The etiology of<unk>  is unknown, but it has been suggested that is due to increased hydrostatic pressure, general weakened connective tissue or as a result of the pulsatile flow of cerebrospinal fluid on weakened spinal dura.<unk>  is common in Marfan syndrome, occurring in 63–92% of people with the syndrome. It may also occur in Ehlers-Danlos Syndrome, neurofibromatosis type I, ankylosing spondylitis, and is associated with spondylolisthesis, vertebral fractures, scoliosis, tumors or trauma. In neurofibromatosis type I, it has been theorized that local infiltration of the dura by plexiform neurofibromas leads to a weakening of the dural allowing the outpouching. A retrospective study found that a majority of<unk>  were associated with nearby plexiform neurofibromas.
Dural ectasia
Arteriovenous fistula
Pseudoaneurysm
Dural arteriovenous fistula
Developmental venous anomaly
Jugular vein ectasia
Collapsed vein
Vascular ring