Unlike classical angina pectoris, traditional cardiovascular risk factors are not thought to be significantly associated with<unk> . The exception to this is with smoking, which is known to be a modifiable risk factor for vasospastic angina. There are several risk factors that are thought to precipitate, or trigger, episodes of<unk> . Many of these factors work by exerting effects on the autonomic nervous system. One of the mechanisms through which this occurs is via increasing sympathetic nervous system activity. The resulting increased sympathetic outflow leads to vasoconstrictive effects on blood vessels. For example, cocaine use can trigger vasospasm in coronary arteries through its actions on adrenergic receptors causing vasoconstriction. Exercise, cold weather, physical activity or exertion, mental stress, hyperventilation are additional precipitating factors.
Coronary vasospasm
Coronary ischemia
Coronary occlusion
Myocardial stunning
Cerebral vasospasm
Hyperdynamic circulation
Coronary steal
Coronary thrombosis