Severe ocular pain, blepharospasm, reduced visual acuity
Altered ocular pH (normal = 7.0-7.2)
Appearance
Conjunctival injection OR blnnching
Chemosis, hemorrhage, epithelial defects
Corneal loss OR edema
Perilimbal ischemia (white ring around iris)
Concerning due to co-location of corneal stem cell layer (re-epitheliazation relies on migration of limbal stem cells)
Conjunctival injection OR blnnching
Chemosis, hemorrhage, epithelial defects
Corneal loss OR edema
Concerning due to co-location of corneal stem cell layer (re-epitheliazation relies on migration of limbal stem cells)
Rapid penetration through cornea and anterior chamber
Causes liquefaction necrosis from hydroxyl ions combining with fatty acids
Elevated IOP may be seen in alkali injury
Acute injury liquefaction necrosis and ischemia which can be graded on degree of limbal and corneal involvement
Early sequelae corneal edema and elevated IOP
Later injury after one week - corneal breakdown, ulceration, perforation because of collagenase activity exceeds synthesis
Injury related to length of exposure and concentration of chemical
Causes coagulation necrosis and protein precipitation thus limits depth of penetration
Delayed complications corneal and conjunctival scarring, vascularization, glaucoma, uveitis
Most exposures produce only simple irritation
Caustic keratoconjunctivitis
Caustic ingestion
Epidemic keratoconjunctivitis
Caustic eye exposure 
Gonorrheal conjunctivitis
Caustic dermal burn
Allergic conjunctivitis
Airbag-related burns