<unk>  generally elevates the concentration of glucose in the blood by promoting gluconeogenesis and glycogenolysis.<unk>  also decreases fatty acid synthesis in adipose tissue and the liver, as well as promoting lipolysis in these tissues, which causes them to release fatty acids into circulation where they can be catabolised to generate energy in tissues such as skeletal muscle when required. Glucose is stored in the liver in the form of the polysaccharide glycogen, which is a glucan (a polymer made up of glucose molecules). Liver cells (hepatocytes) have<unk>  receptors. When<unk>  binds to the<unk>  receptors, the liver cells convert the glycogen into individual glucose molecules and release them into the bloodstream, in a process known as glycogenolysis. As these stores become depleted,<unk>  then encourages the liver and kidney to synthesize additional glucose by gluconeogenesis.<unk>  turns off glycolysis in the liver, causing glycolytic intermediates to be shuttled to gluconeogenesis.<unk>  also regulates the rate of glucose production through lipolysis.<unk>  induces lipolysis in humans under conditions of insulin suppression (such as diabetes mellitus type 1).<unk>  production appears to be dependent on the central nervous system through pathways yet to be defined. In invertebrate animals, eyestalk removal has been reported to affect<unk>  production. Excising the eyestalk in young crayfish produces<unk> -induced hyperglycemia.
Glucagon
Proglucagon
Activated charcoal 
Insulin 
Insulin
Dasiglucagon
Acetylcysteine
Sodium bicarbonate