Unlike testosterone and androstenedione, androgens produced by the<unk><unk> , i.e. DHT and 11-oxyandrogens, cannot be converted by aromatase into estrogens. The<unk><unk>  is not always considered in the clinical evaluation of patients with hyperandrogenism. Ignoring this<unk>  may lead to diagnostic pitfalls and confusion, for example, in late onset congenital adrenal hyperplasia, where testosterone levels may be normal amid the symptoms of hyperandrogenism like hirsutism and acne.
Androgen backdoor pathway
Natural apophyseal glides
Androgen-binding protein
Progestin-induced virilization
Androgen suppression
Adipose-derived hormones
Pharmacokinetics of estradiol
Pharmacokinetics of testosterone