<unk>  is the pattern of damage associated with the destruction of adipose tissue by trauma, hypoxia, or lipase digestion (e.g. pancreatitis). In the classic case of<unk>  in severe acute pancreatitis, the<unk>  arises because adipocytes in the peritoneum are broken and digested by inappropriately activated pancreatic enzymes. Stored triglycerides in the adipocytes are released and split by pancreatic lipases into<unk> ty acids and glycerol molecules. The resultant<unk> ty acids react with extracellular calcium to make calcium soaps (i.e.<unk> ty acid salts) that give<unk>  its characteristic chalky-white appearance.<unk>  is an example of dystrophic calcification because the calcification occurs at normal serum calcium levels. Besides pancreatitis,<unk>  is also associated with conditions such as pancreatic carcinoma and pancreatic trauma. Although the peripancreatic region is the most commonly affected site in pancreatic disease (due to direct contact with enzymes), associated<unk>  can occur around the body in subcutaneous tissue, hand and foot joints, and bone marrow. These extrapancreatic complications are known as pancreatic panniculitis. Beyond saponification and calcification,<unk>  concludes with fibrosis and the formation of grey-yellow scar tissue. It is also possible for calcification to occur around the edges of a<unk> ty necrotic lesion, enclosing the<unk>  in a cyst. These "oil cysts" may persist for months to years without undergoing fibrosis. This is often the case in breast<unk> , where the underlying oil cysts can be visualised using sonography, X-Ray, CT, or MRI.
Fat necrosis
Liquefactive necrosis
Caseous necrosis
Coagulative necrosis
Infibulation
Fibrinoid necrosis
Pulp necrosis
Fibrosis