Other conditions such as Liddle's<unk>  can mimic the clinical features of AME, so diagnosis can be made by calculating the ratio of free urinary cortisol to free urinary cortisone. Since AME patients create less cortisone, the ratio will much be higher than non-affected patients. Alternatively, one could differentiate between the two<unk> s by administering a potassium-sparing diuretic. Patients with Liddle's<unk>  will only respond to a diuretic that binds the ENaC channel, whereas those with AME will respond to a diuretic that binds to ENaC or the<unk>  receptor.
Apparent mineralocorticoid excess syndrome
Glucocorticoid remediable aldosteronism
Syndrome of inappropriate antidiuretic hormone secretion
Cerebral salt-wasting syndrome
Familial hyperaldosteronism
Primary aldosteronism
Pseudo-Cushing's syndrome
Pseudohyperaldosteronism