<unk>  (eccrine) The pathophysiology of<unk> s remains largely unknown. Familial patterns presenting in an autosomal-dominant pattern suggest a genetic link that can result in varying genetic aberrations in lesions, specifically chromosome 16q22. The most commonly accepted theory is that<unk> s are benign growths that arise from the intraepidermal portion of eccrine ducts. Another theory suggests that<unk> s are a reactive hyperplasia rather than a true neoplasm resulting after inflammatory processes such as eczema. A hamartomatous process possibly could explain eruptive<unk> s. A hamartoma of pluripotent stem cells could precede the pathological process.<unk> s may also be under hormonal influence, explaining the female predominance.
Syringoma
Thecoma
Spiradenoma
Pilomatricoma
Cylindroma
Apudoma
Hamartoma
Ameboma