<unk>  is usually symptomless at birth. It usually manifests a few weeks after birth.<unk>  is an acyanotic congenital heart defect, aka a left-to-right shunt, so there are no signs of cyanosis in the early stage. However, uncorrected<unk>  can increase pulmonary resistance leading to the reversal of the shunt and corresponding cyanosis. * Pansystolic (Holosystolic) murmur along lower left sternal border (depending upon the size of the defect) +/- palpable thrill (palpable turbulence of blood flow). Heart sounds are normal. Larger<unk> s may cause a parasternal heave, a displaced apex beat (the palpable heartbeat moves laterally over time, as the heart enlarges). An infant with a large<unk>  will fail to thrive and become sweaty and tachypnoeic (breathe faster) with feeds. The restrictive<unk> s (smaller defects) are associated with a louder murmur and more palpable thrill (grade IV murmur). Larger defects may eventually be associated with pulmonary hypertension due to the increased blood flow. Over time this may lead to an Eisenmenger's syndrome the original<unk>  operating with a left-to-right shunt, now becomes a right-to-left shunt because of the increased pressures in the pulmonary vascular bed.
Ventricular septal defect
Heart septal defect
Atrioventricular septal defect
Atrial septal defect
Aortopulmonary septal defect
Tricuspid atresia
Double aortic arch
Double inlet left ventricle