<unk>  <unk>  is an autosomal dominant type of<unk>  migraine that typically includes weakness of half the body which can last for hours, days, or weeks. It can be accompanied by other symptoms, such as ataxia, coma, and paralysis. Migraine attacks may be provoked by minor head trauma. Some cases of minor head trauma in patients with<unk>  migraine can develop into delayed cerebral edema, a life-threatening medical emergency. Clinical overlap occurs in some<unk>  patients with episodic ataxia type 2 and spinocerebellar ataxia type 6, benign<unk>  infantile epilepsy, and alternating<unk>  of childhood. Three genetic loci for<unk>  are known.<unk> 1, which accounts for about 50% of<unk>  patients, is caused by mutations in a gene coding for the P/Q-type calcium channel α subunit, CACNA1A.<unk> 1 is also associated with cerebellar degeneration.<unk> 2, which accounts for less than 25% of cases, is caused by mutations in the /-ATPase gene ATP1A2.<unk> 3 is a rare subtype of<unk>  and is caused by mutations in a sodium channel α-subunit coding gene, SCN1A. These three subtypes do not account for all cases of<unk> , suggesting the existence of at least one other locus <unk> 4). Also, nonfamilial cases of<unk>  migraine are seen, termed sporadic<unk>  migraine. These cases seem to have the same causes as the<unk>  cases and represent de novo mutations. Sporadic cases are also clinically identical to<unk>  cases with the exception of a lack of known family history of attacks.
Familial hemiplegic migraine
Hemiplegic migraine
Sporadic hemiplegic migraine
Mixed tension migraine
Cluster headache
Acephalgic migraine
Red wine headache
Trigeminal neuralgia