<unk>  <unk>  is a form of kidney damage in which there has been recent exposure to medical imaging<unk>  material without another clear cause for the acute kidney injury.<unk>  is classically defined as a serum creatinine increase of at least 25% and/or an absolute increase in serum creatinine of 0.5 mg/dL after using iodine<unk>  agent without another clear cause for acute kidney injury, but other definitions have also been used. Despite extensive speculation, the actual occurrence of<unk>  has not been demonstrated in the literature. The mechanism of<unk>  is not entirely understood, but is thought to include direct damage from reactive oxygen species,<unk><unk>  increase in urine output, increased oxygen consumption, changes in dilation and narrowing of the blood vessels to the kidneys, and changes in urine viscosity. Analysis of observational studies has shown that radiocontrast use in CT scanning is not causally related to changes in kidney function. Given the increasing doubts about the contribution of radiocontrast to acute kidney injury, the American College of Radiology has proposed the name Post-Contrast Acute Kidney Injury (PC-AKI) does not imply a causal role, with<unk>  or<unk><unk>  Acute Kidney Injury (CI-AKI) reserved for the rare cases where radiocontrast is likely to be causally related.
Contrast-induced nephropathy
Analgesic nephropathy
Reflux nephropathy
Shunt nephritis
IgA nephropathy
Diabetic nephropathy
Benign nephrosclerosis
Acute uric acid nephropathy