<unk> es are myopathies that result from defects in biochemical<unk> m that primarily affect muscle. They are generally genetic defects that interfere with muscle's ability to create energy. At the cellular level,<unk> es lack some kind of enzyme that prevent the chemical reactions necessary to create adenosine triphosphate (ATP). This prevents the muscle cells from being able to function properly and disrupts communication between joints and bones. Some people with a<unk>  never develop symptoms due to the body's ability to produce enough ATP through alternative pathways. In the event more ATP is needed from the affected pathway, the lack of it becomes an issue and symptoms develop. People with a<unk>  often experience symptoms such as progressive muscle weakness, fatigue, pain and cramping after exercise, and considerable breakdown of muscle tissue. The degree of symptoms varies greatly from person to person and is dependent on the severity of enzymatic defect. In extreme cases it can lead to Rhabdomyolysis.
Metabolic myopathy
Inflammatory myopathy
Nemaline myopathy
Brody myopathy
Acquired non-inflammatory myopathy
Centronuclear myopathy
Polymyositis
Hereditary inclusion body myopathy