DYSF The cause of this myopathy is very hard to determine because it can be a mutation in any of at least eight genes and not all are known yet. These mutations can be inherited from one parent, autosomal dominant, or from both parents, autosomal recessive. There are eight known types of<unk> . Types + Classic<unk>  muscular dystrophies Type Eponym Inheritance OMIM Gene Locus Gene also implicated in: Late adult-onset type 1 Welander AD TIA1 2p13 Late adult-onset type 2a - Finnish (tibial) Udd AD TTN 2q31.2 Late adult-onset type 2b Markesbery–Griggs AD ZASP 10q23.2 Early adult-onset type 1 Nonaka AR GNE 9p13.3 Early adult-onset type 2 Miyoshi AR DYSF 2p13.3-p13.1 limb-girdle muscular dystrophy type 2B.<unk>  with anterior tibial onset (DMAT) DMAT can be considered a variant of Miyoshi. Early adult-onset type 3 Laing (Gower) AD MYH7 14q11.2 AD = autosomal dominant; AR = autosomal recessive + Myofibrillar myopathies classifiable as<unk>  Type Eponym Inheritance OMIM Gene Locus Gene also implicated in: Desmin — adult onset (MFM1) Hereditary inclusion-body myositis type 1 AD αB-crystallin — early - mid adult (MFM2) AD ZASP— late adult (MFM4) AD Scapuloperoneal AD MFM = myofibrillary myopathy; AD = autosomal dominant; AR = autosomal recessive + Other<unk>  muscular dystrophies Type Eponym Inheritance OMIM Gene Locus Gene also implicated in:<unk>  with vocal cord and pharyngeal weakness AD MATR3 5q31 Amyotrophic lateral sclerosis 21 (ALS21). One study suggests that all cases are ALS, justifying reclassification.
Distal myopathy
Calpainopathy
Hereditary neuralgic amyotrophy
Nemaline myopathy
Brody myopathy
Inflammatory myopathy
Cuban neuropathy
Metabolic myopathy