The NIH states: "The causes of most cases of<unk>  are still open to debate. Some researchers suggest that certain people may be more sensitive to the body’s normal release of the hormone epinephrine, which causes many of the symptoms of hypoglycemia. Others believe deficiencies in glucagon secretion might lead to<unk> . Several other hormones are responsible for modulating the body's response to insulin, including cortisol, growth hormone and sex hormones. Untreated or under-treated hormonal disorders such as adrenal insufficiency (see also Addison's disease) or growth hormone deficiency can therefore sometimes cause insulin hypersensitivity, and<unk> . Stomach bypass surgery or hereditary fructose intolerance are believed to be causes, albeit uncommon, of<unk> . Myo-inositol or 1D-chiro-inositol withdrawal can cause temporary<unk> . There are several kinds of<unk> : # Alimentary hypoglycemia (consequence of dumping syndrome; it occurs in about 15% of people who have had stomach surgery) # Hormonal hypoglycemia (e.g., hypothyroidism) # Helicobacter pylori-induced gastritis (some reports suggest this bacteria may contribute to the occurrence of<unk>  # Congenital enzyme deficiencies (hereditary fructose intolerance, galactosemia, and leucine sensitivity of childhood) # Late hypoglycemia (occult diabetes; characterized by a delay in early insulin release from pancreatic beta-cells, resulting in initial exaggeration of hyperglycemia during a glucose tolerance test) "Idiopathic<unk> " is a term no longer used because researchers now know the underlying causes of<unk>  and have the tools to perform the diagnosis and the pathophysiological data explaining the mechanisms. To check if there is real hypoglycemia when symptoms occur, neither an oral glucose tolerance test nor a breakfast test is effective; instead, a hyperglucidic breakfast test or ambulatory glucose testing is the current standard. The body requires a relatively constant input of glucose, a sugar produced upon digestion of carbohydrates, for normal functioning. Glucagon and insulin are among the hormones that ensure a normal range of glucose in the human body. Upon consumption of a meal, blood sugar normally rises, which triggers pancreatic cells to produce insulin. This hormone initiates the absorption of the just-digested blood glucose as glycogen into the liver for metabolism or storage, thereby lowering glucose levels in the blood. In contrast, the hormone glucagon is released by the pancreas as a response to lower than normal blood sugar levels. Glucagon initiates uptake of the stored glycogen in the liver into the bloodstream so as to increase glucose levels in the blood. Sporadic, high-carbohydrate snacks and meals are deemed the specific causes of sugar crashes. The “crash” one feels is due to the rapid increase and subsequent decline of blood sugar in the body system as one begins and ceases consumption of high-sugar foods. More insulin than is actually needed is produced in response to the large, rapid ingestion of sugary foods.
Reactive hypoglycemia
Hereditary fructose intolerance
Ketotic hypoglycemia
Insulinoma
Hyperinsulinemic hypoglycemia
Diabetic hypoglycemia
Idiopathic hypoglycemia
Stress hyperglycemia