Most orally administered anabolic steroids function by having an alkylated 17α-carbon atom, which prevents first-pass metabolism by the liver. This approach however results in the AAS having hepatotoxicity.<unk>  is not 17α-alkylated; instead it has increased oral bioavailability due to its cyclopentenyl ether group. After ingestion, the inactive<unk>  is transformed into boldenone.<unk>  itself has very few androgenic effects, and most of what it does have are a result of its conversion to boldenone and its metabolites. Because of high doses necessary for androgenic effects, cost and inconvenience meant that<unk>  never proved to be commercially successful, and its clinical applications were fulfilled by alternative, more effective, AAS. Its illicit usage in bodybuilding and athletics likewise proved limited, though drug tests are still used to detect its metabolites as it remains a banned substance for most competitive sports.<unk> , via boldenone, can be transformed into estrogens, and hence may have some estrogenic activity.
Quinbolone
Tibolone
Oxendolone
Diprobase
Revefenacin
Penicillin
Mycophenolic acid
Oxytetracycline