In early dumping syndrome, pancreatic glucagon is augmented in the early postprandial period, probably through stimulation the catecholamines involved in the generalized autonomic surge induced by the osmotic load, but at 120 min, when most of the hypoglycemias are encountered, pancreatic glucagon is no longer detectable, likely through inhibition by GLP-1. Incretins including GLP1 and GIP also bring in the late dumping effects including the insulin rise and the reactive hypoglycemia.
Oxyhyperglycemia
Hypoglycemia
Hyperglycemia
Hyperinsulinemic hypoglycemia
Hyperinsulinemia
Ketotic hypoglycemia
Hyperproinsulinemia
Glycemia