This phenomenon is thus iodine-induced hyperthyroidism, typically presenting in a patient with endemic goiter (due to iodine deficiency), who relocate to an iodine-abundant geographical area. Also, people who have Graves disease, toxic multinodular goiter, or various types of thyroid adenoma are also at risk of<unk><unk>  effect when receiving iodine, because the thyroid will then not respond to the negative feedback from increased thyroid hormones. The source of iodine may be from the diet, administration of the iodinated contrast for medical imaging, or amiodarone (an antiarrhythmic drug). The hyperthyroidism usually develops over 2 to 12 weeks following iodine administration. In some ways the<unk>  is the opposite of the Wolff–Chaikoff effect, which refers to the short period of thyroid-hormone suppression which happens in normal persons and in persons with thyroid disease, when comparatively large quantities of iodine or iodide are ingested. However, (unlike the Wolff-Chaikoff effect), the<unk><unk>  effect does not occur in persons with normal thyroid glands, as thyroid hormone synthesis and release in normal persons is controlled by pituitary TSH secretion (which does not allow hyperthyroidism when extra iodine is ingested).
Jod-Basedow phenomenon
Renbök phenomenon
Tullio phenomenon
Mizuo–Nakamura phenomenon
Koebner phenomenon
Kanavel's cardinal signs
Palla's sign
Carnett's sign