Classical risk factors for developing<unk>  are: * Polycystic ovary syndrome * A previous diagnosis of<unk>  or prediabetes, impaired glucose tolerance, or impaired fasting glycaemia * A family history revealing a first-degree relative with type 2<unk>  * Maternal age – a woman's risk factor increases as she gets older (especially for women over 35 years of age). *Paternal age – one study found that a father's age over 55 years was associated with<unk>  * Ethnicity (those with higher risk factors include African-Americans, Afro-Caribbeans, Native Americans, Hispanics, Pacific Islanders, and people originating from South Asia) * Being overweight, obese or severely obese increases the risk by a factor 2.1, 3.6 and 8.6, respectively. * A previous pregnancy which resulted in a child with a macrosomia (high birth weight: >90th centile or >4000 g (8 lbs 12.8 oz)) * Previous poor obstetric history * Other genetic risk factors: There are at least 10 genes where certain polymorphism are associated with an increased risk of<unk> , most notably TCF7L2. In addition to this, statistics show a double risk of<unk> M in smokers. Polycystic ovarian syndrome is also a risk factor, although relevant evidence remains controversial. Some studies have looked at more controversial potential risk factors, such as short stature. About 40–60% of women with<unk> M have no demonstrable risk factor; for this reason many advocate to screen all women. Typically, women with<unk> M exhibit no symptoms (another reason for universal screening), but some women may demonstrate increased thirst, increased urination, fatigue, nausea and vomiting, bladder infection, yeast infections and blurred vision.
Gestational diabetes
Gestational hypertension
Drugs in pregnancy
Low Birth Weight
Postpartum infections
Pregnancy vegetarianism
Young blood transfusion
Lotus birth