<unk>  does not have any concrete evidence of epidemiology due to its rare nature. The most concrete evidence that is identified lies in the hands of genetics due to its autosomal dominant pattern of inheritance and the mutation present on chromosome 1. However, there is no evidence to show population favorability, distribution patterns, risk factors, environmental factors, etc.
WNT4 deficiency
Janus kinase 3 deficiency
MECP2 duplication syndrome
GATA2 deficiency
ZC4H2 deficiency
DOCK8 deficiency
NGLY1 deficiency
8p23.1 duplication syndrome