Patients with<unk>  have variable outcomes, either being recognised as developing acute kidney injury, the abrupt loss in renal failure, or being undiagnosed. As the deposition of calcium<unk>  crystals are detected at the renal tubules following the use of OSP, the symptoms of<unk>  are similar to acute tubular necrosis, an intrinsic renal injury. For example, events including diarrhea, vomiting, dehydration, sepsis, and hypotension following the colonoscopy, can indicate the risk of<unk>  and raise the concern for acute tubular necrosis. The results of hypotension and dehydration are dry mucous membrane, decreased skin turgor, and cool extremities, which can be used to notify the abnormal renal perfusion. As there is a gap between the first administration of OSP and recognisable symptoms, many<unk>  incidences are overlooked and not proceeded to biopsy for further investigation. The transient hyperphosphatasemia, the electrolyte disorder with elevated<unk>  level in the blood, is found to be correlated with the use of OSP after colonoscopy. Significant electrolyte abnormalities including hypocalcemia, hypernatremia, and hypomagnesemia are also the outcomes of the use of OSP. As these detection tests are mostly operated at the laboratory level,<unk>  incidents are widely under-recognized and overlooked. Depiction of different adverse renal conditions.<unk>  or deposition of calcium<unk>  within renal tubules belongs to the urinary stone problems.
Phosphate nephropathy
Analgesic nephropathy
Sickle cell nephropathy
IgA nephropathy
Myeloma cast nephropathy
Diabetic nephropathy
Reflux nephropathy
Malarial nephropathy