Pharmacodynamics<unk>  shows high progestogenic activity. With sublingual administration in women, it has at least 150 times the potency of sublingual progesterone and 50 times the potency of sublingual ethisterone. It also has 10 times the potency of injected progesterone via this route. The oral potency of<unk>  in terms of endometrial transformation is similar to that of norethisterone. It has been reported to inhibit ovulation in women. In addition to its progestogenic activity,<unk>  has anabolic and androgenic activity and can produce effects associated with this activity. It has a high ratio of anabolic to androgenic activity. The anabolic potency of<unk>  is similar to that of norethandrolone and is much greater than that of nandrolone or metandienone. It is also greater than that of ethylestrenol.<unk>  has been found to increase nitrogen retention, a measure of anabolic effect, at a dosage of 30 mg/day. Analogously to nandrolone and norethandrolone, 5α-dihydronormethandrone, the 5α-reduced metabolite of<unk> , shows reduced affinity for the androgen receptor relative to<unk> . Its affinity for the androgen receptor is specifically about 33 to 60% of that of<unk> .<unk>  has estrogenic activity via aromatization into methylestradiol. Pharmacokinetics<unk>  is metabolized by aromatase into methylestradiol in small quantities, similarly to methyltestosterone and metandienone. The metabolites of<unk>  have not been well-studied, but 5α-dihydronormethandrone is a likely metabolite formed by 5α-reductase. The pharmacokinetics of<unk>  have been reviewed.
Normethandrone
Chloromethylandrostenediol
Desoxymethyltestosterone
Algestone acetonide
Ethyltestosterone
Androstanolone propionate
Cloxotestosterone acetate
Vinyltestosterone