<unk> -induced<unk>  is caused by over-activation of the body's natural clotting system. This decreases clotting factor availability, thus impairing hemostasis. The exact mechanism by which this is accomplished varies greatly<unk>  to<unk> . Some<unk> s cause something akin to disseminated intravascular coagulation, while others lack the microthrombi characteristic in this disorder. Procoagulant metalloproteinases in the<unk>  promote a<unk><unk>  by activating prothrombin, factor V, factor X or thrombin-like enzymes (fibrinogenases).<unk>  induced<unk>  may also be accompanied by a thrombotic microangiopathy consisting of thrombocytopenia (low platelets), microangiopathic hemolytic anemia and acute kidney injury.
Venom-induced consumption coagulopathy
Vitamin K deficiency bleeding
Vitamin K deficiency
Heparin-induced thrombocytopenia
Sticky platelet syndrome
Pseudo gray platelet syndrome
Disseminated intravascular coagulation
Factor VII deficiency