The cause of<unk>  is not known. It may be caused by persistent bacterial, viral, amoebal, or parasitic infections. Folic acid deficiency, effects of malabsorbed fat on intestinal motility, and persistent small intestinal bacterial overgrowth may combine to cause the disorder. A link between small intestinal bacterial overgrowth and<unk>  has been proposed to be involved in the aetiology of post-infectious irritable bowel syndrome (IBS). Intestinal immunologic dysfunction, including deficiencies in secretory immunoglobulin A (IgA), may predispose people to malabsorption and bacterial colonization, so<unk>  may be triggered in susceptible individuals following an acute enteric infection.
Tropical sprue
Environmental enteropathy
Congenital tufting enteropathy
Coeliac disease
Food protein-induced enterocolitis syndrome
Autoimmune enteropathy
Hereditary diffuse gastric cancer
Schnitzler syndrome