<unk>  can occur sporadically in families or be inherited in an autosomal dominant manner. Two genes associated with<unk>  are BMPR1A and SMAD4. Gene testing may be useful when trying to ascertain which non-symptomatic family members may be at risk of developing polyps, however having a known familial mutation would be unlikely to change the course of treatment. A known mutation may also be of use for affected individuals when they decide to start a family as it allows them reproductive choices. While mutations in the gene PTEN were also thought to have caused<unk> , it is now thought that mutations in this gene cause a similar clinical picture to<unk>  but are actually affected with Cowden<unk>  or other phenotypes of the PTEN hamartoma tumor<unk> .
Juvenile polyposis syndrome
Familial adenomatous polyposis
Serrated polyposis syndrome
Hereditary diffuse gastric cancer
Hereditary nonpolyposis colorectal cancer
MUTYH-associated polyposis
Peutz–Jeghers syndrome
Ogden syndrome