Ataxia, unspecified [R27.0], Reason for Study: ^Reason: Eval L frontal lesion w/ contrast (T1 w only needed, brain wo already done) History: RLE apraxia Brain MRINo evidence of acute ischemic or hemorrhagic lesion.Patchy and somewhat confluent FLAIR/T2 high signal intensities on bilateral periventricular white matter and centrum semiovale indicating nonspecific small vessel ischemic disease, unchanged since prior scan.The ventricles, sulci and cisterns are symmetric and unremarkable. There is no mass, mass effect, edema, midline shift, intra or extra-axial fluid collection/hemorrhage, restricted diffusion/acute ischemia, or abnormal contrast enhancement. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.Cervical spine MRIThe cranial cervical junction is normal.The spinal cord signal intensity is normal.There is no abnormal enhancement.There is subtle kyphotic angulation of the cervical spine at C45.Degenerative changes are follows;C23: Minimal focal bulging of disc central to right which abuts thecal sac. No neuroforaminal stenosis.C34: Diffuse bulging of disc which abuts thecal sac results minimal spinal stenosis. No neuroforaminal stenosis.C45: There is broad based disc protrusion central and slightly eccentric toward right which abuts thecal sac and narrows right side lateral recess. Mild to moderate spinal canal stenosis and moderate neuroforaminal stenosis on the right side are seen.C56: Diffuse broad based disc protrusion which abuts thecal sac, mild to moderate spinal canal stenosis.C67: Diffuse bulging of disc, otherwise unremarkable.C7T1: Diffuse bulging of disc, otherwise unremarkable.
1. No evidence of acute ischemic or hemorrhagic lesion. No abnormal enhancement.2. Non specific small vessel ischemic disease, unchanged.3. Multilevel various degree degenerative changes of cervical spine as described above.