Adenosine is known to increase coronary blood flow in patients with spontaneously visible collaterals, as a result of a reduction in the coronary collateral vascular resistance and peripheral vascular resistance of the recipient vessel (19,20).
Sample text:```Previous studies revealed that acylcarnitines were elevated early after APAP treatment and that their elevation was indicative of mitochondrial damage and dysfunction.(15,19) In the present study these observations were confirmed, as palmitoylcarnitine was elevated by toxic doses of APAP and maintained at normal levels (compared with untreated controls) by Wy-14,643 pretreatment.```
Sample intent: result
Sample text:```They demonstrated by in vitro and ex vivo studies that ATP1A1 mutants showed loss of pump activity and strongly reduced affinity for potassium, causing inappropriate depolarization of the cells (Beuschlein et al. 2013).```
Sample intent: method



Sample text:```The inter-relationships between K1, K1O and anticoagulants in dogs and humans have been previously investigated [25, 26].```
Sample intent: background



Sample text:```The admission assessment included determining the risk factors for cerebral infarction,13,14 clinical ischemic stroke category (National Institute of Neurological Disorders and Stroke1), and disease severity using the National Institutes of Health Stroke Scale.15
545
Abbreviations
AF = Atrial fibrillation
LAA = Left atrial appendage
LAA eV = LAA emptying flow velocity
LAD = Left atrial dimension
LAWV = LAA peak wall velocity
ROC = Receiver operating characteristic
SEC = Spontaneous echocardiographic contrast
TEE = Transesophageal echocardiography
TTE = Transthoracic echocardiography
We defined patients who had no histories of AF and no documented AF on continuous electrocardiographic monitoring during hospitalization as the sinus rhythm group, patients who had histories of AF before admission and/or continuously documented AF on continuous electrocardiographic monitoring during hospitalization as having chronic AF, and patients who had AF at admission and recovered to normal sinus rhythm during hospitalization and/or showed sinus rhythm at admission and documented transient AF on continuous electrocardiographic monitoring during hospitalization as having paroxysmal AF.```
Sample intent: background



Sample text:```These are in keeping with earlier studies on the steadystate response (2, 5, 8), step-function response, (3, 5, 6, 14) and sinusoidal response ( 14) of the various vascular beds.```
Sample intent: result



Sample text:```Trials were considered eligible if they satisfied the following inclusion criteria: (1) RCTs comparing RIC versus no conditioning in patients with AMI; (2) patients were treated by primary PCI or thrombolysis; (3) RIC was induced by intermittent limb ischemia–reperfusion; and (4) trials at least reported one of the following outcome measures, including enzymatic myocardial infarction size as assessed by serum peak creatine```
Sample intent: background
Sample text:```In line with previous results (3, 19, 20, 30), we found that during muscle contraction ERK1/2 phosphorylation was increased and was not affected by CaMKII inhibition.```
Sample intent: result



Sample text:```Further studies are still needed to exclude other purines such as ATP and related nucleotides, due to their significant release from bladder urothelium [8,9], and since ATP in some species can inhibit or relax the bladder [33–35].```
Sample intent: method



Sample text:```Catecholamines, and particularly DA, are believed to increase the efficiency of cortical processing by augmenting the signal-tonoise ratio or gain within cortical networks [4,37–39].```
Sample intent: method



Sample text:```These results corroborate studies that have demonstrated an enhancement of other types of reward with Dz-specific agonists [6,17,21,52].```
Sample intent: result



Sample text:```In this study, the proposed method is based on atrial activity extraction using an echo state network (ESN) re­ cently introduced as a unified solution to the problem of QRST cancellation in the presence of substantial variation in beat morphology and/or occasional ectopic beats [7].```
Sample intent: background



Sample text:```In line with our results, several previous studies demonstrated the anti-proliferative effect of PPARs agonists in several cell types [11-15].```
Sample intent: result



Sample text:```Basal ATP concentrations in the bulk medium of unstimulated CFs are low, but the concentrations of released nucleotides near the cell surface may be sufficient to activate adjacent P2Y receptors and contribute to basal signaling (48, 49).```
Sample intent: method



Sample text:```Trials were considered eligible if they satisfied the following inclusion criteria: (1) RCTs comparing RIC versus no conditioning in patients with AMI; (2) patients were treated by primary PCI or thrombolysis; (3) RIC was induced by intermittent limb ischemia–reperfusion; and (4) trials at least reported one of the following outcome measures, including enzymatic myocardial infarction size as assessed by serum peak creatine```
Sample intent: background



Sample text:```Previous studies revealed that acylcarnitines were elevated early after APAP treatment and that their elevation was indicative of mitochondrial damage and dysfunction.(15,19) In the present study these observations were confirmed, as palmitoylcarnitine was elevated by toxic doses of APAP and maintained at normal levels (compared with untreated controls) by Wy-14,643 pretreatment.```
Sample intent: result



Sample text:```…has been described variously, as intramural coronary artery, mural coronary artery, coronary artery overbridging, and most commonly myocardial bridging (MB) (Canyigit et al. 2009; Lazoura et al. 2010; Mohlenkamp et al. 2002; Montaudon et al. 2007; Ozbag et al. 2002; Ozbag and Kervancioglu 2004).```
Sample intent: method