<unk>  can affect the health of the mother as well as the child before and after delivery.<unk>  disorders are prevalent in women of child-bearing age and for this reason commonly present as a pre-existing<unk>  in pregnancy, or after childbirth. Uncorrected<unk>  dysfunction in pregnancy has adverse effects on fetal and maternal well-being. The deleterious effects of<unk>  dysfunction can also extend beyond pregnancy and delivery to affect neurointellectual development in the early life of the child. Due to an increase in thyroxine binding globulin, an increase in placental type 3 deioidinase and the placental transfer of maternal thyroxine to the fetus, the demand for<unk>  hormones is increased during pregnancy. The necessary increase in<unk>  hormone production is facilitated by high human chorionic gonadotropin (hCG) concentrations, which bind the TSH receptor and stimulate the maternal<unk>  to increase maternal<unk>  hormone concentrations by roughly 50%. If the necessary increase in<unk>  function cannot be met, this may cause a previously unnoticed (mild)<unk>  disorder to worsen and become evident as gestational<unk><unk> . Currently, there is not enough evidence to suggest that screening for<unk>  dysfunction is beneficial, especially since treatment<unk>  hormone supplementation may come with a risk of overtreatment. After women give birth, about 5% develop postpartum<unk> itis which can occur up to nine months afterwards. This is characterized by a short period of hyperthyroidism followed by a period of hypothyroidism; 20–40% remain permanently hypothyroid.
Thyroid disease in pregnancy
Drugs in pregnancy
Thyroid disease in women
Teenage pregnancy
Cannabis in pregnancy
Pre-existing disease in pregnancy
High-risk pregnancy
Sexual activity during pregnancy