Acquired Acquired<unk>  or dyschromatopsia is a condition associated with damage to the diencephalon—primarily the thalamus of the mid brain—or the cerebral cortex—the new brain—specifically the fourth visual association area, V4 which receives information from the parvocellular pathway involved in color processing. Thalamic<unk>  or dyschromatopsia is caused by damage to the thalamus; it is most frequently caused by tumor growth since the thalamus is well protected from external damage. Cerebral<unk>  is a form of acquired color blindness that is caused by damage to the cerebral cortex of the brain, rather than abnormalities in the cells of the eye's retina. It is most frequently caused by physical trauma, hemorrhage or tumor tissue growth. Congenital The known causes of the congenital forms of<unk>  are due to malfunction of the retinal phototransduction pathway. Specifically, this form of<unk>  seems to result from the inability of cone cells to properly respond to light input by hyperpolarizing. Known genetic causes of this include mutations in the cone cell cyclic nucleotide-gated ion channels CNGA3 (ACHM2) and CNGB3 (ACHM3), the cone cell transducin, GNAT2 (ACHM4), subunits of cone phosphodiesterase PDE6C (ACHM5, OMIM 613093) and PDEH (ACHM6, OMIM 610024), and ATF6 (ACHM7, OMIM 616517).
Achromatopsia
Cerebral polyopia
Ocular dysmetria
Macropsia
Xanthopsia
Oscillopsia
Akinetopsia
Cyanopsia