In<unk> , echocardiography typically gives non-specific and only occasional findings of endocardium thickening, left ventricular hypertrophy, left ventricle dilation, and involvement of the mitral and/or tricuspid valves. However, in acute necrotizing<unk> , echocardiography usually gives diagnostically helpful evidence of a non-enlarged heart with a thickened and poorly contracting left ventricle. Gadolinium-based cardiac magnetic resonance imaging is the most useful non-invasive procedure for diagnosing<unk> . It supports this diagnosis if it shows at least two of the following abnormalities: a) an increased signal in T2-weighted images; b) an increased global myocardial early enhancement ratio between myocardial and skeletal muscle in enhanced T1 images and c) one or more focal enhancements distributed in a non-vascular pattern in late enhanced T1-weighted images. Additionally, and unlike in other forms of myocarditis,<unk>  may also show enhanced gadolinium uptake in the sub-endocardium. However, the only definitive test for<unk>  is cardiac muscle biopsy showing the presence of<unk>  infiltration. Since the disorder may be patchy, multiple tissue samples taken during the procedure improve the chances of uncovering the pathology but in any case, negative results do not exclude the diagnosis.<unk>  coronary periarteritis<unk>  coronary periarteritis is an extremely rare heart disorder caused by extensive<unk>  infiltration of the adventitia and periadventitia, i.e. the soft tissues, surrounding the coronary arteries. The intima, tunica media, and tunica intima layers of these arteries remain intact and are generally unaffected. Thus, this disorder is characterized by episodes of angina, particularly Prinzmetal's angina, and chaotic heart arrhythmias which may lead to sudden death. The disorder is considered distinct from<unk>  as well as other forms of inflammatory arterial disorders in that it is limited to the coronary artery system.
Eosinophilic myocarditis
Idiopathic giant-cell myocarditis
Coxsackievirus-induced cardiomyopathy
Viral cardiomyopathy
Amyloid cardiomyopathy
Alcoholic cardiomyopathy
HIV associated cardiomyopathy
Tuberculous pericarditis