GSS is part of a group of diseases called<unk>  spongiform<unk> es. These diseases are caused by prions, which are a class of pathogenic proteins that are resistant to proteases. These prions then form clusters in the brain, which are responsible for the neurodegenerative effects seen in patients. The P102L mutation, which causes a substitution of proline to a leucine in codon 102, has been found in the prion protein gene (PRNP, on chromosome 20) of most affected individuals. Therefore, it appears this genetic change is usually required for the development of the disease.
types of Transmissible spongiform encephalopathy
symptom of Transmissible spongiform encephalopathy
medical cause of Japanese encephalitis
medical cause of Metachromatic leukodystrophy
symptoms of Rabies
Marburg acute multiple sclerosis
medical cause of Hepatic encephalopathy
complications of West Nile fever