For the clinical diagnosis of CNH, it is essential that the symptoms, particularly respiratory alkalosis, persist while the patient is both awake and asleep. The presence of<unk>  during sleep excludes any possible emotional or psychogenic causes for the sustained<unk> . There must also be no evidence of drug or metabolic causes, including cardiac or pulmonary disease, or recent or current use of respiration-stimulating drugs. While a positive diagnosis of CNH in adult cases should be reserved only until all other possible causes of tachypnea have been eliminated, CNH should be suspected in any alert child presenting with unexplained<unk>  and hypocarbia leading to respiratory alkalosis. Once CNH is determined to be a possible cause of<unk> , lesions and their location in the brain are verified using magnetic resonance imaging (MRI). CNH lesions are confirmed through the use of magnetic resonance imaging(MRI) technology.
Central neurogenic hyperventilation
Central sleep apnea
Exercise-induced bronchoconstriction
Hypoxic ventilatory response
Hypoxic pulmonary vasoconstriction
Central hypoventilation syndrome
Intermittent hypoxia
Histotoxic hypoxia