Schematic and FMRI of cortical areas involved in pain processing which are similar to areas that are involved in<unk> s.<unk> s are the result of a dysfunctional somatosensory and a dysfunctional awareness regions of the brain.<unk>  sensory input is produced and conducted through the spinal cord and thalamus and it is received at the primary somatosensory cortex. Once it has reached the primary somatosensory cortex, it is distributed across the brain and it will not be processed unless it is important and one pays close attention to the information based on a specific context. Consciousness to these specific<unk>  sensations is generated only through multiple feedback loops passing through higher cortical areas such as secondary somatosensory area, parietal, insular cortex and premotor areas. The intensity of the<unk>  stimulus is directly proportional to the area of the primary somatosensory region activated. A feedback mechanism from different cortical areas results in the awareness of touch. Even with complete sensory deprivation, discrete<unk>  memories can trigger spontaneous firing of impaired neurons. Therefore, individuals with various psychiatric disorders are more prone to<unk> s than normal individuals. :<unk> s are especially possible due to faulty sensory integration of neuronal signals in the primary and secondary somatosensory system with neuronal signals in the parietal cortex, insular cortex and premotor cortex. Moreover, the posterior insula is responsible for mental body schema representation and can produce<unk>  if defected. Additionally, the regions of the brain involved in<unk> s are similar to the regions of the brain involved in pain.
Tactile hallucination
Autophobia
Pain asymbolia
Neophobia
Lexical–gustatory synesthesia
Emotional detachment
Hallucination
Automatism 