<unk>  must be distinguished from stress hyperglycemia, hyperglycemia due to excessive intravenous glucose, or new-onset<unk>  of another type. Because it is not unusual for<unk>  treatment to precipitate type 1 or type 2<unk>  in a person who is already in the process of developing it, it is not always possible to determine whether apparent<unk>  will be permanent or will go away when the<unk> s are finished. More commonly undiagnosed cases of type 2<unk>  are brought to clinical attention with corticosteroid treatment because subclinical hyperglycemia worsens and becomes symptomatic. Generally,<unk>  without preexisting type 2<unk>  will resolve upon termination of corticosteroid administration.<unk>  does not occur with other<unk>  hormones, such as anabolic<unk> s or sex<unk> s because these other categories of<unk> s have actually shown to have positive effects on glucose metabolism. Criteria The diagnostic criteria for<unk>  are those of<unk>  (fasting glucoses persistently above 125 mg/dl (7 mM) or random levels above 200 mg/dl (11 mM)) occurring in the context of high-dose glucocorticoid therapy. Insulin levels are usually detectable, and sometimes elevated, but inadequate to control the glucose. In extreme cases the hyperglycemia may be severe enough to cause nonketotic hyperosmolar coma.
Steroid diabetes
Ketosis-prone diabetes
Diet in diabetes
Diabetic hypoglycemia
Maturity onset diabetes of the young
Reactive hypoglycemia
Galmic
Cushing's syndrome