The neuromotor manifestation of the<unk>  resembles reflexes initiated by vestibular stimuli. Vestibular stimuli activate primitive reflexes in human infants, such as the asymmetric tonic neck reflex, Moro reflex, and parachute reflex, which are likely mediated by vestibular nuclei in the brainstem. The lateral vestibular nucleus (LVN; Deiter’s nucleus) has descending efferent fibers in the vestibulocochlear nerve distributed to the motor nuclei of the anterior column and exerts an excitatory influence on ipsilateral limb extensor motor neurons while suppressing flexor motor neurons. The anatomical location of the LVN, adjacent to the cerebellar peduncles (see cerebellum), suggests that mechanical forces to the head may stretch the cerebellar peduncles and activate the LVN. LVN activity would manifest as limb extensor activation and flexor inhibition, defined as a<unk> , while flexion of the contralateral limb is likely mediated by crossed inhibition necessary for pattern generation. In simpler terms, the shock of the trauma manually activates the nerves that control the muscle groups<unk> ble for raising the arm. These muscle groups are activated by stimuli in infants for instincts such as grabbing for their mothers or breaking their falls. The LVN has neurons that connect it to motor neurons inside grey matter in the spinal cord, and sends signals to one side of the body that activate motor neurons that cause extension, while suppressing motor neurons that cause flexing. The LVN is located near the connection between the brain and the brain stem, which suggests that excessive force to the head may stretch this connection and thus activate the LVN. The neurons that are stimulated suppress neighboring neurons, which prevents neurons on the other side of the body from being stimulated.
Fencing response
Burst suppression
Premonitory urge
Withdrawal reflex
Prepulse inhibition
Mood congruence
Reflex
Disinhibition