<unk>  has been shown to be a potent inhibitor of the aromatic hydroxylation and/or N-dealkylation-mediated metabolism of many substances including, but not limited to octopamine, amphetamine, methamphetamine, fenfluramine, phenelzine, tranylcypromine, trimipramine, and fluoxetine, likely via inactivating cytochrome P450 enzymes. It also inhibits its own metabolism. On account of these interactions, caution should be used when combining<unk>  with other drugs. As an example, when administered with amphetamine or methamphetamine,<unk>  increases their brain concentrations and prolongs their terminal half-lives by 2- to 3-fold, strongly augmenting both their physiological effects and neurotoxicity in the process.
Iprindole
Sertindole
Pirlindole
Butriptyline
Selegiline
Bromocriptine
Iobitridol
Rimonabant