<unk>  Spreading Depolarizations appear and spread over the cortex. Histopathology at low magnification of a<unk>  on H&E stain, showing pallor in the infarcted area due to edema. Histopathology at high magnification of a normal neuron, and a<unk>  at approximately 24 hours on H&E stain: The neurons become hypereosinophilic and there is an infiltrate of neutrophils. There is slight edema and loss of normal architecture in the surrounding neuropil. Symptoms of<unk>  are determined by the parts of the brain affected. If the infarct is located in the primary motor cortex, contralateral hemiparesis is said to occur. With brainstem localization, brainstem syndromes are typical: Wallenberg's syndrome, Weber's syndrome, Millard–Gubler syndrome, Benedikt syndrome or others.<unk> s will result in weakness and loss of sensation on the opposite side of the body. Physical examination of the head area will reveal abnormal pupil dilation, light reaction and lack of eye movement on opposite side. If the<unk>  occurs on the left side brain, speech will be slurred. Reflexes may be aggravated as well.
Cerebral infarction
Brain ischemia
Stroke
Coma
Brain metastasis
Brain abscess
Intracerebral hemorrhage
Subarachnoid hemorrhage