The pathophysiology of<unk>  remains unknown, although an allergic reaction, trauma, and an autoimmune process have all been implicated as possible causes. The disease is manifested by an abnormal proliferation of lymphoid follicles and vascular endothelium. Peripheral eosinophilia and the presence of eosinophils in the inflammatory infiltrate suggest it may be a hypersensitivity reaction. Some evidence has indicated TH2 lymphocytes may also play a role, but further investigation is needed.<unk>  is generally limited to the skin, lymph nodes, and salivary glands, but patients with<unk>  and nephrotic syndrome have been reported. The basis of this possible association is unclear.
Kimura's disease
Darier's disease
Milroy's disease
Tornwaldt's disease
Leiner's disease
Dieterich's disease
Mortimer's disease
Bright's disease