<unk>  causes fibrosis and scarring of the kidneys, which accounts for the symptoms observed. The kidneys also often have corticomedullary cysts. * Inability to conserve sodium because of defect of tubules leading to polyuria and polydipsia. * Anemia is attributed to a deficiency of erythropoietin production by failing kidneys. * Growth retardation, malaise and pallor are secondary to anemia. * No hypertension as<unk>  is a salt-losing enteropathy.
Juvenile nephronophthisis
Medullary cystic kidney disease
Glomerulocystic kidney disease
Cystic kidney disease
Medullary sponge kidney
Autosomal dominant polycystic kidney disease
Doege–Potter syndrome
Autosomal recessive polycystic kidney disease