<unk>  has an abrupt onset and a monophasic course. Symptoms usually begin 1–3 weeks after infection. Major symptoms include fever, headache, nausea and vomiting, confusion, vision impairment, drowsiness, seizures and coma. Although initially the symptoms are usually mild, they worsen rapidly over the course of hours to days, with the average time to maximum severity being about four and a half days. Additional symptoms include hemiparesis, paraparesis, and cranial nerve palsies.<unk>  in COVID-19 Neurological symptoms were the main presentation of COVID-19, which did not correlate with the severity of respiratory symptoms. The high incidence of<unk>  with hemorrhage is striking. Brain inflammation is likely caused by an immune response to the disease rather than neurotropism. CSF analysis was not indicative of an infectious process, neurological impairment was not present in the<unk>  phase of the infection, and neuroimaging findings were not typical of classical toxic and metabolic disorders. The finding of bilateral periventricular relatively asymmetrical lesions allied with deep white matter involvement, that may also be present in cortical gray-white matter junction, thalami, basal ganglia, cerebellum, and brainstem suggests an<unk>  demyelination process. Additionally, hemorrhagic white matter lesions, clusters of macrophages related to axonal injury and<unk> -like appearance were also found in subcortical white matter.
Acute disseminated encephalomyelitis
Acute flaccid myelitis
Viliuisk encephalomyelitis
Saint Louis encephalitis
Powassan encephalitis
La Crosse encephalitis
Eastern equine encephalitis
Bickerstaff brainstem encephalitis