In order for<unk> ses to occur, the fungus must directly contact the skin. Likelihood of infection is increased if the skin integrity is compromised, as in minor breaks. The fungi use various proteinases to establish infection in the keratinized stratum corneum. Some studies also suggest that a class of proteins called LysM coat the fungal cell walls to help the fungi evade host cell immune response. The course of infection varies between each case, and may be determined by several factors including: "the anatomic location, the degree of skin moisture, the dynamics of skin growth and desquamation, the speed and extent of the inflammatory response, and the infecting species." The ring shape of<unk>  lesions result from outward growth of the fungi. The fungi spread in a centrifugal pattern in the stratum corneum, which is the outermost keratinized layer of the skin. For nail infections, the growth initiates through the lateral or superficial nail plates, then continues throughout the nail. For hair infections, fungal invasion begins at the hair shaft. Symptoms manifest from inflammatory reactions due to the fungal antigens. The rapid turnover of desquamation, or skin peeling, due to inflammation limits<unk> ses, as the fungi are pushed out of the skin.<unk> ses rarely cause serious illness, as the fungi infection tends to be limited to the superficial skin. The infection tends to self-resolve so long as the fungal growth does not exceed inflammatory response and desquamation rate is sufficient. If immune response is insufficient, however, infection may progress to chronic inflammation.
Dermatophyte
Speculum 
Diatragacanth
Papule
Schistocyte
Leper colony
Emerods
Microgenia