The recognised forms of<unk>  are all due to ineffective insulin production or release by pancreatic beta cells. Several of the defects are mutations of transcription factor genes. One form is due to mutations of the glucokinase gene. For each form of<unk> , multiple specific mutations involving different amino acid substitutions have been discovered. In some cases, there are significant differences in the activity of the mutant gene product that contribute to variations in the clinical features of the diabetes (such as degree of insulin deficiency or age of<unk> .
Maturity onset diabetes of the young
Ketosis-prone diabetes
Leucine-sensitive hypoglycemia of infancy
Cystic fibrosis–related diabetes
Lipoatrophic diabetes
Diet in diabetes
Kosaki overgrowth syndrome
Idiopathic hypoglycemia