<unk>  The pathophysiological characteristics of<unk>  are due to a thickened, fibrotic pericardium that forms a non-compliant shell around the heart. This shell prevents the heart from expanding when blood enters it. This results in significant respiratory variation in blood flow in the chambers of the heart. During inspiration, pressure in the thoracic cavity decreases but is not relayed to the left atrium, subsequently a reduction in flow to the left atrium and ventricle happens. During diastole, less blood flow in left ventricle allows for more room for filling in right ventricle and therefore a septal shift occurs. During expiration, the amount of blood entering the left ventricle will increase, allowing the interventricular septum to bulge towards the right ventricle, decreasing the right heart ventricular filing.
Constrictive pericarditis
Tuberculous pericarditis
Acute pericarditis
Ischemic cardiomyopathy
Uremic pericarditis
Myopericarditis
Arrhythmogenic cardiomyopathy
Aicardi syndrome