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Central nervous system oxygen toxicity manifests as symptoms such as visual changes (especially tunnel vision), ringing in the ears (tinnitus), nausea, twitching (especially of the face), irritability (personality changes, anxiety, confusion, etc.), and dizziness.entral nervous system oxygen toxicity manifests as symptoms such as visual changes (especially tunnel vision), ringing in the ears (tinnitus), nausea, twitching (especially of the face), irritability (personality changes, anxiety, confusion, etc.), and dizziness.
Nine TMA patients had received CNI, 3 of them concomitantly received an mTOR-inhibitor. CNI toxicity is implicated in most patients with renal failure after transplant of other organs and may play a role in the development of TMA, the most serious complication.However, decreased renal function should not be routinely ascribed to CNI.enal failure in these patients has been found to be a major risk factor for patient survival, mainly after heart and lung transplantation. Calcineurin-inhibitor (CNI) toxicity is a well-known consequence of rejection prevention therapy in renal and nonrenal transplantation.