One particularity of<unk>  is the long latent phase, during which the disease progresses but is completely asymptomatic. In most cases,<unk>  is detected with concomitant hypertension or coronary artery disease. One of the earliest signs is mild left ventricular diastolic dysfunction with little effect on ventricular filling. Also, the<unk>  patient may show subtle signs of<unk>  related to decreased left ventricular compliance or left ventricular hypertrophy or a combination of both. A prominent “a” wave can also be noted in the jugular venous pulse, and the cardiac apical impulse may be overactive or sustained throughout systole. After the development of systolic dysfunction, left ventricular dilation and symptomatic heart failure, the jugular venous pressure may become elevated, the apical impulse would be displaced downward and to the left. Systolic mitral murmur is not uncommon in these cases. These changes are accompanied by a variety of electrocardiographic changes that may be associated with<unk>  in 60% of patients without structural heart disease, although usually not in the early asymptomatic phase. Later in the progression, a prolonged QT interval may be indicative of fibrosis. Given that<unk> 's definition excludes concomitant atherosclerosis or hypertension, there are no changes in perfusion or in atrial natriuretic peptide levels up until the very late stages of the disease, when the hypertrophy and fibrosis become very pronounced.
Diabetic cardiomyopathy
Amyloid cardiomyopathy
Alcoholic cardiomyopathy
Ischemic cardiomyopathy
Viral cardiomyopathy
Dilated cardiomyopathy
Noncompaction cardiomyopathy
Cardiac amyloidosis