Potential explanations include # Atypical immune recognition of autologous skin antigens # Stimulation of normal T cells by changing skin constituents # Lower threshold for skin irritation # Spreading of infectious antigens causing a secondary response # Hematogenous dissemination of cytokines from the primary site of inflammation
Id reaction
MChip
DirectHit
Endotype
Cue reactivity
Fernandez reaction
Agar dilution
Next-generation matrix