Sequencing the DNA of several patients suggested a mutation in the<unk> <unk> . The presence of this mutation was confirmed by sequencing this<unk>  in additional patients. This<unk>  encodes a prostaglandin transporter. As non-steroidal anti-inflammatory drugs inhibit prostaglandin synthesis this helps to explain the known similarity in the histological findings between these conditions. The identification of this mutation suggests that prostaglandin replacement may be helpful but this will need to be tested in a clinical trail.
Chronic enteropathy associated with SLCO2A1 gene
Non-celiac gluten sensitivity
Attenuated familial adenomatous polyposis
Hereditary diffuse gastric cancer
Lysinuric protein intolerance
Congenital disorder of glycosylation type IIc
Hereditary fructose intolerance
Gluten-sensitive enteropathy–associated conditions