Those affected with<unk>  can have diagnosis achieved via noting an increase of erythrocyte sedimentation rate, as well as the following: * Genetic test * Radiological findings * Clinical findings
Deficiency of the interleukin-1–receptor antagonist
Leukocyte adhesion molecule deficiency
Terminal complement pathway deficiency
Neutrophil-specific granule deficiency
Acquired C1 esterase inhibitor deficiency
Mendelian susceptibility to mycobacterial disease
CD25 deficiency
Immunodeficiency with hyperimmunoglobulin M