Stellate cell The pathophysiology of<unk>  is indicated by increased vascular resistance via different causes; additionally, hepatic stellate cells and myofibroblasts are activated. Increased endogenous vasodilators in turn promote more blood flow in the portal veins. Nitric oxide is an endogenous vasodilator and it regulates intrahepatic vascular tone (it is produced from L-arginine). Nitric oxide inhibition has been shown in some studies to increase<unk>  and hepatic response to norepinephrine.
Portal hypertension
Hypertensive heart disease
Orthostatic hypertension
Pulmonary hypertension
Portopulmonary hypertension
Secondary hypertension
Portal vein thrombosis
Nephrotic syndrome