Urine pregnancy
CBC
Chemistry
LFTs
Hepatocyte injury: AST, ALT, alk phos
Hepatocyte catabolic activity: Bilirubin
Coags
Hepatocyte synthetic function
Albumin
Hepatocyte synthetic function
Ammonia
Hepatocyte catabolic activity
Acute hepatitis panel
Lipase
Urinalysis
?US vs. CT
?Retic count
?Haptoglobin/LDH
?APAP/ASA/Utox/ETOH
Hepatocyte injury: AST, ALT, alk phos
Hepatocyte catabolic activity: Bilirubin
Hepatocyte synthetic function
Hepatocyte synthetic function
Hepatocyte catabolic activity
Transaminases  in hundreds associated with mild injury; thousands suggests extensive injury
Elevations <5x normal typical of alcoholic liver disease
AST:ALT ratio > 2 common in acute alcoholic hepatitis (alcohol stimulates AST production)
May be normal in end-stage liver failure
ALT more specific marker of hepatocyte injury than AST
Mild to moderate elevations accompany virtually all hepatobiliary disease
Elevations > 4x normal suggest cholestasis
Elevation in setting of hepatitis suggestive of alcoholic etiology
Moderate elevations are seen in all hepatocellular disorders and cirrhosis
Hemolysis results in elevation of LDH and unconjugated bili
Elevation does NOT correlate with acute worsening of hepatic function in cirrhotic patient
Serves as marker of generalized decline than as diagnostic tool or therapeutic end point
Marker of synthetic function
Correlation between PT prolongation and clinical outcome in fulminant liver disease
Marker of synthetic function
Half-life is 3 weeks so less useful than PT in evaluating fulminant liver disease
Low levels also seen in malnutrition
Half-life is 3 weeks so less useful than PT in evaluating fulminant liver disease
Jaundice
Hemolytic
Hematuria 
G6PD
Hematuria
Drug related
Nausea and vomiting
Autoimmune hemolytic anemia