Anatomical illustration of human neuroanatomy There is no general consensus on the definition of CBS. Predominant factors correlated with CBS are a decrease of<unk>  acuity,<unk>  field loss, and elderly age. While characteristic features of<unk>  hallucinations are not specifically linked to the anatomical site of the ocular injury, they usually match to the location of<unk>  loss. The most commonly accepted theory for Charles Bonnet Syndrome proposes that extreme<unk>  impairment promotes sensory deafferentation, leading to disinhibition, thus resulting in sudden neural firings of the<unk>  cortical regions. A few studies record that<unk>  hallucinations are likely to be concentrated in the blind regions. Functional magnetic resonance imaging (fMRI) of Charles Bonnet Syndrome patients displays a relationship between<unk>  hallucinations and activity in the ventral occipital lobe. A connection between age-related macular degeneration (AMD) and colored<unk>  hallucinations has been presented. Color vision signals travel through the parvocellular layers of the lateral geniculate nucleus (LGN), later transmitting down the color regions of the ventral<unk>  pathway. Due to cone photoreceptor damage located in the macula, there is a significant reduction of<unk>  input to the<unk>  association cortex, stirring endogenous activation in the color areas and thus leading to colored hallucinations. Patients with CBS alongside macular degeneration exhibit hyperactivity in the color areas of the<unk>  association cortex (as shown in fMRIs). Those who have significant ocular disease yet maintain<unk>  acuity may still be susceptible to CBS. The Deep Boltzmann Machine (DBM) is a way of utilizing an undirected probabilistic process in a neural framework. Researchers argue that the DBM has the ability to model features of cortical learning, perception, and the<unk>  cortex (the locus of<unk>  hallucinations). Compelling evidence details the role homeostatic operations in the cortex play in regards to stabilizing neuronal activity. By using the DBM, researchers show that when sensory input is absent, neuron excitability is influenced, thus potentially triggering complex hallucinations. Acetylcholine Pathway A short-term change in the levels of feedforward and feedback flows of information may intensely affect the presence of hallucinations. In periods of drowsiness, CBS related hallucinations are more prone to arise. Disrupting cortical homeostatic processes after vision has been lost may prevent or setback the emergence of hallucinations. At varying stages of the cortical grading, acetylcholine (ACh) may impact the balance of thalamic and intracortical inputs as well as the balance in between bottom-up and top-down. Particularly in CBS, a shortage of acetylcholine at cortical locations should correspond to the onset of hallucinations. The syndrome can also develop after bilateral optic nerve damage due to methyl alcohol poisoning.
Visual release hallucinations
Wandering 
Musical hallucinations
Bullying
Self-hatred
Somatic experiencing
Callous and unemotional traits
Fear of trains