Some risk factors associated with the development of<unk> s are modifiable.<unk> s are more common in obese women.<unk> s are dependent on estrogen and progesterone to grow and therefore relevant only during the reproductive years. Diet Diets high in fruits and vegetables tend to lower the risk of developing<unk> s. Fibers, vitamin A, C and E, phytoestrogens, carotenoids, meat, fish, and dairy products are of unclear effect. Normal dietary levels of vitamin D may reduce the risk of developing<unk> s. Genetics Fifty percent of<unk> s demonstrate a genetic abnormality. Often a translocation is found on some chromosomes.<unk> s are partly genetic. If a mother had<unk> s, risk in the daughter is about three times higher than average. Black women have a 3-9 times increased chance of developing<unk> s than white women. Only a few specific genes or cytogenetic deviations are associated with<unk> s. 80-85% of<unk> s have a mutation in the mediator complex subunit 12 (MED12) gene. Familial leiomyomata A syndrome (Reed's syndrome) that causes<unk>  leiomyomata along with cutaneous leiomyomata and renal cell cancer has been reported. This is associated with a mutation in the gene that produces the enzyme fumarate hydratase, located on the long arm of chromosome 1 (1q42.3-43). Inheritance is autosomal dominant.
Uterine fibroid
Uterine inversion
Leiomyoma
Cervical pregnancy
Endometrioid tumor
Ovarian pregnancy
Pelvic lipomatosis
Endometriosis