<unk> ness in children and young adults nearly always results from below-average growth in childhood, while<unk> ness in older adults usually results from loss of height due to kyphosis of the spine or collapsed vertebrae from osteoporosis. The most common causes of<unk>  in childhood are constitutional growth delay or familial<unk> . From a medical perspective, severe<unk> ness can be a variation of normal, resulting from the interplay of multiple familial genes. It can also be due to one or more of many abnormal conditions, such as chronic (prolonged) growth hormone or thyroid hormone deficiency, malnutrition, disease of a major organ system, mistreatment, treatment with certain drugs, chromosomal deletions. Human growth hormone (HGH) deficiency may occur at any time during infancy or childhood, with the most obvious sign being a noticeable slowing of growth. The deficiency may be genetic. Among children without growth hormone deficiency,<unk>  may be caused by Turner syndrome or Noonan syndrome, chronic kidney disease, being small for gestational age at birth, Prader–Willi syndrome, Wiedemann-Steiner syndrome, or other conditions. Genetic skeletal dysplasias also known as osteochondrodysplasia usually manifest in<unk> -limbed disproportionate<unk> . When the cause is unknown, it is called idiopathic<unk> .<unk>  can also be caused by the bone plates fusing at an earlier age than normal, therefore stunting growth. Normally, the bone age is the same as the biological age but for some people, it is older. For many people with advanced bone ages, they hit a growth spurt early on which propels them to average height but stop growing at an earlier age. However, in some cases, people who are naturally<unk> er combined with their advanced bone age, end up being even<unk> er than the height they normally would have been because of their stunted growth.
Short stature
Adipomastia
Stunted growth
Failure to thrive
Hyperdontia
Trichomegaly
Anisomastia
Muscle dysmorphia