Location of the<unk> pendix in the digestive system Drawing of<unk> . Acute<unk>  seems to be the result of a primary obstruction of the<unk> pendix. Once this obstruction occurs, the<unk> pendix becomes filled with mucus and swells. This continued production of mucus leads to increased pressures within the lumen and the walls of the<unk> pendix. The increased pressure results in thrombosis and occlusion of the small vessels, and stasis of lymphatic flow. At this point, spontaneous recovery rarely occurs. As the occlusion of blood vessels progresses, the<unk> pendix becomes ischemic and then necrotic. As bacteria begin to leak out through the dying walls, pus forms within and around the<unk> pendix (suppuration). The result is<unk> pendiceal rupture (a 'burst<unk> pendix') causing peritonitis, which may lead to sepsis and in rare cases, death. These events are responsible for the slowly evolving abdominal pain and other commonly associated symptoms. The causative agents include bezoars, foreign bodies, trauma, intestinal worms, lymphadenitis and, most commonly, calcified fecal deposits that are known as<unk> pendicoliths or fecaliths. The occurrence of obstructing fecaliths has attracted attention since their presence in people with<unk>  is higher in developed than in developing countries. In addition an<unk> pendiceal fecalith is commonly associated with complicated<unk> . Fecal stasis and arrest may play a role, as demonstrated by people with acute<unk>  having fewer bowel movements per week compared with healthy controls. The occurrence of a fecalith in the<unk> pendix was thought to be attributed to a right-sided fecal retention reservoir in the colon and a prolonged transit time. However, a prolonged transit time was not observed in subsequent studies. Diverticular disease and adenomatous polyps was historically unknown and colon cancer was exceedingly rare in communities where<unk>  itself was rare or absent, such as various African communities. Studies have implicated a transition to a Western diet lower in fibre in rising frequencies of<unk>  as well as the other aforementioned colonic diseases in these communities. And acute<unk>  has been shown to occur antecedent to cancer in the colon and rectum. Several studies offer evidence that a low fiber intake is involved in the pathogenesis of<unk> . This low intake of dietary fiber is in accordance with the occurrence of a right-sided fecal reservoir and the fact that dietary fiber reduces transit time.
Appendicitis
Cholecystitis
Cervicitis
Psoas muscle abscess
Gastroenterocolitis
Abdominal aortic aneurysm
Proctocolitis
Adenitis