The pathophysiology of<unk> es is better understood at the cellular level with advances in molecular techniques. Mutant proteins can disturb cardiac function in the contractile apparatus (or mechanosensitive complexes). Cardiomyocyte alterations and their persistent responses at the cellular level cause changes that are correlated with sudden cardiac death and other cardiac problems.
Cardiomyopathy
Dilated cardiomyopathy
Amyloid cardiomyopathy
Myopathy
Alcoholic cardiomyopathy
Cardiac amyloidosis
Arrhythmogenic cardiomyopathy
Diabetic cardiomyopathy