Human motor cortex The motor tract. In<unk> , the spine and the proximal portion of nerves are usually the abnormal sites of limb weakness.<unk>  resulting from upper extremity impairments following a stroke occurs due to direct damage to the primary motor cortex, primary somatosensory cortex, secondary sensorimotor cortex, sensorimotor cortical areas, subcortical structures, and/or the corticospinal tract. It is often found that impairments following stroke are either caused by damage to the same or adjacent neurological structures. A combination of these impairments is more likely than just one in isolation. Damage to the corticospinal system results in an inability to activate muscles with enough force or in a coordinated manner, which can lead to paresis, loss of fractional movement, and abnormal muscle tone. Damage to the somatosensory cortical areas causes loss of somatosensation which results in an impaired ability to monitor movement. Considering<unk>  as it relates to cerebral palsy, in premature infants, the most common cause of cerebral palsy is periventricular hemorrhagic infarction. In term infants, the underlying causes are often cerebral malformations, cerebral infarction, and intracerebral hemorrhage. Delayed crawling or walking are the usual concerns that arise in infants with paralysis of the limb. In these cases, abnormalities of the legs are the main focus of the attention.
Monoplegia
Cycloplegia
Neuromyotonia
Myelokathexis
Synkinesis
Neuroglycopenia
Central chromatolysis
Autonomic dysreflexia