Severe procedural complications during a<unk>  procedure, including catastrophic bleeding or direct liver injury, are relatively uncommon. In the hands of an experienced physician, operative mortality is less than 1%. On the other hand, up to 25% of patients who undergo<unk>  will experience transient post-operative hepatic encephalopathy caused by increased<unk>  passage of nitrogen from the gut. A less common, but more serious complication, is hepatic ischemia causing acute liver failure. While healthy livers are predominantly oxygenated by portal blood supply, long-standing portal hypertension results in compensatory hypertrophy of and increased reliance on the hepatic artery for oxygenation. Thus, in people with advanced liver disease the shunting of portal blood away from hepatocytes is usually well tolerated. However, in some cases suddenly shunting portal blood flow away from the liver may result in acute liver failure secondary to hepatic ischemia. Acute hepatic dysfunction after<unk>  may require emergent closure of the shunt. A rare but serious complication is persistent<unk>  infection, also known as endotipsitis. Lastly, the<unk>  may become blocked by a blood clot or in-growth of endothelial cells and no longer function. This has been significantly reduced with the use of polytetrafluoroethylene (PTFE)–covered stents.
Transjugular intrahepatic portosystemic shunt
Distal splenorenal shunt procedure
Portosystemic shunt
Hepatoportoenterostomy
Bidirectional Glenn procedure
Percutaneous hepatic perfusion
Peritoneovenous shunt
Isolated hepatic perfusion