The exact causes of<unk>  are unknown. However, it is pathologically connected to the following conditions: * Endogenous diseases: schizophrenia (especially catatonic schizophrenia). * Exogenous organic diseases: infectious (encephalitis), intoxication (by hallucinogens, for example), traumatic brain injury, epilepsy, delirium tremens. Most often, this pathology is noted in schizophrenia<unk>  catatonia), but sometimes it is described with organic brain lesions and intoxications. There is no self-consciousness disorder in<unk>  with exogenous diseases. There are no catatonia phenomena, and the syndrome ends more rapidly.<unk>  in exogenous organic diseases is evidence of a severe deterioration in the patient's response to the organic disease, and its transition to amential syndrome or mental fog is an even more unfavorable symptom. Exogenous organic<unk>  is different from schizophrenic<unk> . In most cases, the described exogenous psychoses occupy an intermediate position between delirium and<unk> , with a rapid and dynamic development of symptoms and an increase in psychotic symptoms occurring during the evening (as is typical for delirium),. Clinical resolution of such psychoses after deep sleep also points to a case of delirium. All of this allows us to relate these diagnostic categories to have the variants of delirium ("fantastic delirium"). Hallucinogens (LSD, hashish, ketamine) and hormonal preparations (for example, corticosteroids) may cause exogenous<unk> .
Oneiroid syndrome
Galloway Mowat syndrome
Schimmelpenning syndrome
Setleis syndrome
Carey Fineman Ziter syndrome
Omenn syndrome
Leschke syndrome
Mungan syndrome