It ' s racist to give black peoples chicken in the US . <| sep |> Thanks for clarifying that you can ' t rule out a Stinger causing this sort of damage . <| sep |> The debate was ended 20 years ago when the early UFC tournaments proved that a pure grappler will almost always win against a pure striker . <| sep |> Jihan ' s still a little coward <| sep |> Based AutisticHis anger revealed the truth <| sep |> No problem . Specifically , it ' s a Ginsu Bagel Knifebagel . html <| sep |> tending to concentrate within a family . Correlation . Mother and father overeat , kid will likely overeat as well . Is there a fat gene that has been traced through a pedigree ? If so , then sure that ' s more conclusive but right now they are studying phenotypes . Have you ever studied basic biology ? A phenotype is what you can see , a genotype is the actual DNA coding for a phenotype . However , phenotypes , especially overall body condition , are influenced by external factors . Also , the genotype would control where fat is stored , not how much . This is like saying that a family of competitive swimmers has a genetic predisposition to muscular shoulders . Their actions influenced their phenotype . same energy intake and same activity level will make one person obese but the other not . Do you really have any studies to support this ? There ' s already a ton of evidence showing that people maintained on the same diet and exercise level will not have significantly different body composition . This is just one example of the contribution of diet on weight . my argument is the large wealth of scientific data which conclusively and undeniably has established at this point that there is a strong genetic link with obesity . I ' m in my fifth year of a Ph . D . in cell biology so I know that it ' s scientifically irresponsible to make such sweeping claims . We never have all the data , theories are never undeniable . The link you provided is a news article FYI , as somebody who has published research papers , the general public and media almost never interpret research properly . I read the study from the link you sent . Did you read and understand it ? They found that the additive effect of SNPs which is a single change in a base pair in a DNA strand account for 30 % of BMI variation . First of all , changing a single base pair only has a finite amount of outcomes . The most common type of SNP doesn ' t do anything to the genetic and therefore protein product . If a single amino acid in a sequence gets changed , this may interrupt protein folding and may change the function of a protein but this is rare . A lot of the time there ' s a redundancy the BP that gets swapped out is replaced by something that codes for the same amino acid anyway . Not only that , they don ' t even know if the genes affected have anything to do with metabolism since they used the entire genome ! These SNPs might be involved in something totally unrelated to metabolism . There are way too many unknowns to say this is undeniable evidence showing obesity has a genetic component . Also , one of the most wellknown genetic factors regulating obesity is leptin . Even then , it has to do with appetite not regulating things like glycolysis . It has nothing to do with defying thermodynamics . Your body is free to decide for itself how much of its energy intake it wants to convert to fat , how much it wants to poop out again and how much it wants to burn and in what way and how efficiently it wants to burn it . Please pick up a book on biochemistry .