Intravascular hemolysis caused by increased sensitivity of RBCs to hemolytic action of complement
Abnormal PIG-A gene > defect in GPI-linked anchor >  partial/complete absence of GPI-linked proteins (mainly CD55 and CD59) > increased sensitivity of RBCs to hemolytic action of complement
Hemolysis precipitated by stresses that activate complement (eg infection)<unk>  hemolysis attributed to intestinal absorption of lipopolysaccharide (strong activator of complement) at night<unk>  hemolysis attributed to intestinal absorption of lipopolysaccharide (strong activator of complement) at night
Paroxysmal nocturnal hemoglobinuria
Disseminated Intravascular Coagulation 
Hereditary spherocytosis
Thrombotic Thrombocytopenic Purpura 
Myelophthistic anemia
Hemolytic Uremic Syndrome 
Thalassemia
HELLP syndrome