The second study showed that mouse models expressing the human form of mutant ATPase inhibitory factor 1 (hIF1), which causes a sustained inhibition of ATP synthase, were more resilient to neuronal death after excitotoxic damage.
ການສຶກສາຄັ້ງທີສອງສະແດງໃຫ້ເຫັນວ່າຕົວແບບຫນູສະແດງຮູບແບບຂອງມະນຸດຂອງປັດໄຈ inhibitory mutant ATPase 1 (hIF1), ເຊິ່ງເຮັດໃຫ້ການຍັບຍັ້ງ ATP synthase ສະຫນັບສະຫນູນຢ່າງຕໍ່ເນື່ອງ, ມີຄວາມທົນທານຕໍ່ການເສຍຊີວິດ neuronal ຫຼັງຈາກຄວາມເສຍຫາຍ excitotoxic.