The pathophysiology behind<unk>  remains speculative. Several studies indicate that insula and sylvian fissures also known as lateral sulcus could be related to inducing<unk> . Their location is found to coincide with the locations of the abdomen on the Sensory homunculus. Any pathophysiological changes in the M2 portion of cerebral artery which flows through lateral sulcus are associated with the<unk> es of the temporal and parietal regions. The<unk>  symptoms are believed to be associated with the transmission of impulses from the temporal lobe to the dorsal motor nucleus of the vagus nerve via the dense direct projections. The hypothalamus is also believed to induce sympathetic pathways from the amygdala in the medial temporal lobe to the GI tract to trigger such symptoms. Cerebral tumors have been assessed to be a plausible cause of<unk> . Right parietal and occipital encephalomalacia, biparietal atrophy , and bilateral perisylvian polymicrogyria has been possibly associated with ictal<unk>  pain associated with<unk> .
Abdominal epilepsy
Epilepsy in children
Sudden unexpected death in epilepsy
Epilepsy syndromes
Photosensitive epilepsy
Epilepsy
Generalized epilepsy
Temporal lobe epilepsy