Not much is understood about how<unk>  produces its anti-anxiety effects, but rat studies have shown that the possible mechanism of action is by way of increased turnover of cathecholamines. In addition to serotonin it has also been shown to decrease epinephrine, dopamine, and norepinephrine turnover by antagonizing their receptors. When given intravenously in doses of 20–40 mg there are no significant differences in efficacy. Oral doses exceeding 10 mg three times daily do not increase the effects of the drug. Assuming serotonin postsynaptic antagonism is the main mechanism by which<unk>  carries out its effects, studies have shown it to have a half maximal inhibitory concentration(IC50) value of 115 mM at the serotonin receptor.
Benzoctamine
Tesofensine
Bethanechol
Theobromine
Pseudoephedrine
L-Norpseudoephedrine
Ephedrine
Scopolamine