<unk> s may result from diseases acquired during life, or from genetic conditions. Hypertension, smoking, alcoholism, and obesity are associated with the development of brain<unk> s. Cocaine use has also been associated with the development of<unk> s. Other acquired associations with<unk> s include head trauma and infections. Genetic associations Coarctation of the aorta is also a known risk factor, as is arteriovenous malformation. Genetic conditions associated with connective tissue disease may also be associated with the development of<unk> s. This includes: * autosomal dominant polycystic kidney disease, * neurofibromatosis type I, * Marfan syndrome, * multiple endocrine neoplasia type I, * pseudoxanthoma elasticum, * hereditary hemorrhagic telangiectasia and * Ehlers-Danlos syndrome types II and IV. Specific genes have also had reported association with the development of<unk> s, including perlecan, elastin, collagen type 1 A2, endothelial nitric oxide synthase, endothelin receptor A and cyclin dependent kinase inhibitor. Recently, several genetic loci have been identified as relevant to the development of<unk> s. These include 1p34–36, 2p14–15, 7q11, 11q25, and 19q13.1–13.3.
Intracranial aneurysm
Infectious intracranial aneurysm
Subarachnoid hemorrhage
Cerebral vasculitis
Cerebral atherosclerosis
Cerebral infarction
Intracranial hemorrhage
Carotid artery dissection