<unk>  can be diagnosed before, during or after birth if there is a family history of the condition. Several options are available to parents. If there is no family history of<unk> , it is usually only diagnosed when a child begins to walk or crawl. They may experience joint bleeds or easy bruising. Mild<unk>  may only be discovered later, usually after an injury or a dental or surgical procedure. Before pregnancy Genetic testing and counselling are available to help determine the risk of passing the condition onto a child. This may involve testing a sample of tissue or blood to look for signs of the genetic mutation that causes<unk> . During pregnancy A pregnant woman with a history of<unk>  in her family can test for the<unk>  gene. Such tests include: * chorionic villus sampling (CVS): a small sample of the placenta is removed from the womb and tested for the<unk>  gene, usually during weeks 11–14 of pregnancy * amniocentesis: a sample of amniotic fluid is taken for testing, usually during weeks 15–20 of pregnancy There is a small risk of these procedures causing problems such as miscarriage or premature labour, so the woman may discuss this with the doctor in charge of her care. After birth If<unk>  is suspected after a child has been born, a blood test can usually confirm the diagnosis. Blood from the umbilical cord can be tested at birth if there's a family history of<unk> . A blood test will also be able to identify whether a child has<unk>  A or B, and how severe it is. Classification There are several types of<unk> :<unk>  A,<unk>  B,<unk>  C, parahaemophilia, acquired<unk>  A, and acquired<unk>  B.<unk>  A, is a recessive X-linked genetic disorder resulting in a deficiency of functional clotting Factor VIII.<unk>  B, is also a recessive X-linked genetic disorder involving a lack of functional clotting Factor IX.<unk>  C, is an autosomal genetic disorder involving a lack of functional clotting Factor XI.<unk>  C is not completely recessive, as heterozygous individuals also show increased bleeding. The type of<unk>  known as parahaemophilia is a mild and rare form and is due to a deficiency in factor V. This type can be inherited or acquired. A non-genetic form of<unk>  is caused by autoantibodies against factor VIII and so is known as acquired<unk>  A. It is a rare but potentially life-threatening bleeding disorder caused by the development of autoantibodies (inhibitors) directed against plasma coagulation factors. Acquired<unk>  can be associated with cancers, autoimmune disorders and following childbirth.
Haemophilia
Pedophilia
Acquired haemophilia
Exophilia
Dysfibrinogenemia
Von Willebrand disease
Harris platelet syndrome
Quebec platelet disorder