<unk> , like any other AAS, has many side effects. The strong androgenic nature of<unk>  facilitates its tendency to produce virilization and this is why it is not recommended for women for physique- or performance-enhancing purposes. The side effects of<unk>  are similar to other AAS; however, the negative side effects that are specifically facilitated by<unk>  are as follows. Androgenic<unk>  has androgenic activity. Specific to the androgenic properties of trenbolone, common side effects of the AAS use include oily skin, acne, seborrhea, increased facial/body hair growth, and accelerated scalp hair loss. These side effects strongly rely on an individual's genetics and may not always occur in every individual. Men susceptible to hair loss related illnesses, such as baldness have a higher chance of becoming permanently bald with the use of<unk> . In women, voice deepening, hirsutism, clitoral enlargement, and virilization in general may occur. Hypogonadism<unk>  contributes greatly to muscle mass and feed efficiency; however, administration of the AAS suppresses natural testosterone production; i.e., it has the potential to cause hypogonadism. This is a common effect of all AAS; the only difference is the variation in how much they suppress in comparison to others. Cardiovascular Administration of any AAS can lead to cardiovascular issues.<unk>  can have a negative and strong impact on cholesterol through suppressing both high-density lipoprotein (HDL) cholesterol ("good" cholesterol) and increasing low-density lipoprotein (LDL) cholesterol ("bad" cholesterol). When compared to oral AAS,<unk>  has a stronger negative effect on cholesterol levels. This negative effect is much more severe with the use of injectable AAS, particularly<unk> . "Tren cough" The exact mechanisms underlying the cause of the tren cough is not known. However,<unk> 's androgenic effect activates a variety of lipid-like active compounds which are called prostaglandins. Many of these prostaglandins are inflammatory and vasoconstrictive. Prostaglandins are signalled through two varying pathways cyclooxygenase (COX) (Also known as: prostaglandin-endoperoxide synthase) and lipoxygenases (LOX) (also known as: EC 1.13.11.34, EC 1.13.11.33, etc.). Vasoconstriction of the muscular wall of the bronchus in the lungs is what triggers this cough reaction. Trenbolone increases an inflammatory mediator peptide called bradykinin which facilitates the dilation of blood vessels. The bradykinin peptide is well known to promote a cough reaction associated with ACE inhibitor medications prescribed for hypertension. Estrogenic and progestogenic No form of trenbolone, including<unk> , is estrogenic. Excess fluid retention is not possible with the administration of this AAS as a result of its not being estrogenic as opposed to testosterone. However, due to trenbolone's potent progestogenic activity, gynecomastia, which is characterized by development and swelling of breast tissue, may still be possible. Stimulation of estrogenic mechanisms are enforced by progestogenic activity as<unk>  and its compounds bind with high affinity to the progesterone receptor. It has been assumed that gynecomastia as a result of trenbolone use is due to a buildup of the hormone prolactin; however, a variety of studies conclude that it is the progestogenic activity of trenbolone promoting this and not prolactin. Trenbolone also has a negative impact on blood pressure, but it does not appear to negatively affect most healthy adult men in this way.
Trenbolone acetate
Stenbolone acetate
Metenolone acetate
Metenolone
Prebediolone acetate
Trenbolone enanthate
Trestolone
Cortisone acetate