The pathophysiology of a tremor is not completely understood, but what is understood can be explained by two principles. First, the complete absence of structural changes in the neuronal loops results in hyperexcitability and rhythmic movement. Second, is the permanent structural neurodegeneration. The corticostriatothalamocortical hap is a neuronal network that is associated with the integration of different muscle groups. It activates complex movement programs and guarantees continuous movement that cannot be terminated by small external influences. Another important circuit involving<unk>  is the Guillain-Mollaret Triangle, which includes the red nucleus, the inferior olivary nucleus (ION), and the dentate nucleus. This circuit controls voluntary precision movements. The ION is the most important of these components in the formation of tremors. In normal/healthy individuals' ION neurons, calcium channels regulate normal oscillatory depolarizations. This pacemaker affects processing and coordination of cerebellar precision movements and motor learning. Damages, both physical and chemical, to the ION affects the Guillain-Mollaret Triangle and leads to tremors.
Holmes tremor
Intention tremor
Focal dystonia
Myoclonic dystonia
Coprographia
Periodic paralysis
Myopathic gait
Parkinsonian gait