text: <unk>  arthritis most commonly develop as a result of hematogenous spreading of bacteria into the synovial membrane, that induces inflammatory reactions. Eventually, release of cytokines and activation of both host humoral and immunological response along with bacterial virulence factors damages articular surface and cartilage.
option_0: Septic arthritis pathophysiology
option_1: Bursitis pathophysiology
option_2: Rheumatoid arthritis pathophysiology
option_3: Psoriatic arthritis pathophysiology
option_4: Ankylosing spondylitis pathophysiology
option_5: Osteomyelitis pathophysiology
option_6: Osteoarthritis pathophysiology
option_7: Vertebral osteomyelitis pathophysiology