The pathophysiology of<unk>  leads to changes in the structure of the kidney that are most noticeable in the tubular tissue. If the<unk>  is longstanding and severe, the glomerular filtration rate in the affected kidneys never recovers and (prerenal) kidney failure is the result. Changes include: * Fibrosis * Tubular cell size (decrease) * Thickening of Bowman capsule * Tubulosclerosis * Glomerular capillary tuft (atrophy)
Renal artery stenosis
Renovascular hypertension
Juxtaglomerular cell tumor
Hypertensive kidney disease
Benign nephrosclerosis
Polycystic kidney disease
Renal vein thrombosis
Primary aldosteronism