AMPK substrate important for exercise capacity and skeletal muscle function. Required for normal contraction-induced signaling.
(Microbial infection) Upon Epstein-Barr virus (EBV) infection, suppresses viral BZLF1 expression and subsequent EBV reactivation by interacting with JUN and inhibiting its transcriptional activitor activity on BZLF1 Z promoter.
Subcellular locations: Nucleus
Expressed in skeletal muscle.