Intracutaneous injection of pilocarpine (sweat gland stimulant) is known to evoke no sweat response, indicating that lesions are on the post-synaptic side of the nerve-sweat gland junction. The proposed pathomechanisms of<unk>  include: * A deficit within muscarinic cholinergic receptors of the eccrine sweat glands. * Interference in acetylcholine transmission to cholinergic receptors. * A cross-reactive immune response which interferes with cholinergic transmission in the eccrine glands. * Components of an immediate-type allergy (based on the dramatic resumption of axon reflex sweating following glucocorticoid treatment).
Idiopathic pure sudomotor failure
Vitreous touch syndrome
Mongolian idiocy
Ecotropism
Phantom eye syndrome
Somatoparaphrenia
Trigeminal autonomic cephalgia
Trigeminal trophic syndrome