The Pt presents with _ likely due to a corneal abrasion seen on fluorescein staining of eye. The Pt is otherwise well-appearing without evidence of retained foreign body, corneal ulcer_, globe rupture, or superimposed infection. Prescribed antibiotics and instructed the Pt to follow up closely with ophthalmology and avoid wearing contacts_.
Patient presents with Scleral injection. No recent eye trauma or suspected microtrauma (dust, sand, etc). Negative Seidel sign, no sign of corneal abrasion/ulcer. No history of discharge so less likely bacterial or viral conjunctivitis. No significant photophobia. IOP is _ so doubt acute angle closure glaucoma. Given history and exam I have low suspicion for globe rupture, uveitis, HSV keratitis, Endopthalmitist, Foreign Body. Patient likely has allergic conjunctivitis and was prescribed _.
Presentation consistent with subconjunctival hemorrhage.
Given history and exam I have low suspicion for corneal abrasion or ulcer, globe rupture, uveitis, HSV keratitis, Endopthalmitis, Retinal Detachment, Angle Closure Glaucoma, Foreign Body, hyphema.
Given history of flashers and floaters with acute visual acuity loss and ocular ultrasound findings, presentation is concerning for Retinal Detachment vs Vitreous Hemorrhage vs Posterior Vitreous Detachment. Given vision loss is painless I have low suspicion for normally painful syndromes such as Corneal Abrasion/Ulcer, Complex Migraine, Globe Rupture, Acute Angle Glaucoma, Uveitis, Endopthalmitis, Iritis. Additionally, given presentation I have low suspicion for other painless syndromes such as Amaurosis Fugax, CRAO, CRVO, or Stroke.
Given history of painless vision loss and exam with afferent pupillary defect and significantly reduced visual acuity presentation is concerning for CRAO vs CRVO. Vision is unilateral with no other focal neuro deficits so doubt stroke, patient exam and history make retinal detachment, vitreous hemorrhage, posterior vitreous detachment lower on<unk> ial. Given painless vision loss low suspicion for normally painful syndromes such as corneal abrasion/ulcer, complex migraine, globe rupture, acute angle closure glaucoma, optic neuritis, temporal arteritis, uveitis, endophthalmitis, iritis.
Patient presents with nontraumatic painful, unilateral vision loss for which the initial<unk> ial is optic neuritis, temporal arteritis, acute angle closure glaucoma, endophthalmitis, and uveitis. Given patient had increased IOP and concerning ocular exam likely cause is acute angle closure glaucoma. No foreign body sensation or FB on exam so doubt corneal abrasion/ulcer. No recent eye trauma or suspected microtrauma with no signs of inflammation or injection with no significant photophobia so doubt globe rupture, uveitis, endophthalmitis. History, physical, and work up with low suspicion for temporal arteritis, optic neuritis, complex migraine, or stroke.
Patient presents with nontraumatic painful, unilateral vision loss for which the initial<unk> ial is optic neuritis, temporal arteritis, acute angle closure glaucoma, endophthalmitis, and uveitis. Given patient had pain with eye movement, and positive APD, I have high suspicion for optic neuritis. Normal IOP so doubt acute angle closure glaucoma. No foreign body sensation or FB on exam so doubt corneal abrasion/ulcer. No recent eye trauma or suspected microtrauma with no signs of inflammation or injection with no significant photophobia so doubt globe rupture, uveitis, endophthalmitis. History, physical, and work up with low suspicion for temporal arteritis, complex migraine, or stroke.
MDM for different chief complaints
Critical care documentation
Combative patient
Wilderness preparedness
ACEP clinical policies
Expedited partner therapy
SCVMC:CUSTODY CARE
FALLS protocol