Abnormally elevated levels of<unk>  may be caused by pancreatic tumors, such as<unk> oma, symptoms of which include necrolytic migratory erythema, reduced amino acids, and hyperglycemia. It may occur alone or in the context of multiple endocrine neoplasia type 1. Elevated<unk>  is the main contributor to hyperglycemic ketoacidosis in undiagnosed or poorly treated type 1 diabetes. As the beta cells cease to function, insulin and pancreatic GABA are no longer present to suppress the freerunning output of<unk> . As a result,<unk>  is released from the alpha cells at a maximum, causing a rapid breakdown of glycogen to glucose and fast ketogenesis . It was found that a subset of adults with type 1 diabetes took 4 times longer on average to approach ketoacidosis when given somatostatin (inhibits<unk>  production) with no insulin. Inhibiting<unk>  has been a popular idea of diabetes treatment, however, some have warned that doing so will give rise to brittle diabetes in patients with adequately stable blood glucose. The absence of alpha cells (and hence<unk>  is thought to be one of the main influences in the extreme volatility of blood glucose in the setting of a total pancreatectomy.
Glucagon
Proglucagon
Activated charcoal 
Insulin 
Insulin
Dasiglucagon
Acetylcysteine
Sodium bicarbonate