The production of antibodies in Graves' disease is thought to arise by activation of CD4+ T-cells, followed by B-cell recruitment into the thyroid. These B-cells produce antibodies specific to the thyroid antigens. In Hashimoto's thyroiditis, activated CD4+ T-cells produce interferon-γ, causing the thyroid cells to display MHC class II molecules. This expands the autoreactive T-cell repertoire and prolongs the inflammatory response. While<unk>  antibodies are used to track the presence of autoimmune thyroiditis, they are generally not considered to contribute directly to the destruction of the thyroid.
Antithyroid autoantibodies
Antiganglioside antibodies
Anti-malignin antibody
Antisperm antibodies
Anti-gliadin antibodies
Anti–Saccharomyces cerevisiae antibody
Anti-apolipoprotein antibodies
Autoantibody