Agents that have been implicated in<unk> s include cefaclor, amoxicillin, sulfonamides, tetracyclines, ciprofloxacin, nonsteroidal anti-inflammatory drugs, barbiturates, carbamazepine, propranolol, thiouracil, and allopurinol. Metabolites of these drugs might bind with tissue proteins inappropriately, eliciting an acute inflammatory response that typically develops 7–14 days after initiation of the offending agent. Acute hepatitis B will sometimes be complicated with this syndrome, which often resolves with the onset of jaundice.
Serum sickness-like reaction
Papular purpuric gloves and socks syndrome
Acute cutaneous lupus erythematosus
Disseminated disease
Levamisole induced necrosis syndrome
Relapsing fever
Adult-onset Still's disease
Tick-borne lymphadenopathy