The causes of<unk>  lead to variations in symptoms, but a common core of symptoms can be seen regardless of etiology. Causes of<unk>  include<unk>  agenesis, dysplasia and hypoplasia,<unk>  stroke, tumor, cerebellitis, trauma, and neurodegenerative diseases (such as progressive supranuclear palsy and multiple system atrophy).<unk>  can also be seen in children with prenatal, early postnatal, or developmental lesions. In these cases there are lesions of the cerebellum resulting in<unk>  and affect deficits. The severity of<unk>  varies depending on the site and extent of the lesion. In the original report that described this syndrome, patients with bihemispheric infarction, pancerebellar disease, or large unilateral posterior inferior<unk>  artery (PICA) infarcts had more<unk>  deficits than patients with small right PICA infarcts, small right anterior interior<unk>  artery infarcts or superior<unk>  artery (SCA) territory. Overall, patients with damage to either the posterior lobe of the cerebellum or with bilateral lesions had the greatest severity of symptoms, whereas patients with lesions in the anterior lobe had less severe symptoms. In children, it was found that those with astrocytoma performed better than those with medulloblastoma on neuropsychological tests. When diagnosing a patient with<unk> , medical professionals must remember that<unk>  has many different causes.
Cerebellar cognitive affective syndrome
Post-encephalitic parkinsonism
Dejerine–Roussy syndrome
Dopamine-responsive dystonia
Bickerstaff brainstem encephalitis
Wernicke–Korsakoff syndrome
Paraneoplastic cerebellar degeneration
Parkinson-plus syndrome