In patients with hypertension, diagnostic clues pointing to<unk>  can be found on routine labwork. These include low serum potassium (hypokalemia), elevated serum sodium (hypernatremia), and elevated serum bicarbonate (metabolic alkalosis). Urine studies may show elevated urine potassium (kaliuresis). To further differentiate between hyperaldosteronism and<unk> , studies including plasma renin activity (PRA) and plasma aldosterone concentration (PAC) can be obtained.<unk>  will exhibit low levels of both PRA and PAC while hyperaldosteronism will demonstrate elevated PAC. Confirmatory tests to diagnose the specific forms of<unk>  vary depending on the cause. The genetic conditions such as Liddle's syndrome and CAH can be confirmed with genetic tests for the affected genes. CAH can also be confirmed by analyzing enzyme levels following ACTH stimulation testing. AME can be diagnosed with a 24 hour urine collection exhibiting an increased ratio of urinary cortisol to urinary cortisone.
Pseudohyperaldosteronism
Familial hyperaldosteronism
Primary aldosteronism
Pseudohypoaldosteronism
Hyperaldosteronism
Glucocorticoid remediable aldosteronism
Apparent mineralocorticoid excess syndrome
Hypoaldosteronism