The affected individual may not realize that they have a<unk>  problem and may complain of becoming "clumsy" or "muddled" when performing familiar tasks such as setting the table or simple DIY. Anosognosia, a lack of awareness of the deficit, is common and can cause therapeutic resistance. In some agnosias, such as prosopagnosia, awareness of the deficit is often present; however shame and embarrassment regarding the symptoms can be a barrier in admission of a deficiency. Because agnosias result from brain lesions, no direct treatment for them currently exists, and intervention is aimed at utilization of coping strategies by patients and those around them. Sensory compensation can also develop after one modality is impaired in agnostics General principles of treatment: * restitution * repetitive training of impaired ability * development of compensatory strategies utilizing retained cognitive functions Partial remediation is more likely in cases with traumatic/vascular lesions, where more focal damage occurs, than in cases where the deficit arises out of anoxic brain damage, which typically results in more diffuse damage and multiple cognitive impairments. However, even with forms of compensation, some afflicted individuals may no longer be able to fulfill the requirements of their occupation or perform common tasks, such as, eating or navigating. Agnostics are likely to become more dependent on others and to experience significant changes to their lifestyle, which can lead to depression or adjustment disorders.
Associative visual agnosia
Apperceptive agnosia
Cortical visual impairment
Integrative agnosia
Visuospatial dysgnosia
Asomatognosia
Autotopagnosia
Auditory verbal agnosia