<unk>  is highly toxic, with as little as 6 mL being enough to induce severe lead poisoning. The hazards of<unk> 's lead content are heightened due to the compound's volatility and high lipophilicity, enabling it to easily cross the blood-brain barrier and accumulate in the limbic system, frontal cortex, and hippocampus, making chelation therapy ineffective. Early symptoms of acute exposure to<unk>  can manifest as irritation of the eyes and skin, sneezing, fever, vomiting, and a metallic taste in the mouth. Later symptoms of acute<unk>  poisoning include pulmonary edema, anemia, ataxia, convulsions, severe weight loss, delirium, irritability, hallucinations, nightmares, fever, muscle and joint pain, swelling of the brain, coma, and damage to cardiovascular and renal organs. Chronic exposure to<unk>  can cause long-term negative effects such as memory loss, delayed reflexes, neurological problems, insomnia, tremors, psychosis, loss of attention, and an overall decrease in IQ and cognitive function. The carcinogenity of<unk>  is debatable. It is believed to harm the male reproductive system and cause birth defects. Concerns over the toxicity of lead eventually led to the ban on<unk>  in automobile gasoline in many countries. Some neurologists have speculated that the lead phaseout may have caused average IQ levels to rise by several points in the US (by reducing cumulative brain damage throughout the population, especially in the young). For the entire US population, during and after the<unk>  phaseout, the mean blood lead level dropped from 16 μg/dL in 1976 to only 3 μg/dL in 1991. The US Centers for Disease Control considered blood lead levels "elevated" when they were above 10 μg/dL.
Tetraethyllead
Tetrachlorodecaoxide
Hydrogen fluoride
Lewis lead
Nickel tetracarbonyl
Selenium disulfide
Dichloroacetic acid
Triphenylchloroethylene