<unk>  binds to a distinct site which is associated with Cl− ionophores at GABAA receptors. This increases the length of time which the Cl− ionopores are open, thus causing an inhibitory effect. Metabolism of<unk>  is primarily hepatic via demethylation and oxidation. Side-chain oxidation is the primary means of metabolism involved in the termination of the drug's biological activity.
Methohexital
Dianethole
Iopentol
Metharbital
Doxylamine
Difelikefalin
Dextromoramide
Suxamethonium chloride