The<unk>  is one of two major mechanisms postulated to explain the underlying cause of atherosclerosis and coronary heart disease (CHD), the other being the lipid<unk> . Although an ongoing debate involving connection between dietary lipids and CHD sometimes portrays the two<unk>  as being opposed, they are in no way mutually exclusive. Moreover, since the discovery of the role of LDL cholesterol (LDL-C) in the pathogenesis of atherosclerosis, the two<unk>  have become tightly linked by a number of molecular and cellular processes.
Chronic endothelial injury hypothesis
Endothelial activation
Vasculogenic mimicry
Microvasculature remodeling
Reactive perforating collagenosis
Graft-versus-tumor effect
Arteriosclerosis, Thrombosis, and Vascular Biology
Endothelial dysfunction