Mechanism of action Like niacin,<unk>  acts on the niacin receptor 1, inhibiting the enzyme triglyceride lipase. This reduces the concentration of fatty acids in the blood plasma and their inflow into the liver. Consequently, VLDL cholesterol production in the liver is reduced, which leads indirectly to a reduction in LDL and increase in HDL cholesterol. Pharmacokinetics<unk>  is completely absorbed from the gut. It is not bound to blood plasma proteins and not metabolized. Elimination occurs in two phases, the first having a half-life of two hours, the second of 12 to 14 hours. The substance is eliminated via the kidney.
Acipimox
Clostebol propionate
Tezacaftor
Estropipate
Ladostigil
Progabide
Maralixibat chloride
Clostebol caproate