There is no specific imaging finding for<unk> , and the diagnosis is a clinicopathological one, based on presentation and histological findings. Microscopic pathology At the microscopic level,<unk>  shows a proliferation of dermal fibroblasts and dendritic cells, thickened collagen bundles, increased elastic fibers, and deposits of mucin. More recent case reports have described the presence of sclerotic bodies (also known as elastocollagenous balls) in skin biopsies from<unk>  patients. While not universally present, this finding is believed to be unique to patients exposed to gadolinium, although not necessarily limited to areas involved by<unk> . Differential diagnosis The differential diagnoses for<unk>  include diffuse cutaneous or limited cutaneous<unk>  sclerosis, scleromyxedema, lipodermatosclerosis, scleroedema diabeticorum, graft versus host disease, eosinophilic fasciitis; eosinophilia-myalgia syndrome; porphyria cutanea tarda, and other disorders. The nearly universal absence of facial skin involvement in<unk> , presence of yellow plaques on the sclera of the eyes, absence of Raynaud’s phenomenon, and other differences in presentation can aid the proper diagnosis. History of exposure to GBCAs would favor<unk>  as the differential diagnosis.
Nephrogenic systemic fibrosis
Goodpasture syndrome
HIV-associated nephropathy
Eosinophilic granulomatosis with polyangiitis
Granulomatosis with polyangiitis
Thin basement membrane disease
Mixed connective tissue disease
IgA vasculitis