<unk>  can be asymptomatic in many patients who are carriers. Female carriers have been found to more frequently experience symptoms. Enzyme activity was shown to be reduced by 35–65% depending on the severity of the deficiency. Abnormal red blood cell breakdown (hemolysis) in<unk> PGD deficiency can be symptomatic in a number of ways, including the following: * Neonatal jaundice, * Possibility of leading to kernicterus due to a hyperbilirubinemia * Hemolytic crises in response to: * Illness and infections * Certain drugs * Certain foods * Certain chemicals * In extreme cases, kidney failure
6-phosphogluconate dehydrogenase deficiency
Glutaminase deficiency
Ribose-5-phosphate isomerase deficiency
Pyruvate kinase deficiency
Malonyl-CoA decarboxylase deficiency
Pyruvate carboxylase deficiency
3-Methylcrotonyl-CoA carboxylase deficiency
D-glycerate dehydrogenase deficiency