A distinct feature of the<unk>  is its prolonged clinical evolution, with relapsing episodes and autoimmune dysfunction. If diagnosed in its early stages, the symptoms respond well to antibiotics. If the disease evolves to a chronic phase, it can potentially cause oligoarthritis, cognitive impairment, meningoencephalitis and erythema nodosum, with the patient risking to develop both articular and neurological sequelae. The neurological manifestations of BYS were first described by<unk>  et al. including patients with peripheral neuritis, meningitis and cranial neuritis (facial nerve palsy, diplopia and deafness). Likely transmission vectors of BYS belong to the Amblyomma and Rhipicephalus genera, which could help to explain all the particularities observed in BYS versus LD. Some features of BYS also resemble those found in the Southern tick-associated rash illness (STARI, also known as Masters' disease), which is found in the Southern USA.
Baggio–Yoshinari syndrome
DeSanto-Shinawi syndrome
Keppen–Lubinsky syndrome
Barakat-Perenthaler syndrome
Pelger–Huët anomaly
Aagenaes syndrome
Koolen–De Vries syndrome
GRACILE syndrome