160 px The mechanism of this condition is apparently controlled by(or due to) the SLC2A10 gene. The molecular genetic pathogenesis finds that SLC2A10 encodes GLUT10(in nuclear membrane, or the endoplasmic reticulum, the later of which GLUT10 transports DHA into).Clinically speaking, according to one review, the condition of<unk>  is seen more with the advance of age.
Arterial tortuosity syndrome
Charcot–Bouchard aneurysm
Monckeberg's arteriosclerosis
Diabetic angiopathy
Angiolathyrism
Mycotic aneurysm
Cirsoid aneurysm
Carotid artery dissection