Risk factors include pregnancy and the postpartum period. Evidence suggests that estrogen- and progesterone-related vascular changes affect the coronary<unk> es during this period, contributing to<unk> . Some case reports and case series suggest associations with autoimmune inflammatory diseases, but there have not been larger studies to explore this relationship. Underlying heritable conditions such as fibromuscular dysplasia and connective-tissue disorders (e.g., Marfan syndrome, Ehlers–Danlos syndrome, and Loeys–Dietz syndrome) are associated with<unk> , but<unk>  otherwise lacks a significant genetic component.<unk>  triggers may include severe physical or emotional stress, but many cases have no obvious cause.
Spontaneous coronary artery dissection
Subacute bacterial endocarditis
Left ventricular thrombus
Libman–Sacks endocarditis
Coronary artery aneurysm
Nonbacterial thrombotic endocarditis
Coronary steal
Primary ventricular fibrillation