Initially, the symptoms of<unk>  are indistinguishable from those of neonatal jaundice, a usually harmless condition commonly seen in infants. However, infants with<unk>  develop progressive conjugated jaundice, pale white stools, and dark urine. Some infants fail to thrive as there will be a degree of fat and fat-soluble vitamin malabsorption (e.g. Vitamin K). This may cause a bleeding tendency. Eventually, and usually after 2 months, cirrhosis with portal hypertension will develop. If left untreated,<unk>  can lead to liver failure. Unlike other forms of jaundice, however,<unk> -related cholestasis mostly does not result in kernicterus, a form of brain damage resulting from liver dysfunction. This is because in<unk> , the liver, although diseased, is still able to conjugate bilirubin, and conjugated bilirubin is unable to cross the blood–brain<unk> rrier.
Biliary atresia
Vanishing bile duct syndrome
Neonatal cholestasis
Congenital hepatic fibrosis
Neonatal jaundice
Progressive familial intrahepatic cholestasis
Biliary disease
Choledochal cysts