<unk>  (AAG) is a rare form of dysautonomia in which the patient’s immune system produces ganglionic anti-nicotinic acetylcholine receptor (AChR) antibodies, inhibiting ganglionic AChR currents and impairing transmission in<unk>  ganglia. Symptoms onset can be acute, subacute or gradual.
Autoimmune autonomic ganglionopathy
Primary autonomic failure
Neurogenic inflammation
Visual pathway lesions
Small fiber peripheral neuropathy
Lower motor neuron lesion
Functional neurologic disorder
Upper motor neuron lesion