The CT scan of a patient with<unk>  (left): The follow-up CT (right) after 7 years shows full remission. The pathogenesis of<unk>  is unknown. Several hypotheses are given, such as it arises from sinusoidal epithelial damage, increased sinusoidal pressure due to obstruction in blood outflow from the liver, or hepatocellular necrosis. Two morphologic patterns of<unk> <unk>  were described by Yanoff and Rawson. In the<unk> lebectatic type, the blood-filled spaces are lined with endothelium and are associated with aneurysmal dilatation of the central vein; in the parenchymal type, the spaces have no endothelial lining and they usually are associated with haemorrhagic parenchymal necrosis. Some consider both patterns to be one process, initiated by focal necrosis of liver parenchyma, observed in parenchymal type, progressing into formation of fibrous wall and endothelial lining around haemorrhage of<unk> lebectatic type. Fibrosis, cirrhosis, regenerative nodules, and tumours may also be seen.
Peliosis hepatis
Amoebic liver abscess
Hepatolithiasis
Hepatic artery thrombosis
Focal fatty liver
Pyogenic liver abscess
Perihepatitis
Liver abscess