<unk>  <unk>  is an intermediate form of diabetes that has some characteristics of type 1 and some of type 2 diabetes. Type 1 diabetes involves autoimmune destruction of pancreatic beta cells which create insulin. This occurs earlier in a person's life, leading to patients being insulin dependent, and the lack of natural insulin makes patients<unk>  to a condition called diabetic ketoacidosis (DKA). Type 2 diabetes is different in that it is usually caused by insulin resistance in the body in older patients leading to beta cell burnout over time, and is not<unk>  to DKA.<unk>  is a condition that involves DKA like type 1, but occurs later in life and can regain beta cell function like type 2 diabetes. However, it is distinct from latent autoimmune diabetes of adults (LADA), a form of type 1 sometimes referred to as type 1.5 that does not occur with DKA. There are also distinctions to be made between<unk>  and LADA as patients who exhibit<unk>  symptoms can regain beta cell function similar to type 2 diabetics whereas LADA will not exhibit this reclamation of beta cell function.<unk>  is readily diagnosible because it presents a single characteristic, ketoacidosis, which confirms it as<unk> .<unk>  comes in four forms depending upon the presence or absence of β-cell autoantibodies (A+ or A−) and β-cell functional reserve (β+ or β−). Other styles of classification have been used for<unk> , including styles incorporating BMI, but the Aβ system has been found to have the highest accuracy and predictive value of all the systems utilized. The autoantibodies used to diagnose the A+ subtypes of<unk>  include the autoantibodies detected in patients with type 1 diabetes, including Glutamic Acid Decarboxylase 65 (GAD65), Zinc Transporter T8 (ZnT8), Islet Antigen-2 (IA-2), and HLA class II type 1 diabetes susceptibility alleles.
Ketosis-prone diabetes
Diabetic hypoglycemia
Lifestyle causes of type 2 diabetes
Idiopathic hypoglycemia
Steroid diabetes
Lipoatrophic diabetes
Non-islet cell tumor hypoglycemia
Reactive hypoglycemia