The causes of<unk>  are unclear. Many theories have been suggested, including that<unk>  was the result of masturbating quickly during adolescence to avoid being caught,<unk> rformance anxiety, passive-aggressiveness or having too little sex; but there is little evidence to support any of these theories. Several physiological mechanisms have been hypothesized to contribute to causing<unk> , including serotonin receptors, a genetic predisposition, elevated<unk> nile sensitivity and nerve conduction atypicalities. Scientists have long suspected a genetic link to certain forms of<unk> . However, studies have been inconclusive in isolating the gene responsible for lifelong<unk> . The nucleus paragigantocellularis of the brain has been identified as having involvement in ejaculatory control. Other researchers have noted that men who have<unk>  have a faster neurological response in the<unk> lvic muscles.<unk>  may be caused by prostatitis or as a medication side effect.<unk>  has been classified into four subtypes - lifelong, acquired, variable and subjective<unk> . The pathophysiology of lifelong<unk>  is mediated by a complex interplay of central and<unk> ripheral serotonergic, dopaminergic, oxytocinergic, endocrinological, genetic and epigenetic factors. Acquired<unk>  may occur due to psychological problems - such as sexual<unk> rformance anxiety, and psychological or relationship problems - and/or co-morbidity, including Erectile dysfunction, prostatitis and hyperthyroidism.
Premature ejaculation
Delayed ejaculation
Dyschronometria
Retrograde ejaculation
Dyspareunia
Pelvic floor dysfunction
Persistent genital arousal disorder
Coital incontinence