The force of a blow to the orbit is dissipated by a fracture of the surrounding bone, usually the orbital floor and/or the medial orbital wall. In<unk>  fractures, the medial wall is fractured indirectly. When an external force is applied to the orbital cavity from an object whose diameter is larger than that of the orbit, the orbital contents are retropulsed and compressed. The consequent sudden rise in intraorbital pressure is transmitted to the walls of the orbit, which ultimately leads to fractures of the thin medial wall and/or orbital floor. Theoretically, this mechanism should lead to more fractures of the medial wall than the floor, since the medial wall is slightly thinner (0.25 mm vs 0.50 mm). However, it is known that pure<unk>  fractures most frequently involve the orbital floor. This may be attributed to the honeycomb structure of the numerous bony septa of the ethmoid sinuses, which support the lamina papyracea, thus allowing it to withstand the sudden rise in intraorbital hydraulic pressure better than the orbital floor. In children, the flexibility of the actively developing floor of the orbit fractures in a linear pattern that snaps backward. This is commonly referred to as a trapdoor fracture. The trapdoor can entrap soft-tissue contents, thus causing permanent structural change that requires surgical intervention.
Orbital blowout fracture
Orbital apex syndrome
Nasal fracture
Orbital emphysema
Le Fort fracture of skull
Nasal septal hematoma
Labyrinthine fistula
Orbital cellulitis