Although it was marketed as being safer,<unk>  has most of the pharmacological effects and dangers of barbiturates and acts at the barbiturate binding site (though it is less sedating at effective doses). It is reported to have some anticonvulsant properties against absence seizures, but can exacerbate generalized tonic-clonic seizures.<unk> 's mechanism of action is not completely known. It has been shown in animal studies to have effects at multiple sites in the central nervous system, including the thalamus and limbic system.<unk>  binds to GABAA receptors which interrupts neuronal communication in the reticular formation and spinal cord, causing sedation and altered perception of pain.<unk>  has the ability to activate currents even in the absence of GABA. This relatively unique property makes<unk>  exceptionally dangerous when used in combination with other GABA-mediated drugs (including alcohol). It is also a potent adenosine reuptake inhibitor. Related drugs include carisoprodol and tybamate (prodrugs of<unk> , felbamate, mebutamate, and methocarbamol.
Meprobamate
Felbamate
Lamotrigine
Cenobamate
Topiramate
Disulfiram
Retigabine
Tiagabine