The proposed mechanism of the characteristic haemolytic anaemia in<unk>  is due to alteration of the red cell metabolism, namely pyruvate kinase instability leaving them susceptible to circulating hemolysin such as lysolecithin. Changes in membrane lipid compositions such as increased cholesterol and polyunsaturated fatty acid (PUFA) have been reported during the hemolytic phase.
Zieve's syndrome
Naegeli–Franceschetti–Jadassohn syndrome
Galloway Mowat syndrome
Löffler's syndrome
Grinspan's syndrome
Gardner's syndrome
Hapnes Boman Skeie syndrome
Binder's syndrome