Pyrethroids, including<unk> , disrupt the functioning of the nervous system in an organism. They are fast-acting axonic excitotoxins, the toxic effects of which are mediated by preventing the closure of the voltage-gated sodium channels in axonal membranes. The sodium channel is a membrane protein with a hydrophilic interior. This interior is shaped precisely to allow sodium ions to pass through the membrane, enter the axon, and propagate an action potential. The binding of the insecticide to the protein keeps the channels in their open state, so the nerves cannot repolarize, thereby paralyzing the organism. One consequence of the mode of action is that<unk>  has useful knockdown properties. That is, affected larvae rapidly cease feeding and may fall off the crop; flying insects drop to the ground. Paralysis and death follows provided the insect has absorbed a sufficient dose.
Cyhalothrin
Diflorasone diacetate
Clobetasone
Ipriflavone
Flumetasone
Fluticasone furoate
Tralomethrin
Fluticasone propionate