<unk>  first develops with damage to the arterial wall, followed by platelet aggregation at the site of injury, recruitment of inflammatory cells, proliferation and migration of vascular smooth muscle cells, and collagen deposition. Mechanical injury of arterials due to stretching of arterial walls with a balloon catheter results in the recruitment of cells such as monocytes, macrophages, and neutrophils to the site of injury. Macrophages in particular express many growth factors, cytokines, and enzymes that facilitate vascular smooth muscle cell migration and proliferation. C-reactive protein is a systemic inflammatory mediator correlated with<unk> , but it is still unknown if this protein is a marker of increased risk or a causative agent of the condition.
Neointimal hyperplasia
Intimal hyperplasia
Neovascularization
Endothelial activation
Hydrodilatation
Infibulation
Glomerulation
Angiomatosis