<unk>  is rare, but usually occurs in women who have had multiple pregnancies or in people who have undergone previous<unk> s. The precise mechanism leading to<unk>  is unknown, but it most commonly occurs in individuals whose platelets lack the HPA-1a antigen (old name: PLA1). The patient develops antibodies to the HPA-1a antigen leading to platelet destruction. In some cases, HPA-5b has also been implicated. It is unclear why alloantibodies attack the patient's own, as well as any transfused allogeneic random donor platelets. Probable explanation for this is that the recipient's platelets acquire the phenotype of donor's platelet by binding of the soluble antigens from the donor onto the recipient's platelet.
Post-transfusion purpura
Nonthrombocytopenic purpura
Transfusion-dependent anemia
Venom-induced consumption coagulopathy
Acute hemolytic transfusion reaction
Heparin-induced thrombocytopenia
Hyperviscosity syndrome
Thrombocytopenic purpura