ntractions such as stimulants like caffeine, chocolate, low-potassium states, alcohol, and age and conduction systems, anti-arrhythmics, street drugs, lack of oxygen at high altitudes, mitral valve prolapse, and so on–the list is long. Initial interventions include eliminating the cause if possible, or normalizing electrolytes such as potassium. potassium.
Most cardiologists do not treat premature ventricular contractions (PVC’s) unless they are happening frequently and on a regular basis, like more than six times a minute in couplets, triplets, or in short runs. . By stabilizing the membrane of the electrical conduction system, Q10 can make it harder for arrhythmias to occur in the first place.
The complexity of the cardiac energy system is clear. The bottom line is that patients need to be aware of the vital importance of energy metabolism in cardiac disease, both from the standpoint of accelerating adequate ATP turnover, as well as reserve, and maintaining the size of the energy pool itself.
The triad of cardiac supplements, including Q10 magnesium , L-carnitine, pqq and d-ribose are critical for establishing the energy health of the heart and to assist the ailing heart in its quest to supply the energy it needs.
In summary, metabolic cardiology is a fascinating new concept.
Attention is given to the energy demands of the diseased heart, and it is often missed in conventional cardiology practice. Disease states such as heart disease, heart failure, or cardiomyopathy are associated with low cardiac energy production.
Optimal cardiovascular function is dependent on maintaining adequate levels and energy reserves. Metabolic cardiology highlights the importance of sustaining key enzymatic and biochemical reactions that revitalize the energy charge in failing hearts. Efforts to support the metabolic needs of the heart have been well documented, yet are not known to most cardiologists. These therapies have an excellent safety profile, and are a key factor in light of the finding that the fourth leading cause of death in the United States is properly-prescribed medications.
The importance of supporting energy production in the heart cells and preservation of the mitochondria is essential. The heart contains more energy per gram than any other organ, and the chemical energy that fuels the heart comes primarily from ATP; ATP reserves must be maintained. In diastolic dysfunction, which is an early sign of heart failure, despite the presence of normal systolic function and preserved ejection fraction, high concentrations of ATP are needed and can reverse this early heart disease. In failing hearts, such as heart failure, these energetic changes become even more profound as the left ventricle proceeds from hypertrophy to dilatation.
Cardiac energetics also provides important prognostic information in the patients for heart failure, in determining the heart contractility reserve. It is especially necessary prior to surgery and/or angioplasty to protect the patient.
Cardiovascular function depends on the operational capacity of the heart cell to generate energy, to expand and contract. Insufficient myocardial energy contributes significantly to congestive heart failure. Literally, heart failure is energy-starved heart. Although there may be several causes of myocardial dysfunction, the energy deficiency in the heart plays a significant role.
It is no longer enough that physicians focus on fluid retention aspects of pump failure, giving a diuretic. Diuretic therapies target the kidney indirectly, to relieve the heart failure without addressing the root cause. Metabolic solutions, on the other hand, treat the heart muscle directly. Metabolic cardiology supports the biochemical interventions that can be employed to directly improve energy, and therefore energy metabolism in the heart cells; d-ribose, PQQ, co-enzyme Q10, carnitine, and magnesium all promote cardiac energy metabolism and help normalize myocardial energy reserve. All of these interventions positively impact the cardiac systolic and diastolic function. Acknowledging this 