Brain lesions<unk>  may be an acquired deficit from lesions in the posterior side of the visual cortex. Lesions more often cause gross<unk> . The neurons of the middle temporal cortex respond to moving stimuli and hence the middle temporal cortex is the motion-processing area of the cerebral cortex. In the case of LM, the brain lesion was bilateral and symmetrical, and at the same time small enough not to affect other visual functions. Some unilateral lesions have been reported to impair motion perception as well.<unk>  through lesions is rare, because damage to the occipital lobe usually disturbs more than one visual function.<unk>  has also been reported as a result of traumatic brain injury. Transcranial magnetic stimulation Inconspicuous<unk>  can be selectively and temporarily induced using transcranial magnetic stimulation (TMS) of area V5 of the visual cortex in healthy subjects. It is performed on a 1 cm² surface of the head, corresponding in position to area V5. With an 800-microsecond TMS pulse and a 28 ms stimulus at 11 degrees per second, V5 is incapacitated for about 20–30 ms. It is effective between −20 ms and +10 ms before and after onset of a moving visual stimulus. Inactivating V1 with TMS could induce some degree of<unk>  60–70 ms after the onset of the visual stimulus. TMS of V1 is not nearly as effective in inducing<unk>  as TMS of V5. Alzheimer's disease Besides memory problems, Alzheimer's patients may have varying degrees of<unk> . This could contribute to their marked disorientation. While Pelak and Hoyt have recorded an Alzheimer's case study, there has not been much research done on the subject yet. Antidepressants Inconspicuous<unk>  can be triggered by high doses of certain antidepressants with vision returning to normal once the dosage is reduced.
Akinetopsia
Palinopsia
Photopsia
Pelopsia
Dysmorphopsia
Oscillopsia
Agraphesthesia
Cyanopsia