The shaft of normal<unk>  (left) and<unk>  in<unk>  (right). The<unk>  is normal in quantity and is usually silvery-blond or straw-colored. It is disorderly, stands out from the scalp, cannot be combed flat, but it can be controlled by braiding methods. This is caused by mutations in one of three possible genes; PADI3, TGM, or TCHH. These genes code for proteins involved in<unk>  shaft formation and improvement often occurs in later childhood.<unk>  is mainly autosomal recessive, but it can also be autosomal dominant because there are other involved genes that have yet to be identified. By early adulthood, phenotypic symptoms of UHS spontaneously improve or disappear. The<unk>  of people with UHS will eventually lie flat and appear near normal in texture by adolescence or early adulthood for unknown reasons. Basmanav et al. have suggested that either different, non-mutated isoforms of the<unk> -forming enzymes are produced as we age, or that aging-related mechanistic changes in<unk>  such as greater diameter and length help resolve UHS.
Uncombable hair syndrome
X-linked hypertrichosis
Dual-role transvestism
Prepubertal hypertrichosis
Adiposogenital dystrophy
Paraphrenia
Latrodectism
Panner disease