<unk>  has been shown to form as a result of tumor formation, especially in the posterior third ventricle, trauma, lesions, cerebral infarction, encephalitis as a result of herpes simplex, and Landau-Kleffner syndrome. The exact location of damage which results in pure word deafness is still under debate, but the planum temporale, posterior STG, and white matter damage to the acoustic radiations (AR) have all been implicated.<unk>  is rarely diagnosed in its pure form.<unk>  can both present as the result of acute damage or as chronic, progressive degeneration over time. Cases have been documented that result from severe acute head trauma resulting in bilateral temporal lobe damage. In contrast,<unk>  has also been documented to present progressively over several years. In one such case, the patient exhibited progressive word deafness over a 9-year period but did not exhibit any other cognitive of mental deterioration. MRIs showed cortical atrophy in the left superior temporal lobe region. In childhood,<unk>  can also be caused by Landau-Kleffner syndrome, also called acquired epileptic aphasia. It is often the first symptom of this disease. A review of 45 cases suggested a relationship between prognosis and age of onset with poorer prognosis for those with earlier onset. In extremely rare cases,<unk>  has been known to present as a symptom of neurodegenerative disease, such as Alzheimer's disease. In such cases<unk>  is a symptom that is typically followed by more severe neurological symptoms typical of Alzheimer's disease.
Auditory verbal agnosia
Visuospatial dysgnosia
Apperceptive agnosia
Integrative agnosia
Hemispatial neglect
Phonagnosia
Associative visual agnosia
Social-emotional agnosia