<unk>  refers to shingles of the geniculate ganglion, a portion of the facial nerves. After the initial infection known as chickenpox, varicella zoster virus lies dormant in nerve cells in the body, where it is kept in check by the immune system. If the immune system is suppressed in any way (for example during an illness, while experiencing stress or undergoing chemotherapy), the virus travels to the end of the nerve cell If the nerve cells affected lie within the facial nerves, it causes the symptoms described above and is referred to as<unk>  or simply RHS.<unk>  is estimated to account for 12% of all facial nerve paralysis. It occurs in both immunocompetent and immunocompromised individuals with immunocompromised patients often having more severe disease presentation. RHS may occur in any age group with cases reported in patients ranging in age from 3 months to 82 years. The affected ganglion is responsible for the movements of facial muscles, the touch sensation of a part of ear and ear canal, the taste function of the frontal two-thirds of the tongue, and the moisturization of the eyes and the mouth. The syndrome specifically refers to the combination of this entity with weakness of the muscles activated by the facial nerve. In isolation, the latter is called Bell's palsy. However, as with shingles, the lack of lesions does not definitely exclude the existence of a herpes infection. Even before the eruption of vesicles, varicella zoster virus can be detected from the skin of the ear.
Ramsay Hunt syndrome type 2
Bell's palsy
Ramsay Hunt syndrome type 1
Tolosa–Hunt syndrome
Robinow syndrome
Autosomal dominant porencephaly type I
Seaver Cassidy syndrome
Dandy–Walker malformation