Although the mechanism of action of<unk>  is unknown, research involving humans and animals has shown that<unk>  does not achieve anti-inflammatory response by stimulation of the adrenal or pituitary gland, but it has shown<unk>  restrains prostaglandin synthetase in vitro and reduces plasma levels of prostaglandin E, possibly causing the anti-inflammatory response. When tested in rats<unk>  prevented experimentally stimulated polyarthritis and reduced inflammation. In patients with rheumatoid arthritis or osteoarthritis<unk>  restrained disease activity as efficiently as aspirin and indometacin, although the occurrence of mild gastrointestinal adverse effects and tinnitus was lower in patients treated with<unk>  than it was with aspirin-treated patients and the occurrence of adverse effects of the central nervous system was lower with<unk>  than it was with indometacin.
Tolmetin
Cromoglicic acid
Tralomethrin
Cyfluthrin
Lisocabtagene maraleucel
Betamethasone dipropionate
Coralmyn
Vilanterol