Reason: are there any abnormalities? History: term infant with perinatal depression, post cooling. 41 weeks gestational age, L GA, convulsions, respiratory distress with pneumothorax. There is evidence of restricted diffusion within the right posterolateral thalamus, the bilateral midthalami, and the corpus callosum without cortical involvement. There is also a small focus of susceptibility within the right posterolateral thalamus, representing microscopic blood products.The ventricles and sulci are normal in size. The cerebellar tonsils are in normal position. The posterior cerebellar vermis may be hypoplastic versus anatomic variation, with a prominent posterior fossa CSF space. There is fluid adjacent to the torcula and the right transverse sinus which displaces the adjacent sinuses, perhaps representing an effusion due to molding of the skull. The myelination patterns are appropriate for the patient's gestational age. There is no evidence of definite intracranial hemorrhage. There are no masses, mass effect or midline shift. The pituitary gland is normal in size. Flow voids are present within the major vessels indicating patency. The paranasal sinuses and mastoid air cells are clear. There is a 1.1 cm subgaleal hematoma extending across the midline parietal scalp.
1. Findings consistent with hypoxemic ischemic injury, likely profound given the involvement of the posterolateral thalamus.2. Small parietal subgaleal hematoma.3. T1 hypointense fluid adjacent to the torcula, which likely represents an effusion due to skull molding. No evidence of intracranial hematoma.