 Images are motion degraded. Given this caveat:There are tiny punctate foci of restricted diffusion with matching ADC hypointensity within bilateral cerebellar hemispheres as well as supratentorial brain, most notable involving the right parietal lobe and posterior right frontal lobe.Hemorrhagic products are noted within the patient's shunt tract as well as within the known falcine comment bilateral occipital horn, and left cerebellar hemorrhages.Confluent T2 hyperintensity is present within the white matter as well as pons without associated restricted diffusion, susceptibility abnormality, or enhancement. Chronic lacunar infarcts are noted involving bilateral thalami.Note is again made of left occipital lobe encephalomalacia. The cerebellar tonsils are in normal position. The pituitary gland is normal in size. There are no extraaxial fluid collections or subdural hematomas. Flow voids are present within the major vessels indicating patency. There is no abnormal enhancement within the brain.
1.The study is motion degraded limiting sensitivity of the study.2.There are tiny punctate foci of restricted diffusion with matching ADC hypointensity within bilateral cerebellar hemispheres as well as supratentorial brain, most notable involving the right parietal lobe and posterior right frontal lobe. Given the vascular distribution, these most likely represent an embolic origin.3.Redemonstration of intracranial hemorrhages.4.Advanced chronic small vessel ischemic disease.5.Left occipital lobe encephalomalacia.