The following are examples of potential causes for reactive lymphadenopathies, all of which have predominantly follicular patterns: * Rheumatoid arthritis * Sjögren syndrome * IgG4-related disease (IgG4-related lymphadenopathy) * Kimura disease * Toxoplasmosis * Syphilis * Castleman disease * Progressive transformation of germinal centers (PTGC) Microorganisms can infect lymph nodes by causing pain and inflammation including redness and tenderness. Bacterial, fungal and viral infections including Bartonella, Staphylococcal, Granulomatous, Adenoviral and Lyme disease are all associated with<unk> . Other autoimmune related diseases that are associated are rheumatoid arthritis, systemic lupus, dermatomyositis and Sjögren syndrome. Immunoglobulin G- related diseases are immune-mediated fibroinflammatory conditions that affect many organs in the body. Chronic inflammatory disorders such as Kimura disease are also the outcome of inflamed or enlarged lymph nodes. Parasitic bacterias such as Toxoplasma gondii or Treponema pallidum can cause enlargement of the lymph nodes as well. Other related causes such as lymphoproliferative conditions known as Castleman disease, or progressive transformation of germinal centers cause lymph node enlargement. Environmental conditions that may also play a role are animal or insect exposure, chronic medication usage and immunization status. The patient's occupational history such as metal work or coal mining may expose them to silicon or beryllium.<unk>  is common in children and young adults, but is not limited to any age; it is also common among the elderly and is non-sex specific. Children often experience reactive lymph nodes when they are younger due to new exposure of environmental pathogens, even without development of an infection. Clinically,<unk>  lymphadenopathy is usually restricted to a single area on the body, but can also be on several parts of the body as well.<unk>  is one of the most common types of lymphadenopathies and can be associated with paracortical and sinus<unk> .
Follicular hyperplasia
Lymphoid hyperplasia
Perifollicular mucinosis
Microglandular hyperplasia
Hyperkeratinization
Pseudopolyps
Xanthogranulomatous inflammation
Reticular formation