As a derivative of EDTA,<unk>  chelates iron and thus reduces the number of metal ions complexed with anthracycline and, consequently, decrease the formation of superoxide radicals. The exact chelation mechanism is unknown, but it has been postulated that<unk>  can be converted into ring-opened form intracellularly and interfere with iron-mediated free radical generation that is in part thought to be responsible for anthryacycline induced cardiomyopathy. It was speculated that<unk>  could be used for further investigation to synthesize new antimalarial drugs.
Dexrazoxane
Trenbolone acetate
Metenolone
Ibandronic acid
Finerenone
Trenbolone
Mitotane
Trestolone