Disturbances of phenyalanine metabolism have a relatively high incidence in Slavonic populations in comparison with other populations (Thalhammer et al., 1975). The aim of this study is to find out whether the incidence of cystinuria (McKusick 22010) among newborns, normals or mentally retarded populations also differs from the findings of Levy (1973), Smith and Procopis (1975) and Moore et al. (1972).
GUTHRIE'S1 2 3 recently introduced bacterial inhibition assay provides a simple and practical mass screening method for phenylketonuria. Although phenylketonuria is the best known it is by no means the only inborn error of amino acid metabolism characterized by the accumulation of large amounts of one or more amino acids in the blood. Ten such disorders have been reported in the literature.4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 These are listed in Table 1, together with their most striking clinical features. Several others are known to be under investigation. All except tyrosinosis have been associated with mental retardation in some if not all persons affected. This report describes . . .
Every function of n inputs can be efficiently computed by a complete network of n processors in such a way that: If no faults occur, no set of size t n /2 of players gets any additional information (other than the function value), Even if Byzantine faults are allowed, no set of size t n /3 can either disrupt the computation or get additional information. Furthermore, the above bounds on t are tight!