Diabetes is a chronic disease, i.e. it cannot be cured, but symptoms and complications can be minimized with proper treatment. Diabetes can lead to elevated blood sugar levels, which in turn can lead to damage to the heart, blood vessels, kidneys, eyes and nerves. There are very few studies on how to treat<unk> , specifically, which is probably due to difficulties in classifying and diagnosing the disease.<unk>  patients often do not need insulin treatment immediately after being diagnosed because their own insulin production decreases more slowly than T1D patients, but in the long run they will need it. About 80% of all<unk>  patients initially misdiagnosed with type 2 (and who have GAD antibodies) will become insulin-dependent within 3 to 15 years (according to differing<unk>  sources). The treatment for Type 1 diabetes/LADA is exogenous insulin to control glucose levels, prevent further destruction of residual beta cells, reduce the possibility of diabetic complications, and prevent death from diabetic ketoacidosis (DKA). Although<unk>  may appear to initially respond to similar treatment (lifestyle and medications) as type 2 diabetes, it will not halt or slow the progression of beta cell destruction, and people with<unk>  will eventually become insulin-dependent. People with<unk>  have insulin resistance similar to long-term type 1 diabetes; some studies showed that people with<unk>  have less insulin resistance, compared with those with type 2 diabetes; however, others have not found a difference. A Cochrane systematic review from 2011 showed that treatment with Sulphonylurea did not improve control of glucose levels more than insulin at 3 nor 12 months of treatment. This same review actually found evidence that treatment with Sulphonylurea could lead to earlier insulin dependence, with 30% of cases requiring insulin at 2 years. When studies measured fasting C-peptide, no intervention influenced its concentration, but insulin maintained concentration better compared to Sulphonylurea. The authors also examined a study utilizing Glutamic Acid Decarboxylase formulated with aluminium hydroxide (GAD65), which showed improvements in C-peptide levels that were maintained for 5 years. Vitamin D with insulin also demonstrated steady fasting C-peptide levels in the vitamin group, with the same levels declining in the insulin-only group at a 12-month follow-up. One study examining the effects of Chinese remedies on fasting C-peptide on a 3 month follow-up did not show a difference compared to insulin alone. Still, it is important to highlight that the studies available to be included in this review presented considerable flaws in quality and design.
Latent autoimmune diabetes in adults
Hepatitis C and HIV coinfection
Lifestyle causes of type 2 diabetes
Smoking in South Korea
Type 3c diabetes
Steroid use in Bollywood
Tuberculosis in relation to HIV
Genetic causes of type 2 diabetes