In regards to mechanism, there are three genes: GP1BA, GP1BB and GP9 that are involved (due to mutations). These mutations do not allow the GPIb-IX-V complex to bind to the von Willebrand factor, which in turn is what would help platelets adhere to a site of injury which eventually helps stop bleeding.
Bernard–Soulier syndrome
Fryns-Aftimos syndrome
Harris platelet syndrome
Ehlers–Danlos syndromes
Quebec platelet disorder
Keutel syndrome
Von Willebrand disease
Flammer syndrome