Descriptions of human<unk>  are few. A patient receiving prolonged parenteral nutrition acquired a syndrome described as ‘acquired<unk> .’ This syndrome, exacerbated by methionine administration, was characterized by high blood methionine, low blood uric acid, and low urinary uric acid and sulfate concentrations. The patient suffered mental disturbances that progressed to a coma. Pathological changes occurring in individuals with a genetic disease that results in a sulfite oxidase (a molybdoenzyme)<unk>  include increased plasma and urine sulfite, sulfate, thiosulfate, S-sulfocysteine and taurine; seizures, and brain atrophy/lesions; dislocated lenses; and death at an early age..
Molybdenum deficiency
Boron deficiency 
Molybdenum cofactor deficiency
Vitamin D deficiency
Vitamin D toxicity
Copper deficiency
Fluorine deficiency
Biotin deficiency