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The main target organs where parathyroid hormone exerts its effects are the bones and the kidneys. When needed, parathyroid hormone is released by the parathyroid glands into the blood and causes the bones to release calcium and increase levels in the bloodstream.
Cell-surface receptors are optimal targets for exploitation in ligand-directed systems for the targeted delivery of therapeutics to tumors. Here, we introduce a strategy for capitalizing on the overexpression of one such receptor, somatostatin receptor type 2 (SSTR2), in neuroendocrine tumors (NETs) of the pancreas. We will prepare phage-based gene delivery constructs targeted by octreotide, a somatostatin peptide-analogue known to selectively bind SSTR2 in pancreatic NETs.