otype in the non-pathological SVZ during maturing by accumulating dense systems and long functions abundant with intermediate filaments. These features resemble the hypocellular difference level from the adult individual SVZ where neurogenic capability and neuroblast migration can be decreased (Guerrero-Cazares et al. 2011 Quinones-Hinojosa et al. 2006 Sanai et al. 2011 2004 Furthermore we discovered that the ependymal level from the aged SVZ provided cells coexpressing GFAP and S100 markers. This selecting was previously defined in older mice recommending that astrocytes could transform into ependymal cells to mediate ependymal fix (Luo et al. 2008 Nevertheless our outcomes indicate these GFAP/S100 positive cells correspond certainly to ependymal cells.