TS has been reported almost exclusively in immunocompromised patients, primarily organ transplant recipients on regimens of immunosuppressive drugs, and also in patients with hematolymphoid malignancies. As of 2016 there were no case reports in the literature describing cases of TS in patients with HIV-AIDS. There is compelling evidence that<unk>  is caused by a polyomavirus called<unk>  polyomavirus (TSPyV) or Human polyomavirus 8. There is evidence that exposure to the virus is common among healthy adults; estimates of seroprevalence (that is, prevalence of detectable antibodies against viral proteins) in immunocompetent adults range from 70 to 80% in different sample populations. TSPyV infects the skin, but viral DNA is rarely detectable there in asymptomatic individuals even if they possess antibodies to the virus indicating exposure. It is not known whether TS represents new primary infection or opportunistic reactivation of a latent infection.
Trichodysplasia spinulosa
Adiposogenital dystrophy
Encephalocraniocutaneous lipomatosis
Pachyonychia congenita
Lipoatrophia annularis
Mandibuloacral dysplasia
Thanatophoric dysplasia
Genu recurvatum