<unk>  (or ADIS) refers to the induction of cellular<unk>  as a result of<unk><unk>  therapy. ADIS is observed in prostate cancer cells that are dependent on<unk> s for cell proliferation.<unk>  withdrawal<unk>  cells to undergo cellular<unk>  by up-regulating intracellular reactive oxygen species (ROS) that cause DNA damage. ADIS is maintained through the up-regulation of the cell cycle regulator p16ink4a.
Androgen deprivation-induced senescence
Antiestrogen withdrawal response
Congenital estrogen deficiency
Side effects of bicalutamide
Endocrine therapy resistance in breast cancer
Stimulus-triggered acquisition of pluripotency
Inborn errors of steroid metabolism
Thyroid hormone resistance