Multiple sclerosis [340], Reason for Study: ^Reason: new ms flare History: BL leg weakness Brain MRIThere is no evidence of acute ischemic or hemorrhagic lesion.There are multiple variable sized FLAIR high signal intensity lesions on subcortical white matter and bilateral corpus callosum consisted with multiple sclerosis.There is no evidence of abnormal enhancement.Comparing to prior scan, there is no significant interval change in terms of the size, number, distribution and signal intensity of identifiable lesions.The ventricles, sulci and cisterns are symmetric and unremarkable. The midline structures and cranial-cervical junction are normal. Normal flow voids are identified in the major intracranial vessels. The paranasal sinuses and mastoid air cells are clear.C-SpineThere are multifocal high signal intensities of spina cord on both T2 and STIR images compatible with multiple sclerosis involvement. The lesions were seen on from C2-C3 to C4-C5 level and T1-T2 to T2-T3 level.There is no evidence of abnormal enhancement.The cranial-cervical junction is normal. There is normal cervical lordosis. The cervical spine alignment is anatomic. The vertebral body height is preserved. The bone marrow and end plates demonstrate a normal MR appearance. The epidural space, thecal sac, and spinal cord are preserved with no evidence of spinal canal/neural foraminal narrowing or myelopathy. The intervertebral discs demonstrate normal T2 intensity and height with no evidence of disc herniation. The paraspinal soft tissues are unremarkable.There is no evidence of abnormal enhancement, cord compression or mass.
Multifocal intracranial and spinal cord FLAIR high signal intensity lesions consistent with multiple sclerosis lesions.No significant interval change of intracranial lesions since prior scan.No evidence of abnormal enhancement of those lesions.