Biochemical estimation revealed that APE administration increased lipid peroxidation by two folds accompanied by a two-fold decline in the glutathione contents at 8 h post-APE treatment.
APE treatment reduced the activities of glutathione peroxidase, glutathione-S-transferase, superoxide dismutase, and catalase at 8 h by 2.1 , 2.3 , 2, and 3.5 folds, respectively.