Treatment of<unk>  focuses on preventing further attacks and relieving acute symptoms. Avoiding carbohydrate-rich meals, strenuous exercise and other identified triggers, and taking acetazolamide or another carbonic anhydrase inhibitor, may help prevent attacks of weakness. Some patients also take potassium-sparing diuretics such as spironolactone to help maintain potassium levels. Paralysis attacks can be managed by drinking one of various potassium salts dissolved in water (debate exists over which, if any one in particular, is best used, but potassium chloride and bicarbonate are common). Rapidly absorbed boluses of liquid potassium are generally needed to abort an attack, but some patients also find positive maintenance results with time-released potassium tablets. IV potassium is seldom justified unless the patient is unable to swallow. Daily potassium dosage may need to be much higher than for potassium replacement from simple<unk> : 100-150 mEqs of potassium is often needed to manage daily fluctuations in muscle strength and function. Perioperatively, prevention includes avoiding neuromuscular blockade, avoid excessive hyperventilation, warm the patient, provide adequate hydration, avoid glucose infusions, do not give diuretics, and closely monitor the electrocardiogram for signs of<unk> . Normal saline is the preferred IV solution for patients with familial<unk> . Glucose containing solutions may cause weakness. Additionally, the high chloride content can cause a mild acidosis which would be preferred over alkalosis.
Hypokalemic periodic paralysis
Sleep paralysis
Hyperkalemic periodic paralysis
Potassium-aggravated myotonia
Thyrotoxic periodic paralysis
Thyrotoxic myopathy
Alcoholic polyneuropathy
Glycine encephalopathy