The first recorded cases of<unk>  in humans were as recent as 1977, 1978, and 1979. They were reported by Ashford, a British parasitologist who discovered three cases while working in Papua New Guinea. Ashford found that the parasite had very late sporulation, from 8–11 days, making the illness difficult to diagnose. When examining feces, the unsporulated oocysts can easily be mistaken for fungal spores, and thus can be easily overlooked. In 2007, Indian researchers published a case report that found an association between Cyclospora infection and Bell's palsy. This was the first reported case of Bell’s palsy following chronic Cyclospora infection. In addition to other extra-intestinal reports,<unk>  might be involved in either reversible neuronal damage or other unknown mechanisms to lead to Guillain-Barré syndrome or Bell's palsy. In 2010, a report of Cyclospora transmission via swimming in the Kathmandu Valley was published in the Journal of Institute of Medicine. The researchers found that openly defecated human stool samples around the swimmer's living quarters and near the swimming pool were positive for Cyclospora. However, they did not find the parasite in dog stool, bird stool, cattle dung, vegetable samples, or water samples. They concluded that pool water contaminated via environmental pollution might have caused the infection, as the parasite can resist chlorination in water.<unk>  infections have been well reported in Nepal. In one study, Tirth Raj Ghimire, Purna Nath Mishra, and Jeevan Bahadur Sherchan collected samples of vegetables, sewage, and water from ponds, rivers, wells, and municipal taps in the Kathmandu Valley from 2002 to 2004. They found Cyclospora in radish, cauliflower, cabbage, and mustard leaves, as well as sewage and river water. This first epidemiological study determined the seasonal character of<unk>  outbreaks in Nepal during the rainy season, from May to September.<unk>  in AIDS patients At the beginning of the AIDS epidemic in the early 1980s,<unk>  was identified as one of the most important opportunistic infections among AIDS patients. In 2005, Ghimire and Mishra reported a case of<unk>  in a patient with low hemoglobin and suggested that this coccidian might be involved in reducing hemoglobin due to lack of immune system. In 2006, their groups published a paper about the role of<unk>  in HIV/AIDS patients and non-HIV/AIDS patients in the Kathmandu Valley. In 2008, Indian researchers published a report about the epidemiology of Cyclospora in HIV/AIDS patients in Kathmandu. They examined samples of soil, river water, sewage, chicken stool, dog stool, and stool in the streets, and found them positive for Cyclospora. They also evaluated several risk factors for<unk>  in AIDS patients.
Cyclosporiasis
Isosporiasis
Paragonimiasis
Metagonimiasis
Necatoriasis
Mansonelliasis
Bertielliasis
Rhinosporidiosis