<unk>  may have a genetic basis. Published reports have indicated fatal occurrences that appeared to possess a familial pattern, more to the point, a germline mutation. The pathophysiology of<unk><unk>  culminates in occlusion of the pulmonary blood vessels. This could be due to edematous tissue (sclerotic fibrous tissue). Thickening is identified in lobular septal veins, also dilatation of lymphatics happens. Furthermore, alveolar capillaries become dilated (due to back-pressure).
Pulmonary venoocclusive disease
Pulmonary alveolar proteinosis
Chronic thromboembolic pulmonary hypertension
Pulmonary sequestration
Bronchiolitis obliterans
Idiopathic pulmonary haemosiderosis
Pulmonary hyalinizing granuloma
Acute eosinophilic pneumonia