Some individuals may benefit from diet modification, such as a reduced fat diet, following cholecystectomy. The liver produces bile and the gallbladder acts as reservoir. From the gallbladder, bile enters the intestine in individual portions. In the absence of a gallbladder, bile enters the intestine constantly, but in small quantities. Thus, it may be insufficient for the digestion of fatty foods.<unk>  treatment depends on the identified violations that led to it. Typically, the patient is recommended a dietary restriction table with fatty foods, enzyme preparations, antispasmodics, and sometimes cholagogue. If the pain is caused by biliary microlithiasis, oral ursodeoxycholic acid can alleviate the condition. A trial of bile acid sequestrant therapy is recommended for bile acid diarrhea. Functional dyspepsia is subdivided into Epigastric Distress Syndrome (EPS) and Post-Prandial Distress Syndrome (PDS). Treatment for EPS and PDS can both include proton pump inhibitors and dopamine antagonists. Tricyclic antidepressants have also been proven effective for nausea, vomiting, early satiety, impaired motility and other related symptoms. When investigation reveals no abnormalities within the abdominal cavity, the attending physician may consider Anterior cutaneous nerve entrapment syndrome (ACNES) as a possible cause. ACNES may present with pseudovisceral symptoms, including nausea, bloating, diarrhea and early satiety.
Postcholecystectomy syndrome
Gallstone ileus
Abdominal compartment syndrome
Abdominal aortic aneurysm
Superior mesenteric artery syndrome
Pellegrini–Stieda syndrome
Spontaneous bacterial peritonitis
Nutcracker syndrome