Although the etiology is unconfirmed, transient<unk>  is known to be caused by increased levels of ammonia in the blood stream, as well as a failure of the urea cycle to convert enough of the ammonia into urea. Since transamination of proteins is a leading producer of ammonia, protein restriction may be recommended as a therapy to reduce the symptoms of the episode. THAN can also be treated by avoiding amino acids in TPN or total parenteral nutrition or by giving a high caloric diet to limit catabolism of the tissues and therefore to minimize the breakdown of endogenous protein. The most common treatments are dialysis (both peritoneal and hemodialysis), sodium benzoate, and arginine. Sodium Benzoate combines with glycine to be excreted in the form of hippuric acid. The goal of these treatments is to convert nitrogen to a compound that can be excreted more easily.
Transient hyperammonemia of the newborn
Permanent neonatal diabetes
Neonatal hepatitis
Hemolytic disease of the newborn
Hemolytic disease of the newborn 
Transient neonatal diabetes
Neonatal diabetes
Neonatal hypoglycemia