Mechanistically, the causes of<unk>  are two-fold: widening of the pelvic<unk>  hiatus and descent of pelvic<unk>  below the pubococcygeal line, with specific organ prolapse, graded relative to the hiatus. People with an inherited deficiency in their collagen type may be more likely to develop<unk> . Additionally, people with congenitally weak connective tissue and fascia are at an increased risk for stress urinary incontinence and pelvic organ prolapse. Recent literature demonstrates that defects in endopelvic fascia and compromised levator ani muscle function have been categorized as important etiologic factors in the development of<unk> . Some circumstances are clearly associated with collagen defects. These include vaginally delivering a child, being post-menopausal, and being of advanced age. Some lifestyle behaviors can lead to<unk> . This includes avoiding urinating or bowel movements, obesity, use of muscle relaxants or narcotics, and use of antihistamines or anticholinergics. Using muscle relaxants or narcotics can lead to increased smooth and skeletal muscle relaxation, potentially related to urinary incontinence. Antihistamines and anticholinergics have additive effects that lead to urinary hesitancy and retention, ultimately leading to<unk> . Urinary incontinence can also affect athletes, especially those in sports that require high impact such as jumping. Gymnasts, for example, report a high prevalence of urinary incontinence. Studies show that athletes in sports requiring high spinal stability may also suffer from this condition, as the activation of abdominal wall muscles can cause urinary alterations during activities. In some cases, sexual abuse can also be associated with chronic pelvic pain and<unk> .<unk>  can result after pelvic radiation, as well as other treatments for gynecological cancers.
Pelvic floor dysfunction
Delayed ejaculation
Premature ejaculation
Pelvic girdle pain
Pelvic pain
Dyspareunia
Pelvic organ prolapse
Coital incontinence