One of the important manifestations of acute rheumatic fever,<unk>  is similarly caused by an autoimmune response following infection by group A β-hemolytic streptococci. Two cross reactive streptococcal antigens have been identified, the M protein and N-acetyl-beta-D-glucosamine, whereby infection leads to autoantibodies being produced against host tissues (molecular mimicry) causing a variety of streptococcal related diseases including<unk>  but also rheumatic heart disease and nephritic syndrome. Autoantibodies against basal ganglia proteins have been found in<unk>  but these are non-specific. Dopamine receptor autoantibodies have been reported to correlate with clinical symptoms. Whether these antibodies represent an epi-phenomenon or are pathogenic, remains to be proven.
Sydenham's chorea
Huntington's disease
Benign hereditary chorea
Parkinson-plus syndrome
Wernicke–Korsakoff syndrome
Huntington's disease-like syndrome
X-linked dystonia parkinsonism
Stratton Parker syndrome