EILO may arise because of a relative mechanical 'insufficiency' of the<unk>  structures that should act to maintain glottic patency. It has been proposed that a narrowing at the<unk>  inlet during the state of high airflow (e.g. when running fast), can act to cause a pressure drop across the larynx which then acts to 'pull' the<unk>  structures together. The Bernoulli principle states that increasing airflow through a tube creates increasing negative pressures within that tube. Complex neuromuscular functioning is required to maintain<unk>  opening and to allow the larynx to achieve a great number of tasks (i.e. speaking, airway protection, swallowing). It is thus also possible that EILO may arise as form a degree of neuromuscular failure. A small heredity study indicated that an autosomal dominant model of inheritance with variable expressivity and reduced penetrance in males may be relevant; because in ten families studied, there was at least one affected person in every generation in which both parents were examined. Further work is needed to determine if structural deficiencies in the<unk>  tissue of individuals with EILO are present.
Exercise-induced laryngeal obstruction
Laryngopharyngeal reflux
Odontoma dysphagia syndrome
Esophageal motility disorder
Retropharyngeal abscess
Subglottic stenosis
Parapharyngeal abscess
Esophageal food bolus obstruction