It authorizes the holder to teach in the area of specialization listed on the credential in the following settings: special day classes, special schools, home/hospital settings, correctional facilities, nonpublic schools and agencies, and resource rooms.
14â16 The neuronal loss in Chagas heart disease has been postulatedto occur predominantly during the acute phase of the infec-tion, with participation of 3 mechanisms: direct parasitism ofneurons, degeneration caused by periganglionic inflamma-tion, and antineuronal autoimmune reaction.nly inthe late 1940s, through improved specific serological tests todetect antibodies against T cruzi, the clinical syndromes ofmegaesophagus, megacolon, and heart failure, by then ofunknown cause, were ascribed to Chagas disease, and the firstreport of cardiac neuronal damage appeared in the literature.
Stanley B. Prusiner. Bovine spongiform encephalopathy (BSE) and human Creutzfeldt-Jakob disease (CJD) are among the most notable central nervoussystem degenerative disorders caused by prions. CJD may presentas a sporadic, genetic, or infectious illness.rions are transmissibleparticles that are devoid of nucleic acid and seem to be composedexclusively of a modified protein (PrP Sc). The normal, cellular prion protein (PrP C) is converted into PrP Sc through a posttranslational process during which it acquiresa high-sheet content.