<unk>  is a clinical entity which is manifested by progressive motor and sensory changes in: * legs * incontinence * back of leg pain * scoliosis In order to understand the pathophysiology that is involved in a<unk><unk>  cord, the reduction/oxidation ratio has to be used in vivo of cytochrome alpha and alpha 3 to signal the oxidative metabolic functioning in humans. Studies have found that marked metabolic and electrophysiological susceptibility to hypoxic stress to the lumbar and sacral portion of the<unk>  cord under traction with various weights. Similar effects were found in redox behavior of<unk><unk>  cord during the surgical procedures to repair it. This can be due to impairment of mitochondrial oxidative metabolism under constant or intermittent stretching. The act of prolonged stretching can lead to structural damage to the neural perikarya and eventually the axons in neurons. The untethering process can improve the oxidative metabolism and can help to repair injured neurons.
Tethered spinal cord syndrome
Posterior cord syndrome
Say–Meyer syndrome
Central cord syndrome
Madras motor neuron disease
Upper motor neuron syndrome
Nicolau–Balus syndrome
Syringomyelia