<unk>  is an inherited genetic syndrome, but the way in which this disorder is inherited is unclear. Frequently (17-49%) those with<unk>  have relatives with a history of epileptic seizures. There is also a higher rate of females showing<unk>  symptoms than males. The majority of<unk>  cases have an onset in early childhood to puberty. According to voxel-based morphometry there are abnormalities in grey matter volume in cortical as well as subcortical construction. Proton magnetic resonance spectroscopy (MRS) revealed that there is dysfunction of thalamic neurons. Integrity of white matter (WM) was interrupted by in the corpus callosum and frontal WM tracts. Further MRI divulged that there are abnormality in both GM and WM structure tract. Voxel-based morphometry (VBM), is one of the most extensively used structural MRI analytic methods. It is used to detect detects regionally specific differences in GM volume or concentration on a whole-brain voxel-wise. In a study with patients,<unk>  showed an increase in nasofrontal GM in<unk>  patients relative to matched controls, suggesting the possibility that<unk>  patients may have structural abnormalities of the frontal lobe.<unk>  is the characteristically genetic origin, the variant which causes the disease need to first positional cloning and lately next-generation sequencing. There are six genes GABRA1, GABRD, EFHC1, BRD2, CASR, and ICK are considered as major vulnerable alleles for<unk> .
Juvenile myoclonic epilepsy
Progressive myoclonus epilepsy
Benign infantile epilepsy
Childhood absence epilepsy
Benign familial infantile epilepsy
Musicogenic epilepsy
Northern epilepsy syndrome
Vertiginous epilepsy