Mechanism of action<unk>  selectively forms highly stable complexes with copper and proteins. These complexes are then believed to be primarily excreted via the bile, restoring the normal excretion route of copper that is impaired in patients with Wilson's disease. The binding and excretion mechanism is stable; whereas many de-coppering agents form unstable complexes that are excreted via urine. Bis-choline tetrathiomolybdate
Tiomolibdic acid
Tricresyl phosphate
Penicilloic acid
Disufenton sodium
Dimethyl methylphosphonate
Gadobenic acid
Sucroferric oxyhydroxide
Theramine