A fasting blood sugar level of ≥ 7.0 mmol / L is used in the general diagnosis of diabetes. There are no clear guidelines for the diagnosis of<unk> , but the criteria often used are that the patient should develop the disease in<unk> ood, not need insulin treatment for the first 6 months after diagnosis and have autoantibodies in the blood. Glutamic acid decarboxylase autoantibody (GADA), islet cell autoantibody (ICA), insulinoma-associated (IA-2) autoantibody, and zinc transporter autoantibody (ZnT8) testing should be performed in order to correctly diagnose diabetes. Persons with<unk>  typically have low, although sometimes moderate, levels of C-peptide as the disease progresses. Those with insulin resistance or type 2 diabetes are more likely to have high levels of C-peptide due to an over production of insulin. Autoantibodies Destruction of Glutamate decarboxylase (pictured here) via autoantibodies is strongly linked with<unk>  type 1 diabetes. Glutamic acid decarboxylase autoantibodies (GADA), islet cell autoantibodies (ICA), insulinoma-associated (IA-2) autoantibodies, and zinc transporter autoantibodies (ZnT8) are all associated with<unk> ; GADAs are commonly found in cases of diabetes mellitus type 1. The presence of islet cell complement fixing autoantibodies also aids in a differential diagnosis between<unk>  and type 2 diabetes. Persons with<unk>  often test positive for ICA, whereas type 2 diabetics only seldom do. Persons with<unk>  usually test positive for glutamic acid decarboxylase antibodies, whereas in type 1 diabetes these antibodies are more commonly seen in<unk>  rather than in children. In addition to being useful in making an early diagnosis for type 1 diabetes mellitus, GAD antibodies tests are used for differential diagnosis between<unk>  and type 2 diabetes and may also be used for differential diagnosis of gestational diabetes, risk prediction in immediate family members for type 1, as well as a tool to monitor prognosis of the clinical progression of type 1 diabetes.
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