The theoretical<unk>  is distinct from schizophrenia and induced by the use of current (dopaminergic) antipsychotics by the depletion of dopamine and related to the known side effect caused by their long-term use,<unk>  dyskinesia. In addition to dopaminergic upregulation in the nigrostriatal tracts, many investigators have suggested that dopaminergic upregulation may occur in mesolimbic or mesocortical tracts, leading to a worsening of psychosis beyond the original level. This phenomenon has been called<unk> ' or 'supersensitivity psychosis'.<unk>  was researched in 1978 and 1989, and sporadic research continues. Some studies have found it to be associated with psychotic depression and potentially, dissociation. For people with any<unk>  conditions clozapine remains an option but since it can create blood dyscrasias, which require frequent blood work, as well as other severe side effects, it is used increasingly less in clinical practice. Although<unk>  continues to be studied, it still has not been established as a fact but it is known that the study classes of antipsychotics such as the NMDA receptor modulators (glutamate antagonists) in not creating<unk>  dyskinesia will not create this condition.
Tardive psychosis
Unitary psychosis
Post-schizophrenic depression
Myxedema psychosis
Pseudoneurotic schizophrenia
Substance-induced psychosis
Secondary mania
Dancing mania