Negatively regulates the G2-M phase transition, possibly by promoting cyclin B1/CCNB1 and CDK1 proteasomal degradation and thereby preventing their accumulation during interphase.
Subcellular locations: Cytoplasm, Nucleus
A higher concentration is observed in the cytoplasm compared to the nucleus.
Seems to be expressed in prostate and less abundantly in adult brain, fetal liver, and fetal kidney.