A few studies have shown that Rasip1 is necessary to maintain the integrity of EC cell–cell adhesions in vitro [7, 12, 14].
Sample text:```Because previous studies indicated that hexapeptides bind the junction core (29, 32), we tested two junctions, one with an AT-rich core and the other with a GC-rich core (Fig.```
Sample intent: result
Sample text:```Interestingly, MHC I and ERAP1 were coordinately expressed in normal and, to a lesser extent, neoplastic cells and tumor lesions (13, 14).```
Sample intent: method



Sample text:```Consistent with the notion that Derlin-1 acts in the retrotranslocation [21,22,26,48–51], misfolded proteins accumulate in the ER in derlin-1 mutants, indicative of disrupted ERAD.```
Sample intent: method



Sample text:```Endothelial cell cultures and capillary tubeswere generated as previously described (5, 11).```
Sample intent: background



Sample text:```Isolation of cardiomyocytes by standard enzymatic techniques and patch-clamp recordings were performed as described previously [42].```
Sample intent: background



Sample text:```b-catenin normally associates with E-cadherin and with a-catenin and thereby participates in the regulation of cell-to-cell adhesion (Vleminckx and Kemler 1999; Miyoshi and Takai 2005).```
Sample intent: method
Sample text:```Our results confi rm previous studies that reached the same conclusion (Boushey et al 1994; Vermeulen et al 2000; Till et al 2005; Marcucci et al 2005).```
Sample intent: result



Sample text:```Intriguingly, using MPRs-deficient MEF cells, mouse and human CLN5 have been shown to reach lysosomes in an MPRindependent manner [12].```
Sample intent: background



Sample text:```Because previous studies indicated that hexapeptides bind the junction core (29, 32), we tested two junctions, one with an AT-rich core and the other with a GC-rich core (Fig.```
Sample intent: result



Sample text:```Consistent with the notion that Derlin-1 acts in the retrotranslocation [21,22,26,48–51], misfolded proteins accumulate in the ER in derlin-1 mutants, indicative of disrupted ERAD.```
Sample intent: method



Sample text:```b-catenin normally associates with E-cadherin and with a-catenin and thereby participates in the regulation of cell-to-cell adhesion (Vleminckx and Kemler 1999; Miyoshi and Takai 2005).```
Sample intent: method



Sample text:```Endothelial cell cultures and capillary tubeswere generated as previously described (5, 11).```
Sample intent: background



Sample text:```Interestingly, MHC I and ERAP1 were coordinately expressed in normal and, to a lesser extent, neoplastic cells and tumor lesions (13, 14).```
Sample intent: method



Sample text:```For experiments measuring the effect of Sch-28080 and ouabain on pHi recovery in ICs, only basolateral Cl removal was used to differentiate IC types (15, 17).```
Sample intent: background



Sample text:```In line with our results, several previous studies demonstrated the anti-proliferative effect of PPARs agonists in several cell types [11-15].```
Sample intent: result



Sample text:```Our results further confirm previous findings on the differential regulatory mechanisms for Hoxa3 and Hoxb3 expression in r5 and r6 by kreisler (Manzanares et al., 1997, 1999a,b, 2001).```
Sample intent: result