Ataxia. New onset seizure. Rule out intracranial hemorrhage, ischemia or tumor. History of cerebral palsy, ADHD and autism. There is no evidence of intracranial hemorrhage, mass, or acute infarct. Increased T2 FLAIR signal in the deep biparietal white-matter may represent terminal zones of myelination, a normal variation. Asymmetrically increased T2/FLAIR signal involving the right temporal lobe, including the hippocampal formation, and part of the right frontal and parietal lobes is probably artifactual. The temporal horn of the right lateral ventricle is asymmetrically enlarged but without definite architecture distortion or volume loss of the hippocampus. These findings are unchanged since the previous examination of 2013. Otherwise, the ventricles and basal cisterns are normal in size and configuration. There is no midline shift or herniation. The major cerebral flow voids are intact. The orbits and scalp soft tissues are grossly unremarkable. The adenoids and nasal turbinate mucosa are enlarged. There are also prominent lateral retropharyngeal lymph nodes. There is also mild ethmoid mucosal thickening. The mastoid air cells are clear. There is no destructive skull lesion.
1.No evidence of acute intracranial hemorrhage, mass, or acute infarct.2.Asymmetric enlargement of the temporal horn of the right lateral ventricle without definite signal or morphologic abnormality of the right hippocampus might be developmental. Increased deep parietal white-matter T2 signal may reflect terminal zones of myelination, which is a normal variation.