medial rectus, superior rectus, inferior rectus, and inferior oblique muscles, while trochlear nerve (CNIV) and abducens nerve (CNVI) paralysis affect superior oblique muscle and lateral rectus muscle respectively. 300x300px (ptosis) and nonaligned eyes (strabismus) are observed, showing paresis of CNIII, CNIV and CNVI.|300x300px The typical symptoms of RPON are<unk>  headaches and ipsilateral paralysis of the extraocular muscles <unk>  that are responsible for controlling eye movements. People with RPON experience different severity of pain, duration of symptoms, and frequency of attacks, which are also dependent on the treatment they received. The attacks of RPON usually occur on the same side of the head in subsequent episodes. Headache The most common type of headache is migraine-like, usually accompanied by symptoms such as nausea, vomiting, photophobia, or phonophobia. Less commonly, RPON can occur without migrainous headache nor accompanied by the classical migrainous symptoms. The frequency of headaches can range from several times per month to once in several years, while the duration varies from several days to a week, which lasts longer than that of a typical migraine.<unk> <unk>  usually occurs either concurrently or within 1 week after the onset of headache, and less commonly up to 2 weeks. The ocular manifestation of RPON depend on which ocular cranial nerve is being affected. CNIII is involved in the vast majority of cases, but the involvement of CNVI and CNIV is also reported in studies. Typical ocular signs involving CNIII include outwards and downwards drifting of eyes (strabismus), difficulty in controlling inward and upward eye movements, drooping of eyelid (ptosis) and double vision. Pupil dilation (mydriasis) and reduction in pupillary light reflexes can also be observed as pupillomotor fibers are also affected by CNIII paralysis. Rare cases involve abducens nerve paralysis affects lateral eye movement while trochlear nerve paralysis affects vertical eye movement. Similar to headache,<unk>  usually resolve gradually and completely over time, but recovery may be incomplete after several episodes.
Recurrent painful ophthalmoplegic neuropathy
Acute visual loss
Visual impairment due to intracranial pressure
Persistent edema of rosacea
Recurrent corneal erosion
Diffuse unilateral subacute neuroretinitis
Chronic progressive external ophthalmoplegia
Facial onset sensory and motor neuropathy