<unk>  cannot be localized to a specific hemisphere or cerebral area because drawing and<unk>  tasks require both perceptual and motor functioning. It has been linked to parietal lesions in the left and right hemisphere, stroke and Alzheimer's disease. Initially, researchers tried to isolate the cause to left hemisphere lesions in the parietal lobe because of its similarities to Gerstmann syndrome; however, lesions in the dorsal stream also result in visual agnosia and a piecemeal drawing. Although<unk>  can result from lesions in any part of the brain, it is most commonly associated with lesions in the parietal-occipital lobes.<unk>  is common after right parietal stroke and it continues after visuospatial symptoms have subsided. Patients with posterior and parietal lobe lesions tend to have the most severe symptoms. In Alzheimer's disease research, the AT8 antibody has proven to be an early indicator of tau protein pathology.<unk>  patients with the most AT8 pathology were least able to copy an image, while those best able to had the least neuritic pathology. Therefore, figure copying ability is highly correlated with Alzheimer's disease pathology.
Constructional apraxia
Ideomotor apraxia
Hemimotor neglect
Visuospatial dysgnosia
Neurapraxia
Pseudodyssynergia
Integrative agnosia
Dyssynergia