Usually manifesting itself between 20 and 40 years of age, it is characterized by pain, paresthesia, muscular weakness and autonomic dysfunction. In its terminal state, the kidneys and the heart are affected.<unk>  is characterized by the systemic deposition of amyloidogenic variants of the transthyretin protein, especially in the peripheral nervous system, causing a progressive sensory and motor polyneuropathy.
Familial amyloid polyneuropathy
Familial amyloid neuropathy
MGUS polyneuropathy
Distal hereditary motor neuropathy type V
Muscular dystrophy
Charcot–Marie–Tooth disease
Hereditary sensory and autonomic neuropathy type I
Hereditary inclusion body myopathy