Risk is highest within first 24 hours (2.5-4%), particularly within first 6 hours
Usually diagnosed by CT after acute deterioration in neuro status
Only aneurysm treatment is effective in preventing rebleeding
Leading cause of death and disability after rupture
Typically begins no earlier than day three after hemorrhage
Characterized by decline in neuro status
Aggressive treatment can only be started after aneurysm has been treated
treatment for symptomatic vasospasm: Triple-H therapy (hemodilution + induced hypertension (pressors) + hypervolemia), balloon angioplasty, or intra-arterial vasodilators[citation needed]
Studies have not provided strong evidence of benefit Triple-H therapy[citation needed]
treatment for symptomatic vasospasm: Triple-H therapy (hemodilution + induced hypertension (pressors) + hypervolemia), balloon angioplasty, or intra-arterial vasodilators[citation needed]
Studies have not provided strong evidence of benefit Triple-H therapy[citation needed]
Most likely related to the release of catecholamines due to hypoperfusion of hypothalamus
Ischemia
Elevated troponin (20-40% of cases)
ST segment depression
Rhythm disturbances
Torsades, A-fib/flutter
QT prolongation
Deep, symmetric TWI
Prominent U waves
Elevated troponin (20-40% of cases)
ST segment depression
Torsades, A-fib/flutter
Consider ventricular drain placement for deteriorating LOC + no improvement within 24hr
Hyponatremia is seen in 10%-40% of the patients with<unk>  who are admitted to the ICU.
Cerebral Salt Wasting and SIADH are the two most common causes
Subarachnoid hemorrhage
Intra-axial
Subdural hemorrhage
Hemorrhagic stroke 
Ischemic stroke
Traumatic intracerebral hemorrhage
Hemorrhagic stroke
Extra-axial