The pathophysiology of<unk>  is dependent on the underlying disease process, which can vary depending on what condition the<unk>  is secondary to. More specifically, different diseases (many of which are mentioned above in the Causes section) affect different segments of the glomerulus and cause disease-specific segments of the glomerulus to become inflamed. Most often, it is dependent on what part of the glomerulus is damaged by antibody-antigen complex (immune complex) deposition. In all cases, however, the inflammatory processes in the glomerulus cause the capillaries to swell and the pores between podocytes become large enough that inappropriate contents in the blood plasma (i.e. red blood cells, protein, etc.) will begin to spill into the urine. This causes a decrease in glomerular filtration rate (GFR) and, if left untreated over time, will eventually produce uremic symptoms and retention of sodium and water in the body, leading to both edema and hypertension.
Nephritic syndrome
Nephrotic syndrome  Nephrotic syndrome  1
Papillorenal syndrome
Culture-bound syndrome
Hepatorenal syndrome
Conorenal syndrome
Emmonsiosis
Cardiorenal syndrome