Although to date there have not been any studies specific to<unk>  in humans, two studies in animals and known renal physiology provide a likely mechanism. The elevated blood pressure seen in<unk>  is thought to be caused by the activation of the renin–angiotensin–aldosterone system (RAAS). Compression of the renal parenchyma alters intrarenal blood flow, resulting in a localized decrease in blood flow to the affected nephrons. This decrease in blood flow is recognized by the juxtaglomerular (JG) cells and promotes the cleavage of prorenin into renin. Renal hematoma locations with corresponding renal anatomy. Each kidney is surrounded by two layers of connective tissue, the inner renal capsule and outer renal (Gerota's) fascia. These layers thus form two potential spaces where fluid can form, the small subcapsular space and the large perinephric space. The renal capsule is fibrous and does not easily expand. Thus, even a small collection of blood in the subcapsular space can cause mass effect on the renal parenchyma.
Page kidney
Multicystic dysplastic kidney
Horseshoe kidney
Medullary sponge kidney
Crossed renal ectopia
Ectopic kidney
Glomerulocystic kidney disease
Medullary cystic kidney disease