Stellate cell The pathophysiology of portal hypertension is indicated by increased vascular resistance via different causes; additionally, hepatic stellate cells and myofibroblasts are activated. Increased endogenous vasodilators in turn promote more blood flow in the portal veins. Nitric oxide is an endogenous vasodilator and it regulates intrahepatic vascular tone (it is produced from L-arginine). Nitric oxide inhibition has been shown in some studies to increase portal hypertension and hepatic response to norepinephrine.
complications of Primary biliary cholangitis
complications of Gallstone
complications of Amoebiasis
complications of Hepatitis A
complications of Hepatitis E
complications of Cholera
medical cause of Pancreatitis
complications of Hepatitis