<unk>  can be grouped into NID A and NID B, with the "A" form affecting the sympathetic innervation, and the "B" version affecting the parasympathetic innervation. In 2002 Martucciello and colleagues published the first analysis of associated anomalies in<unk>  population is an important clinical approach to investigate possible pathogenetic correlations. Two recessive syndromes were identified (3 families). The first was characterized by NID B, intestinal malrotation, and congenital short bowel, the second by NID B, short stature, mental retardation, and facial dysmorphism. In this study, gastrointestinal anomalies accounted for 67.4% of all associated disorders. These data suggest a strong correlation between<unk>  and intestinal development.
Intestinal neuronal dysplasia
Autoimmune gastrointestinal dysmotility
Small intestinal bacterial overgrowth
Foreign body in alimentary tract
Exocrine pancreatic insufficiency
Non-celiac gluten sensitivity
Small intestine neuroendocrine tumor
Familial multiple intestinal atresia