<unk>  alters both the structure and function of the<unk>  as a result of the direct neurotoxic effects of alcohol intoxication or acute alcohol withdrawal. Increased alcohol intake is associated with<unk>  to<unk>  regions including the frontal lobe, limbic system, and cerebellum, with widespread cerebral atrophy, or<unk>  shrinkage caused by neuron degeneration. This<unk>  can be seen on neuroimaging scans. Frontal lobe<unk>  becomes the most prominent as alcoholics age and can lead to impaired neuropsychological performance in areas such as problem solving, good judgement, and goal-directed behaviors. Impaired emotional processing results from<unk>  to the limbic system. This may lead to troubles recognizing emotional facial expressions and “interpreting nonverbal emotional cues". Binge drinking, or heavy episodic drinking, can lead to<unk>  in the limbic system that occurs after a relatively short period of time. This<unk><unk>  increases the risk of alcohol-related dementia, and abnormalities in mood and cognitive abilities. Binge drinkers also have an increased risk of developing alcohol use disorder. Alcohol use disorder is also associated with many other health problems including memory disorders, high blood pressure, muscle weakness, heart problems, anaemia, low immune function, liver disease, disorders of the digestive system, and pancreatic problems. It has also been correlated with depression, unemployment, and family problems with an increased risk of domestic abuse. Gender and parental history of alcohol use disorder and binge drinking has an influence on susceptibility to alcohol dependence as higher levels are typically seen in males and in those with a family history.
Alcohol-related brain damage
Alcohol intoxication
Combined drug intoxication
Chronic solvent-induced encephalopathy
Alcohol withdrawal syndrome
Lithium toxicity
Toxic encephalopathy
Steroid dementia syndrome