This is a real-world medical entrance exam question, please give the true answer based on the question and selection. Topic name: None, Subject name: Unknown
[{'content': 'Which is not a pathologic feature H. pylori chronic gastritis? \nSelect and explain. \nA. Eosinophilic infiltrates, B. Intraephithelial neutrophil deposits, C. Affects intestinal gland formation in stomach, D. Subepithelial plasma cell deposits', 'role': 'user'}, {'content': 'Answer: A \nExplanation: (Refer: Robbins Pathologic Basis of Disease, 8thedition, pg no: 776)\nH. Pylori chronic gastritis\nMorphology\n\nOrganism is concentrated within the superficial mucus overlyingepithelial cells.\nH. pylori may be present in foci of pyloric metaplasia withinchronically injured duodenum or gastric-type mucosa withinBarrett esophagus.\nIn stomach, H. pylori are typically found in the Antrum.\nWhen viewed endoscopically, H. pylori-infected antral mucosais usually erythematous and has a coarse or even nodularappearance\nThe inflammatory infiltrate generally includes variable number of neutrophils within the lamina propria,including some thatcross the basement membrane to assume an intraepitheliel location and accumulate in the lumen of gastric pits to createpit abscesses.\nSuperficial lamina propria has large numbers of plasma cells and increased numbers of lymphocytes and macrophages.\nIntraepithelial neutrophils and subepithelial plasma cellsare characteristic of H. pylori gastritis.\nWhen intense, inflammatory infiltrates may create thickenedrugal folds, mimicking early infiltrative lesions.\nLong-standing cases involve body and fundus, and the mucosa becomes atrophic.\nLymphoid aggregates, some with germinal centers, are frequently present and represent an induced form of mucosa- associated lymphoid tissue, or MALT, that has the potentialto transform into lymphoma.\n\nCharacteristics of Helicobacter pylori-associated and Autoimmune Gastritis', 'role': 'assistant'}]