Factors that identify etiology of the patient include age, duration of lymphadenopathy, external exposures, associated symptoms and location on the body. +Medications negatively associated with lymphadenopathies Atenolol Hydralazine Penicillins Pyrimethamine (Daraprim) Captopril Allopurinol Phenytoin (Dilantin) Quinidine Carbamazepine (Tegretol) Primidone (Mysoline Trimethoprim/sulfamethoxazole Beta blockers such as Atenolol or ACE inhibitors like Captopril, can cause certain lymphadenopathies for some individuals. Captopril is an analog to proline and completely inhibits angiotensin converting enzymes (ACE) and as a result decreases angiostatin II production. It can also inhibit tumor angiogenesis through MMPs and endothelial cell migration, which can ultimately cause lymph node enlargement. Carbamazepine is an anticonvulsant that works by decreasing nerve impulses that are brought on by seizures. Some of the more serious side effects are allergic skin reactions and low blood cell counts. Other anticonvulsant medications, Phenytoin and Primidone, can also cause lymph node enlargement due to changes in the blood after drug administration. Other medications such as Pyrimethamine, Quinidine and Trimethoprim/ sulfamethoxazole antibiotics also change blood chemistry after administration and can cause lymph node enlargement. Hydralazine and Allopurinol are medications that are prescribed to patients to lower their blood pressure and are vasodilators causing the blood cells to dilate. A family history, regional exam and epidemiological cues are usually the most useful information to utilize for treatment options because it can help classify the patient's condition as either low risk, or high risk. Low risk, meaning the patient is not exposed to malignancy or serious disease and high risk means that they are. If the patient is not at risk for malignancy or serious illness, it is recommended for the physician to observe any changes or symptom resolutions within 3- 4 weeks. If the lymphadenopathy does not resolve the next step would be getting a biopsy. Proceeding a tissue sample, an effective treatment for<unk>  is surgical removal of the lesion after an initial conformation of the disease based on the patients biopsy results.
Follicular hyperplasia
Lymphoid hyperplasia
Perifollicular mucinosis
Microglandular hyperplasia
Hyperkeratinization
Pseudopolyps
Xanthogranulomatous inflammation
Reticular formation