The precise cause of<unk>  is unknown, but several mechanisms are thought to contribute. One may be a decrease in the level of circulating factor X, a clotting factor necessary for coagulation. The proposed mechanism for this decrease in factor X is that circulating<unk>  fibrils bind and inactivate factor X. Another contributing factor may be enhanced fibrinolysis, the breakdown of clots. Subendothelial deposits of<unk>  may weaken blood vessels and lead to the extravasation of blood.<unk>  deposits in the gastrointestinal tract and liver may also play a role in the development of<unk> .
Amyloid purpura
Roussy–Lévy syndrome
Solar purpura
Drug-induced purpura
Golfer's vasculitis
Obstructive purpura
Henoch–Schönlein purpura
Venom-induced consumption coagulopathy