<unk> es are reported in endemic areas, such as Southeast Asia, India, and Sub-Saharan Africa. The pathogenesis of acute kidney injury in severe<unk> is unspecific and multifactorial—it affects fewer than 4.8 percent of cases, but reports a high risk of mortality (15 to 45 percent). Histologic evidence shows a large combination of pathogenic mechanisms at play—acute tubular necrosis, interstitial nephritis and glomerulonephritis. Risk factors for<unk>  acute kidney injury include delayed diagnosis, high parasitemia, and clinical presentation of oliguria, low blood pressure, severe anemia, and jaundice. In addition, patients already suffering from diarrhea, hepatitis, or respiratory distress have a worse prognosis.
Malarial nephropathy
IgA nephropathy
Shunt nephritis
Mesoamerican nephropathy
Analgesic nephropathy
Uddanam nephropathy
Sickle cell nephropathy
HIV-associated nephropathy