<unk>  on computed tomography (left) and cavernous transformation of the<unk> <unk>  after 1 year (right) The diagnosis of<unk>  is usually made with imaging confirming a clot in the<unk> <unk> ; ultrasound is the least invasive method and the addition of Doppler technique shows a filling defect in blood flow.<unk>  may be classified as either occlusive or nonocclusive based on evidence of blood flow around the clot. An alternative characterization based on site can be made: Type 1 is limited to the main<unk> <unk> , Type 2 involves only a<unk> <unk>  branch (2a, or 2b if both branches are affected), and Type 3 if clot is found throughout both areas. Determination of condition severity may be derived via computed tomography (CT) with contrast, magnetic resonance imaging (MRI), or MR angiography (MRA). Those with chronic<unk>  may undergo upper endoscopy (esophagogastroduodenoscopy, EGD) to evaluate the presence of concurrent dilated<unk> s (varices) in the stomach or esophagus. Other than perhaps slightly elevated transaminases, laboratory tests to evaluate liver function are typically normal. D-dimer levels in the blood may be elevated as a result of fibrin breakdown. On duplex ultrasound, demonstration of echogenic material within the<unk> <unk> , complete or partial absence of colour flow in the<unk> <unk> , presence of collateral vessels around the<unk> <unk>  or gall bladder that bypass the<unk> <unk> .
Portal vein thrombosis
Phlebothrombosis
Hepatic artery thrombosis
Deep vein thrombosis
Septic thrombophlebitis
Varicose veins
Pseudothrombophlebitis
Superficial vein thrombosis