<unk>  is probably genetically determined, though conventional genetic influences may be less important than other mechanisms. Usually, there is no family history of similar seizures, although siblings with<unk>  or<unk>  and rolandic epilepsy or, less common,<unk>  and idiopathic childhood occipital epilepsy of Gastaut have been reported. There is a high prevalence of febrile seizures (about 17%). SCN1A mutations have been reported in a child and in 2 siblings with relatively early onset of seizures, prolonged time over which many seizures have occurred, and strong association of seizures with febrile precipitants even after the age of 5 years. However, no such mutations were found in another couple of siblings and many other cases with typical<unk> . These data indicate that SCN1A mutations when found contribute to a more severe clinical phenotype of<unk> .
Panayiotopoulos syndrome
Fryns-Aftimos syndrome
Sotos syndrome
Cortes Lacassie syndrome
Patau syndrome
Lelis syndrome
Berk–Tabatznik syndrome
Tangier disease