identified on the FLAIR and T2 images. Punctate focus of T2 and FLAIR signal hyperintensity is present in the right basal ganglia. Several punctate foci of signal hyperintensity are present in the pons.There is a punctate focus of signal hypointensity on susceptibility imaging in the right cerebellar hemisphere. This most likely represents a microhemorrhage and was also present on the previous exam. Another punctate focus of susceptibility is present in the left insular cortex which was also present on the prior exam. Initial location these microhemorrhages do not establish a particular pattern and are of unknown significance.Normal vascular flow voids are present in the distal carotid and vertebral arteries, the basilar artery and the proximal anterior, middle and posterior cerebral arteries as well as the internal cerebral veins and superior sagittal sinus.The visualized portions of the paranasal sinuses are clear. The visualized portions of the mastoid air cells are clear. The visualized portions of the orbits are intact. The eyeball lenses are thin.
1.Stable ventriculomegaly. Possibility of normal pressure hydrocephalus cannot be excluded.2.Periventricular and subcortical white matter lesions of a mild degree and scattered lesions in the brainstem and right basal ganglia are nonspecific. At this age they are most likely vascular related. 3.Subdural effusions are stable compared to prior exam. This could be related to atrophy.