* Complete motor paralysis below the level of the lesion due to interruption of the corticospinal tract * Loss of pain and temperature sensation at and below the level of the lesion due to interruption of the spinothalamic tract * Retained proprioception and vibratory sensation due to intact dorsal columns * Autonomic dysfunction may be present and can manifest as hypotension (either orthostatic or frank hypotension), sexual dysfunction, and/or bowel and bladder dysfunction * Areflexia, flaccid internal and external anal sphincter, urinary retention and intestinal obstruction may also be present in individuals with<unk>  cord<unk> . Symptoms usually occur very quickly and are often experienced within one hour of the initial damage. MRI can detect the magnitude and location of the damage 10–15 hours after the initiation of symptoms. Diffusion-weighted imaging may be used as it is able to identify the damage within a few minutes of symptomatic onset. Clinical features include paraparesis or quadriparesis (depending on the level of the injury) and impaired pain and temperature sensation. Complete motor paralysis below the level of the lesion due to interruption of the corticospinal tract, and loss of pain and temperature sensation at and below the level of the lesion. Proprioception and vibratory sensation is preserved, as it is in the dorsal side of the<unk>  cord.
Anterior spinal artery syndrome
Vascular myelopathy
Lateral medullary syndrome
Occipital horn syndrome
Medial medullary syndrome
Central cord syndrome
Posterior cord syndrome
Acute flaccid myelitis