Since the clinical manifestations of each form of<unk>  are unique and depend to a large extent on the underlying enzyme defects, their precursor retention and defective products, the therapeutic goal of<unk>  is to replenish insufficient adrenal hormones and suppress excess of precursors. Treatment of all forms of<unk>  may include any of: # Supplying enough glucocorticoid to reduce hyperplasia and overproduction of androgens or mineralocorticoids # Providing replacement mineralocorticoid and extra salt if the person is deficient # Providing replacement testosterone or estrogens at puberty if the person is deficient # Additional treatments to optimize growth by delaying puberty or delaying bone maturation If<unk>  is caused by the deficiency of the 21-hydroxylase enzyme, then treatment aims to normalize levels of main substrate of the enzyme - 17α-hydroxyprogesterone.
Congenital adrenal hyperplasia
Polycystic ovary syndrome
Lipoid congenital adrenal hyperplasia
Late onset congenital adrenal hyperplasia
Persistent adrenarche syndrome
Congenital iodine deficiency syndrome
Congenital hypothyroidism
Congenital hyperinsulinism