<unk>  arthritis most commonly develop as a result of hematogenous spreading of bacteria into the synovial membrane, that induces inflammatory reactions. Eventually, release of cytokines and activation of both host humoral and immunological response along with bacterial virulence factors damages articular surface and cartilage.
Septic arthritis pathophysiology
Bursitis pathophysiology
Rheumatoid arthritis pathophysiology
Psoriatic arthritis pathophysiology
Ankylosing spondylitis pathophysiology
Osteomyelitis pathophysiology
Osteoarthritis pathophysiology
Vertebral osteomyelitis pathophysiology